Search results for "Helium"

showing 10 items of 1689 documents

Gathering of aging and estrogen withdrawal in vascular dysfunction of senescent accelerated mice.

2010

The aim of this work was to characterize a mouse model of experimental menopause and cardiovascular aging that closely reflects menopause in women. Senescence accelerated mouse (SAM)-Resistant type 1 (SAMR1, n=30) and SAM-Prone type 8 (SAMP8, n=30) were separated at 5months of age into three groups: 1) sham-operated (Sham); 2) ovariectomized (Ovx); and 3) ovariectomized chronically-treated with estrogen (Ovx+E2). Contractile responses to KCl (60mM) and thromboxane A(2) were greater in aorta from SAMP8 mice compared with SAMR1 in all groups. Neither ovariectomy nor estrogen replacement modified the contractile responses from SAMR1 mice. Conversely, in Ovx SAMP8 the increased maximal contract…

SenescenceAgingmedicine.medical_specialtyEndotheliummedicine.drug_classThromboxaneOvariectomyVasodilator AgentsDown-RegulationIn Vitro TechniquesNitric OxideBiochemistryReceptors Thromboxane A2 Prostaglandin H2Thromboxane A2chemistry.chemical_compoundMiceEndocrinologyInternal medicineGeneticsmedicineAnimalsVasoconstrictor AgentsEnzyme InhibitorsMolecular BiologyAortaEstradiolChemistryEstrogen Replacement TherapyAging PrematureEstrogensCell Biologymedicine.diseaseAcetylcholineMenopauseVasodilationEndocrinologymedicine.anatomical_structureNG-Nitroarginine Methyl EsterEstrogenVasoconstriction15-Hydroxy-11 alpha9 alpha-(epoxymethano)prosta-513-dienoic AcidOvariectomized ratBlood VesselsFemalehormones hormone substitutes and hormone antagonistsAcetylcholinemedicine.drugExperimental gerontology
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Paradoxical effect of l-arginine: Acceleration of endothelial cell senescence

2009

We have recently shown that inhibition of nitric oxide (NO) synthesis by asymmetrical dimethylarginine (ADMA) accelerated endothelial cell (EC) senescence which was prevented by coincubation with L-arginine; however the effect of long-term treatment of l-arginine alone on senescence of ECs have not been investigated. Human ECs were cultured in medium containing different concentrations of L-arginine until senescence. L-Arginine paradoxically accelerated senescence indicated by inhibiting telomerase activity. Moreover, L-arginine decreased NO metabolites, increased peroxynitrite, and 8-iso-prostaglandin F(2alpha) formation. In old cells, the mRNA expression of human amino acid transporter (h…

SenescenceArginineEndotheliumBiophysicsBiologyArginineNitric OxideBiochemistryNitric oxidechemistry.chemical_compoundmedicineHumansCationic Amino Acid Transporter 2Molecular BiologyCells CulturedCellular SenescenceCationic Amino Acid Transporter 1ArginaseEndothelial CellsCell BiologyTransfectionMolecular biologyArginaseEndothelial stem cellOxidative Stressmedicine.anatomical_structurechemistryEndothelium VascularPeroxynitriteBiochemical and Biophysical Research Communications
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Telomere length and cardiovascular disease

2010

SummaryTelomeres are structures composed of deoxyribonucleic acid repeats that protect the end of chromosomes, but shorten with each cell division. They have been the subject of many studies, particularly in the field of oncology, and more recently their role in the onset, development and prognosis of cardiovascular disease has generated considerable interest. It has already been shown that these structures may deteriorate at the beginning of the atherosclerotic process, in the onset and development of arterial hypertension or during myocardial infarction, in which their length may be a predictor of outcome. As telomere length by its nature is a marker of cell senescence, it is of particula…

SenescenceGenetic Markersmedicine.medical_specialtyCell divisionDiseaseBioinformaticsmedicine.disease_causeTélomèreInternal medicinemedicineAnimalsHumansFacteurs de risque cardiovasculairesMyocytes CardiacMyocardial infarctionRisk factorCellular SenescenceCardiovascular risk factorsMaladies cardiovasculairesbusiness.industryStress oxydatifGeneral MedicineTelomeremedicine.diseaseTelomereEndocrinologyCardiovascular diseasesGene Expression RegulationGenetic markerOxidative stressEndothelium VascularbusinessCardiology and Cardiovascular MedicineOxidative stressArchives of Cardiovascular Diseases
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In vivo Microperfusion Studies: Validation and Potential Uses in Critical Patients

2010

Recent studies have suggested an increase in sepsis rates, this increase is due to several factors, such as the increase in the average age of the population, the growing number of invasive procedures and use of immunosuppressive drugs. The increase in sepsis incidence causes localized endothelial damage in various regions, it was this aspect that led us to focus the studies on the endothelium and in vivo microperfusion investigations in order to contribute to the scientific knowledge in this field. The present study will meticulously analyze this current and complex issue that affects on a daily basis, everyone involved in critical patient care.

Sepsismedicine.anatomical_structureEndotheliumbusiness.industryIn vivoMedicineGeneral MedicineEndothelium vascular epithelium microcircol sepsisPharmacologybusinessmedicine.disease
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Modulatory Role of Endothelial and Nonendothelial Nitric Oxide in 5-Hydroxytryptamine-induced Contraction in Cerebral Arteries after Subarachnoid Hem…

1996

OBJECTIVE : Endothelial dysfunction is claimed to play a role in the pathogenesis of delayed cerebral vasospasm after subarachnoid hemorrhage (SAH). We have examined the effect of experimental SAH on the modulatory action of endothelial and nonendothelial nitric oxide (NO) in the contractile response of goat middle cerebral artery to 5-hydroxytryptamine (5-HT). METHODS : We compared the 5-HT-induced contractile responses of cerebral arteries from control goats and from goats with SAH that had been experimentally induced 3 days earlier by delivery of autologous arterial blood into the subarachnoid space. Contractile responses were examined by recording the isometric tension in isolated cereb…

Serotoninmedicine.medical_specialtySubarachnoid hemorrhageEndotheliumCerebral arteriesNitric OxideCerebral vasospasmReference Valuesmedicine.arteryInternal medicinemedicineAnimalscardiovascular diseasesEndothelial dysfunctionVascular diseasebusiness.industryGoatsCerebral ArteriesSubarachnoid Hemorrhagemedicine.diseasenervous system diseasesmedicine.anatomical_structureEndocrinologyVasoconstrictionAnesthesiaMiddle cerebral arteryFemaleSurgeryEndothelium VascularNeurology (clinical)businessBlood vesselNeurosurgery
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RUOLO DELLA LEPTINA NELLE CELLULE EPITELIALI NASALI

2014

Leptin is involved in the lung epithelial homeostasis. Its role in the nasal tract is largely unknown. Allergic rhinitis (AR) is induced by the allergen exposure leading to a consequential structural abnormalities in the nasal epithelium. Topical corticosteroids are recommended as first-line therapy in AR. Parietaria pollen is one of the most important allergenic sources in the southern Europe. In vitro on human nasal epithelial cell line RPMI 2650, we aimed to determine whether allergen stimulation acts on leptin/leptin receptor pathway and how Fluticasone Furoate (FF) influences this pathway. The effects of the major allergen rPar j 1, of FF, of leptin and of TGFβ1 on cell proliferation a…

Settore BIO/14 - Farmacologiafluticasone furoaterPar j 1.allergic rhinitiepitheliumleptin
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Recent Patents on Oxidative Stress-Related Biomarkers in Chronic Heart Failure: The Central Role of Endothelium and Myeloperoxidase

2013

Endothelial cells are key modulators of multiple physiological processes, and their impairment may result in the generation of endothelial dysfunction and ultimately cardiovascular diseases. Under physiologic conditions, reactive oxygen and nitrogen mediators of endothelial cells act to propagate signals driven by different stimuli, by forming molecules with a longer half-life like hydrogen peroxide. Reactive oxygen species (ROS) are constantly produced as a consequence of aerobic metabolism. Under physiologic conditions, their tendency to cause oxidative damage is counterbalanced by the action of antioxidants or oxidant-scavenging enzymes. An imbalance in favour of oxidants leads to oxidat…

Settore BIO/16 - Anatomia UmanaClinical BiochemistryMolecular MedicineChronic heart failure coronary artery disease diagnostic markers endothelial dysfunction endothelium myeloperoxidase oxidative stress prognostic biomarkers serum assaysRecent Patents on Biomarkers
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Effects of oxidative stress on primary human endothelial cells. Potential active contribution of endothelium to inflammatory heart diseases

2007

Settore BIO/16 - Anatomia UmanaMPO endothelium endothelial dysfunction oxidative stress chronic heart failure
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Endurance training damages small airway epithelium in mice.

2005

Settore BIO/16 - Anatomia UmanaSettore MED/10 - Malattie Dell'Apparato RespiratorioTraining bronchial epithelium inflammatory cellsSettore BIO/09 - Fisiologia
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Chaperones in disease: quantitative changes in chronic obstructive pulmonary disease (COPD)

2010

Settore BIO/16 - Anatomia Umanachaperones COPD lung cells epithelium hsp10 hsp60 hsp27 hsp40 hsp70 hsp90 cigarette smoke
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