Search results for "Healthspan"

showing 4 items of 4 documents

Toxicological assessment of mesoporous silica particles in the nematode Caenorhabditis elegans

2018

[EN] Here we report the toxicological evaluation of mesoporous silica particles (MSPs) in the nematode C. elegans. Specifically, we have investigated the effect of bare micro- (M0) and nano-sized (N0) MSPs, and their corresponding functionalized particles with a starch derivative (Glu-N) (M1 and N1, respectively) on C. elegans ageing parameters. The toxicity of MSPs, their impact on C. elegans lifespan, movement capacity, progeny and ability to survive upon exposure to acute oxidative stress were assessed. This study demonstrated that both size particles assayed (M0 and N0), labeled with rhodamine and monitored through fluorescence microscopy, are ingested by the nematode. Moreover, toxicit…

TECNOLOGIA DE ALIMENTOSNematodesLongevityNanoparticle02 engineering and technology010402 general chemistrymedicine.disease_cause01 natural sciencesBiochemistrySurface FunctionalizationRhodaminechemistry.chemical_compoundQUIMICA ORGANICAIn vivoToxicity TestsOral intakemedicineAnimalsParticle SizeCaenorhabditis elegansGeneral Environmental ScienceLifespanChemistryQUIMICA INORGANICAStarchMesoporous silicaSilicon Dioxide021001 nanoscience & nanotechnology0104 chemical sciencesOxidative StressMesoporous SilicaNanotoxicologyToxicityBiophysicsNanoparticlesHealthspan0210 nano-technologyMesoporous materialOxidative stressEnvironmental Research
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Moderate overexpression of glucose-6-phosphate dehydrogenase improves healthspan in mice. Implications in skeletal muscle regeneration

2019

An enormous increment in the average lifespan of diverse populations occurred worldwide in the 20th century. In the last 10–15 years, the importance has changed from extending average and even maximal longevity to favoring healthy aging, and many researchers have focused their studies on the aim at extending healthspan. Prolonging lifespan without taking care of improving healthspan can derive in a long time of living with disabilities and constitutes a risk factor for the old population to suffer a higher prevalence of aging-related diseases. Indeed, modern disease treatments frequently diminish mortality without any effect on the deterioration of overall health. Understanding the mechanis…

regenerationagingUNESCO::CIENCIAS MÉDICASoxidative stresshealthspanskeletal muscle:CIENCIAS MÉDICAS [UNESCO]g6pd
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Improvements to Healthspan Through Environmental Enrichment and Lifestyle Interventions: Where Are We Now?

2020

Environmental enrichment (EE) is an experimental paradigm that is used to explore how a complex, stimulating environment can impact overall health. In laboratory animal experiments, EE housing conditions typically include larger-than-standard cages, abundant bedding, running wheels, mazes, toys, and shelters which are rearranged regularly to further increase stimulation. EE has been shown to improve multiple aspects of health, including but not limited to metabolism, learning and cognition, anxiety and depression, and immunocompetence. Recent advances in lifespan have led some researchers to consider aging as a risk factor for disease. As such, there is a pressing need to understand the pro…

0301 basic medicineGerontologymedia_common.quotation_subjectContext (language use)ReviewDiseasehealthspanlcsh:RC321-57103 medical and health sciences0302 clinical medicinemedicineQuality (business)hypothalamuslcsh:Neurosciences. Biological psychiatry. Neuropsychiatrymedia_commonEnvironmental enrichmentHPA axisGeneral NeuroscienceLongevityCognitionMedical research030104 developmental biologyBDNFhealthy agingenvironmental enrichmentAnxietymedicine.symptomPsychologylifespan030217 neurology & neurosurgeryNeuroscienceFrontiers in Neuroscience
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Glucose 6-P dehydrogenase delays the onset of frailty by protecting against muscle damage.

2021

Background: Frailty is a major age-associated syndrome leading to disability. Oxidative damage plays a significant role in the promotion of frailty. The cellular antioxidant system relies on reduced nicotinamide adenine dinucleotide phosphate (NADPH) that is highly dependent on glucose 6-P dehydrogenase (G6PD). The G6PD-overexpressing mouse (G6PD-Tg) is protected against metabolic stresses. Our aim was to examine whether this protection delays frailty. Methods: Old wild-type (WT) and G6PD-Tg mice were evaluated longitudinally in terms of frailty. Indirect calorimetry, transcriptomic profile, and different skeletal muscle quality markers and muscle regenerative capacity were also investigate…

medicine.medical_specialtyAging[SDV]Life Sciences [q-bio]Respiratory chainOxidative phosphorylationDiseases of the musculoskeletal systemGlucosephosphate DehydrogenaseMitocondrisLipid peroxidation03 medical and health scienceschemistry.chemical_compoundMice0302 clinical medicineEnvellimentPhysiology (medical)Internal medicineAdipocytemedicineNADPHAnimalsOrthopedics and Sports MedicineRespiratory exchange ratio030304 developmental biologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesDisabilityFrailtybusiness.industryMusclesQM1-695Skeletal muscleGlucose 1-DehydrogenaseGlutathioneOriginal Articles3. Good healthMitochondriamedicine.anatomical_structureEndocrinologyGlucosechemistryRC925-935Human anatomyHealthspanOriginal ArticleAntioxidantbusinessReactive oxygen species030217 neurology & neurosurgeryJournal of cachexia, sarcopenia and muscle
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