Search results for "INFORMATICS"

showing 10 items of 2542 documents

Cytotoxicity, anti-angiogenic, apoptotic effects and transcript profiling of a naturally occurring naphthyl butenone, guieranone A

2012

Abstract Background Malignant diseases are responsible of approximately 13% of all deaths each year in the world. Natural products represent a valuable source for the development of novel anticancer drugs. The present study was aimed at evaluating the cytotoxicity of a naphtyl butanone isolated from the leaves of Guiera senegalensis, guieranone A (GA). Results The results indicated that GA was active on 91.67% of the 12 tested cancer cell lines, the IC50 values below 4 μg/ml being recorded on 83.33% of them. In addition, the IC50 values obtained on human lymphoblastic leukemia CCRF-CEM (0.73 μg/ml) and its resistant subline CEM/ADR5000 (1.01 μg/ml) and on lung adenocarcinoma A549 (0.72 μg/m…

Cell cycle checkpointCytotoxicityApoptosisMicroarrayBiologyBioinformaticslcsh:RC254-282BiochemistryAngiogensismedicineCytotoxic T cellDoxorubicinlcsh:QH573-671CytotoxicityMolecular Biologylcsh:CytologyResearchCell BiologyCell cyclelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensMolecular biologyChorioallantoic membraneCell cultureApoptosisGuieranone APharmacogenomicsmedicine.drugCell Division
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Risk Factors and Molecular Features Associated with Bladder Cancer Development

2017

Bladder cancer remains a global epidemiologic problem, with a strong male predominance and association with tobacco smoking. However, several other risk factors have also been associated with development of this disease, which is characterized by alterations in multiple molecular pathways. Development of the more prevalent, less aggressive, recurrent, noninvasive tumors is characterized by constitutive activation of the Ras–MAPK pathway. The less common but more aggressive invasive tumors, which have a higher mortality rate, are characterized by alterations in the p53 and retinoblastoma pathways. Alterations in pathways involved in cell-cycle regulation, apoptosis, cell signaling, angiogene…

Cell signalingBladder cancerRetinoblastomabusiness.industryAngiogenesisMortality rate030232 urology & nephrologyDiseaseCell cyclemedicine.diseaseBioinformatics03 medical and health sciences0302 clinical medicineApoptosis030220 oncology & carcinogenesismedicinebusiness
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Molecular evolution of the metazoan extracellular matrix: cloning and expression of structural proteins from the demosponges Suberites domuncula and …

2000

One crucial event during evolution to multicellularity was the development of either direct cell–cell contact or indirect interaction via extracellular matrix (ECM) molecules. The identification of those polypeptides provides conclusive data on the phylogenetic relationship of metazoan phyla and helps us to understand the position of the Metazoa among the other kingdoms. Recently it became evident that the ECM of sponges is amazingly complex; it is composed of fibrous molecules, e.g., collagen, and their corresponding receptors, which are highly similar to those existing in other metazoan phyla. While these data already support the view of monophyly of Metazoa, additional studies are requir…

Cell signalingDNA ComplementaryDermatopontinMolecular Sequence DataGene ExpressionBiologyBioinformaticsTransplantation AutologousExtracellular matrixEvolution MolecularMyotrophinGeneticsAnimalsAmino Acid SequenceCloning MoleculareducationGrowth SubstancesMolecular BiologyPeptide sequenceEcology Evolution Behavior and SystematicsPhylogenyCell Aggregationeducation.field_of_studyExtracellular Matrix ProteinsBase SequenceSequence Homology Amino AcidReceptor Protein-Tyrosine Kinasesbiology.organism_classificationRecombinant ProteinsCell biologyPoriferaSuberites domunculaTransplantationChondroitin Sulfate ProteoglycansIntercellular Signaling Peptides and ProteinsCollagenCarrier ProteinsCell Adhesion MoleculesFunction (biology)Journal of molecular evolution
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Fusion of bone-marrow-derived cells with Purkinje neurons, cardiomyocytes and hepatocytes

2003

Recent studies have suggested that bone marrow cells possess a broad differentiation potential, being able to form new liver cells, cardiomyocytes and neurons1,2. Several groups have attributed this apparent plasticity to ‘transdifferentiation’3,4,5. Others, however, have suggested that cell fusion could explain these results6,7,8,9. Using a simple method based on Cre/lox recombination to detect cell fusion events, we demonstrate that bone-marrow-derived cells (BMDCs) fuse spontaneously with neural progenitors in vitro. Furthermore, bone marrow transplantation demonstrates that BMDCs fuse in vivo with hepatocytes in liver, Purkinje neurons in the brain and cardiac muscle in the heart, resul…

Cell typeCell signalingBone Marrow CellsBiologyBioinformaticsGiant CellsModels BiologicalCell FusionMicePurkinje CellsmedicineAnimalsMyocyteMyocytes CardiacProgenitor cellBone Marrow TransplantationMultidisciplinaryCell fusionStem CellsTransdifferentiationCell DifferentiationCell cycleCell biologyMice Inbred C57BLmedicine.anatomical_structureHepatocytesBone marrow
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Derivation of clinical-grade human embryonic stem cells.

2006

Embryonic stem cells proliferate in vitro while maintaining an undifferentiated state, and are capable of differentiating into most cell types under appropriate conditions. These properties imply great potential in the treatment of various diseases and disabilities. In fact, the first clinical trials with hESC for treating spinal cord injuries will begin next year. However, therapeutic application of human embryonic stem cell derivatives is compromised by the exposure of existing lines to animal and human components, with the subsequent risk of contamination with retroviruses and other pathogens, which can be transmitted to patients. The scientific community is striving to avoid the use of …

Cell typeCellular differentiationCell Culture TechniquesObstetrics and GynecologyClinical gradeCell DifferentiationBiologyBioinformaticsEmbryo MammalianEmbryonic stem cellCell LineTotipotent stem cellReproductive MedicineCell cultureImmunologyAnimalsHumansStem cellTotipotent Stem CellsDevelopmental BiologyReproductive biomedicine online
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MicroRNAs: Promising New Antiangiogenic Targets in Cancer

2014

[EN] MicroRNAs are one class of small, endogenous, non-coding RNAs that are approximately 22 nucleotides in length; they are very numerous, have been phylogenetically conserved, and involved in biological processes such as development, differentiation, cell proliferation, and apoptosis. MicroRNAs contribute to modulating the expression levels of specific proteins based on sequence complementarity with their target mRNA molecules and so they play a key role in both health and disease. Angiogenesis is the process of new blood vessel formation from preexisting ones, which is particularly relevant to cancer and its progression. Over the last few years, microRNAs have emerged as critical regulat…

Cell typeDOWN-REGULATIONArticle SubjectAngiogenesisHUMAN BREAST-CANCERMIR-200 FAMILYlcsh:MedicineAngiogenesis InhibitorsReview ArticleBiologyBioinformaticsGeneral Biochemistry Genetics and Molecular BiologyNUCLEAR EXPORTTUMOR ANGIOGENESISNeovascularizationMicroprocessor complexSMALL RNASDownregulation and upregulationNeoplasmsmicroRNAGene expressionmedicineAnimalsHumansMolecular Targeted TherapyPrecision MedicineIN-VIVOGENE-EXPRESSIONGeneral Immunology and MicrobiologyNeovascularization PathologicCell growthlcsh:RMICROBIOLOGIAGeneral MedicineMICROPROCESSOR COMPLEXMicroRNAsENDOTHELIAL GROWTH-FACTORCancer researchmedicine.symptom
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HSP60 expression during carcinogenesis: a molecular “Proteus” of carcinogenesis?

2005

Sir, I read with much interest the comprehensive review by Ciocca and Calderwood (2005) in which they analyze the diagnostic, prognostic, predictive, and treatment values of a number of Hsps in oncology. Concerning the implications of Hsps in cancer diagnosis in particular, they report that since “Hsps are overexpressed in a wide range of malignant cells and tissues … Hsp detection is not useful in diagnostic immunopathology.” Nevertheless, “Hsp expression levels can help indicate the presence of abnormal changes during the process of carcinogenesis.” The authors support this remark by reference to several papers by our group (Cappello et al 2002–2003, 2003a, 2003b, 2003c) in which we demon…

Cellular differentiationBiologymedicine.disease_causeBioinformaticsBiochemistryAdjuvants ImmunologicNeoplasmsGene expressionmedicineAnimalsHumansNeoplasmHeat-Shock ProteinsCancerCell BiologyHsp60Prognosismedicine.diseaseSquamous metaplasiaImmune SystemLetter from the EditorCancer researchBiomarker (medicine)HSP60CarcinogenesisCell Stress & Chaperones
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Can Alzheimer disease be a form of type 3 diabetes?

2012

Alzheimer disease (AD) and metabolic syndrome are two highly prevalent pathological conditions of Western society due to incorrect diet, lifestyle, and vascular risk factors. Recent data have suggested metabolic syndrome as an independent risk factor for AD and pre-AD syndrome. Furthermore, biological plausibility for this relationship has been framed within the “metabolic cognitive syndrome” concept. Due to the increasing aging of populations, prevalence of AD in Western industrialized countries will rise in the near future. Thus, new knowledge in the area of molecular biology and epigenetics will probably help to make an early molecular diagnosis of dementia. An association between metabo…

Central Nervous SystemAgingmedicine.medical_specialtySingle-nucleotide polymorphismType 2 diabetesBiologyBioinformaticsPolymorphism Single NucleotideSHIP2 ADAlzheimer DiseaseRisk FactorsDiabetes mellitusInternal medicinemedicineDiabetes MellitusDementiaHumansInsulinEpigeneticsRisk factorLife StyleAgedSettore MED/04 - Patologia GeneraleMetabolic SyndromeInositol Polyphosphate 5-PhosphatasesSyndromeModels Theoreticalmedicine.diseasePhosphoric Monoester HydrolasesEndocrinologySettore MED/26 - NeurologiaGeriatrics and GerontologyAlzheimer's diseaseMetabolic syndromeCognition DisordersSignal TransductionRejuvenation research
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Molecular evidence for the inverse comorbidity between central nervous system disorders and cancers detected by transcriptomic meta-analyses.

2014

There is epidemiological evidence that patients with certain Central Nervous System (CNS) disorders have a lower than expected probability of developing some types of Cancer. We tested here the hypothesis that this inverse comorbidity is driven by molecular processes common to CNS disorders and Cancers, and that are deregulated in opposite directions. We conducted transcriptomic meta-analyses of three CNS disorders (Alzheimer's disease, Parkinson's disease and Schizophrenia) and three Cancer types (Lung, Prostate, Colorectal) previously described with inverse comorbidities. A significant overlap was observed between the genes upregulated in CNS disorders and downregulated in Cancers, as wel…

Central Nervous SystemCancer ResearchGene ExpressionDiseaseComorbidityBioinformaticsProstate cancer0302 clinical medicineNeoplasmsGenetics (clinical)0303 health sciencesWnt signaling pathwayParkinson DiseaseAlzheimer's diseasePeptidylprolyl Isomerase[SDV.BIBS]Life Sciences [q-bio]/Quantitative Methods [q-bio.QM]3. Good health[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Alzheimer's diseaseResearch ArticleSignal Transductionlcsh:QH426-470[SDV.CAN]Life Sciences [q-bio]/CancerProtein degradationBiology03 medical and health sciencesAlzheimer Disease[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]medicineGeneticsCancer GeneticsHumansGene NetworksMolecular BiologyBiologyEcology Evolution Behavior and Systematics030304 developmental biologyPeptidylprolyl isomeraseGene Expression ProfilingCancerComputational Biologymedicine.diseaseColorectal cancerComorbidityMalariaNIMA-Interacting Peptidylprolyl IsomeraseMeta-analysislcsh:GeneticsGene Expression RegulationImmunologySchizophrenia[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologie030217 neurology & neurosurgery
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Therapeutic Modulation of Urinary Bladder Function: Multiple Targets at Multiple Levels

2015

Storage dysfunction of the urinary bladder, specifically overactive bladder syndrome, is a condition that occurs frequently in the general population. Historically, pathophysiological and treatment concepts related to overactive bladder have focused on smooth muscle cells. Although these are the central effector, numerous anatomic structures are involved in their regulation, including the urothelium, afferent and efferent nerves, and the central nervous system. Each of these structures involves receptors for—and the urothelium itself also releases—many mediators. Moreover, hypoperfusion, hypertrophy, and fibrosis can affect bladder function. Established treatments such as muscarinic antago…

Central Nervous Systemmedicine.medical_specialtyUrinary BladderPopulationCentral nervous systemMuscarinic Antagonistsurologic and male genital diseasesToxicologyBioinformaticsMuscle hypertrophyNeurons EfferentFibrosisInternal medicinemedicineAnimalsHumansNeurons AfferentUrotheliumeducationPharmacologyeducation.field_of_studyUrinary bladderbusiness.industryUrinary Bladder DiseasesMuscle SmoothAdrenergic beta-AgonistsHyperplasiamedicine.diseasefemale genital diseases and pregnancy complicationsUrodynamicsTreatment OutcomeEndocrinologymedicine.anatomical_structureOveractive bladderAdrenergic alpha-1 Receptor AntagonistsUrological AgentsUrotheliumbusinessSignal TransductionAnnual Review of Pharmacology and Toxicology
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