Search results for "Host-Pathogen Interaction"

showing 10 items of 189 documents

Experimental virus evolution reveals a role of plant microtubule dynamics and TORTIFOLIA1/SPIRAL2 in RNA trafficking.

2014

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ArabidopsisPlant ScienceMicrotubulesRNA Transport//purl.org/becyt/ford/1 [https]INFECTIONTobacco mosaic virusTOBACCO-MOSAIC-VIRUSMovement proteinCytoskeletonCytoskeletonGeneticsCoat proteinMultidisciplinaryTRANSGENIC PLANTSQREXPERIMENTAL EVOLUTIONARABIDOPSISBiological Evolution3. Good healthCell biologyMacromolecular assemblyTobacco Mosaic VirusMICROTUBULESMedical MicrobiologyTobamovirusesViral Pathogensdynamic plasticityHost-Pathogen InteractionsMedicineTobacco mosaic viruscortical microtubuleCellular Structures and OrganellesCortical microtubuleARABIDOPSIS CORTICAL MICROTUBULESCell wallsMicrotubule-Associated ProteinsCIENCIAS NATURALES Y EXACTASResearch ArticleEvolutionary ProcessesSciencePlant Cell BiologyPlant PathogensORGANIZATIONBiologyMicrobiologyPlant Viral PathogensCiencias BiológicasMOVEMENT PROTEINComplexesMicrotubuleEvolutionary Adaptation//purl.org/becyt/ford/1.6 [https]Microbial PathogensPlant DiseasesEvolutionary BiologyArabidopsis ProteinsBotánicaRNABiology and Life SciencesCell BiologyPlant PathologyTMVCytoplasmMutationRNAVirologíaHELICAL GROWTHPloS one
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Invasion Ability and Disease Dynamics of Environmentally Growing Opportunistic Pathogens under Outside-Host Competition

2014

Most theories of the evolution of virulence concentrate on obligatory host-pathogen relationship. Yet, many pathogens replicate in the environment outside-host where they compete with non-pathogenic forms. Thus, replication and competition in the outside-host environment may have profound influence on the evolution of virulence and disease dynamics. These environmentally growing opportunistic pathogens are also a logical step towards obligatory pathogenicity. Efficient treatment methods against these diseases, such as columnaris disease in fishes, are lacking because of their opportunist nature. We present a novel epidemiological model in which replication and competition in the outside-hos…

Bacterial Diseases0106 biological sciencesPopulation ModelingDisease01 natural sciencesTheoretical EcologyMedicine and Health SciencesPathogenPOPULATIONmedia_common0303 health scienceseducation.field_of_studyPREDATIONMultidisciplinaryEcologyTransmission (medicine)EcologySimulation and ModelingQRCHANNEL CATFISHEVOLUTIONARY DYNAMICSCOMMUNITYInfectious DiseasesHost-Pathogen Interactions1181 Ecology evolutionary biologyMedicineAlgorithmsResearch ArticleTRANSMISSIONSciencemedia_common.quotation_subjecteducationPopulationVirulenceOpportunistic InfectionsBiologyResearch and Analysis Methods010603 evolutionary biologyCompetition (biology)03 medical and health sciences14. Life underwaterParasite EvolutioneducationEvolutionary dynamicsta413030304 developmental biologyEvolutionary BiologyMathematical ModelingSTABILITYMORTALITYEcology and Environmental SciencesBiology and Life SciencesComputational BiologyFLAVOBACTERIUM-COLUMNAREOutbreakModels TheoreticalEmerging Infectious DiseasesEvolutionary Ecologyta1181VIRULENCEParasitologyInfectious Disease ModelingPLoS ONE
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Type-IIA secreted phospholipase A2 is an endogenous antibiotic-like protein of the host.

2010

International audience; Type-IIA secreted phospholipase A(2) (sPLA(2)-IIA) has been proposed to play a role in the development of inflammatory diseases. It has been shown to release arachidonic acid, the precursor of proinflammatory eicosanoids, to hydrolyze phospholipids of pulmonary surfactant, and to bind to specific receptors located on cell surface membranes. However, the most established biological role of sPLA(2)-IIA is related to its potent bactericidal property in particular toward Gram-positive bacteria. This enzyme is present in animal and human biological fluids at concentrations sufficient to kill bacteria. Human recombinant sPLA(2)-IIA is able to kill Gram-positive bacteria at…

Bacterial Toxinsmedicine.disease_causeGroup II Phospholipases A2BiochemistryMicrobiologyAnthraxMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineImmune systemPhospholipase A2PhosphatidylcholinemedicineAnimalsHumansEscherichia coli030304 developmental biologyAntigens Bacterial0303 health sciencesPhospholipase AArachidonic AcidbiologyDrug Resistance MicrobialPathogenic bacteriaGeneral Medicinebiology.organism_classificationAnti-Bacterial Agents3. Good healthBacillus anthracisBiochemistrychemistryBacillus anthracisHost-Pathogen Interactionsbiology.protein[SDV.IMM.VAC]Life Sciences [q-bio]/Immunology/VaccinologyBacteria030215 immunology
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Host–virus evolutionary dynamics with specialist and generalist infection strategies: Bifurcations, bistability, and chaos

2019

In this work, we have investigated the evolutionary dynamics of a generalist pathogen, e.g., a virus population, that evolves toward specialization in an environment with multiple host types. We have particularly explored under which conditions generalist viral strains may rise in frequency and coexist with specialist strains or even dominate the population. By means of a nonlinear mathematical model and bifurcation analysis, we have determined the theoretical conditions for stability of nine identified equilibria and provided biological interpretation in terms of the infection rates for the viral specialist and generalist strains. By means of a stability diagram, we identified stable fixed…

BistabilityPopulationGeneral Physics and AstronomyDynamical Systems (math.DS)Fixed pointParameter spaceBiologyGeneralist and specialist speciesModels Biological01 natural sciencesStability (probability)010305 fluids & plasmas0103 physical sciencesFOS: MathematicsHumansQuantitative Biology::Populations and EvolutionComputer SimulationMathematics - Dynamical SystemsQuantitative Biology - Populations and Evolution010306 general physicsEvolutionary dynamicseducationMathematical Physicseducation.field_of_studyApplied MathematicsDegenerate energy levelsPopulations and Evolution (q-bio.PE)Statistical and Nonlinear Physics3. Good healthNonlinear DynamicsEvolutionary biologyFOS: Biological sciencesHost-Pathogen InteractionsVirusesVirus Physiological Phenomena
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HCV genotype 5: an orphan virus

2013

HCV genotype 5 (HCV-5) is the least known HCV genotype. It is found mainly in South Africa and in restricted areas of Belgium, Spain, France, Syria and Greece. Sporadic cases are reported worldwide. The main modes of transmission are blood transfusion and iatrogenic causes. Little is known about its origin, but various studies have elucidated its spread worldwide. In endemic areas, patients infected with HCV-5 are on average older and have a higher viral load and more advanced fibrosis than those infected with non-HCV-5 genotypes. The current standard of care for HCV-5 chronic infection is 48 weeks of dual therapy with pegylated interferon plus ribavirin. ‘Favourable’ Il28B polymorphisms a…

Blood transfusionGenotypemedicine.medical_treatmentHepacivirusAntiviral Agentschemistry.chemical_compoundPegylated interferonGenotypemedicinePrevalenceHumansPharmacology (medical)PharmacologyTransmission (medicine)business.industryRibavirinvirus diseasesHepatitis Cmedicine.diseaseHepatitis Cdigestive system diseasesChronic infectionInfectious DiseasesTreatment OutcomechemistryImmunologyHost-Pathogen InteractionsbusinessViral loadmedicine.drug
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Stochastic Episodes of Latent Cytomegalovirus Transcription Drive CD8 T-Cell “Memory Inflation” and Avoid Immune Evasion

2021

Acute infection with murine cytomegalovirus (mCMV) is controlled by CD8+ T cells and develops into a state of latent infection, referred to as latency, which is defined by lifelong maintenance of viral genomes but absence of infectious virus in latently infected cell types. Latency is associated with an increase in numbers of viral epitope-specific CD8+ T cells over time, a phenomenon known as “memory inflation” (MI). The “inflationary” subset of CD8+ T cells has been phenotyped as KLRG1+CD62L- effector-memory T cells (iTEM). It is agreed upon that proliferation of iTEM requires repeated episodes of antigen presentation, which implies that antigen-encoding viral genes must be transcribed du…

CD4-Positive T-LymphocytesGene Expression Regulation Viral0301 basic medicineMuromegaloviruslatent infectionTime FactorsTranscription Geneticeffector memory CD8+ T cellsAntigen presentationImmunologyBiologyVirusImmediate-Early Proteins03 medical and health sciences0302 clinical medicineImmune systemImmunityAnimalsCytotoxic T cellImmunology and AllergyLatency (engineering)Antigens ViralLungGenememory inflationlatencyOriginal Researchimmune evasionMice Inbred BALB CStochastic ProcessesModels ImmunologicalHerpesviridae InfectionsRC581-607VirologyVirus LatencyDisease Models Animalvirus reactivationantigen presentationPhenotype030104 developmental biologyHost-Pathogen Interactionsgene expressionFemaleVirus ActivationImmunologic diseases. AllergyImmunologic MemoryCD8030215 immunologyFrontiers in Immunology
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Evolutionary convergence and nitrogen metabolism in Blattabacterium strain Bge, primary endosymbiont of the cockroach Blattella germanica.

2009

Bacterial endosymbionts of insects play a central role in upgrading the diet of their hosts. In certain cases, such as aphids and tsetse flies, endosymbionts complement the metabolic capacity of hosts living on nutrient-deficient diets, while the bacteria harbored by omnivorous carpenter ants are involved in nitrogen recycling. In this study, we describe the genome sequence and inferred metabolism of Blattabacterium strain Bge, the primary Flavobacteria endosymbiont of the omnivorous German cockroach Blattella germanica. Through comparative genomics with other insect endosymbionts and free-living Flavobacteria we reveal that Blattabacterium strain Bge shares the same distribution of functio…

Cancer Researchfood.ingredientlcsh:QH426-470NitrogenBlochmanniaZoologyCockroachesEvolution Molecular03 medical and health sciencesBlattabacteriumfoodSymbiosisEnterobacteriaceaePhylogeneticsAmmoniabiology.animalBotanyGeneticsAnimalsAmino AcidsSymbiosisMolecular BiologyGenetics (clinical)Ecology Evolution Behavior and SystematicsPhylogeny030304 developmental biology0303 health sciencesGerman cockroachCockroachbiologyPhylogenetic treeEvolutionary Biology/Evolutionary and Comparative Genetics030306 microbiologyAntsBacteroidetesfungiGenomicsbiochemical phenomena metabolism and nutritionbiology.organism_classificationGenetics and Genomics/Microbial Evolution and Genomicslcsh:GeneticsGenetics and Genomics/Genome ProjectsEvolutionary Biology/Microbial Evolution and GenomicsHost-Pathogen InteractionsBacteriaGenome BacterialMetabolic Networks and PathwaysResearch ArticlePLoS genetics
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The transcription factor Interferon Regulatory Factor 4 is required for the generation of protective effector CD8 + T cells

2013

Robust cytotoxic CD8 + T-cell response is important for immunity to intracellular pathogens. Here, we show that the transcription factor IFN Regulatory Factor 4 (IRF4) is crucial for the protective CD8 + T-cell response to the intracellular bacterium Listeria monocytogenes . IRF4-deficient ( Irf4 −/− ) mice could not clear L. monocytogenes infection and generated decreased numbers of L. monocytogenes -specific CD8 + T cells with impaired effector phenotype and function. Transfer of wild-type CD8 + T cells into Irf4 −/− mice improved bacterial clearance, suggesting an intrinsic defect of CD8 + T cells in Irf4 −/− mice. Following transfer into wild-type recipients, Irf4 −/− CD8 + T cells bec…

Cellular differentiationGene ExpressionEomesoderminBiologyMiceInterleukin 21AnimalsCytotoxic T cellListeriosisIL-2 receptorAntigen-presenting cellSTAT4Cell ProliferationMice KnockoutMultidisciplinaryCell DifferentiationBiological SciencesListeria monocytogenesMolecular biologyMice Inbred C57BLHost-Pathogen InteractionsInterferon Regulatory FactorsImmunologyPositive Regulatory Domain I-Binding Factor 1CD8T-Lymphocytes CytotoxicTranscription FactorsProceedings of the National Academy of Sciences
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Cellular interactions ofCandida albicanswith human oral epithelial cells and enterocytes

2010

The human pathogenic fungus Candida albicans can cause systemic infections by invading epithelial barriers to gain access to the bloodstream. One of the main reservoirs of C. albicans is the gastrointestinal tract and systemic infections predominantly originate from this niche. In this study, we used scanning electron and fluorescence microscopy, adhesion, invasion and damage assays, fungal mutants and a set of fungal and host cell inhibitors to investigate the interactions of C. albicans with oral epithelial cells and enterocytes. Our data demonstrate that adhesion, invasion and damage by C. albicans depend not only on fungal morphology and activity, but also on the epithelial cell type an…

Cellular differentiationImmunologyEndocytosisMicrobiologyMicrobiologyCell Line TumorVirologyCandida albicansmedicineHumansCandida albicansbiologyCell DifferentiationEpithelial CellsPathogenic fungusbiology.organism_classificationEpitheliumCorpus albicansCell biologyEnterocytesmedicine.anatomical_structureMicroscopy FluorescenceCaco-2Cell cultureHost-Pathogen InteractionsMicroscopy Electron ScanningCaco-2 CellsCellular Microbiology
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Validation of the Tetracycline Regulatable Gene Expression System for the Study of the Pathogenesis of Infectious Disease

2011

Understanding the pathogenesis of infectious disease requires the examination and successful integration of parameters related to both microbial virulence and host responses. As a practical and powerful method to control microbial gene expression, including in vivo, the tetracycline-regulatable system has recently gained the favor of many investigative groups. However, some immunomodulatory effects of the tetracyclines, including doxycycline, could potentially limit its use to evaluate host responses during infection. Here we have used a well-established murine model of disseminated candidiasis, which is highly dependent on both the virulence displayed by the fungal cells and on the host im…

ChemokineScienceImmunologyVirulenceMycologyPathogenesisKidneyResponse ElementsMicrobiologyMicrobiologyPathogenesisMiceGene Expression Regulation FungalCandida albicansGene expressionmedicineAnimalsPromoter Regions GeneticCandida albicansBiologyImmunity to InfectionsProtein Synthesis InhibitorsDoxycyclineMultidisciplinaryVirulencebiologyQCandidiasisImmunityRTetracyclinebiology.organism_classificationDisseminated CandidiasisDisease Models AnimalInfectious DiseasesMedical MicrobiologyInfectious disease (medical specialty)DoxycyclineHost-Pathogen InteractionsMutationImmunologybiology.proteinCytokinesMedicineChemokinesSpleenResearch Articlemedicine.drug
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