Search results for "INTERFERON-GAMMA"

showing 10 items of 352 documents

Analysis of the immune response induced by a single xenoantigen in vivo

2004

Transgenic mice expressing human major histocompatibility complex (MHC) class II molecules would provide a valuable model system for studying murine anti-human MHC immune response. We have previously shown that skin from HLA-DR1 transgenic mice was rejected by control littermates and spleen cells from rejecting mice were able to proliferate to donor cells. The aim of this paper is to analyze the mechanism of recognition of this xenoantigen and the possible involvement of antibody response in anti-HLA-DR1 immune response. Control littermates were immunized with spleen cells from HLA-DR1 transgenic (TG) mice; at indicated times, xenoantigen-specific proliferation and IFNgamma production was a…

Malemedicine.drug_classTransgeneT-LymphocytesImmunologyEpitopes T-Lymphocytechemical and pharmacologic phenomenaSpleenMice TransgenicHuman leukocyte antigenMonoclonal antibodyMajor histocompatibility complexImmunoglobulin GInterferon-gammaMiceImmune systemAntigens HeterophilemedicineImmunology and AllergyAnimalsHumansCell ProliferationbiologyHLA-DR1 AntigenMolecular biologyPeptide Fragmentsmedicine.anatomical_structureImmunoglobulin MImmunoglobulin GImmunologyAntibody Formationbiology.proteinFemaleAntibodySpleen
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PLASMA LEVELS OF TUMOR NECROSIS FACTOR a AND INTERFERON g IN SICILIAN CHILDREN WITH MEDITERRANEAN SPOTTED FEVER

1997

The plasma levels of tumor necrosis factor-alpha and interferon-gamma were measured in 53 consecutive children with serologically confirmed Mediterranean spotted fever and were found to be increased during the acute phase compared with the convalescent phase (tumor necrosis factor-alpha mean 32.17 vs. 4.12 pg/ml, P < 0.0001; interferon-gamma mean 84.17 vs. 2.65 pg/ml, P = 0.0006). Plasma levels of both cytokines were higher in patients with a typical exanthema rather than those with a very mild or no exanthema; tumor necrosis factor-alpha levels were significantly lower in the latter (tumor necrosis factor-alpha 32.17 vs. 9.85 pg/ml, P < 0.0001; interferon-gamma 84.17 vs. 38.14 pg/ml, P = 0…

Malemedicine.medical_specialtyAdolescentmedicine.medical_treatmentClinical BiochemistryInflammationBiologyBoutonneuse FeverInterferon-gammaInternal medicinemedicineHumansInterferon gammaChildSicilyHematologyTumor Necrosis Factor-alphaSodiumInfantmedicine.diseaseSpotted feverBoutonneuse feverC-Reactive ProteinEndocrinologyRickettsiosisCytokineChild PreschoolImmunologyFemaleTumor necrosis factor alphamedicine.symptommedicine.drug
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Angiotensin II-induced vascular dysfunction depends on interferon-γ-driven immune cell recruitment and mutual activation of monocytes and NK-cells.

2013

Objective— Immune cells contribute to angiotensin II (ATII)–induced vascular dysfunction and inflammation. Interferon-γ (IFN-γ), an inflammatory cytokine exclusively produced by immune cells, seems to be involved in ATII-driven cardiovascular injury, but the actions and cellular source of IFN-γ remain incompletely understood. Approach and Results— IFN-γ −/− and Tbx21 −/− mice were partially protected from ATII-induced (1 mg/kg per day of ATII, infused subcutaneously by miniosmotic pumps) vascular endothelial and smooth muscle dysfunction, whereas mice overexpressing IFN-γ showed constitutive vascular dysfunction. Absence of T-box expressed in T cells (T-bet), the IFN-γ transcription factor…

Malemedicine.medical_specialtyAdoptive cell transfermedicine.medical_treatmentInflammationBiologyMonocytesInterferon-gammaMiceRandom AllocationImmune systemReference ValuesInternal medicinemedicineAnimalsVascular DiseasesVascular recruitmentVascular tissueAortaAngiotensin IIAngiotensin IIKiller Cells NaturalMice Inbred C57BLDisease Models AnimalOxidative StressCytokineEndocrinologyInterleukin 12Endothelium Vascularmedicine.symptomCardiology and Cardiovascular MedicineArteriosclerosis, thrombosis, and vascular biology
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The chemiluminescence activity of peripheral blood mononuclear cells during acute experimental allergic encephalomyelitis

1987

The spontaneous chemiluminescence activity (CL-A) of peripheral mononuclear cells (MNC) was examined in Lewis rats with acute experimental allergic encephalomyelitis (EAE), compared to rats immunized with complete adjuvant (n = 11) and healthy animals (n = 16). In rats with EAE, CL-A increased sharply 8-9 days after immunization (3420 +/- 3124 counts/10 s, n = 16) at the time of flattening of the weight curve. This CL-A peak was compared to that of animals immunized with complete adjuvant: 765 +/- 441 counts/10 s (P = 0.01) and healthy rats: 450 +/- 172 counts/10 s (P = 0.0001). After this initial peak in EAE rats, CL-A decreased almost to normal values when animals lost weight (746 +/- 251…

Malemedicine.medical_specialtyPathologyEncephalomyelitis Autoimmune ExperimentalEncephalomyelitisImmunologyCentral nervous systemPeripheral blood mononuclear cellInterferon-gammaImmunopathologyInternal medicineParalysismedicineAnimalsImmunology and AllergyLymphocytesbusiness.industryBrainmedicine.diseaseRatsRespiratory burstPeripheralEndocrinologymedicine.anatomical_structureNeurologyRats Inbred LewAcute DiseaseLuminescent MeasurementsLeukocytes MononuclearNeurology (clinical)medicine.symptombusinessInfiltration (medical)Journal of Neuroimmunology
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Influence of HMB Supplementation and Resistance Training on Cytokine Response to Resistance Exercise

2014

The purpose of this study was to determine the effects of a multinutritional supplement including amino acids, β-hydroxy-β-methylbutyrate (HMB), and carbohydrates on cytokine responses to resistance exercise and training.Seventeen healthy, college-aged men were randomly assigned to a Muscle Armor™ (MA; Abbott Nutrition, Columbus, OH) or placebo supplement group and 12 weeks of resistance training. An acute resistance exercise protocol was administered at 0, 6, and 12 weeks of training. Venous blood samples at pre-, immediately post-, and 30-minutes postexercise were analyzed via bead multiplex immunoassay for 17 cytokines.After 12 weeks of training, the MA group exhibited decreased interfer…

Malemedicine.medical_specialtyStrength trainingmedicine.medical_treatmentInterleukin-1betaMedicine (miscellaneous)Immune functionGranulocytePlaceboBody Mass IndexInterferon-gammaYoung AdultDouble-Blind MethodMuscle damageInternal medicineMyokineMyokinemedicineDietary CarbohydratesValeratesHumansMicronutrientsAmino Acidsta315Chemokine CCL4Immune function; Muscle damage; Myokine; Nutrition; Strength trainingChemokine CCL2NutritionNutrition and DieteticsInterleukin-13business.industryInterleukin-6MonocyteBody WeightInterleukin-8InterleukinResistance TrainingVenous bloodHealthy VolunteersInterleukin-10Cytokinemedicine.anatomical_structureEndocrinologyNutrition AssessmentImmunologyDietary SupplementsCytokinesStrength trainingbusiness
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Comparison of intramuscular and subcutaneous administration of a herpes zoster live-attenuated vaccine in adults aged ≥50 years: a randomised non-inf…

2015

AbstractZostavax® is a live, attenuated varicella zoster virus (VZV) vaccine developed specifically for the prevention of HZ and PHN in individuals aged ≥50 years. During the clinical development of Zostavax, which was mainly in the US, the vaccine was administrated by the subcutaneous (SC) route. In Europe, many healthcare professionals prefer administering vaccines by the intramuscular (IM) route. This was an open-label, randomised trial conducted in 354 subjects aged ≥50 years. The primary objectives were to demonstrate that IM administration is both non-inferior to SC administration in terms of 4-week post-vaccination geometric mean titres (GMTs), and elicits an acceptable geometric mea…

Malemedicine.medical_specialtySub-cutaneous administration routeEnzyme-Linked Immunospot AssayHerpesvirus 3 HumanInjections SubcutaneousVaricella zoster virus vaccinePainmedicine.disease_causeAntibodies ViralVaccines AttenuatedHerpes ZosterInjections Intramuscularlaw.inventionShinglesInterferon-gammaRandomized controlled triallawImmunology and Microbiology(all)Internal medicinemedicineEdemaHerpes Zoster VaccineHumansAdverse effectAgedRandomised controlled trialAged 80 and overAttenuated vaccineIntention-to-treat analysisGeneral VeterinaryGeneral Immunology and Microbiologybusiness.industryELISPOTImmunogenicityVaccinationPublic Health Environmental and Occupational HealthVaricella zoster virusIntramuscular administration routeMiddle Agedmedicine.diseaseveterinary(all)Infectious DiseasesErythemaImmunologyMolecular MedicineFemalebusinessShinglesVaccine
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Enhanced serum levels of soluble HLA class I molecules are induced by treatment with recombinant interferon-gamma (IFN-γ)

1991

SUMMARY In order to investigate serum levels of soluble HLA class I antigens after single injection of various doses of recombinant IFN-γ (rIFN-γ) and to correlate the changes observed to beta-2-microglobulin serum levels, we studied five patients with metastasizing renal cell carcinoma. Each patient received three treatment cycles of 10, 100 and 500 μg rIFN-γ three times at weekly intervals. The treatment cycles were separated by a therapy-free interval of 2 weeks. The order of dose levels was randomly assigned to each patient. Serum levels of soluble HLA class I proteins were measured by an ELISA in samples drawn immediately before and 4, 24, 48, 72 and 168 h after each administration of …

Malemedicine.medical_specialtymedicine.medical_treatmentImmunologyHuman leukocyte antigenlaw.inventionInterferon-gammalawInternal medicinemedicineHumansImmunology and AllergyInterferon gammaCarcinoma Renal CellKidneyDose-Response Relationship DrugBeta-2 microglobulinbusiness.industryHistocompatibility Antigens Class IRadioimmunoassayRecombinant Interferon GammaMiddle AgedRecombinant Proteinsmedicine.anatomical_structureCytokineEndocrinologySolubilityImmunologyRecombinant DNADrug Evaluationbeta 2-MicroglobulinbusinessResearch Articlemedicine.drugClinical and Experimental Immunology
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Targeting transcription factor Stat4 uncovers a role for interleukin-18 in the pathogenesis of severe lupus nephritis in mice

2011

Polymorphisms in the transcription factor Stat4 gene have been implicated as risk factors for systemic lupus erythematosus. Although some polymorphisms have a strong association with autoantibodies and nephritis, their impact on pathophysiology is still unknown. To explore this further we used signal transducers and activators of transcription 4 (Stat4) knockout MRL/MpJ-Fas(lpr)/Fas(lpr) (MRL-Fas(lpr)) mice and found that they did not differ in survival or renal function from Stat4-intact MRL-Fas(lpr) mice. Circulating interleukin (IL)-18 levels, however, were elevated in Stat4-deficient compared to Stat4-intact mice, suggesting that this interleukin might contribute to the progression of l…

Malemusculoskeletal diseasesMice Inbred MRL lprchronic inflammationLupus nephritisKidneyInterleukin-23ArticleProinflammatory cytokineOligodeoxyribonucleotides AntisenseGene Knockout TechniquesInterferon-gammaMiceimmune system diseasesmedicineAnimalsskin and connective tissue diseasesSTAT4DNA PrimersAutoimmune diseaseMice Knockoutlupus nephritisMice Inbred BALB CBase Sequencebusiness.industryGene Transfer TechniquesInterleukin-18InterleukinGlomerulonephritishemic and immune systemsSTAT4 Transcription Factormedicine.diseaseInterleukin-12chronic glomerulonephritisNephrologyImmunologyInterleukin 18FemalebusinessNephritisKidney International
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T-bet as a possible therapeutic target in autoimmune disease

2002

The prominent role of pro-inflammatory cytokines produced by T helper-1 (T(H1)) cells in regulating autoimmune responses in vitro and in vivo has been demonstrated. Recent observations of T cell polarisation by regulatory transcription factors--especially T-bet (T-box expressed in T cells)--raise the question of their influence in controlling autoimmune diseases. Here, the authors summarise recent observations of the role of T-bet in controlling chronic inflammatory and autoimmune diseases and discuss the implications of these findings for future therapeutic approaches.

Mice Inbred MRL lprTranscription GeneticTransgeneT cellCellular differentiationClinical BiochemistryMice TransgenicLymphocyte ActivationAutoimmune DiseasesInterferon-gammaMiceTh2 CellsCrohn DiseaseDrug DiscoverymedicineAnimalsLupus Erythematosus SystemicIL-2 receptorIntestinal MucosaMice KnockoutPharmacologyAutoimmune diseaseLupus erythematosusbusiness.industryZAP70Cell DifferentiationTh1 CellsColitisInflammatory Bowel Diseasesmedicine.diseaseCeliac DiseaseDisease Models Animalmedicine.anatomical_structureCTLA-4ImmunologyCytokinesMolecular MedicineT-Box Domain ProteinsbusinessTranscription FactorsExpert Opinion on Therapeutic Targets
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Hepatocytes of double-transgenic mice expressing high levels of hepatitis B virus e antigen and interferon-gamma are not injured by HBeAg specific au…

2000

Seroconversion from HBeAg to alphaHBe of persons chronically infected by HBV is usually associated with a transient exacerbation of liver disease and subsequent normalization of liver histology. It is speculated that these clinico-pathological features may be due to the activation of cytodestructive mechanisms by alphaHBe antibodies. The aim of the present study was to investigate the pathogenic potential of alphaHBe antibodies in a transgenic mouse model. Therefore, alphaHBe autoantibodies were elicited in double-transgenic mice expressing high amounts of HBeAg and interferon-gamma in the liver. Interferon-gamma has reviously been shown to play an important role in the development of hepat…

Mice Transgenicmedicine.disease_causeTransfectionCell LineLiver diseaseInterferon-gammaMiceInterferonAntibody SpecificityVirologymedicineAnimalsInterferon gammaHepatitis B e AntigensSeroconversionHepatitis B AntibodiesProtein PrecursorsAutoantibodiesHepatitis B virusbiologyViral Core Proteinsvirus diseasesInterferon-alphaGeneral Medicinemedicine.diseasebiology.organism_classificationFlow CytometryHepatitis BVirologydigestive system diseasesHepadnaviridaeHBeAgLiverImmunologybiology.proteinAntibodymedicine.drugArchives of virology
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