Search results for "Immune response"

showing 10 items of 184 documents

A whole blood test to measure SARS-CoV-2-specific response in COVID-19 patients

2021

Objectives To examine whether specific T-cell-responses to SARS-CoV-2 peptides can be detected in COVID-19 using a whole-blood experimental setting, which may be further explored as potential diagnostic tool. Methods We evaluated IFN-γ levels after stimulating whole-blood with spike and remainder-antigens peptides megapools (MP) derived from SARS-CoV-2 sequences; IL-1β, IL-1RA, IL-2, IL-4, IL-5, IL-6, IL-7, IL-8, IL-9, IL-10, IL-12p70, IL-13, IL-15, IL-17A, eotaxin, basic FGF, G-CSF, GM-CSF, IFN-γ, IP-10, MCP-1, MIP-1α, MIP-1β, PDGF, RANTES, TNF-α, VEGF were also evaluated. Results IFN-γ-response to spike and remainder-antigens MPs was significantly increased in 35 COVID-19-patients compare…

Male0301 basic medicinemedicine.medical_treatmentBasic fibroblast growth factorchemistry.chemical_compound0302 clinical medicineT-cell based testsMedicine030212 general & internal medicineIFN-γAntigens ViralMacrophage inflammatory proteinbiologyInterleukinGeneral MedicineMiddle AgedWhole bloodVascular endothelial growth factorInfectious Diseasesmedicine.anatomical_structureSpike Glycoprotein CoronavirusCytokinesOriginal ArticleFemalePlatelet-derived growth factor receptorAdultMicrobiology (medical)Specific response030106 microbiologyCOVID-19 Serological TestingInterferon-gamma03 medical and health sciencesTh2 CellsAntigenHumansImmune responseAgedSARS-CoV-2business.industryGrowth factorMonocyteCOVID-19Multiplex analysisTh1 CellsSerology responsechemistryImmunoglobulin GImmunologybiology.proteinbusinessClinical Microbiology and Infection
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Lung injury does not aggravate mechanical ventilation-induced early cerebral inflammation or apoptosis in an animal model.

2018

INTRODUCTION:The acute respiratory distress syndrome is not only associated with a high mortality, but also goes along with cognitive impairment in survivors. The cause for this cognitive impairment is still not clear. One possible mechanism could be cerebral inflammation as result of a "lung-brain-crosstalk". Even mechanical ventilation itself can induce cerebral inflammation. We hypothesized, that an acute lung injury aggravates the cerebral inflammation induced by mechanical ventilation itself and leads to neuronal damage. METHODS:After approval of the institutional and state animal care committee 20 pigs were randomized to one of three groups: lung injury by central venous injection of …

MaleARDSCritical Care and Emergency MedicinePulmonologySwinePhysiologymedicine.medical_treatmentVentilator-Induced Lung InjuryInterleukin-1betalcsh:MedicineApoptosisPathology and Laboratory MedicineHippocampusPositive-Pressure RespirationRandom Allocation0302 clinical medicineAnimal CellsImmune PhysiologyMedicine and Health Scienceslcsh:ScienceImmune ResponseAcute Respiratory Distress SyndromeTidal volumeCerebral CortexNeuronsCognitive ImpairmentRespiratory Distress SyndromeInnate Immune SystemMultidisciplinarymedicine.diagnostic_testCognitive NeurologyBrainGeneral MedicineLung InjuryNeurologyAnesthesiaBreathingCytokinesTumor necrosis factor alphamedicine.symptomAnatomyCellular TypesGeneral Agricultural and Biological SciencesResearch ArticleHistologyCognitive NeuroscienceImmunology10208 Institute of NeuropathologyInflammation610 Medicine & healthGenetics and Molecular BiologyGlial Cells1100 General Agricultural and Biological SciencesLung injury03 medical and health sciencesSigns and SymptomsRespiratory Failure1300 General Biochemistry Genetics and Molecular BiologyDiagnostic MedicinemedicineAnimalsMicroglial CellsMechanical ventilationInflammation1000 Multidisciplinarybusiness.industryInterleukin-6Tumor Necrosis Factor-alphalcsh:RBiology and Life Sciences030208 emergency & critical care medicineCell BiologyMolecular Developmentmedicine.diseaseRespiration ArtificialBronchoalveolar lavage030228 respiratory systemImmune SystemCellular NeuroscienceGeneral Biochemistry570 Life sciences; biologyCognitive Sciencelcsh:QbusinessDevelopmental BiologyNeurosciencePloS one
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Antiretroviral therapy abrogates association between arginase activity and HIV disease severity

2010

AbstractArginase-induced L-arginine deprivation is emerging as a key mechanism for the downregulation of immune responses. We hypothesised that arginase activity increases with disease severity in HIV-seropositive patients. Our results show that peripheral blood mononuclear cells (PBMCs) from 23 HIV-seropositive patients with low CD4+ T cell counts (≤350 cells/μl) expressed significantly more arginase compared with 21 patients with high CD4+ T cell counts. Furthermore, we found a significant association between the two principal prognostic markers used to monitor HIV disease (CD4+ T cell count and plasma viral load) and PBMC arginase activity in antiretroviral therapy naïve patients but not…

MaleAnti-HIV AgentsT cellT cellsCD4 cell countL-arginineHIV InfectionsArgininePeripheral blood mononuclear cellSeverity of Illness Index03 medical and health sciences0302 clinical medicineImmune systemImmunopathologymedicineHumansImmune response030304 developmental biology0303 health sciencesbiologyArginasebusiness.industryPublic Health Environmental and Occupational HealthHIVGeneral MedicineViral Loadbiology.organism_classification3. Good healthCD4 Lymphocyte CountArginaseInfectious Diseasesmedicine.anatomical_structureSociety Meeting PaperLentivirusImmunologyHIV-1Leukocytes MononuclearParasitologyFemaleViral diseasebusinessViral load030217 neurology & neurosurgeryTransactions of the Royal Society of Tropical Medicine and Hygiene
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Phase I trial of thymidylate synthase poly-epitope peptide (TSPP) vaccine in advanced cancer patients.

2015

Thymidylate synthase (TS) poly-epitope peptide (TSPP) is a 27-mer peptide vaccine containing the amino acidic sequences of three epitopes with HLA-A2.1-binding motifs of TS, an enzyme overexpressed in cancer cells, which plays a crucial role in DNA repair and replication. Based on the results of preclinical studies, we designed a phase Ib trial (TSPP/VAC1) to investigate, in a dose escalation setting, the safety and the biological activity of TSPP vaccination alone (arm A) or in combination with GM-CSF and IL-2 (arm B) in cancer patients. Twenty-one pretreated metastatic cancer patients, with a good performance status (ECOG ≤ 1) and no severe organ failure or immunological disease, were enr…

MaleCancer Researchmedicine.medical_treatmentImmunologyPharmacologyThymidylate synthaseCancer VaccinesCTLsNeoplasmsCancer vaccineMedicineImmunology and AllergyHumansImmune responseAdverse effectAgedbiologyPerformance statusbusiness.industryPhase Ib trialCancerImmunotherapyThymidylate SynthaseMiddle Agedmedicine.diseaseCancer vaccine; CTLs; Immune response; Immunotherapy; Phase Ib trial; Cancer Research; Oncology; Immunology; Immunology and AllergyOncologyCTLImmunologyToxicityVaccines Subunitbiology.proteinPeptide vaccineFemaleCancer vaccineImmunotherapybusinessCancer immunology, immunotherapy : CII
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Analysis of Mycobacterium tuberculosis-Specific CD8 T-Cells in Patients with Active Tuberculosis and in Individuals with Latent Infection

2009

CD8 T-cells contribute to control of Mycobacterium tuberculosis infection, but little is known about the quality of the CD8 T-cell response in subjects with latent infection and in patients with active tuberculosis disease. CD8 T-cells recognizing epitopes from 6 different proteins of Mycobacterium tuberculosis were detected by tetramer staining. Intracellular cytokines staining for specific production of IFN-gamma and IL-2 was performed, complemented by phenotyping of memory markers on antigen-specific CD8 T-cells. The ex-vivo frequencies of tetramer-specific CD8 T-cells in tuberculous patients before therapy were lower than in subjects with latent infection, but increased at four months a…

MaleEpitopes T-Lymphocytelcsh:MedicineCD8-Positive T-LymphocytesEpitopeDiagnostic RadiologyInfectious Diseases/Bacterial InfectionsSpectrum Analysis TechniquesCellular typesCytotoxic T celllcsh:ScienceImage Cytometryeducation.field_of_studyMultidisciplinarybiologyRadiology and ImagingImmune cellsInfection ImagingMiddle AgedFlow CytometryActinobacteriaPhenotypeSpectrophotometryCytokinesWhite blood cellsFemaleCytophotometryResearch Articlemedicine.drugInterleukin 2Cell biologyBlood cellsTuberculosisImaging TechniquesImmunologyPopulationT cellsCytotoxic T cellsResearch and Analysis MethodsMycobacterium tuberculosisDiagnostic MedicineImmunology/Immunity to InfectionsHLA-A2 AntigenmedicineHumansTuberculosiseducationMedicine and health sciencesHLA-A AntigensBacteriaFluorimetrylcsh:ROrganismsBiology and Life SciencesMycobacterium tuberculosisbiology.organism_classificationmedicine.diseaseVirologyRetractionAnimal cellsImmunology/Immune ResponseImmunologyMycobacterium tuberculosis CD8 T cells Tuberculosis Latent Infectionlcsh:QCD8MycobacteriumPLoS ONE
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Immunogenetics, Gender, and Longevity

2007

In this article we discuss relevant data on aging, longevity, and gender with particular focus on inflammation gene polymorphisms which could affect an individual's chance to reach the extreme limit of human life. The present review is not an extensive revision of the literature, but rather an expert opinion based on selected data from the authors' laboratories. In 2000-2005 in the more developed regions, the life expectancy at birth is 71.9 years for men (78.3 in Japan) and 79.3 years for women (86.3 in Japan). Indeed, gender accounts for important differences in the prevalence of a variety of age-related diseases. Considering people of far-advanced age, demographic data document a clear-c…

MaleGerontologyAgingmedia_common.quotation_subjectLongevityPopulationDiseaseGeneral Biochemistry Genetics and Molecular BiologyHistory and Philosophy of ScienceHLA AntigensImmunogeneticsHumansMedicineSex Ratioeducationmedia_commonInflammationeducation.field_of_studySuccessful agingbusiness.industryGeneral NeuroscienceMortality rateAging Immune response Inflammation LongevityLongevityInfectious disease (medical specialty)Life expectancyFemalebusinessDeveloped countryAnnals of the New York Academy of Sciences
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CANDIDA ALBICANS INDUCES SELECTIVE DEVELOPMENT OF MACROPHAGES AND MONOCYTE DERIVED DENDRITIC CELLS BY A TLR2 DEPENDENT SIGNALLING

2011

As TLRs are expressed by haematopoietic stem and progenitor cells (HSPCs), these receptors may play a role in haematopoiesis in response to pathogens during infection. We have previously demonstrated that in in vitro defined conditions inactivated yeasts and hyphae of Candida albicans induce HSPCs proliferation and differentiation towards the myeloid lineage by a TLR2/MyD88 dependent pathway. In this work, we showed that C. albicans invasive infection with a low virulence strain results in a rapid expansion of HSPCs (identified as LKS cells: Lin(-) c-Kit(+) Sca-1(+) IL-7R alpha(-)), that reach the maximum at day 3 post-infection. This in vivo expansion of LKS cells in TLR2(-/-) mice was del…

MaleImmune CellsCellular differentiationImmunologylcsh:MedicineMycologyMicrobiologyMonocytesMiceCandida albicansAnimalsLymphopoiesisProgenitor celllcsh:ScienceCandida albicansBiologyImmune ResponseCells CulturedMice KnockoutMultidisciplinarybiologyMacrophageslcsh:RFungal DiseasesCandidiasisCell DifferentiationHematologyDendritic CellsFlow Cytometrybiology.organism_classificationToll-Like Receptor 2Corpus albicansHematopoiesisCell biologyHost-Pathogen InteractionToll-Like Receptor 4HaematopoiesisTLR2Infectious DiseasesMyeloid Differentiation Factor 88Medicinelcsh:QFemaleStem cellInfectious Disease ModelingResearch ArticleSignal Transduction
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Blockade of MIF-CD74 Signalling on Macrophages and Dendritic Cells Restores the Antitumour Immune Response Against Metastatic Melanoma.

2018

Mounting an effective immune response against cancer requires the activation of innate and adaptive immune cells. Metastatic melanoma is the most aggressive form of skin cancer. While immunotherapies have shown a remarkable success in melanoma treatment, patients develop resistance by mechanisms that include the establishment of an immune suppressive tumor microenvironment. Thus, understanding how metastatic melanoma cells suppress the immune system is vital to develop effective immunotherapies against this disease. In this study, we find that macrophages (MOs) and dendritic cells (DCs) are suppressed in metastatic melanoma and that the Ig-CDR-based peptide C36L1 is able to restore MOs and …

MaleModels MolecularImmunologyMelanoma Experimentalchemical and pharmacologic phenomenaModels Biologicalimmune responseMiceStructure-Activity RelationshipT-Lymphocyte SubsetsAnimalsdendritic cellsNeoplasm MetastasisReceptors ImmunologicMacrophage Migration-Inhibitory FactorsMelanomaOriginal ResearchMacrophagesHistocompatibility Antigens Class IIImmunitypeptide-based immunotherapyAntigens Differentiation B-LymphocyteCD74macrophage migration inhibitory factorPeptidesProtein BindingSignal Transductionmetastatic melanomaFrontiers in immunology
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Local increase of arginase activity in lesions of patients with cutaneous leishmaniasis in Ethiopia.

2012

Background Cutaneous leishmaniasis is a vector-borne disease that is in Ethiopia mainly caused by the parasite Leishmania aethiopica. This neglected tropical disease is common in rural areas and causes serious morbidity. Persistent nonhealing cutaneous leishmaniasis has been associated with poor T cell mediated responses; however, the underlying mechanisms are not well understood. Methodology/Principal Findings We have recently shown in an experimental model of cutaneous leishmaniasis that arginase-induced L-arginine metabolism suppresses antigen-specific T cell responses at the site of pathology, but not in the periphery. To test whether these results translate to human disease, we recruit…

MalePathologyCD3 ComplexBiopsyAntigens CD8Antigens CD3Antigens CD40302 clinical medicineINFECTIONSUPPRESSOR-CELLSAETHIOPICAChildImmune ResponseSOUTH-WESTERN ETHIOPIAIN-VIVOSkin0303 health sciencesbiologyPARASITOLOGYlcsh:Public aspects of medicine11 Medical And Health SciencesMiddle Aged3. Good healthArginaseInfectious Diseasesmedicine.anatomical_structureCD4 AntigensMedicineFemalemedicine.symptomLife Sciences & BiomedicineResearch ArticleNeglected Tropical DiseasesEXPRESSIONAdultmedicine.medical_specialtylcsh:Arctic medicine. Tropical medicineAdolescentlcsh:RC955-962CD8 AntigensT cellImmunology030231 tropical medicineLeishmaniasis CutaneousPeripheral blood mononuclear cellImmunomodulationLesionYoung Adult03 medical and health sciencesLeishmania aethiopicaCutaneous leishmaniasisTropical MedicineParasitic DiseasesL-ARGININE METABOLISMmedicineACTIVATED GRANULOCYTESHumansBiology030304 developmental biologyScience & TechnologyNITRIC-OXIDEArginasebusiness.industryPublic Health Environmental and Occupational HealthLeishmaniasislcsh:RA1-127006 Biological Sciencesbiology.organism_classificationmedicine.diseaseMICEImmunologyLeukocytes MononuclearEthiopiabusinessCD8PLoS Neglected Tropical Diseases
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The effect of biological sealants and adhesive treatments on matrix metalloproteinase expression during renal injury healing

2017

Background Renal injuries are relatively common in cases of abdominal trauma. Adhesives and sealants can be used to repair and preserve damaged organs. Using a rat model, this study explores the activity of different matrix metalloproteinases (MMP) during the healing of renal injuries treated by two biological adhesives (TachoSil and GelitaSpon) and a new synthetic elastic cyanoacrylate (Adhflex). Methods Renal traumatic injuries were experimentally induced in 90 male Wistar rats by a Stiefel Biopsy Punch in the anterior aspect of the left kidney. Animals were divided into five groups: 1, sham non-injured (n = 3); 2, non-treated standard punch injury (n = 6); 3, punch injury treated with Ta…

MalePathologyCritical Care and Emergency MedicinePhysiology030232 urology & nephrologylcsh:MedicineMatrix metalloproteinasePathology and Laboratory MedicineKidneyMMP8Biochemistrylaw.invention0302 clinical medicinelawMedicine and Health Scienceslcsh:ScienceImmune ResponseTrauma MedicineKidneyMultidisciplinaryProteasesTachoSilEnzymesmedicine.anatomical_structureTraumatic injuryCyanoacrylate030220 oncology & carcinogenesisPhysical SciencesAnatomyTraumatic InjuryResearch Articlemedicine.medical_specialtyHistologyMaterials ScienceImmunologyFibrin Tissue Adhesive03 medical and health sciencesSigns and SymptomsDiagnostic MedicineAdhesivesTissue RepairmedicineAnimalsRats WistarMaterials by AttributeInflammationWound Healingbusiness.industrylcsh:RBiology and Life SciencesProteinsKidneysRenal Systemmedicine.diseaseMatrix MetalloproteinasesAbdominal traumaEnzymologyMetalloproteaseslcsh:QPhysiological ProcessesbusinessWound healingCollagensPLOS ONE
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