Search results for "Hepatocytes"

showing 10 items of 224 documents

Cryopreservation of rat, dog and human hepatocytes: influence of preculture and cryoprotectants on recovery, cytochrome P450 activities and induction…

2006

Several cryopreservation protocols for hepatocytes have been proposed over the past few years, but their effectiveness varies greatly as a function of the characteristics of the method used. One factor in the success of cryopreservation is the quality of cells before freezing. The results suggest that the cryopreservation of hepatocytes in a medium containing polyvinylpyrrolidone (PVP), in addition to DMSO, constitutes a convenient means of long-term storage of hepatocytes for preparing primary cultures to be used in drug metabolism studies. The combined use of the two cryoprotectants is particularly critical for low-viability cell suspensions. An interesting alternative to increase cell vi…

MaleHot TemperatureCryoprotectantHealth Toxicology and MutagenesisCellCombined useDrug Evaluation PreclinicalToxicologyBiochemistryCryopreservationRats Sprague-DawleyCryoprotective AgentsDogsCytochrome P-450 Enzyme SystemmedicineAnimalsHumansDimethyl SulfoxideViability assayCells CulturedCryopreservationPharmacologybiologyPovidoneCytochrome P450General MedicineRatsCell biologyEnzyme Activationmedicine.anatomical_structureBiochemistryHepatocytesbiology.proteinDrug metabolismXenobiotica
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Vaccenic and elaidic acid equally esterify into triacylglycerols, but differently into phospholipids of fed rat liver cells.

2011

Elaidic acid (trans-9-C18:1 or trans-9) is assumed to exert atherogenic effects due to its double bond configuration. The possibility that trans-9 and vaccenic acid (trans-11-C18:1 or trans-11), its positional isomer, were biochemically equivalent and interchangeable compounds, was investigated by reference to their cis-isomers through esterification-related activities using rat liver cells and subcellular fractions. In hepatocytes, both trans-C18:1 were incorporated to the same extent in triacylglycerols, but trans-9 was more esterified than trans-11 into phospholipids (P < 0.05). Glycerol-3-phosphate acyltransferase activity in microsomes was lower with trans-11 than with trans-9, while t…

MaleLipoproteinsPhospholipidCell Culture TechniquesVaccenic acidGene ExpressionOleic AcidsBiochemistrychemistry.chemical_compoundIsomerismMicrosomesAnimalsRats WistarPhospholipidsTriglycerideschemistry.chemical_classificationEsterificationCholesterolOrganic ChemistryFatty acidCell BiologyElaidic acidMitochondriaRatsEnzymeCholesterolchemistryBiochemistryLiverTherapeutic EquivalencyAcyltransferaseGlycerol-3-Phosphate O-AcyltransferaseMicrosomeHepatocyteslipids (amino acids peptides and proteins)Oleic AcidLipids
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Hepatocytes cultured in alginate microspheres: an optimized technique to study enzyme induction.

2004

An important application of hepatocyte cultures is identification of drugs acting as inducers of biotransformation enzymes that alter metabolic clearance of other therapeutic agents. In the present study we optimized an in vitro system with hepatocytes cultured in alginate microspheres that allow studies of enzyme induction with excellent sensitivity. Induction factors obtained with standard inducers, such as 3-methylcholanthrene or phenobarbital, were higher compared to those with conventional hepatocyte co-cultures on collagen coated dishes. This is illustrated by activities of 7-ethoxyresorufin-O-deethylase (EROD) after incubation with 5 microM 3-methylcholanthrene (3-MC), a standard ind…

MaleLiver cytologyAlginatesCell Culture TechniquesBiologyToxicologySensitivity and SpecificityHydroxylationRats Sprague-Dawleychemistry.chemical_compoundGlucuronic AcidIn vivomedicineCytochrome P-450 CYP1A1AnimalsTechnology PharmaceuticalInducerEnzyme inducerCells CulturedGlutathione TransferaseHexuronic AcidsReproducibility of ResultsReference StandardsIn vitroCoculture TechniquesMicrospheresRatsmedicine.anatomical_structureBiochemistrychemistryLiverCell cultureHepatocyteEnzyme InductionPhenobarbitalCytochrome P-450 CYP2B1biology.proteinHepatocytesMethylcholanthreneToxicology
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Neonatal Livers: A Source for the Isolation of Good-Performing Hepatocytes for Cell Transplantation

2014

Hepatocyte transplantation is an alternative therapy to orthotopic liver transplantation for the treatment of liver diseases. However, the supply of hepatocytes is limited given the shortage of organs available to isolate good-functioning quality cells. Neonatal livers may be a potential source alternative to adult livers to obtain good-performing hepatic cells for hepatocyte transplantation, which has not yet been explored profoundly. High-yield preparations of viable hepatocytes were isolated from 1- to 23-day-old liver donors, cryopreserved, and banked. Cell integrity and functional quality assessment were performed after thawing. Neonatal hepatocytes showed better postthawing recovery …

MaleLiver cytologyCellBiomedical Engineeringlcsh:MedicineCell SeparationBiologyCryopreservationAndrologymedicineHumansProgenitor cellCells CulturedCryopreservationTransplantationlcsh:RInfant NewbornCell BiologyLiver TransplantationTransplantationmedicine.anatomical_structureLiverApoptosisHepatocyteHepatocytesHepatic stellate cellFemaleCell Transplantation
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Toxicological profile of cereulide, the Bacillus cereus emetic toxin, in functional assays with human, animal and bacterial cells

2007

International audience; Some strains of the endospore-forming bacterium Bacillus cereus produce a heat-stable ionophoric peptide, cereulide, of high human toxicity. We assessed cell toxicity of cereulide by measuring the toxicities of crude extracts of cereulide producing and non-producing strains of B. cereus, and of pure cereulide, using cells of human, animal and bacterial origins. Hepatic cell lines and boar sperm, with cytotoxicity and sperm motility, respectively, as the end points, were inhibited by <= 1 nM of cereulide present as B. cereus extract. RNA synthesis and cell proliferation in HepG2 cells was inhibited by 2 nM of cereulide. These toxic effects were explainable by the acti…

MaleLuminescenceSwineCytotoxicityBacillus cereusCYP1A1Toxicologymedicine.disease_causeHepa-1Ames testPotassium carrierchemistry.chemical_compoundMiceDepsipeptidesBioassayRNA Neoplasm0303 health sciencesbiologyMotilityAliivibrio fischeriSpermatozoaAmes testCereusBiochemistry[SDV.TOX]Life Sciences [q-bio]/ToxicologySperm MotilityBiological AssayERODBioluminescenceHepG2CereulideCell SurvivalBacterial ToxinsVibrio fischeriHEp-2Microbiology03 medical and health sciencesBacillus cereusCell Line TumorIonophoremedicineAnimalsHumansRNA synthesis030304 developmental biologyCell ProliferationDose-Response Relationship Drug030306 microbiologyToxinMutagenicity TestsfungiMicronucleus assayCereulidecomet test (SCG)biology.organism_classificationComet assaychemistryHepatocytesbacteriaBoar spermGenotoxicityGenotoxicity
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Metabolic pathways of 4-bromo-2,5-dimethoxyphenethylamine (2C-B): analysis of phase I metabolism with hepatocytes of six species including human

2004

Abstract 4-Bromo-2,5-dimethoxyphenethylamine (2C-B) is a psychoactive designer drug of abuse that is sold under the street names “Venus”, “Bromo”, “Erox”, “XTC” or “Nexus”. Concern has been raised because only little is known about its toxicity and metabolism in humans. In the present study we incubated 2C-B with human, monkey, dog, rabbit, rat and mouse hepatocytes to identify the metabolites formed and to determine possible toxic effects as evidenced by an ATP assay. Our data allow construction of the main metabolic pathways of 2C-B. Oxidative deamination results in the 2-(4-bromo-2,5-dimethoxyphenyl)-ethanol (BDMPE) and 4-bromo-2,5-dimethoxyphenylacetic acid (BDMPAA) metabolites. Additio…

MaleMetaboliteDeaminationMice Inbred StrainsBiologyToxicologyGas Chromatography-Mass SpectrometryRats Sprague-DawleyMicechemistry.chemical_compoundAdenosine TriphosphateDogsSpecies SpecificitymedicineAnimalsHumansCells CulturedDemethylationDose-Response Relationship DrugMolecular Structure25-Dimethoxy-4-MethylamphetamineIllicit DrugsOxidative deaminationMetabolismMiddle AgedRatsMacaca fascicularisMetabolic pathwaymedicine.anatomical_structurechemistryBiochemistryDeaminationHepatocyteHepatocytesRabbitsOxidation-ReductionDrug metabolismToxicology
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Dehydroepiandrosterone up-regulates the Adrenoleukodystrophy-related gene (ABCD2) independently of PPAR alpha in rodents

2007

International audience; X-linked adrenoleukodystrophy (X-ALD) is a neurodegenerative disease caused by mutations in the ABCD1 gene, which encodes a peroxisomal ABC transporter, ALDP, supposed to participate in the transport of very long chain fatty acids (VLCFA). The adrenoleukodystrophyrelated protein (ALDRP), which is encoded by the ABCD2 gene, is the closest homolog of ALDP and is considered as a potential therapeutic target since functional redundancy has been demonstrated between the two proteins. Pharmacological induction of Abcd2 by fibrates through the activation of PPARa has been demonstrated in rodent liver. DHEA, the most abundant steroid in human, is described as a PPARa activat…

MalePEROXISOMEProhormonePeroxisome proliferator-activated receptorATP-binding cassette transporterBiochemistryMice0302 clinical medicineABC TRANSPORTERSPPAR-ALPHAAdrenal GlandsTestisDHEACells Culturedchemistry.chemical_classification0303 health sciencesSex CharacteristicsbiologyBrainGeneral MedicineOrgan SizePeroxisome3. Good healthUp-RegulationLiverAdrenoleukodystrophyFemalemedicine.drugAndrostenediolmedicine.medical_specialtyADRENOLEUKODYSTROPHYATP Binding Cassette Transporter Subfamily D03 medical and health sciencesABCD3Internal medicinemedicineABCD2AnimalsPPAR alpha[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyRats Wistar030304 developmental biologyActivator (genetics)Body Weightnutritional and metabolic diseasesMembrane ProteinsDehydroepiandrosteronemedicine.diseaseRatsMice Inbred C57BLEndocrinologychemistrybiology.proteinHepatocytesATP-Binding Cassette TransportersAcyl-CoA Oxidase030217 neurology & neurosurgery
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Immunopositivity for histone macroH2A1 isoforms marks steatosisassociated hepatocellular carcinoma.

2012

BackgroundHepatocellular carcinoma (HCC) is one of the most common cancers worldwide. Prevention and risk reduction are important and the identification of specific biomarkers for early diagnosis of HCC represents an active field of research. Increasing evidence indicates that fat accumulation in the liver, defined as hepatosteatosis, is an independent and strong risk factor for developing an HCC. MacroH2A1, a histone protein generally associated with the repressed regions of chromosomes, is involved in hepatic lipid metabolism and is present in two alternative spliced isoforms, macroH2A1.1 and macroH2A1.2. These isoforms have been shown to predict lung and colon cancer recurrence but to ou…

MalePathologyMouseBiological Markers/metabolismEpidemiologyTumor Microenvironment/geneticsColorectal cancerGene ExpressionHepatocytes/metabolism/pathologyNonalcoholic SteatohepatitisHistonesFatty Liver/chemically induced/complications/genetics/metabolismMice0302 clinical medicineGastrointestinal CancersTumor MicroenvironmentPathologyProtein IsoformsDiethylnitrosamineSettore MED/49 - Scienze Tecniche Dietetiche ApplicateMice KnockoutRegulation of gene expression0303 health sciencesMultidisciplinaryProtein Isoforms/genetics/metabolismbiologyLiver DiseasesPTEN Phosphohydrolase/deficiency/geneticshepatocellular carcinoma biomarker histone variant steatosis epigeneticsLiver NeoplasmsQFatty liverRHistone ModificationAnimal ModelsImmunohistochemistry3. Good healthHistoneOncology030220 oncology & carcinogenesisHepatocellular carcinomaMedicineEpigeneticsCarcinoma Hepatocellular/etiology/genetics/metabolism/pathologyResearch ArticleGene isoformmedicine.medical_specialtyCarcinoma HepatocellularHistologyClinical Research DesignScienceGastroenterology and HepatologyDiet High-Fat03 medical and health sciencesModel OrganismsDiagnostic MedicineGastrointestinal TumorsGeneticsCancer GeneticsCancer Detection and DiagnosisEarly DetectionmedicineAnimalsHumansAnimal Models of DiseaseObesityddc:612BiologyHistones/genetics/metabolismNutrition030304 developmental biologyCell NucleusCell Nucleus/genetics/metabolism/pathologyTumor microenvironmentbusiness.industryPTEN PhosphohydrolaseCancers and NeoplasmsHepatocellular Carcinomamedicine.diseasedigestive system diseasesFatty LiverBiomarker EpidemiologyGene Expression RegulationHepatocytesbiology.proteinLiver Neoplasms/etiology/genetics/metabolism/pathologySteatosisbusinessBiomarkersGeneral Pathology
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Id2 leaves the chromatin of the E2F4-p130-controlled c-myc promoter during hepatocyte priming for liver regeneration

2006

The Id (inhibitor of DNA binding or inhibitor of differentiation) helix–loop–helix proteins are involved in the regulation of cell growth, differentiation and cancer. The fact that the molecular mechanisms of liver regeneration are not completely understood prompted us to study the fate of Id2 in proliferating liver. Id2 increases in liver regeneration after partial hepatectomy, following the early induction of its gene. Co-immunoprecipitation shows that Id2 forms a complex with E2F4, p130 and mSin3A in quiescent liver and all these components are present at the c-myc promoter as shown using ChIP (chromatin immunoprecipitation). Activation of c-myc during hepatocyte priming (G0–G1 transitio…

MalePriming (immunology)E2F4 Transcription FactorId2Cell cycleBiologyBiochemistryProto-Oncogene Proteins c-mycE2FmedicineAnimalsHistone deacetylaseRats WistarPromoter Regions GeneticE2FMolecular BiologyE2F4Inhibitor of Differentiation Protein 2Cell BiologyMolecular biologyChromatinLiver regenerationLiver RegenerationRatsSpecific Pathogen-Free OrganismsUp-RegulationChromatinC-mycmedicine.anatomical_structureGene Expression RegulationHepatocyteHepatocytesLiver regenerationHistone deacetylaseCarrier ProteinsChromatin immunoprecipitationResearch Article
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Kinetics of tienilic acid bioactivation and functional generation of drug–protein adducts in intact rat hepatocytes

2005

13 pages; Drug-induced autoimmune hepatitis is among the most severe hepatic idiosyncratic adverse drug reactions. Considered multifactorial, the disease combines immunological and metabolic aspects, the latter being to date much better known. As for many other model drugs, studies on tienilic acid (TA)-induced hepatitis have evidenced the existence of bioactivation during the hepatic oxidation of the drug, allowing the identification of the neoantigen of anti-LKM2 autoantibodies and the pathway responsible for its formation. However, most of these results are based on the use of microsomal fractions whose relevance to the liver in vivo still needs to be established. In the more complex int…

MaleTicrynafen[SDV.BC]Life Sciences [q-bio]/Cellular BiologyAutoimmune hepatitisPlasma protein bindingHydroxylationBiochemistryRats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineIn vivoCYP[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologymedicineAnimalsPrimary cultured hepatocytesTienilic acid[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyCytochrome P450 Family 2[SDV.BC] Life Sciences [q-bio]/Cellular BiologyBiotransformationCells Cultured030304 developmental biologyPharmacologyHepatitis0303 health sciencesDrug bioactivationChemistryGlutathionemedicine.diseaseGlutathioneIn vitroRats3. Good health[SDV.TOX] Life Sciences [q-bio]/Toxicologymedicine.anatomical_structureSteroid 16-alpha-HydroxylaseBiochemistryTienilic acid[SDV.TOX]Life Sciences [q-bio]/Toxicology030220 oncology & carcinogenesisHepatocyteHepatocytesAryl Hydrocarbon HydroxylasesDrug–protein adductsProtein Bindingmedicine.drugBiochemical Pharmacology
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