Search results for "INTERLEUKIN-6"

showing 10 items of 370 documents

Interleukin-6 Is a Promising Marker of COVID-19 in Children: A Case Series of 2 Brothers with Severe COVID-19 Pneumonia.

2022

BACKGROUND To date, Coronavirus disease 2019 (COVID-19) remains a global health concern, with fatalities mostly in older age groups with underlying medical conditions, while children are less likely to manifest severe symptoms. CASE REPORT We describe the clinical cases of 2 brothers admitted to our Children's Hospital for persistent fever and cough during the COVID-19 pandemic. Case 1. A 1.5-year-old boy had fever, expiratory dyspnea, desaturation, oxygen saturation 94-96% with O2, and bilateral hissing and crackling rales. His interleukin-6 level in the acute phase of the disease was 100.41 and at the resolution it was 46.2 pg/ml. Treatment with amoxicillin plus clavulanic acid, methylpre…

Settore MED/04 - Patologia GeneraleMaleFeverInterleukin-6SARS-CoV-2SiblingsCOVID-19InfantGeneral MedicinePneumoniaMethylprednisoloneSettore MED/38 - Pediatria Generale E SpecialisticaCoughHumansPandemicsAgedThe American journal of case reports
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Significance of Autologous Interleukin-6 Production in the HA22T/VGH Cell Model of Hepatocellular Carcinoma

2006

Cancer cells may often support their own growth, survival, and drug resistance by autocrine/paracrine loops based on the production of different factors; results from us and others have shown that similar interleukin-6 (IL-6)-related loops are operative in multiple myeloma and prostate or renal cancer. Because this aspect has not been investigated in detail for hepatocellular carcinoma (HCC), we have examined it in HA22T/VGH cells. These differ from other primary liver cancer cell lines (that is, HepG2, HuH-6, and HuH-7) in that enzyme-linked immunosorbent assay (ELISA) showed the HA22T/VGH cells to secrete remarkable amounts of IL-6 (16.8 ng/10(6) cells/24 h); this production, due to const…

Settore MED/09 - Medicina InternaCarcinoma HepatocellularCurcuminCellBiologyautocrine cellgrowth stimulatory loopModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyAntibodiesFlow cytometryParacrine signallingHistory and Philosophy of SciencemedicineCytokine Receptor gp130Tumor Cells CulturedHumansNF-kBRNA Small InterferingReceptorAutocrine signallingNF-k Bmedicine.diagnostic_testCyclohexanonesGeneral Neuroscienceinterleukin-6Cell MembraneLiver NeoplasmsNF-kappa Bhepatocellular carcinomaMolecular biologyReceptors Interleukin-6medicine.anatomical_structureCell cultureCancer cellBenzamidesbiology.proteinSettore BIO/14 - Farmacologiaautocrine cell growth stimulatory loopAntibody
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Novel pathogenic mechanism of microbial metalloproteinases: liberation of membrane-anchored molecules in biologically active form exemplified by stud…

1996

Certain membrane-anchored proteins, including several cytokines and cytokine receptors, can be released into cell supernatants through the action of endogenous membrane-bound metalloproteinases. The shed molecules are then able to fulfill various biological functions; for example, soluble interleukin-6 receptor (sIL-6R) can bind to bystander cells, rendering these cells sensitive to the action of IL-6. Using IL-6R as a model substrate, we report that the metalloproteinase from Serratia marcescens mimics the action of the endogenous shedding proteinase. Treatment of human monocytes with the bacterial protease led to a rapid release of sIL-6R into the supernatant. This effect was inhibitable …

Staphylococcus aureusProteasesmedicine.medical_treatmentImmunologyBiologyMatrix metalloproteinaseMicrobiologyMonocytesSubstrate SpecificityAntigens CDChlorocebus aethiopsmedicineAnimalsHumansReceptorSerratia marcescensMetalloproteinaseProteaseMembrane ProteinsMetalloendopeptidasesBiological activityBacterial InfectionsReceptors InterleukinListeria monocytogenesReceptors Interleukin-6Recombinant ProteinsBlotInfectious DiseasesSolubilityBiochemistryPseudomonas aeruginosaParasitologySignal transductionResearch ArticleSignal TransductionInfection and Immunity
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Selective killing of human monocytes and cytokine release provoked by sphingomyelinase (beta-toxin) of Staphylococcus aureus.

1996

The best-known activity of Staphylococcus aureus sphingomyelinase C, alias beta-toxin, is as a hemolysin that provokes hot-cold lysis of erythrocytes which contain substantial amounts of sphingomyelin in the plasma membrane. Sheep erythrocytes are most susceptible, and we found that one hemolytic unit, representing the toxin concentration that elicits 50% hemolysis of 2.5 X 10(8) erythrocytes per ml, corresponds to 0.05 enzyme units or to approximately 0.25 microg of sphingomyelinase per ml. The cytotoxic action of beta-toxin on nucleated cells has not been described in any detail before, and the present investigation was undertaken to fill this information gap. We now identify beta-toxin a…

Staphylococcus aureusTime FactorsLipopolysaccharideCD14ImmunologyBacterial ToxinsLipopolysaccharide ReceptorsExotoxinsMicrobiologyMonocytesMicrobiologychemistry.chemical_compoundHemolysin ProteinsPhospholipase A2Antigens CDmedicineHumansbiologyCell DeathDose-Response Relationship DrugCytotoxinsMonocyteHemolysinReceptors Interleukinmedicine.diseaseReceptors Interleukin-6HemolysisInfectious Diseasesmedicine.anatomical_structureSphingomyelin PhosphodiesteraseMechanism of actionchemistrybiology.proteinCytokinesParasitologymedicine.symptomSphingomyelinResearch ArticleInterleukin-1
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REGULATORY ELEMENTS OF THE LEUKAEMIA INHIBITORY FACTOR (LIF) PROMOTER IN MURINE BONE MARROW STROMAL CELLS

1999

Leukaemia inhibitory factor (LIF) plays an important role as a haematopoietically active cytokine. As described earlier in a murine model, interleukin 1 (IL-1) induced LIF mRNA and protein expression. We utilized the murine cell line +/+-1.LDA11 to further define regulatory mechanisms of LIF expression in bone marrow stromal cells. The production of LIF mRNA is stimulated by IL-1beta, TNF-alpha, and the cAMP analogue 8-bromoadenosine 3':5'-monophosphate (8BrcAMP). LIF mRNA expression is controlled at the transcriptional level. Different fragments from -542 to -45 bp 5' upstream of the transcriptional start site of the murine LIF gene were fused to the luciferase gene. All LIF-promoter lucif…

Stromal cellRecombinant Fusion Proteinsmedicine.medical_treatmentImmunology8-Bromo Cyclic Adenosine MonophosphateBone Marrow CellsStimulationRegulatory Sequences Nucleic AcidBiologyLeukemia Inhibitory FactorBiochemistryMiceGenes ReportermedicineAnimalsHumansImmunology and AllergyLuciferaseRNA MessengerNuclear proteinPromoter Regions GeneticMolecular BiologyCells CulturedLymphokinesMessenger RNAInterleukin-6Tumor Necrosis Factor-alphaInterleukinHematologyMolecular biologyGrowth InhibitorsRecombinant ProteinsCytokinemedicine.anatomical_structureGene Expression RegulationBone marrowStromal CellsInterleukin-1Cytokine
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Sympathetic neurons can produce and respond to interleukin 6

1998

Neuronal expression of cytokines is an area of active investigation in the contexts of development, disease, and normal neural function. Although cultured rat sympathetic neurons respond very weakly to exogenous interleukin 6 (IL-6), we find that addition of soluble IL-6 receptor (sIL-6R) and IL-6 enhances neuronal survival in the absence of nerve growth factor. Neutralizing monoclonal antibodies against IL-6 block these effects. Addition of IL-6 and sIL-6R also induces a subset of neuropeptide and transmitter synthetic enzyme mRNAs identical to that demonstrated for leukemia inhibitory factor, ciliary neurotrophic factor, and oncostatin M. Both of these effects are duplicated by addition o…

Superior cervical ganglionmedicine.medical_specialtyCell SurvivalRecombinant Fusion ProteinsSuperior Cervical GanglionCiliary neurotrophic factorPC12 CellsRats Sprague-DawleyMiceParacrine signallingContactinsInternal medicinemedicineAnimalsNerve Growth FactorsRNA MessengerInterleukin 6Autocrine signallingNeural Cell Adhesion MoleculesCells CulturedNeuronsMultidisciplinarybiologyInterleukin-6Neuropeptides3T3 CellsBiological SciencesReceptors Interleukin-6RatsCell biologyAutocrine CommunicationNerve growth factorEndocrinologyAnimals Newbornbiology.proteinNeural cell adhesion moleculeLeukemia inhibitory factorCaltech Library ServicesProceedings of the National Academy of Sciences
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Mycoplasma fermentans-derived lipid inhibits class II major histocompatibility complex expression without mediation by interleukin-6, interleukin-10,…

1996

Mycoplasma cause several diseases in man and animals. Some strains can chronically infect humans, leading to fever or inflammatory syndromes such as arthritis, particularly in immunosuppressed patients. A set of pathogenicity factors shared by many mollicutes may be membrane components that activate macrophages to secrete cytokines and other inflammatory mediators. Mycoplasma-derived high molecular weight material (MDHM) is a macrophage-activating amphiphilic lipid which was purified from Mycoplasma fermentans. We studied the influence of MDHM on the expression of major histocompatibility complex (MHC) class II molecules by mouse resident peritoneal macrophages with an ELISA. Highly purifie…

T cellImmunologyAntigen presentationBiologyNitric OxideMajor histocompatibility complexMicrobiologyMiceAntigenTransforming Growth Factor betaInterferonMHC class ImedicineAnimalsImmunology and AllergyMycoplasma fermentansCells CulturedMycoplasma fermentansMice Inbred C3HInterleukin-6Tumor Necrosis Factor-alphaHistocompatibility Antigens Class IIInterleukinbiology.organism_classificationLipidsInterleukin-10Molecular WeightKineticsmedicine.anatomical_structureInterferon Type IImmunologyProstaglandinsbiology.proteinCytokinesFemaleImmunosuppressive Agentsmedicine.drugEuropean Journal of Immunology
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Oleuropein protects against dextran sodium sulfate-induced chronic colitis in mice.

2013

The anti-inflammatory effect of oleuropein (1), the major phenolic secoiridoid in Olea europaea, was evaluated in an experimental model of chronic colitis in mice. Animals were exposed to four repeated cycles of dextran sodium sulfate in drinking water followed by a 7-day rest period. Animals receiving a standard diet supplemented with 0.25% of 1 (equivalent to 500 mg/kg/day) for 56 days exhibited a decrease of inflammatory symptoms, as reflected by improvement of disease activity index and histopathological changes. It was found that 1 decreased inflammatory cell recruitment and the release of inflammatory cytokines interleukin (IL)-1β and IL-6 with increased IL-10 levels in colon tissue. …

T-LymphocytesInterleukin-1betaIridoid GlucosidesAnti-Inflammatory AgentsPharmaceutical ScienceNitric Oxide Synthase Type IIPharmacologyp38 Mitogen-Activated Protein KinasesAnalytical Chemistrychemistry.chemical_compoundMiceOleuropeinOleaDrug DiscoveryAnimalsIridoidsIntestinal MucosaChronic colitisPyransPharmacologybiologyMolecular StructureExperimental modelInterleukin-6Organic ChemistryDextran Sulfatebiology.organism_classificationColitisInterleukin-10Mice Inbred C57BLComplementary and alternative medicinechemistryOleaCyclooxygenase 2Molecular MedicineDextran sodium sulfateJournal of natural products
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Inflammation in Ischemic Stroke Subtypes

2011

Determining the cause of stroke does influence choices for management. categorization of subtypes of ischemic stroke has had considerable study, but definitions are hard to formulate and their application for diagnosis in an individual patient is often problematic. Cerebral ischemia initiates a complex cascade of events at genomic, molecular, and cellular levels, and inflammation is important in this cascade. In 1993 for For the Trial of Org 10172 in Acute Stroke Treatment (TOAST), Adams et al] conducted a placebo-controlled, randomized, blinded study of the low-molecular-weight heparinoid given to patients within 24 hours after stroke and developed a system for diagnosis of subtype of isch…

TOAST Classificationmedicine.medical_specialtySettore MED/09 - Medicina InternaIschemiaInflammationstroke subtype stroke TOASTBrain damageSeverity of Illness IndexBrain IschemiaInternal medicineOutcome Assessment Health CareDrug DiscoverySeverity of illnessmedicineAnimalsHumanscardiovascular diseasesStrokeInflammationPharmacologyInterleukin-6business.industrymedicine.diseaseSurgeryStrokeVenous thrombosisC-Reactive ProteinSubtypes ischemic stroke TOASTEtiologyCardiologyCytokinesmedicine.symptombusinessCurrent Pharmaceutical Design
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The designer cytokine hyper-interleukin-6 is a potent activator of STAT3-dependent gene transcription in vivo and in vitro.

1999

Interleukin-6 (IL-6) triggers pivotal pathways in vivo. The designer protein hyper-IL-6 (H-IL-6) fuses the soluble IL-6 receptor (sIL-6R) through an intermediate linker with IL-6. The intracellular pathways that are triggered by H-IL-6 are not defined yet. Therefore, we studied the molecular mechanisms leading to H-IL-6-dependent gene activation. H-IL-6 stimulates haptoglobin mRNA expression in HepG2 cells, which is transcriptionally mediated as assessed by run-off experiments. The increase in haptoglobin gene transcription correlates with higher nuclear translocation of tyrosine-phosphorylated STAT3 and its DNA binding. As H-IL-6 stimulates STAT3-dependent gene transcription, we compared t…

Therapeutic gene modulationSTAT3 Transcription FactorTranscriptional ActivationTranscription GeneticRecombinant Fusion ProteinsResponse elementE-boxBiologyTransfectionBiochemistryCell LineMiceSp3 transcription factorAntigens CDCytokine Receptor gp130E2F1AnimalsHumansRNA MessengerPhosphorylationMolecular BiologyCell NucleusATF3Sp1 transcription factorMice Inbred C3HMembrane GlycoproteinsHaptoglobinsInterleukin-6Liver receptor homolog-1Biological TransportCell BiologyDNAReceptors InterleukinMolecular biologyReceptors Interleukin-6DNA-Binding ProteinsGene Expression RegulationTrans-ActivatorsTyrosineThe Journal of biological chemistry
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