Search results for "Homeostasis"

showing 10 items of 630 documents

Early Low-Fat Diet Enriched With Linolenic Acid Reduces Liver Endocannabinoid Tone and Improves Late Glycemic Control After a High-Fat Diet Challenge…

2016

International audience; Evidence suggests that alterations of glucose and lipid homeostasis induced by obesity are associated with the elevation of endocannabinoid tone. The biosynthesis of the two main endocannabinoids, N-arachidonoylethanolamine and 2-arachidonoyl-glycerol, which derive from arachidonic acid, is influenced by dietary fatty acids (FAs). We investigated whether exposure to n-3 FA at a young age may decrease tissue endocannabinoid levels and prevent metabolic disorders induced by a later high-fat diet (HFD) challenge. Three-week-old mice received a 5% lipid diet containing lard, lard plus safflower oil, or lard plus linseed oil for 10 weeks. Then, mice were challenged with a…

Blood Glucose0301 basic medicinemedicine.medical_specialty[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and MetabolismMice TransgenicCarbohydrate metabolismBiologyDiet High-FatMice03 medical and health scienceschemistry.chemical_compoundInternal medicineInternal MedicinemedicineAnimalsHomeostasisObesityDiet Fat-RestrictedGlycemic2. Zero hungerdiabetesalpha-Linolenic acidBody WeightFatty liveralpha-Linolenic AcidLipid metabolismLipid Metabolismmedicine.diseaseEndocannabinoid system3. Good healthFatty LiverMice Inbred C57BL[SDV.AEN] Life Sciences [q-bio]/Food and Nutrition030104 developmental biologyEndocrinologyLiverchemistryendocananbinoid systemCarbohydrate MetabolismArachidonic acidlipids (amino acids peptides and proteins)Metabolic syndrome[SDV.AEN]Life Sciences [q-bio]/Food and NutritionEndocannabinoids
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Asperuloside Enhances Taste Perception and Prevents Weight Gain in High-Fat Fed Mice

2021

Asperuloside is an iridoid glycoside found in many medicinal plants that has produced promising anti-obesity results in animal models. In previous studies, three months of asperuloside administration reduced food intake, body weight, and adipose masses in rats consuming a high fat diet (HFD). However, the mechanisms by which asperuloside exerts its anti-obesity properties were not clarified. Here, we investigated homeostatic and nutrient-sensing mechanisms regulating food intake in mice consuming HFD. We confirmed the anti-obesity properties of asperuloside and, importantly, we identified some mechanisms that could be responsible for its therapeutic effect. Asperuloside reduced body weight …

Blood GlucoseLeptinMalecannabinoid (CB) receptor 10301 basic medicineTastePro-Opiomelanocortinfood intakeEndocrinology Diabetes and MetabolismAdipose tissueWeight Gainnutrient-sensing mechanismslcsh:Diseases of the endocrine glands. Clinical endocrinologyCyclopentane MonoterpenesEnergy homeostasisMiceEndocrinology0302 clinical medicineGlucosidesWeight lossInsulinasperuloside; cannabinoid (CB) receptor 1; CD36; FFAR1-4; food intake; nutrient-sensing mechanisms; TAS1R2-3; weight lossReceptorOriginal ResearchLeptindigestive oral and skin physiologyTaste PerceptionGhrelinTAS1R2-3Ghrelinmedicine.symptommedicine.medical_specialtyHypothalamusBiologyDiet High-Fatasperuloside03 medical and health sciencesInternal medicinemedicineAnimalsPyranslcsh:RC648-665Body WeightFFAR1-4030104 developmental biologyEndocrinologyAnti-Obesity Agentsweight lossEnergy IntakeCD36Weight gain030217 neurology & neurosurgery
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Temporal and tissue-specific requirements for T-lymphocyte IL-6 signalling in obesity-associated inflammation and insulin resistance

2017

Low-grade inflammation links obesity to insulin resistance through the activation of tissue-infiltrating immune cells. Interleukin-6 (IL-6) is a crucial regulator of T cells and is increased in obesity. Here we report that classical IL-6 signalling in T cells promotes inflammation and insulin resistance during the first 8 weeks on a high-fat diet (HFD), but becomes dispensable at later stages (after 16 weeks). Mice with T cell-specific deficiency of IL-6 receptor-α (IL-6RαT-KO) exposed to a HFD display improved glucose tolerance, insulin sensitivity and inflammation in liver and EWAT after 8 weeks. However, after 16 weeks, insulin resistance in IL-6RαT-KO epididymal white adipose tissue (EW…

Blood GlucoseMale0301 basic medicinemedicine.medical_specialtyTime FactorsT-LymphocytesT cellScienceGeneral Physics and AstronomyInflammationWhite adipose tissueBiologyDiet High-FatArticleGeneral Biochemistry Genetics and Molecular BiologyMice03 medical and health sciences0302 clinical medicineInsulin resistanceImmune systemInternal medicinemedicineAnimalsHomeostasisObesityReceptorInflammationMice KnockoutMultidisciplinaryInterleukin-6QGeneral ChemistryT lymphocyteLipid Metabolismmedicine.diseaseReceptors Interleukin-6030104 developmental biologymedicine.anatomical_structureEndocrinology030220 oncology & carcinogenesisInsulin Resistancemedicine.symptomHomeostasisSignal TransductionNature Communications
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Evidence for hypothalamic ketone bodies sensing: impact on food intake and peripheral metabolic responses in mice

2016

Monocarboxylates have been implicated in the control of energy homeostasis. Among them, the putative role of ketone bodies produced notably during high-fat diet (HFD) has not been thoroughly explored. In this study, we aimed to determine the impact of a specific rise in cerebral ketone bodies on food intake and energy homeostasis regulation. A carotid infusion of ketone bodies was performed on mice to stimulate sensitive brain areas for 6 or 12 h. At each time point, food intake and different markers of energy homeostasis were analyzed to reveal the consequences of cerebral increase in ketone body level detection. First, an increase in food intake appeared over a 12-h period of brain keton…

Blood GlucoseMale0301 basic medicineobesitynervous-systemPhysiology[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and MetabolismKetone BodiesEnergy homeostasisEatingMicebodiesHomeostasisGlucose homeostasisoxidative stressAgouti-Related ProteinNeuropeptide YPhosphorylationmonocarboxylate transporters2. Zero hunger[ SDV.MHEP.PHY ] Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]fat massHypothalamusKetone bodiesStarvation responseketogenic mediterranean dietweight-lossmedicine.medical_specialtybeta-hydroxybutyrateHypothalamusBiologyDiet High-Fat03 medical and health sciencesInsulin resistancerat-brainPhysiology (medical)Internal medicinemedicine[SDV.MHEP.PHY]Life Sciences [q-bio]/Human health and pathology/Tissues and Organs [q-bio.TO]Animalsglucose homeostasisAdenylate Kinase/metabolism; Agouti-Related Protein/metabolism; Animals; Blood Glucose; Diet High-Fat; Eating/drug effects; Eating/physiology; Energy Metabolism/drug effects; Energy Metabolism/physiology; Gluconeogenesis/drug effects; Gluconeogenesis/physiology; Homeostasis; Hypothalamus/drug effects; Hypothalamus/metabolism; Insulin Resistance/physiology; Ketone Bodies/pharmacology; Male; Mice; Mice Inbred C57BL; Neuropeptide Y/metabolism; Phosphorylation/drug effectsenergy homeostasisAdenylate KinaseGluconeogenesismedicine.diseaseMice Inbred C57BL030104 developmental biologyEndocrinologyGluconeogenesislow-carbohydrateInsulin ResistanceEnergy Metabolism[SDV.AEN]Life Sciences [q-bio]/Food and NutritionHomeostasis
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Acute activation of cannabinoid receptors by anandamide reduces gastrointestinal motility and improves postprandial glycemia in mice.

2015

International audience; The endocannabinoid system (ECS) is associated with an alteration of glucose homeostasis dependent on cannabinoid receptor-1 (CB1R) activation. However, very little information is available concerning the consequences of ECS activation on intestinal glucose absorption. Mice were injected intraperitoneally with anandamide, an endocannabinoid binding both CB1R and CB2R. We measured plasma glucose and xylose appearance after oral loading, gastrointestinal motility, and glucose transepithelial transport using the everted sac method. Anandamide improved hyperglycemia after oral glucose charge whereas glucose clearance and insulin sensitivity were impaired, pointing out so…

Blood GlucoseMaleIndolesCannabinoid receptorMESH : Piperidines[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and Metabolismmedicine.medical_treatmentMESH: EndocannabinoidsMESH : PyrazolesMESH : Receptors CannabinoidMicechemistry.chemical_compoundPiperidinesMESH : IndolesMESH: Receptors CannabinoidMESH: Reverse Transcriptase Polymerase Chain ReactionMESH : Arachidonic AcidsGlucose homeostasisMESH: Gastrointestinal TransitMESH: AnimalsReceptors CannabinoidMESH: IndolesReverse Transcriptase Polymerase Chain ReactionMESH : RatsMESH : Reverse Transcriptase Polymerase Chain ReactionAnandamidePostprandial PeriodEndocannabinoid systemMESH : Gastrointestinal MotilityPostprandialMESH: PiperidinesMESH: Postprandial PeriodMESH: Gastrointestinal MotilityRimonabantMESH : EndocannabinoidsMESH : Gastrointestinal Transitmedicine.medical_specialtyMESH: RatsPolyunsaturated AlkamidesMESH : MaleArachidonic AcidsMESH : Mice Inbred C57BLMESH : Rats WistarMESH: Mice Inbred C57BLInternal medicineMESH : MiceInternal MedicinemedicineAnimalsMESH: Arachidonic AcidsMESH : Polyunsaturated AlkamidesRats WistarGastrointestinal TransitMESH: MiceGastric emptyingMESH: Polyunsaturated AlkamidesGlucose transporterMESH: Rats WistarMESH : Blood GlucoseMESH: MaleRatsMice Inbred C57BL[SDV.AEN] Life Sciences [q-bio]/Food and NutritionEndocrinologychemistryHyperglycemiaMESH : HyperglycemiaMESH: Blood GlucosePyrazolesMESH : AnimalsCannabinoidMESH : Postprandial PeriodGastrointestinal MotilityMESH: Hyperglycemia[SDV.AEN]Life Sciences [q-bio]/Food and NutritionMESH: PyrazolesEndocannabinoids
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Hypothalamic reactive oxygen species are required for insulin-induced food intake inhibition: an NADPH oxidase-dependent mechanism

2009

1939-327X (Electronic) Journal Article Research Support, Non-U.S. Gov't; OBJECTIVE: Insulin plays an important role in the hypothalamic control of energy balance, especially by reducing food intake. Emerging data point to a pivotal role of reactive oxygen species (ROS) in energy homeostasis regulation, but their involvement in the anorexigenic effect of insulin is unknown. Furthermore, ROS signal derived from NADPH oxidase activation is required for physiological insulin effects in peripheral cells. In this study, we investigated the involvement of hypothalamic ROS and NADPH oxidase in the feeding behavior regulation by insulin. RESEARCH DESIGN AND METHODS: We first measured hypothalamic RO…

Blood GlucoseMaleReactive Oxygen Species/*metabolismHypothalamusHomeostasis/drug effects/physiologyInbred C57BLCerebral VentriclesCerebral Ventricles/drug effects/*physiologyMiceHomeostasisInsulinAnimalsBlood Glucose/metabolismHypothalamus/*physiologyInsulin/administration & dosage/blood/*pharmacologyNADPH OxidasesEnergy Intake/drug effects/*physiologyNADPH Oxidase/*metabolismGlutathioneGlutathione/metabolismMice Inbred C57BLOriginal ArticleEnergy IntakeReactive Oxygen SpeciesEnergy MetabolismSignal Transduction
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GLP-2 as Beneficial Factor in the Glucose Homeostasis in Mice Fed a High Fat Diet

2015

Glucagon like peptide-2 (GLP-2) is a gastrointestinal hormone released in response to dietary nutrients, which acts through a specific receptor, the GLP-2 receptor (GLP-2R). The physiological effects of GLP-2 are multiple, involving also the intestinal adaptation to high fat diet (HFD). In consideration of the well-known relationship between chronic HFD and impaired glucose metabolism, in the present study we examined if the blocking of the GLP-2 signaling by chronic treatment with the GLP-2R antagonist, GLP-2 (3-33), leads to functional consequences in the regulation of glucose metabolism in HFD-fed mice. Compared with animals fed standard diet (STD), mice at the 10th week of HFD showed hy…

Blood GlucoseMaleTime FactorsDiet High-FatPeptide FragmentsMice Inbred C57BLDisease Models AnimalHormone Antagonistsobesity insulin resistance pancreatic isletsInsulin-Secreting CellsGlucagon-Like Peptide 2AnimalsHomeostasisInsulinGLP-2; obesity insulin resistance pancreatic isletsGLP-2BiomarkersGlucose Metabolism DisordersSignal Transduction
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The Impact of Amino Acids on Postprandial Glucose and Insulin Kinetics in Humans

2020

Different amino acids (AAs) may exert distinct effects on postprandial glucose and insulin concentrations. A quantitative comparison of the effects of AAs on glucose and insulin kinetics in humans is currently lacking. PubMed was queried to identify intervention studies reporting glucose and insulin concentrations after acute ingestion and/or intravenous infusion of AAs in healthy adults and those living with obesity and/or type 2 diabetes (T2DM). The systematic literature search identified 55 studies that examined the effects of l-leucine, l-isoleucine, l-alanine, l-glutamine, l-arginine, l-lysine, glycine, l-proline, l-phenylalanine, l-glutamate, branched-chain AAs (i.e., l-leucine, l-iso…

Blood GlucoseMaleinsulin secretionobesitymedicine.medical_treatmentAdministration OralReviewType 2 diabetes0302 clinical medicinesystematic reviewInsulinIngestionGlucose homeostasis030212 general & internal medicineInfusions IntravenousNutrition and DieteticsL-ARGININEINTRAVENOUS ARGININEFREE FATTY-ACIDSBLOOD-GLUCOSEdynamicsPostprandial PeriodPostprandialSECRETIONFemaletype 2 diabetesLeucineGROWTH-HORMONElcsh:Nutrition. Foods and food supplyAdultORAL ALANINEmedicine.medical_specialtyMETABOLIC-RESPONSElcsh:TX341-641030209 endocrinology & metabolism03 medical and health sciencesInternal medicinemedicineHumansglucose homeostasisinsulin sensitivityamino acidsbusiness.industryInsulinmedicine.diseaseObesityGlucoseEndocrinologyDiabetes Mellitus Type 2kineticsPLASMA-GLUCAGON RESPONSEtime series dataIsoleucinebusinessFood ScienceINGESTIONNutrients
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Cellular ionic alterations with age: relation to hypertension and diabetes.

2000

BACKGROUND: Cytosolic free calcium (Cai) and magnesium (Mgi) are vital to cellular homeostasis and function. OBJECTIVE: To evaluate cellular divalent cations in normal subjects at different ages and their relationship to ion levels in essential hypertension and diabetes. DESIGN: A cross-sectional study. SETTING: A university hospital in New York. PARTICIPANTS: A total of 103 subjects (32 older, 71.1 ± 1.2 y/o, and 71 young/middle aged subjects, 51.1 ± 2.3 y/o). INTERVENTION: Oral glucose tolerance test. MEASUREMENTS: 19F and 31P NMR spectroscopy were used to measure Cai and Mgi levels in erythrocytes from normal (>65 y/o, n = 11; 65 y/o, n = 9; 65 y/o, n = 12; <65 y/o, n = 15) subjects; the…

Blood GlucoseMalemedicine.medical_specialtyAgingErythrocytesMagnetic Resonance Spectroscopymedicine.medical_treatmentCellular homeostasisEssential hypertensionCytosolInternal medicineDiabetes mellitusmedicineHumansInsulinMagnesiumMagnesium ionAgedAged 80 and overGlucose tolerance testmedicine.diagnostic_testbusiness.industryInsulinAge FactorsGlucose Tolerance TestMiddle Agedmedicine.diseaseCausalityEndocrinologyGlycemic indexBlood pressureCross-Sectional StudiesDiabetes Mellitus Type 2Case-Control StudiesHypertensionCalciumFemaleGeriatrics and GerontologybusinessJournal of the American Geriatrics Society
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One-hour post-load plasma glucose level is associated with a worse metabolic profile in children with GH deficiency

2017

Purpose: In children, the plasma glucose value at 1 h (1hPG) during OGTT higher than 132.5 mg/dl is a predictor of alterations in glucose metabolism. We aimed to metabolically characterize GHD children according to 1hPG levels. Methods: Fifty-one GHD children (35 M, 16 F; mean age 8.6 years), grouped according to 1hPG, were evaluated at diagnosis and after 12 months of GH treatment (GHT) and compared with 50 matched controls at baseline. Auxological parameters, insulin-like growth factor-1 (IGF-1), glucose and insulin during OGTT, lipid profile, the oral disposition index (DIo), the homeostasis model assessment estimate of insulin resistance (Homa-IR), and the insulin sensitivity index (ISI…

Blood GlucoseMalemedicine.medical_specialtyTime FactorsTime FactorEndocrinology Diabetes and Metabolismmedicine.medical_treatment030209 endocrinology & metabolism030204 cardiovascular system & hematologyCarbohydrate metabolismSettore MED/13 - Endocrinologia03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyInsulin resistanceGH treatmentInternal medicineInsulin SecretionHumansInsulinMedicineChildDwarfism PituitaryGrowth hormoneGlucose tolerance testmedicine.diagnostic_testHuman Growth Hormonebusiness.industryCholesterolInsulinCase-control studynutritional and metabolic diseasesGlucose Tolerance TestInsulin sensitivitymedicine.diseaseMetabolismEndocrinologychemistryCase-Control StudiesChild PreschoolMetabolomeFemaleInsulin ResistanceCase-Control StudiebusinessLipid profileHomeostasisHumanJournal of Endocrinological Investigation
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