Search results for "Helium"

showing 10 items of 1689 documents

Characterization of endogenous noradrenaline release from intact and epithelium-denuded rat isolated trachea.

1991

1. Overflow of endogenous noradrenaline (NA) from the in vitro incubated rat trachea evoked by two periods of electrical field stimulation (S1, S2 at 3 or 15 Hz) or by high potassium (60 mM) was determined by high performance liquid chromatography (h.p.l.c.) with electrochemical detection. 2. In the presence of the neuronal uptake inhibitor desipramine, the alpha 2-adrenoceptor antagonist, yohimbine, enhanced the overflow of NA evoked by stimulation at 3 Hz by about 100% suggesting the presence of presynaptic inhibitory autoreceptors on the sympathetic nerves innervating the trachea. 3. When desipramine and yohimbine were present throughout the experiments, the overflow of NA evoked by the …

medicine.medical_specialtyScopolamineNeuromuscular transmissionStimulationTetrodotoxinIn Vitro TechniquesInhibitory postsynaptic potentialEpitheliumchemistry.chemical_compoundNorepinephrineDesipramineInternal medicinemedicineElectrochemistryAnimalsChromatography High Pressure LiquidPharmacologyOxotremorineDesipramineYohimbineMuscle SmoothRats Inbred StrainsEpitheliumElectric StimulationYohimbineRatsTracheaEndocrinologymedicine.anatomical_structurechemistryMuscle TonusTetrodotoxinAutoreceptorPotassiumFemalemedicine.drugResearch Article
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Endothelial modulation of 5-hydroxytryptamine-induced contraction in goat cerebral arteries.

1993

Abstract 1. 1. In isolated goat middle cerebral artery segments, 5-hydroxytryptamine (5-HT, 10 −8 −3 × 10 −5 M) caused concentration-dependent contractions, with EC 50 = 2.1 (1.9−2.5) × 10 −7 M and E max = 60 ± 2% of 50 mM KCl-induced contraction. 2. 2. Mechanical removal of endothelium significantly increased the E max (91 ± 8%) and did not change the EC 50 value of 5-HT-elicited contractions. 3. 3. Incubation of unrubbed arteries with the irreversible inhibitor of EDRF, gossypol (10 −5 M), significantly increased the contractile response to 5-HT ( E max = 77 ± 4%). 4. 4. Incubation of unrubbed arteries with the competitive inhibitor of the NO synthesis, N G -nitro - l -arginine (L-NOARG) …

medicine.medical_specialtySerotoninContraction (grammar)ArginineEndotheliumCerebral arteriesIndomethacinProstacyclinIn Vitro TechniquesArginineNitric OxideNitroarginineMuscle Smooth Vascularchemistry.chemical_compoundInternal medicinemedicine.arteryIsometric ContractionmedicineAnimalsPharmacologyChemistryGoatsEndothelium-derived relaxing factorGossypolAnatomyCerebral ArteriesEpoprostenolEndocrinologymedicine.anatomical_structureMiddle cerebral arteryFemaleEndothelium Vascularmedicine.symptomVasoconstrictionmedicine.drugMuscle ContractionGeneral pharmacology
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Magnesium and Type 2 Diabetes: An Update

2015

The link between magnesium (Mg) deficiency and type 2 diabetes mellitus is well known. Type 2 diabetes is frequently associated with both extracellular and intracellular Mg deficits. A chronic latent Mg deficit or an overt clinical hypomagnesaemia is common in subjects with type 2 diabetes, especially in those with poorly controlled glycemic profiles. Insulin and glucose are important regulators of Mg metabolism. Intracellular Mg plays a key role in regulating insulin action, insulin-mediated-glucose-uptake and vascular tone. Reduced intracellular Mg concentrations result in a defective tyrosine-kinase activity, postreceptorial impairment in insulin action and worsening of insulin resistanc…

medicine.medical_specialtySettore MED/09 - Medicina InternaDiabetes riskbusiness.industryInsulinmedicine.medical_treatmentType 2 Diabetes MellitusType 2 diabetesmedicine.diseaseInsulin resistanceEndocrinologyInternal medicineDiabetes mellitusmedicineMetabolic syndromebusinessMagnesium Diabetes Metabolic syndrome Hypertension Insulin resistance Inflammation Aging Endothelium Hypomagnesaemia MetabolismGlycemicInternational Journal of Diabetes and Clinical Research
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Secretory effect of azodisalicylate (azodisal sodium) on the short circuited mucosa of the rat ileum in vitro.

1988

Azodisalicylate (ADS) is one of the newly developed substitutes of sulphasalazine consisting of two molecules of 5-amino-salicylic acid. Azodisalicylate caused diarrhoea in some patients, apparently caused by an antiabsorptive secretagogue action of this compound. The mechanism of this was studied in the short circuited isolated mucosa of the rat ileum. Mucosal addition of ADS increased the potential difference (PD) and short circuit current (Isc) at a concentration of 1.3.10(-4) mol/l (4 mg/dl) with maximal effects at 1.3.10(-3) mol/l (40 mg/dl). Epithelial resistance was only slightly decreased at the higher concentrations of 40 and 100 mg/dl. Serosal ADS had no effect on electrical param…

medicine.medical_specialtySodiumIndomethacinchemistry.chemical_elementIleumIn Vitro TechniquesBiologyEpitheliumMembrane PotentialsChloridesIntestinal mucosaFurosemideIleumInternal medicinemedicineAnimalsSecretionIntestinal MucosaTranscellularMembrane potentialSodiumElectric ConductivityGastroenterologyStimulation ChemicalRatsAminosalicylic AcidsEndocrinologymedicine.anatomical_structureMechanism of actionchemistrySecretagoguemedicine.symptomResearch ArticleGut
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A systematic review and meta-analysis of the effect of statins on plasma asymmetric dimethylarginine concentrations.

2015

AbstractThe impact of statin therapy on plasma asymmetric dimethylarginine (ADMA) levels has not been conclusively studied. Therefore the aim of the meta-analysis was to assess the effect of statins on circulating ADMA levels. We searched selected databases (up to August 2014) to identify randomized controlled trials (RCTs) that investigate the effect of statins on plasma ADMA concentrations. A weighted meta-regression (WMD) using unrestricted maximum likelihood model was performed to assess the impact of statin dose, duration of statin therapy and baseline ADMA concentrations as potential variables on the WMD between statin and placebo group. In total, 1134 participants in 9 selected RCTs …

medicine.medical_specialtyStatinmedicine.drug_classUrologyPlaceboArgininePlacebo groupArticlelaw.inventionchemistry.chemical_compoundRandomized controlled triallawInternal medicineMedicineHumanscardiovascular diseasesRandomized Controlled Trials as TopicMultidisciplinarybusiness.industrynutritional and metabolic diseasesPublication biasConfidence intervalEndocrinologychemistryMeta-analysisEndothelium VascularHydroxymethylglutaryl-CoA Reductase InhibitorsbusinessAsymmetric dimethylargininePublication BiasScientific reports
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Expression of differentiation antigens and growth-related genes in normal kidney, autosomal dominant polycystic kidney disease, and renal cell carcin…

1992

Cellular differentiation and mRNA levels of genes involved in kidney growth were investigated in normal kidney cells, cyst-lining epithelial cells of polycystic kidney disease, and renal carcinoma cells (RCC). All cells comparatively studied exhibited an antigenic phenotype of proximal tubular cells as shown by the expression of a panel of brush border membrane enzymes and kidney-associated cell surface antigens. The epithelial developmental antigen Exo-1 was expressed in 50% to 80% of cyst-lining epithelia in polycystic kidney tissue and in 20% to 30% of polycystic kidney cells cultured in vitro. Normal kidney cells and RCC were negative under identical culture conditions. The expression o…

medicine.medical_specialtyTGF alphaCellular differentiationAutosomal dominant polycystic kidney diseaseGene ExpressionBiologyKidneyEpitheliumProto-Oncogene Proteins c-mycGrowth factor receptorEpidermal growth factorInternal medicinemedicinePolycystic kidney diseaseHumansRNA MessengerGrowth SubstancesCarcinoma Renal CellCells CulturedKidneyurogenital systemAntibodies MonoclonalTransforming Growth Factor alphamedicine.diseasePolycystic Kidney Autosomal DominantAntigens DifferentiationImmunohistochemistryKidney NeoplasmsErbB ReceptorsEndocrinologymedicine.anatomical_structureGenesNephrologyAntigens SurfaceCancer researchTransforming growth factorAmerican journal of kidney diseases : the official journal of the National Kidney Foundation
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Critical role of dipeptidyl peptidase IV in neuropeptide Y-mediated endothelial cell migration in response to wounding

2001

Recently, we have discovered that neuropeptide Y (NPY), a sympathetic neurotransmitter, is also present in human umbilical endothelial cells (HUVECs), and is potently chemotactic and angiogenic by acting on one or several of Y1-Y5 receptors. In HUVECs, NPY is co-localized with dipeptidyl peptidase IV (DPPIV) which cleaves Tyr(1)-Pro(2) from NPY(1-36) to form NPY(3-36) resulting in the formation of a non-Y1 receptor agonist, which remains angiogenic. Presently we studied the effects of DPPIV's blockade using monoclonal antibodies (mAbs) on migration of HUVECs in response to NPY(1-36) or NPY(3-36) following cell wounding. Both peptides caused similar dose-dependent increases in cell migration…

medicine.medical_specialtyTime FactorsEndotheliumPhysiologyDipeptidyl Peptidase 4Blotting WesternImmunoblottingBiologyBiochemistryDipeptidyl peptidaseUmbilical CordCellular and Molecular NeuroscienceEndocrinologyWestern blotCell MovementInternal medicinemental disordersmedicineHumansNeuropeptide YReceptormedicine.diagnostic_testChemotaxisNeuropeptide Y receptorhumanitiesCell biologyBlotEndothelial stem cellEndocrinologymedicine.anatomical_structureWounds and InjuriesEndothelium VascularPeptides
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Glucose-induced loss of glycosyl-phosphatidylinositol-anchored membrane regulators of complement activation (CD59, CD55) by in vitro cultured human u…

2000

Aims/hypothesis. This study examines whether increased glucose concentrations are responsible for a decreased expression of membrane regulators of complement activation molecules. The effect of high glucose in determining an increase in membrane attack complex deposition on endothelial cells was also investigated. Methods. Endothelial cells were isolated from umbilical cord tissue, cultured in the presence of increased concentrations of glucose, and the expression of CD46, CD55, and CD59 was detected by ELISA (enzyme-linked immunosorbent assay) and by flow cytometry. Glucose-treated endothelial cells were also incubated with antiendothelial cell antibodies and fresh complement to assess the…

medicine.medical_specialtyUmbilical VeinsEndotheliumGlycosylphosphatidylinositolsEndocrinology Diabetes and MetabolismCellCD59 AntigensCD59Complement Membrane Attack ComplexBiologyUmbilical veinMembrane Cofactor ProteinAntigens CDPregnancyInternal medicineInternal MedicinemedicineHumansComplement ActivationCells CulturedMembrane GlycoproteinsCD55 AntigensCD46Cell biologyComplement systemEndothelial stem cellEndocrinologymedicine.anatomical_structureGlucoseFemaleEndothelium VascularComplement membrane attack complexDiabetologia
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Gastrin induces the interaction between human mononuclear leukocytes and endothelial cells through the endothelial expression of P-selectin and VCAM-…

2009

Gastric mucosal inflammation is frequently associated with hypergastrinemia, and a correlation exists between the level of gastrin and degree of gastritis. We have previously observed that gastrin promotes leukocyte-endothelial interactions and contributes to Helicobacter -induced inflammation in the rat mesentery. In the present study, we aimed to evaluate a possible proinflammatory activity of gastrin in humans. The interaction between human leukocytes [U-937 cells, peripheral blood polymorphonuclear (PMN), and peripheral blood mononuclear (PBMC) cells] and human umbilical vein endothelial cells (HUVEC) was analyzed in static and dynamic conditions. The endothelial expression of adhesion …

medicine.medical_specialtyUmbilical VeinsEndotheliumP-selectinPhysiologyLeukocyte adhesion moleculeVascular Cell Adhesion Molecule-1Cell CommunicationBiologyCholecystokinin receptorPeripheral blood mononuclear cellchemistry.chemical_compoundInternal medicineGastrinsmedicineCell AdhesionHumansLeukocyte RollingVCAM-1Cells CulturedGastrinEndothelial CellsCell BiologyFlow CytometryReceptor Cholecystokinin BEndothelial stem cellP-Selectinmedicine.anatomical_structureEndocrinologychemistryMicroscopy FluorescenceLeukocytes MononuclearAmerican journal of physiology. Cell physiology
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Progestogens stimulate prostacyclin production by human endothelial cells.

2005

BACKGROUND: The effects of progestogens on endothelial physiology are poorly studied. Prostacyclin is a potent vasodilator synthesized by two isoforms of cyclooxygenase (COX) in endothelium. We examined the effects of two clinically used progestogens, progesterone and medroxyprogesterone acetate (MPA), on prostacyclin production by cultured human umbilical vein endothelial cells (HUVEC) and the possible role of progesterone receptors and both COX enzymes. METHODS: Cells were exposed to 1-100 nmol/l of either progesterone or MPA and prostacyclin production was measured in culture medium. RESULTS: Both progestogens significantly increased prostacyclin release in a time- and dose-dependent man…

medicine.medical_specialtyUmbilical VeinsEndotheliumProstacyclinMedroxyprogesterone AcetateUmbilical veinInternal medicineProgesterone receptormedicineMedroxyprogesterone acetateHumansCyclooxygenase InhibitorsReceptorCells CulturedNitrobenzenesProgesteroneSulfonamidesbiologyCyclooxygenase 2 InhibitorsDose-Response Relationship DrugEstradiolRehabilitationObstetrics and GynecologyEndothelial CellsMembrane ProteinsEpoprostenolEndothelial stem cellMifepristoneEndocrinologymedicine.anatomical_structureReproductive MedicineCyclooxygenase 2Prostaglandin-Endoperoxide Synthasescardiovascular systembiology.proteinCyclooxygenase 1PyrazolesCyclooxygenaseEndothelium VascularProgestinsReceptors Progesteronemedicine.drugHuman reproduction (Oxford, England)
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