Search results for "Histochemistry"

showing 10 items of 1604 documents

Chemotherapy accelerates immune-senescence and functional impairments of Vδ2pos T cells in elderly patients affected by liver metastatic colorectal c…

2019

Abstract Human (gamma delta) γδ T cells are unconventional innate-like lymphocytes displaying a broad array of anti-tumor activities with promising perspectives in cancer immunotherapy. In this context, Vδ2pos T cells represent the preferential target of several immunotherapy protocols against solid tumors. However, the impact of both aging and chemotherapy (CHT) on Vδ2pos T cells is still unknown. The present study evaluates with multi-parametric flow cytometry the frequencies, terminal differentiation, senescence and effector-functions of peripheral blood and tumor infiltrating Vδ2pos T cells purified from liver metastases (CLM) of patients affected by colorectal cancer (CRC) compared to …

0301 basic medicineSenescenceCancer ResearchColorectal cancermedicine.medical_treatmentImmunologyShort ReportContext (language use)Antineoplastic AgentsCD16lcsh:RC254-282γδ T cellsFlow cytometryImmunophenotyping03 medical and health sciences0302 clinical medicineCancer immunotherapyT-Lymphocyte SubsetsCell Line TumorAntineoplastic Combined Chemotherapy ProtocolsmedicineImmunology and AllergyChemotherapyHumansCellular SenescenceCancerPharmacologymedicine.diagnostic_testbusiness.industryLiver NeoplasmsCD28Receptors Antigen T-Cell gamma-deltaImmunotherapylcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseImmunohistochemistry030104 developmental biologyOncology030220 oncology & carcinogenesisCancer researchMolecular MedicineImmune-senescence/AgingbusinessColorectal NeoplasmsBiomarkersJournal for Immunotherapy of Cancer
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Anatomy, immunohistochemistry, and numerical distribution of human splenic microvessels.

2019

Abstract The microvascular architecture of the spleen plays an important role in the immunological function of this organ. The different types of vessels are related to different reticular cells each with their own immunomodulatory functions. The present study describes an immunohistochemical and morphometric analysis of the various types of vessels in 21 human autopsy non-pathological splenic samples. On an area of 785,656.37 μm2 for each sample, we classified and quantified the type and number of vascular structures, each according to their morphology and immunohistochemical profile, and obtained the ratios between them. The distribution of trabecular vessels and the characteristics of th…

0301 basic medicineSialic Acid Binding Ig-like Lectin 1CD8 AntigensCD34ImmunoglobulinsSpleenAntigens CD3403 medical and health sciencesMucoproteinsTrabecular veinsReticular cellmedicineHumansAdapaleneVeinForensic PathologySinus (anatomy)VenuleChemistryGeneral MedicineAnatomyImmunohistochemistryActinsPlatelet Endothelial Cell Adhesion Molecule-1Arterioles030104 developmental biologymedicine.anatomical_structureMicrovesselsImmunohistochemistry030101 anatomy & morphologyAutopsyAnatomyCell Adhesion MoleculesSplenic ArterySpleenDevelopmental BiologyAnnals of anatomy = Anatomischer Anzeiger : official organ of the Anatomische Gesellschaft
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Characterizing diversity in the tumor-immune microenvironment of distinct subclasses of gastroesophageal adenocarcinomas

2020

Background Gastroesophageal adenocarcinomas (GEAs) are heterogeneous cancers where immune checkpoint inhibitors have robust efficacy in heavily inflamed microsatellite instability (MSI) or Epstein-Barr virus (EBV)-positive subtypes. Immune checkpoint inhibitor responses are markedly lower in diffuse/genome stable (GS) and chromosomal instable (CIN) GEAs. In contrast to EBV and MSI subtypes, the tumor microenvironment of CIN and GS GEAs have not been fully characterized to date, which limits our ability to improve immunotherapeutic strategies. Patients and methods Here we aimed to identify tumor-immune cell association across GEA subclasses using data from The Cancer Genome Atlas (N = 453 GE…

0301 basic medicineT cellmedicine.medical_treatmentAdenocarcinomaArticle03 medical and health sciences0302 clinical medicineImmune systemStomach NeoplasmsTumor MicroenvironmentMedicineHumansTumor microenvironmentbusiness.industryMicrosatellite instabilityHematologyImmunotherapyCell cyclemedicine.diseaseImmunohistochemistry030104 developmental biologymedicine.anatomical_structureOncology030220 oncology & carcinogenesisCancer researchAdenocarcinomaMicrosatellite InstabilitybusinessCD8
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Combined B, T and NK Cell Deficiency Accelerates Atherosclerosis in BALB/c Mice.

2016

This study focused on the unique properties of both the Ldlr knockout defect (closely mimicking the human situation) and the BALB/c (C) inbred mouse strain (Th-2 slanted immune response). We generated two immunodeficient strains with severe combined B- and T-cell immunodeficiency with or without a complete lack of natural killer cells to revisit the role of adaptive immune responses on atherogenesis. C-Ldlr-/- Rag1-/- mice, which show severe combined B- and T-cell immunodeficiency and C-Ldlr-/- Rag1-/- Il2rg-/- mice, which combine the T- and B-cell defect with a complete lack of natural killer cells and inactivation of multiple cytokine signalling pathways were fed an atherogenic Western ty…

0301 basic medicineT-Lymphocyteslcsh:MedicineNK cellsAdaptive ImmunityBiochemistryVascular MedicineMicechemistry.chemical_compoundCellular typesReceptorlcsh:ScienceImmunodeficiencyMice KnockoutB-LymphocytesMice Inbred BALB CMultidisciplinarybiologyT CellsImmune cellsAcquired immune systemLipidsPlaque AtheroscleroticKiller Cells NaturalCholesterolPhenotypeWhite blood cellsFemalelipids (amino acids peptides and proteins)Research ArticleCell biologyBlood cellsLipoproteinsImmunologyResearch and Analysis MethodsBALB/cImmune Deficiency03 medical and health sciencesImmune systemmedicineAnimalsImmunohistochemistry TechniquesTriglyceridesMedicine and health sciencesBiology and life sciencesCholesterolMacrophageslcsh:RImmunologic Deficiency SyndromesWild typeProteinsAtherosclerosisbiology.organism_classificationmedicine.diseaseMolecular biologyHistochemistry and Cytochemistry Techniques030104 developmental biologyAnimal cellsReceptors LDLchemistryImmune SystemMutationImmunologyLDL receptorImmunologic TechniquesClinical Immunologylcsh:QClinical MedicinePLoS ONE
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Alternative Splice Forms of CYLD Mediate Ubiquitination of SMAD7 to Prevent TGFB Signaling and Promote Colitis

2018

Background & Aims The CYLD lysine 63 deubiquitinase gene (CYLD) encodes tumor suppressor protein that is mutated in familial cylindromatosus, and variants have been associated with Crohn disease (CD). Splice forms of CYLD that lack exons 7 and 8 regulate transcription factors and functions of immune cells. We examined the expression of splice forms of CYLD in colon tissues from patients with CD and their effects in mice. Methods We performed immunohistochemical analyses of colon tissues from patients with untreated CD and patients without inflammatory bowel diseases (controls). We obtained mice that expressed splice forms of CYLD (sCYLD mice) without or with SMAD7 (sCYLD/SMAD7 mice) from tr…

0301 basic medicineTranscription FactorBiopsyInbred C57BLTransgenicImmune RegulationSettore MED/12MiceRandom Allocation0302 clinical medicineCrohn DiseaseReference ValuesNeedleIntestinal Mucosaintegumentary systemChemistryBiopsy NeedleGastroenterologyT helper cellFlow CytometryPost-translational ModificationImmunohistochemistryDeubiquitinating Enzyme CYLDCysteine Endopeptidasesmedicine.anatomical_structure030211 gastroenterology & hepatologyTumor necrosis factor alphaSignal TransductionGenetically modified mouseRegulatory T cellTransgeneMice TransgenicSmad7 ProteinTransforming Growth Factor beta103 medical and health sciencesImmune systemmedicineAnimalsHumansCytokine SignalingHepatologyAnimalHEK 293 cellsUbiquitinationMolecular biologyMice Inbred C57BLDisease Models Animal030104 developmental biologyDisease ModelsCytokine Signaling; Immune Regulation; Post-translational Modification; Transcription Factor; Biopsy Needle; Crohn Disease; Cysteine Endopeptidases; Deubiquitinating Enzyme CYLD; Disease Models Animal; Flow Cytometry; Immunohistochemistry; Intestinal Mucosa; Mice Inbred C57BL; Mice Transgenic; Random Allocation; Reference Values; Signal Transduction; Smad7 Protein; Transforming Growth Factor beta1; UbiquitinationTransforming growth factorGastroenterology
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Alterations in Tight- and Adherens-Junction Proteins Related to Glaucoma Mimicked in the Organotypically Cultivated Mouse Retina Under Elevated Press…

2020

Purpose To scrutinize alterations in cellular interactions and cell signaling in the glaucomatous retina, mouse retinal explants were exposed to elevated pressure. Methods Retinal explants were prepared from C57bl6 mice and cultivated in a pressure chamber under normotensive (atmospheric pressure + 0 mm Hg), moderately elevated (30 mm Hg), and highly elevated (60 mm Hg) pressure conditions. The expression levels of proteins involved in the formation of tight junctions (zonula occludens 1 [ZO-1], occludin, and claudin-5) and adherens junctions (VE-cadherin and β-catenin) and in cell-signaling cascades (Cdc42 and activated Cdc42 kinase 1 [ACK1]), as well as the expression levels of the growth…

0301 basic medicineelevated pressureBlotting WesternVimentinReal-Time Polymerase Chain ReactionOccludinRetinaTight JunctionsAdherens junctionMice03 medical and health scienceschemistry.chemical_compoundOrgan Culture Techniques0302 clinical medicineAntigens CDOccludinmedicinecell signalingAnimalscell contactsEye Proteinscdc42 GTP-Binding ProteinReceptorretina explantsmousebeta CateninRetinabiologyTight junctionGlial fibrillary acidic proteinChemistryGlaucomaRetinalAdherens JunctionsProtein-Tyrosine KinasesCadherinsImmunohistochemistryCell biologyMice Inbred C57BLAtmospheric Pressure030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisZonula Occludens-1 Proteinbiology.proteinInvestigative Opthalmology & Visual Science
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RNase H2 Loss in Murine Astrocytes Results in Cellular Defects Reminiscent of Nucleic Acid-Mediated Autoinflammation

2018

Aicardi-Goutières syndrome (AGS) is a rare early onset childhood encephalopathy caused by persistent neuroinflammation of autoimmune origin. AGS is a genetic disorder and >50% of affected individuals bear hypomorphic mutations in ribonuclease H2 (RNase H2). All available RNase H2 mouse models so far fail to mimic the prominent CNS involvement seen in AGS. To establish a mouse model recapitulating the human disease, we deleted RNase H2 specifically in the brain, the most severely affected organ in AGS. Although RNase H2δGFAPmice lacked the nuclease in astrocytes and a majority of neurons, no disease signs were apparent in these animals. We additionally confirmed these results…

0301 basic medicinelcsh:Immunologic diseases. AllergyMaleEncephalomyelitis Autoimmune ExperimentalAicardi–Goutières syndromeRNase PDNA damageImmunologyRibonuclease HFluorescent Antibody TechniqueAicardi-goutières Syndrome ; Cellular Senescence ; Dna Damage ; Interferon Signature ; Rnase H2BiologyNervous System MalformationsAutoimmune Diseases03 medical and health sciencesMiceAutoimmune Diseases of the Nervous SystemNucleic AcidsmedicineImmunology and Allergycellular senescenceAnimalsRibonucleaseNeuroinflammationCells CulturedOriginal ResearchInflammationMice KnockoutInnate immune systemBrainmedicine.diseaseMolecular biologyImmunohistochemistryDisease Models Animal030104 developmental biologymedicine.anatomical_structurePhenotypeinterferon signatureAstrocytesKnockout mousebiology.proteinAicardi–Goutières syndromeDNA damageFemalelcsh:RC581-607RNase H2BiomarkersAstrocyteFrontiers in Immunology
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Migration of Melanoblasts into the Developing Murine Hair Follicle Is Accompanied by Transient c-Kit Expression

2002

Disruption of the c-Kit/stem cell factor (SCF) signaling pathway interferes with the survival, migration, and differentiation of melanocytes during generation of the hair follicle pigmentary unit. We examined c-Kit, SCF, and S100 (a marker for precursor melanocytic cells) expression, as well as melanoblast/melanocyte ultrastructure, in perinatal C57BL/6 mouse skin. Before the onset of hair bulb melanogenesis (i.e., stages 0–4 of hair follicle morphogenesis), strong c-Kit immunoreactivity (IR) was seen in selected non-mela-nogenic cells in the developing hair placode and hair plug. Many of these cells were S100-IR and were ultrastructurally identified as melanoblasts with migratory appearanc…

0301 basic medicinemedicine.medical_specialtyHistologyMorphogenesisStem cell factorBiologyMelanocyteOuter root sheathMice03 medical and health sciencesCell MovementMelanoblastInternal medicineMorphogenesismedicineAnimalsStem Cell Factorintegumentary system030102 biochemistry & molecular biologyStem CellsHair follicleImmunohistochemistryCell biologyMice Inbred C57BLMicroscopy Electron030104 developmental biologyHair follicle morphogenesisDermal papillaemedicine.anatomical_structureEndocrinologyAnimals NewbornMelanocytesAnatomyHair FollicleJournal of Histochemistry & Cytochemistry
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Renal Oncocytomas (Typical and Atypical Variants): A Pathologic, Immunohistochemical, Morphometric, and Flow Cytometric Differential Study of 14 Case…

1996

Eight conventional and six atypical oncocytomas in a series of 147 renal neoplasms were studied. Histopathologic findings revealed exclusively oncocytic cells, but cellular polymorphism was higher in the atypical tumors. Atypical oncocytomas presented focal necrosis, transcapsular invasion, or both. Electron microscopy showed similar findings in all cases. Immunohistochemistry of atypical oncocytomas had higher expression against proliferating cell nuclear antigen and more discontinuous immunostaining against laminin than typical ones. Flow cytometry revealed one or two aneuploid peaks in five typical and two atypical cases, although the latter had a higher proliferative fraction than typi…

0301 basic medicinemedicine.medical_specialtyPathology030102 biochemistry & molecular biologybiologymedicine.diagnostic_testCytogeneticsKaryotypeurologic and male genital diseasesmedicine.diseaseMolecular biologyPathology and Forensic MedicineProliferating cell nuclear antigenRenal neoplasmFlow cytometry03 medical and health sciences030104 developmental biologymedicinebiology.proteinImmunohistochemistrySurgeryOncocytomaAnatomyImmunostainingInternational Journal of Surgical Pathology
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Evaluation of the Ocular Tolerance of Three Tacrolimus Topical Pharmaceutical Preparations by Bovine Corneal Opacity and Permeability Test.

2015

Tacrolimus ocular preparations are commonly employed in autoimmune or inflammatory ocular disorders. However, currently there are not yet approved ocular formulations. Tacrolimus ocular side effects have been reported in clinical use, so the evaluation of different pharmaceutical preparations is mandatory. In this study, the local corneal tolerance and safety profile of three common tacrolimus 0.03% pharmaceutical preparations were evaluated.Corneal irritation and permeability of tacrolimus preparations were evaluated with the bovine corneal opacity and permeability (BCOP) test. Complementary corneal hematoxylin/eosin and immunohistochemistry staining for tight junctions and adherent juncti…

0301 basic medicinemedicine.medical_specialtyPathologygenetic structuresH&E stainCorneal irritationmedicine.disease_causePermeabilityTacrolimusCorneal permeabilityCornea03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineCorneal OpacityOphthalmologymedicineAnimalsDose-Response Relationship Drugbusiness.industryCorneal opacityImmunohistochemistryeye diseasesSensory SystemsTacrolimusOphthalmologySafety profileDisease Models Animal030104 developmental biologyPermeability (electromagnetism)030221 ophthalmology & optometryIrritantsCattlesense organsIrritationOphthalmic SolutionsbusinessImmunosuppressive AgentsCurrent eye research
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