Search results for "Host-pathogen interaction"

showing 10 items of 189 documents

Respiratory syncytial virus inhibits ciliagenesis in differentiated normal human bronchial epithelial cells: effectiveness of N-acetylcysteine.

2012

Persistent respiratory syncytial virus (RSV) infections have been associated with the exacerbation of chronic inflammatory diseases, including chronic obstructive pulmonary disease (COPD). This virus infects the respiratory epithelium, leading to chronic inflammation, and induces the release of mucins and the loss of cilia activity, two factors that determine mucus clearance and the increase in sputum volume. These alterations involve reactive oxygen species-dependent mechanisms. The antioxidant N-acetylcysteine (NAC) has proven useful in the management of COPD, reducing symptoms, exacerbations, and accelerated lung function decline. NAC inhibits RSV infection and mucin release in human A54…

Viral DiseasesPulmonologyChronic Obstructive Pulmonary Diseaseslcsh:MedicineMucin 5ACVirus ReplicationAcetylcysteinePulmonary Disease Chronic ObstructiveTubulinRespiratory systemlcsh:ScienceCells CulturedMultidisciplinaryInterleukin-13Microscopy VideoCell DifferentiationForkhead Transcription FactorsFree Radical Scavengersrespiratory systemHost-Pathogen InteractionLower Respiratory Tract InfectionsInfectious Diseasesmedicine.anatomical_structureInterleukin 13Medicinemedicine.symptomResearch Articlemedicine.drugDrugs and DevicesInflammationBronchiRespiratory Syncytial Virus InfectionsBiologyMicrobiologyAntiviral AgentsUpper Respiratory Tract InfectionsmedicineHumansCiliaBiologyInflammationRespiratory Syncytial Virus InfectionA549 cellMucinlcsh:RImmunityEpithelial CellsAxonemal DyneinsEpitheliumAcetylcysteineGene Expression RegulationRespiratory Syncytial Virus HumanRespiratory InfectionsImmunologyRespiratory epitheliumlcsh:QPLoS ONE
researchProduct

Lepidopteran species have a variety of defence strategies against bacterial infections

2017

The insect immune system has versatile ways of coping with microbial insults. Currently, innate immune priming has been described in several invertebrates, and the first insights into its mechanistic basis have been described. Here we studied infections with two different strains of Serratia marcescens bacteria in two different Lepidopteran hosts. The results reveal fundamental differences between the two hosts, a well-known model organism Galleria mellonella and a non-model species Arctia plantaginis. They differ in their strategies for resisting oral infections; priming their defences against a recurring sepsis; and upregulating immunity related genes as a response to the specific pathoge…

0301 basic medicine6-Toximmune priming030106 microbiologyVirulenceMicrobiologyresistance03 medical and health sciencesImmune systemSpecies SpecificityImmunityisovahakoisaAnimalsimmuniteettigeeniekspressioArctia plantaginisPathogenDefensinEcology Evolution Behavior and SystematicsSerratia marcescensCecropinInnate immune systemtolerancebiologyfungibacterial infectionvirulenssibiology.organism_classificationimmunityextracellular proteaseLepidopteravirulenceGalleria mellonella030104 developmental biologyGalleria mellonellaDefensinHost-Pathogen InteractionsSerratia marcescensgene expressionta1181
researchProduct

Susceptibility of Spodoptera frugiperda and S. exigua to Bacillus thuringiensis Vip3Aa insecticidal protein

2011

The Vip3Aa protein is an insecticidal protein secreted by Bacillus thuringiensis during the vegetative stage of growth. The activity of this protein has been tested after different steps/protocols of purification using Spodoptera frugiperda as a control insect. The results showed that the Vip3Aa protoxin was stable and retained full toxicity after being subjected to common biochemical steps used in protein purification. Bioassays with the protoxin in S. frugiperda and S. exigua showed pronounced differences in LC(50) values when mortality was measured at 7 vs. 10d. At 7d most live larvae were arrested in their development. LC(50) values of "functional mortality" (dead larvae plus larvae rem…

InsecticidesLongevityBacillus thuringiensisInsect pest controlInsect proteaseBacterial ProteinSpodopteraSpodopteraMedian lethal doseMicrobiologyLethal Dose 50Vegetative insecticidal proteinBacterial ProteinsSpecies SpecificityBacillus thuringiensisparasitic diseasesExiguaProtein purificationBotanyAnimalsBacillus thuringiensiBioassayPest Control BiologicalInsecticideEcology Evolution Behavior and SystematicsbiologyAnimalfungiMidgutbiology.organism_classificationBioinsecticideHost-Pathogen InteractionMode of actionLarvaHost-Pathogen InteractionsInstarBiological AssayElectrophoresis Polyacrylamide GelDisease SusceptibilityJournal of Invertebrate Pathology
researchProduct

The Cytoskeletal Adaptor Obscurin-Like 1 Interacts with the Human Papillomavirus 16 (HPV16) Capsid Protein L2 and Is Required for HPV16 Endocytosis.

2016

ABSTRACT The human papillomavirus (HPV) capsid protein L2 is essential for viral entry. To gain a deeper understanding of the role of L2, we searched for novel cellular L2-interacting proteins. A yeast two-hybrid analysis uncovered the actin-depolymerizing factor gelsolin, the membrane glycoprotein dysadherin, the centrosomal protein 68 (Cep68), and the cytoskeletal adaptor protein obscurin-like 1 protein (OBSL1) as putative L2 binding molecules. Pseudovirus (PsV) infection assays identified OBSL1 as a host factor required for gene transduction by three oncogenic human papillomavirus types, HPV16, HPV18, and HPV31. In addition, we detected OBSL1 expression in cervical tissue sections and no…

0301 basic medicineKeratinocytesvirusesImmunologyEndocytic cycleEndocytosisMicrobiologyClathrinCell Line03 medical and health sciencesTransduction (genetics)TetraspaninViral entryVirologyTwo-Hybrid System TechniquesCaveolinHumansHuman papillomavirus 16biologyPapillomavirus InfectionsSignal transducing adaptor proteinOncogene Proteins ViralVirus InternalizationEndocytosisCell biologyVirus-Cell InteractionsCytoskeletal Proteins030104 developmental biologyInsect ScienceGene Knockdown TechniquesHost-Pathogen Interactionsbiology.proteinCapsid ProteinsHeLa CellsJournal of virology
researchProduct

Diabetes mellitus association with coronavirus disease 2019 ( COVID ‐19) severity and mortality: A pooled analysis

2020

Highlights There are ~ 2-fold increased odds of severe coronavirus disease 2019 (COVID-19) and a ~ 2-fold increased risk of odds of mortality in patients with history of diabetes mellitus compared to those without diabetes mellitus. Patients with a history of diabetes mellitus should be closely monitored if they get infected with COVID-19. Results of meta-analysis showing association of diabetes mellitus with severity (Panel A) of disease and mortality (Panel B) in coronavirus disease 2019 (COVID-19) patients.

AdultMalemedicine.medical_specialtyTime FactorsAdolescentendocrine system diseasesEndocrinology Diabetes and MetabolismPneumonia Viralcoronavirus030209 endocrinology & metabolismDisease030204 cardiovascular system & hematologymedicine.disease_causeRisk AssessmentSeverity of Illness IndexCOVID-19 coronavirus diabetes mellitusBetacoronavirusYoung Adult03 medical and health sciences0302 clinical medicineCOVID‐19Risk FactorsCause of DeathInternal medicineDiabetes mellitusPandemicSeverity of illnessResearch LetterHumansMedicineYoung adultPandemicsAgedCoronavirusCause of deathAged 80 and overSARS-CoV-2business.industryCOVID-19Middle AgedPrognosismedicine.diseaseResearch LettersHost-Pathogen Interactionsdiabetes mellitusFemaleCoronavirus InfectionsbusinessRisk assessmentJournal of Diabetes
researchProduct

The transcription factor Interferon Regulatory Factor 4 is required for the generation of protective effector CD8 + T cells

2013

Robust cytotoxic CD8 + T-cell response is important for immunity to intracellular pathogens. Here, we show that the transcription factor IFN Regulatory Factor 4 (IRF4) is crucial for the protective CD8 + T-cell response to the intracellular bacterium Listeria monocytogenes . IRF4-deficient ( Irf4 −/− ) mice could not clear L. monocytogenes infection and generated decreased numbers of L. monocytogenes -specific CD8 + T cells with impaired effector phenotype and function. Transfer of wild-type CD8 + T cells into Irf4 −/− mice improved bacterial clearance, suggesting an intrinsic defect of CD8 + T cells in Irf4 −/− mice. Following transfer into wild-type recipients, Irf4 −/− CD8 + T cells bec…

Cellular differentiationGene ExpressionEomesoderminBiologyMiceInterleukin 21AnimalsCytotoxic T cellListeriosisIL-2 receptorAntigen-presenting cellSTAT4Cell ProliferationMice KnockoutMultidisciplinaryCell DifferentiationBiological SciencesListeria monocytogenesMolecular biologyMice Inbred C57BLHost-Pathogen InteractionsInterferon Regulatory FactorsImmunologyPositive Regulatory Domain I-Binding Factor 1CD8T-Lymphocytes CytotoxicTranscription FactorsProceedings of the National Academy of Sciences
researchProduct

Analysis of Early Host Responses for Asymptomatic Disease Detection and Management of Specialty Crops

2010

The rapid and unabated spread of vector-borne diseases within US specialty crops threatens our agriculture, our economy, and the livelihood of growers and farm workers. Early detection of vector-borne pathogens is an essential step for the accurate surveillance and management of vector-borne diseases of specialty crops. Currently, we lack the tools that would detect the infectious agent at early (primary) stages of infection with a high degree of sensitivity and specificity. In this paper, we outline a strategy for developing an integrated suite of platform technologies to enable rapid, early disease detection and diagnosis of huanglongbing (HLB), the most destructive citrus disease. The re…

Crops AgriculturalCitrusTime FactorsPolymers and PlasticsDisease detectionCitruPlant DiseaseDiseaseBiologyAsymptomaticSettore AGR/07 - Genetica AgrariamedicinePlant DiseasesGeneral Environmental ScienceHost (biology)business.industryRapid expansionSpecialty cropsBiotechnologyHost-Pathogen InteractionHost-Pathogen InteractionsBiological MarkerIdentification (biology)medicine.symptombusinessBiomarkersInfectious agentCritical Reviews™ in Immunology
researchProduct

Interactions among bacterial strains and fluke genotypes shape virulence of co-infection.

2015

Most studies of virulence of infection focus on pairwise host–parasite interactions. However, hosts are almost universally co-infected by several parasite strains and/or genotypes of the same or different species. While theory predicts that co-infection favours more virulent parasite genotypes through intensified competition for host resources, knowledge of the effects of genotype by genotype (G × G) interactions between unrelated parasite species on virulence of co-infection is limited. Here, we tested such a relationship by challenging rainbow trout with replicated bacterial strains and fluke genotypes both singly and in all possible pairwise combinations. We found that virulence (host mo…

Genotypemedia_common.quotation_subjectVirulenceDiplostomum pseudospathaceumTrematode InfectionsFlavobacteriumGeneral Biochemistry Genetics and Molecular BiologyCompetition (biology)facilitationFlavobacterium columnareFish DiseasesFlavobacteriaceae InfectionsGenotypeParasite hostingAnimalsEvolutionary dynamicsResearch ArticlesGeneral Environmental Sciencemedia_commonGeneticsGeneral Immunology and MicrobiologybiologyVirulenceHost (biology)ta1183General Medicinemultiple infectionbiology.organism_classification3. Good healthconcomitant infectionOncorhynchus mykissHost-Pathogen Interactionsta1181epidemiologyTrematodaTrematodaGeneral Agricultural and Biological SciencesFlavobacteriumProceedings. Biological sciences
researchProduct

Host-based lipid inflammation drives pathogenesis in Francisella infection

2017

Mass spectrometry imaging (MSI) was used to elucidate host lipids involved in the inflammatory signaling pathway generated at the host-pathogen interface during a septic bacterial infection. Using Francisella novicida as a model organism, a bacterial lipid virulence factor (endotoxin) was imaged and identified along with host phospholipids involved in the splenic response in murine tissues. Here, we demonstrate detection and distribution of endotoxin in a lethal murine F. novicida infection model, in addition to determining the temporally and spatially resolved innate lipid inflammatory response in both 2D and 3D renderings using MSI. Further, we show that the cyclooxygenase-2-dependent lip…

0301 basic medicineLipopolysaccharideDIVERSITYGene ExpressionLIPOPOLYSACCHARIDEhost-pathogen interactionmedicine.disease_cause01 natural sciencesMass SpectrometryVirulence factorMicechemistry.chemical_compoundlipid inflammationcyclooxygenase pathwayHETEROGENEITYFrancisellaPhospholipidsMice KnockoutMultidisciplinarybiologyTULAREMIABiological SciencesMolecular ImagingHost-Pathogen InteractionsFrancisellalipids (amino acids peptides and proteins)FemaleSignal TransductionLPSHost–pathogen interactionmicrobial pathogenesismass spectrometry imagingDinoprostoneMicrobiologyCyclooxygenase pathwayProinflammatory cytokine03 medical and health sciencesImmune systemIMAGING MASS-SPECTROMETRYmedicineAnimalsBIOSYNTHESISFrancisella novicidaInflammationMacrophages010401 analytical chemistrybacterial infections and mycosesbiology.organism_classificationSurvival AnalysisImmunity Innate0104 chemical sciencesEndotoxinsMice Inbred C57BL030104 developmental biologychemistryCyclooxygenase 2EicosanoidsGram-Negative Bacterial InfectionsSpleenProceedings of the National Academy of Sciences
researchProduct

Co-factors, Microbes, and Immunogenetics in Celiac Disease to Guide Novel Approaches for Diagnosis and Treatment.

2021

Celiac disease (CeD) is a frequent immune-mediated disease that affects not only the small intestine but also many extraintestinal sites. The role of gluten proteins as dietary triggers, HLA-DQ2 or -DQ8 as major necessary genetic predisposition, and tissue transglutaminase (TG2) as mechanistically involved autoantigen, are unique features of CeD. Recent research implicates many cofactors working in synergism with these key triggers, including the intestinal microbiota and their metabolites, nongluten dietary triggers, intestinal barrier defects, novel immune cell phenotypes, and mediators and cytokines. In addition, apart from HLA-DQ2 and -DQ8, multiple and complex predisposing genetic fact…

GlutensTissue transglutaminaseHuman leukocyte antigenDiseaseGut floraImmunologic Testsmedicine.disease_causeBioinformaticsAutoimmunityImmune systemPredictive Value of TestsRisk FactorsGenetic predispositionMedicineAnimalsHumansGenetic Predisposition to DiseaseImmunogenetic PhenomenaIrritable bowel syndromeHepatologybiologyBacteriabusiness.industryfungiGastroenterologynutritional and metabolic diseasesmedicine.diseasebiology.organism_classificationPrognosisGastrointestinal MicrobiomeIntestinesCeliac DiseaseDisease Models AnimalPhenotypeHost-Pathogen Interactionsbiology.proteinbusinessGastroenterology
researchProduct