Search results for "Hemoglobin"

showing 10 items of 504 documents

Correlation between periodontal disease management and metabolic control of type 2 diabetes mellitus: a systematic literature review

2016

Background: Diabetes and periodontal disease share common features in terms of inflammatory responses. Current scientific evidence suggests that treatment of periodontal disease might contribute to glycemic control. The objective of the study is a review of the last three years. Material and Methods: A literature search was performed in the MEDLINE (PubMed), Cochrane, and Scopus databases, for articles published between 01-01-2013 and 30-06-2015, applying the key terms “periodontal disease” AND “diabetes mellitus”. The review analyzed clinical trials of humans published in English and Spanish. Results: Thirteen clinical trials were reviewed, representing a total of 1,912 patients. Three of …

Blood Glucosemedicine.medical_specialtyPathologyMEDLINEOdontologíaReviewMalalties periodontals03 medical and health sciences0302 clinical medicineDiabetes mellitusInternal medicinemedicineHumans030212 general & internal medicineGeneral DentistryPeriodontal DiseasesGlycemicGlycated HemoglobinClinical Trials as TopicOral Medicine and PathologyDiabetisbusiness.industryDiabetesType 2 Diabetes Mellitus030206 dentistrymedicine.disease:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludJadad scaleClinical trialSystematic reviewGlucoseOtorhinolaryngologyDiabetes Mellitus Type 2Metabolic control analysisUNESCO::CIENCIAS MÉDICASGlucosaSurgeryPeriodontal diseasebusiness
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Losartan reduces microalbuminuria in hypertensive microalbuminuric type 2 diabetics.

2001

Background. The aim of the present study was to assess the antialbuminuric effect of losartan in a large number of hypertensive type 2 diabetics. Methods. This was a 6-month, open-label, prospective and multicentre study. A total of 422 patients with type 2 diabetes who were hypertensive [sitting systolic blood pressure (SBP) > 140 mmHg and/or diastolic blood pressure (DBP) ≥90 mmHg] and microalbuminuric [urinary albumin excretion (UAE) 30-300 mg/day] were eligible for the study. After a 2-week run-in period, patients were placed on losartan 50 mg once a day. If the BP did not reach the desired goal (<140/90 mmHg) after a 4-week period, the losartan dose was doubled. In the absence of contr…

Blood Glucosemedicine.medical_specialtySystoleUrologyRenal functionBlood PressureType 2 diabetesLosartanDiabetic nephropathyDiastoleDiabetes mellitusInternal medicinemedicineAlbuminuriaHumansAntihypertensive AgentsGlycated HemoglobinTransplantationbusiness.industryBody Weightmedicine.diseaseAngiotensin IIUric AcidLosartanEndocrinologyBlood pressureHydrochlorothiazideDiabetes Mellitus Type 2NephrologyHypertensionRegression AnalysisMicroalbuminuriaDrug Therapy CombinationbusinessDiabetic Angiopathiesmedicine.drugNephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
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Effects of the dual sodium-glucose linked transporter inhibitor, licogliflozinvsplacebo or empagliflozin in patients with type 2 diabetes and heart f…

2020

Aims Explore the efficacy, safety and tolerability of the dual sodium-glucose cotransporter (SGLT) 1 and 2 inhibitor, licogliflozin in patients with type-2 diabetes mellitus (T2DM) and heart failure. Methods This multicentre, parallel-group phase IIA study randomized 125 patients with T2DM and heart failure (New York Heart Association II-IV; plasma N-terminal pro b-type natriuretic peptide [NT-proBNP] &gt;300 pg/mL) to licogliflozin (2.5 mg, 10 mg, 50 mg) taken at bedtime, empagliflozin (25 mg) or placebo (44 patients completed the study). The primary endpoint was change from baseline in NT-proBNP after 12 weeks. Secondary endpoints included change from baseline in glycated haemoglobin, fas…

Blood Glucosemedicine.medical_specialtyUrologyheart failureType 2 diabetesPlacebo030226 pharmacology & pharmacyBedtimeAnhydridesSGLT2 INHIBITORS03 medical and health sciencespharmacotherapy0302 clinical medicineDouble-Blind MethodGlucosidesDiabetes mellitusmedicineEmpagliflozinHumansHypoglycemic AgentsSorbitolPharmacology (medical)030212 general & internal medicineCOTRANSPORTER 2 INHIBITORSBenzhydryl CompoundsPharmacologyGlycated HemoglobinOUTCOMESbusiness.industrySodiumbiomarkersOriginal Articlesmedicine.diseaseEFFICACYBlood pressureGlucoseTreatment OutcomeTolerabilityDiabetes Mellitus Type 2PRESERVED EJECTION FRACTIONHeart failureSAFETYOriginal Articlebiomarkers heart failure pharmacotherapy type 2 diabetestype 2 diabetesbusinessBritish Journal of Clinical Pharmacology
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Correction of glycaemia and GLUT1 level by mildronate in rat streptozotocin diabetes mellitus model

2011

Anti-ischaemic drug mildronate suppresses fatty acid metabolism and increases glucose utilization in myocardium. It was proposed that it could produce a favourable effect on metabolic parameters and glucose transport in diabetic animals. Rats with streptozotocin diabetes mellitus were treated with mildronate (100 mg/kg daily, per os, 6 weeks). Therapeutic effect of mildronate was monitored by measuring animal weight, concentrations of blood glucose, insulin, blood triglycerides, free fatty acids, blood ketone bodies and cholesterol, glycated haemoglobin per cent (HbA1c%) and glucose tolerance. GLUT1 mRNA and protein expression in kidneys, heart, liver and muscles were studied by means of re…

Blood Glucosemedicine.medical_specialtyendocrine system diseasesmedicine.medical_treatmentClinical BiochemistryBiochemistryStreptozocinDiabetes Mellitus Experimentalchemistry.chemical_compoundInternal medicineDiabetes mellitusDiabetes MellitusmedicineAnimalsBody SizeHypoglycemic AgentsInsulinRNA MessengerRats WistarTriglyceridesGlycated HemoglobinGlucose Transporter Type 1Glucose tolerance testmedicine.diagnostic_testFatty acid metabolismbiologyCholesterolbusiness.industryInsulinFatty AcidsGlucose transporternutritional and metabolic diseasesCell BiologyGeneral MedicineGlucose Tolerance Testmedicine.diseaseRatsEndocrinologychemistrybiology.proteinKetone bodiesGLUT1businessMethylhydrazinesCell Biochemistry and Function
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Biosimilars and Novel Insulins.

2019

Background Insulin therapy is the mainstay of treatment for type 1 diabetes and may be necessary in type 2 diabetes. Current insulin analogues present a more physiological profile, are effective, and with less risk of hypoglycemia, but they are expensive. Biosimilar insulins should offer the advantages of insulin analogues at reduced costs. In addition, current rapid-acting insulin analogues are not fast enough to control excessive postprandial glucose excursions in many patients. Areas of uncertainty Biosimilar insulins demonstrated that are safe and effective, but interchangeability and automatic substitution remain an issue. Ultrafast-acting insulins should reduce postprandial hyperglyce…

Blood Glucosemedicine.medical_specialtyendocrine system diseasesmedicine.medical_treatmentInsulin GlargineType 2 diabetes030204 cardiovascular system & hematologyHypoglycemiaInsulin aspart03 medical and health sciences0302 clinical medicinemedicineInsulin lisproHumansHypoglycemic AgentsPharmacology (medical)030212 general & internal medicineIntensive care medicineBiosimilar PharmaceuticalsRandomized Controlled Trials as TopicPharmacologyGlycated HemoglobinType 1 diabetesInsulin Lisprobusiness.industryInsulin glargineInsulinnutritional and metabolic diseasesBiosimilarGeneral Medicinemedicine.diseaseDiabetes Mellitus Type 1Diabetes Mellitus Type 2Hyperglycemiabusinessmedicine.drugAmerican journal of therapeutics
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Effects Of Combined Strength And Endurance Training On Physical Performance And Biomarkers Of Healthy Young Women

2018

Kyröläinen, H, Hackney, AC, Salminen, R, Repola, J, Häkkinen, K, and Haimi, J. Effects of combined strength and endurance training on physical performance and biomarkers of healthy young women. J Strength Cond Res 32(6): 1554– 1561, 2018—Cardiovascular fitness has decreased and obesity has increased in young adults worldwide during the last 10 years. Therefore, there is an urgent need to find out optimal exercise training programs for improving physical performance and health outcomes, especially among sedentary women. Subjects were 25- to 30-year-old women with a very low physical activity, and 65% of them were overweight (body mass index . 25). They performed endurance and strength traini…

Blood GlucosenaisetHydrocortisonekestävyysharjoitteluIsometric exerciseOverweightliikuntaBody Mass Indexsedentary womenHemoglobinsphysical training0302 clinical medicineSex Hormone-Binding GlobulinTestosteroneOrthopedics and Sports Medicine030212 general & internal medicineta315EstradiolVO2 maxGeneral MedicineLipoproteins LDLfyysinen kuntoCholesterolFemalevoimaharjoittelumedicine.symptomLipoproteins HDLAdultmedicine.medical_specialtyStrength trainingPhysical Therapy Sports Therapy and Rehabilitation03 medical and health sciencesOxygen ConsumptionEndurance trainingmedicineHumansMuscle StrengthExercise physiologyMuscle SkeletalExerciseTriglycerideskehonkoostumusnuoret aikuisetbody compositionhormonesblood lipidsbusiness.industryResistance Training030229 sport sciencesmedicine.diseaseObesityhormonitBicyclingPhysical Endurancephysical fitnessPhysical therapySedentary BehaviorbusinessBody mass indexBiomarkers
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Implanted neonatal human dermal fibroblasts influence the recruitment of endothelial cells in mice

2012

The vascularization of new tissue within a reasonable time is a crucial prerequisite for the success of different cell- and material-based strategies. Considering that angiogenesis is a multi-step process involving humoral and cellular regulatory components, only in vivo assays provide the adequate information about vessel formation and the recruitment of endothelial cells. The present study aimed to investigate if neonatal human dermal fibroblasts could influence in vivo neovascularization. Results obtained showed that fibroblasts were able to recruit endothelial cells to vascularize the implanted matrix, which was further colonized by murine functional blood vessels after one week. The ve…

CD31MalePathologymedicine.medical_specialtyAngiogenesisCell TransplantationBiomedical EngineeringCD34Medicine (miscellaneous)Neovascularization PhysiologicInflammationAntigens CD34BiologyNitric OxideRegenerative MedicineBiomaterialsNeovascularizationHemoglobinsMiceTissue engineeringMicroscopy Electron TransmissionIn vivoReportmedicineAnimalsHumansRegenerationSkinInflammationMatrigelNeovascularization PathologicTissue EngineeringEndothelial CellsGeneral MedicineFibroblastsMice Inbred C57BLPlatelet Endothelial Cell Adhesion Molecule-1Drug CombinationsPhenotypeProteoglycansCollagenLamininmedicine.symptom
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In vitro model for the activation of CD4 and CD8 T cell receptors.

2008

Previously, most models that sought to explain the misregulation of immune cell function assumed molecular similarities between the disease-causing pathogens and the host's proteins. In recent time several different models have been proposed and in this study, these concepts are compared to a new hypothesis proposing another explanation for this immune dysregulation: the possibility that the mislocalization of proteins may be responsible for autoimmune activity. Based on this hypothesis, proteins are recognized as self or non-self depending on where they appear in sufficiently high concentrations. To examine this new idea, the intracellular human proteins beta-actin, GAPDH, and hemoglobin a…

CD4-Positive T-LymphocytesCytoplasmImmunologyReceptors Antigen T-CellAutoimmunityCell SeparationCD8-Positive T-Lymphocytesmedicine.disease_causeLymphocyte ActivationHemoglobinsAlbuminsmedicineExtracellularImmunology and AllergyCytotoxic T cellHumansInsulinReceptorGlyceraldehyde 3-phosphate dehydrogenaseCells CulturedbiologyAlbuminModels ImmunologicalGlyceraldehyde-3-Phosphate DehydrogenasesGeneral MedicineImmune dysregulationFlow CytometryActinsCell biologyProtein Transportbiology.proteinCell activationExtracellular SpaceIntracellularHuman immunology
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Determinants of procalcitonin concentration in acute heart failure.

2014

Age, years 73.1 ± 10.4 Male, n (%) 133 (51.0) Hypertension, n (%) 212 (81.2) Diabetes mellitus, n (%) 126 (49.4) Previous smoker, n (%) 104 (28.2) Ischemic etiology, n (%) 92 (35.3) Peripheral oedema, n (%) 187 (71.7) Previous admission for AHF, n (%) 95 (36.4) Prior use of beta-blockers, n (%) 111 (42.5) Prior use of loop diuretics, n (%) 175 (67.0) Prior use of ACEI/ARB, n (%) 134 (51.3) Heart rate, beats/min 97 ± 28 SBP, mm Hg 148 ± 34 DBP, mm Hg 81 ± 19 Atrial fibrillation, n (%) 119 (45.6) Hemoglobin, g/dl 12.1 ± 2.0 Serum creatinine, mg/dl 1.23 ± 0.57 Urea, mg/dl 60.5 ± 30.1 Sodium, mEq/l 137.8 ± 4.7 NT-proBNP, pg/ml 4813 (6011) Leukocyte count, 10 cells/l 9286 ± 3468 Relative lymphoc…

CalcitoninMalemedicine.medical_specialtyCalcitonin Gene-Related PeptideFibrinogenGastroenterologyProcalcitoninCohort Studieschemistry.chemical_compoundInternal medicineHeart ratemedicineHumansProtein PrecursorsHeart FailureCreatinineEjection fractionbusiness.industryMiddle Agedmedicine.diseasechemistryHeart failureAcute DiseaseUreaFemaleHemoglobinCardiology and Cardiovascular MedicinebusinessBiomarkersmedicine.drugInternational journal of cardiology
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Tumor Hypoxia: Causative Factors, Compensatory Mechanisms, and Cellular Response

2004

Abstract Learning Objectives After completing this course, the reader will be able to: Explain the effect of hypoxia on resistance to treatment. Describe the causes of tumor hypoxia. Characterize cellular response to hypoxia. Access and take the CME test online and receive 1 hour of AMA PRA category 1 credit at CME.TheOncologist.com Hypoxia is a characteristic feature of locally advanced solid tumors resulting from an imbalance between oxygen (O2) supply and consumption. Major causative factors of tumor hypoxia are abnormal structure and function of the microvessels supplying the tumor, increased diffusion distances between the nutritive blood vessels and the tumor cells, and reduced O2 tra…

Cancer ResearchCell SurvivalAnemiamedicine.medical_treatmentPhotodynamic therapyDiseaseNeoplasmsmedicineHumansNeovascularization PathologicTumor hypoxiabusiness.industryAnemiaTumor OxygenationHypoxia (medical)medicine.diseaseAdaptation PhysiologicalCell HypoxiaOxygenRadiation therapyCell Transformation NeoplasticOncologyDrug Resistance NeoplasmImmunologyDisease ProgressionCancer researchHemoglobinmedicine.symptombusinessThe Oncologist
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