Search results for "IL-2"

showing 10 items of 267 documents

Anti-GD3 antibodies are potent activators of human gamma/delta and alpha/beta positive T cells.

1995

The ganglioside GD3 has a variety of biological functions. These include stimulatory effects on proliferation, natural killer activity and cytokine production by freshly isolated peripheral T cells. In this study we have characterized anti-GD3 antibody (MoAb Z21) mediated effects on T cell clones. Our data indicate that alpha/beta TCR CD4+ and CD8+ as well as gamma/delta TCR positive T cells can be stimulated resulting in proliferation and cytokine production. This effect could be blocked by cyclosporin A and did not involve the LFA-3 or CD4 molecule. Apart from IFN-gamma and IL-2 production by T helper 1 and T helper 0 cells we have observed production of IL-4 and IL-10 by T helper 2 cells…

CD3 ComplexT cellReceptors Antigen T-Cell alpha-betaT-LymphocytesImmunologyBiologyLymphocyte ActivationInterleukin 21CD28 AntigensAntigens CDGangliosidesmedicineCytotoxic T cellHumansIL-2 receptorAntigen-presenting cellMembrane GlycoproteinsCD28Antibodies MonoclonalReceptors Antigen T-Cell gamma-deltaGeneral MedicineNatural killer T cellCD58 AntigensMolecular biologymedicine.anatomical_structureImmunologyCD4 AntigensCyclosporinelipids (amino acids peptides and proteins)CD8Scandinavian journal of immunology
researchProduct

LFA-1 Contributes to Signal I of T-Cell Activation and to the Production of Th1 Cytokines

2010

The beta(2) integrins are important for both transendothelial migration of leukocytes and T-cell activation during antigen presentation. In T cells, triggering of leukocyte functional antigen-1 (LFA-1) is required for full activation and T-helper (Th)1/Th2 differentiation. We used CD18-deficient (CD18(-/-)) mice to examine the role of LFA-1 in the activation of T cells. Compared with wild-type controls, CD18(-/-) T cells proliferated normally when stimulated with antibodies against CD3 and CD28, but secreted significantly less IFN-gamma and IL-2 than their wild-type counterparts. However, when T cells were stimulated with dendritic cells (DCs) that provide additional LFA-1 ligation, the pro…

CD3 ComplexT cellchemical and pharmacologic phenomenaDermatologyBiologyBiochemistryAntibodiesMinor Lymphocyte Stimulatory AntigensInterferon-gammaMice03 medical and health sciencesInterleukin 210302 clinical medicineCD28 AntigensCell AdhesionmedicineAnimalsCytotoxic T cellIL-2 receptorAntigen-presenting cellMolecular Biology030304 developmental biologyMice Inbred BALB C0303 health sciencesCD40CD28Cell Differentiationhemic and immune systemsDendritic CellsCell BiologyTh1 CellsIntercellular Adhesion Molecule-1Natural killer T cellLymphocyte Function-Associated Antigen-1Mice Mutant StrainsCell biologyMice Inbred C57BLmedicine.anatomical_structureCD18 Antigensbiology.proteinInterleukin-2Cell DivisionSignal Transduction030215 immunologyJournal of Investigative Dermatology
researchProduct

Differential effects of IL-10 on proliferation and cytokine production of human gamma/delta and alpha/beta T cells.

1994

Gamma/delta TCR bearing T lymphocytes represent a T-cell subset whose functional relevance remains unclear. Nevertheless these T cells may play a role in the early immune response against bacteria. Until now the regulatory mechanisms on this response have not been investigated. The study described here evaluated the immunoregulatory effects of Interleukin-10 on gamma/delta and alpha/beta TCR-positive T-cell clones and freshly isolated peripheral-blood mononuclear cells (PBMC). IL-10 has been shown previously to inhibit lectin and antigen-induced proliferation and cytokine production by alpha/beta T cells. The results outlined below show that rhIL-10 strongly inhibits lectin-induced producti…

CD3 Complexmedicine.medical_treatmentT cellReceptors Antigen T-Cell alpha-betaImmunologyAlpha (ethology)BiologyLymphocyte ActivationInterferon-gammaT-Lymphocyte SubsetsLectinsmedicineHumansIL-2 receptorTumor Necrosis Factor-alphaGrowth factorMacrophagesT-cell receptorReceptors Antigen T-Cell gamma-deltaGeneral MedicineT lymphocyteMolecular biologyRecombinant ProteinsInterleukin-10Interleukin 10Cytokinemedicine.anatomical_structureImmunologyCytokinesInterleukin-2Interleukin-4Scandinavian journal of immunology
researchProduct

Exclusive transduction of human CD4+ T Cells upon systemic delivery of CD4-targeted lentiviral vectors

2015

Abstract Playing a central role in both innate and adaptive immunity, CD4+ T cells are a key target for genetic modifications in basic research and immunotherapy. In this article, we describe novel lentiviral vectors (CD4-LV) that have been rendered selective for human or simian CD4+ cells by surface engineering. When applied to PBMCs, CD4-LV transduced CD4+ but not CD4− cells. Notably, also unstimulated T cells were stably genetically modified. Upon systemic or intrasplenic administration into mice reconstituted with human PBMCs or hematopoietic stem cells, reporter gene expression was predominantly detected in lymphoid organs. Evaluation of GFP expression in organ-derived cells and blood …

CD4-Positive T-Lymphocytes10028 Institute of Medical VirologyCell TransplantationGenetic enhancementAdoptiveMice SCIDImmunotherapy AdoptiveInterleukin 21MiceMice Inbred NODTransduction GeneticBone MarrowLeukocytesImmunology and AllergyCytotoxic T cellIL-2 receptorLuciferasesCells CulturedMice KnockoutHeterologousTumorCulturedForkhead Transcription FactorsAcquired immune systemFlow Cytometry3. Good healthCell biologymedicine.anatomical_structure[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/Virology2723 Immunology and Allergy[SDV.IMM]Life Sciences [q-bio]/ImmunologyImmunotherapyRegulatory T cellCellsKnockoutTransplantation HeterologousImmunologyMononuclearGenetic VectorsGreen Fluorescent Proteins610 Medicine & healthStreptamerThymus GlandBiologySCIDCell LineTransductionGeneticCell Line TumormedicineAnimalsHumansInterleukin 3Transplantation2403 ImmunologyLentivirusGenetic TherapyMolecular biology[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyHEK293 CellsLeukocytes MononuclearInbred NOD570 Life sciences; biologySpleen
researchProduct

B cells participate in thymic negative selection of murine auto-reactive CD4+ T cells.

2010

It is well documented that thymic epithelial cells participate in the process of negative selection in the thymus. In recent years it was reported that also dendritic cells enter the thymus and contribute to this process, thus allowing for the depletion of thymocytes that are specific to peripherally expressed self-antigens. Here we report that also B cells may take part in the elimination of auto-reactive thymocytes. Using a unique mouse model we show that B cells induce negative selection of self-reactive thymocytes in a process that leads to the deletion of these cells whereas regulatory T cells are spared. These findings have direct implication in autoimmunity, as expression of a myelin…

CD4-Positive T-LymphocytesB CellsImmune CellsImmunologyCD1Antigen-Presenting Cellslcsh:MedicineAutoimmunityMice TransgenicThymus GlandBiologyMiceNegative selectionAntigenImmune ToleranceAnimalsIL-2 receptorAntigen-presenting celllcsh:ScienceBiologyClonal AnergyB-LymphocytesMultidisciplinaryCD40Clonal anergyT Cellslcsh:RImmunityCell biologyImmunologyInterleukin 12biology.proteinlcsh:QResearch ArticlePLoS ONE
researchProduct

In vitro generation of CD4+CD25+ regulatory cells from murine naive T cells

2007

CD4+ CD25+ regulatory T cells (Tregs) are crucial for the maintenance of immunological tolerance. Recent data indicate that Tregs not only develop in the thymus during ontogeny but can also differentiate from naive T cells in the periphery. The following protocol describes a method by which Tregs are generated in vitro by stimulation of naive T cells in the presence of transforming growth factor beta (Ti-Tregs). In vitro-induced regulatory T cells express markers of conventional Treg such as CD25 and the genetic program committing transcription factor FoxP3. Functionally the in vitro-generated Ti-Tregs suppress T-cell activation and proliferation while in vivo these cells have been proven t…

CD4-Positive T-LymphocytesCD3 ComplexT-Lymphocytesmedicine.medical_treatmentchemical and pharmacologic phenomenaBiologyBioinformaticsT-Lymphocytes RegulatoryGeneral Biochemistry Genetics and Molecular BiologyMiceInterleukin 21CD28 AntigensmedicineAnimalsCytotoxic T cellIL-2 receptorInterleukin 3Mice Inbred BALB CInterleukin-2 Receptor alpha SubunitFOXP3hemic and immune systemsTransfectionImmunotherapyTransforming growth factor betaCell biologyCD4 Antigensbiology.proteinBiomarkersNature Protocols
researchProduct

Isolation of CD4+ T cells from murine lungs: a method to analyze ongoing immune responses in the lung.

2007

The regulation of the cellular immune response in lung diseases is not yet fully understood. Isolating different subsets of immune cells directly from the lung is therefore an indispensable method of gaining detailed knowledge on the function of these cells in this organ. This protocol describes a method of isolating and magnetically labeling CD4+ lung T cells, which are then loaded and retained on the column while all other cells run through it (positive selection). The yield of this isolation is approximately 5 x 10(5) to 1.5 x 10(6) CD4+ cells from a murine lung. These cells can be further investigated by several methods such as flow cytometry, western blot analysis, RT-PCR, immunostaini…

CD4-Positive T-LymphocytesCD40biologyStreptamerCell SeparationMolecular biologyGeneral Biochemistry Genetics and Molecular BiologyInterleukin 21Micebiology.proteinInterleukin 12Cytotoxic T cellAnimalsIL-2 receptorAntigen-presenting cellLungInterleukin 3Nature protocols
researchProduct

A Key Regulatory Role of the Transcription Factor NFATc2 in Bronchial Adenocarcinoma via CD8+ T Lymphocytes

2009

AbstractThe Ca2+-regulated calcineurin/nuclear factor of activated T cells (NFAT) cascade controls alternative pathways of T-cell activation and peripheral tolerance. Here, we describe reduction of NFATc2 mRNA expression in the lungs of patients with bronchial adenocarcinoma. In a murine model of bronchoalveolar adenocarcinoma, mice lacking NFATc2 developed more and larger solid tumors than wild-type littermates. The extent of central tumor necrosis was decreased in the tumors in NFATc2(−/−) mice, and this finding was associated with reduced tumor necrosis factor-α and interleukin-2 (IL-2) production by CD8+ T cells. Adoptive transfer of CD8+ T cells of NFATc2(−/−) mice induced transforming…

CD4-Positive T-LymphocytesCancer ResearchAdoptive cell transferTranscription GeneticTransplantation HeterologousMice TransgenicReceptors Nerve Growth FactorAdenocarcinomaCD8-Positive T-LymphocytesBiologyReceptors Tumor Necrosis FactorTransforming Growth Factor beta1Interferon-gammaMiceGlucocorticoid-Induced TNFR-Related ProteinAnimalsHumansIL-2 receptorInterleukin-7 receptorMice Inbred BALB CReceptors Interleukin-7NFATC Transcription FactorsTumor Necrosis Factor-alphaBronchial NeoplasmsInterleukin-2 Receptor alpha SubunitPeripheral toleranceForkhead Transcription FactorsNFATCalcineurinDisease Models AnimalOncologyCancer researchInterleukin-2Tumor necrosis factor alphaCD8Cancer Research
researchProduct

The role of CD8+ T cells and their local interaction with CD4+ T cells in myelin oligodendrocyte glycoprotein35-55-induced experimental autoimmune en…

2013

Abstract T cells have an essential role in the induction of multiple sclerosis and its animal model experimental autoimmune encephalomyelitis (EAE). Although for CD4+ T cells it is well established that they contribute to the disease, less is known about the role of CD8+ T cells. Our aim was to determine the individual contribution of CD4+ and CD8+ T cells in myelin oligodendrocyte glycoprotein (MOG)35–55–induced EAE. We investigated MOG35–55–activated CD8+ T cells to clarify their potential to induce or attenuate EAE. We monitored the behavior of CD8+ T cells and their interaction with CD4+ T cells directly at the site of inflammation in the CNS using intravital imaging of the brainstem of…

CD4-Positive T-LymphocytesCentral Nervous SystemEncephalomyelitis Autoimmune ExperimentalT cellImmunologyMedizinCell CommunicationCD8-Positive T-LymphocytesLymphocyte ActivationInterleukin 21MiceCell MovementmedicineImmunology and AllergyCytotoxic T cellAnimalsIL-2 receptorInflammationMice KnockoutCD40biologyCD28Molecular biologyPeptide FragmentsMice Inbred C57BLmedicine.anatomical_structureImmunologybiology.proteinInterleukin 12Myelin-Oligodendrocyte GlycoproteinCD8Journal of immunology (Baltimore, Md. : 1950)
researchProduct

Characterization of lung γδ T cells following intranasal infection with Mycobacterium bovis bacillus Calmette-Guérin

2002

The lungs are considered to have an impaired capacity to contain infection by pathogenic mycobacteria, even in the presence of effective systemic immunity. In an attempt to understand the underlying cellular mechanisms, we characterized the gammadelta T cell population following intranasal infection with Mycobacterium bovis bacillus Calmette-Guerin (BCG). The peak of gammadelta T cell expansion at 7 days postinfection preceded the 30 day peak of alphabeta T cell expansion and bacterial count. The expanded population of gammadelta T cells in the lungs of BCG-infected mice represents an expansion of the resident Vgamma2 T cell subset as well as an influx of Vgamma1 and of four different Vdelt…

CD4-Positive T-LymphocytesCytotoxicity ImmunologicT cellImmunologyGene Rearrangement delta-Chain T-Cell Antigen ReceptorEpitopes T-Lymphocytechemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationLymphocyte DepletionInterleukin 21MiceAntigenT-Lymphocyte SubsetsmedicineImmunology and AllergyCytotoxic T cellAnimalsTuberculosisIL-2 receptorAntigen-presenting cellLungAdministration IntranasalCells CulturedGene Rearrangement gamma-Chain T-Cell Antigen ReceptorT-cell receptorhemic and immune systemsReceptors Antigen T-Cell gamma-deltaAcquired immune systemFlow CytometryMycobacterium bovisMice Inbred C57BLstomatognathic diseasesmedicine.anatomical_structureImmunologyCytokinesCell DivisionT-Lymphocytes Cytotoxic
researchProduct