Search results for "Immune system"

showing 10 items of 2885 documents

Dual anti-oxidant and anti-inflammatory actions of the electrophilic cyclooxygenase-2-derived 17-oxo-DHA in lipopolysaccharide- and cigarette smoke-i…

2014

Abstract Background 17-Oxo-DHA is an endogenous electrophilic derivative of the omega-3 fatty acid docosahexaenoic acid (DHA) which is generated in activated macrophages by the action of cyclooxygenase-2. Methods The ability of 17-oxo-DHA to control inflammation and oxidative stress was tested in human macrophages (THP-1) and bronchial epithelial cell line (16HBE) stimulated with cigarette smoke extract (CSE) and lipopolysaccharide (LPS). All data were further confirmed using primary bronchial epithelial cells, alveolar macrophages and peripheral blood mononuclear cells. Results 17-Oxo-DHA was a strong inducer of the anti-oxidant response promoting Nrf2 nuclear accumulation, leading to the …

LipopolysaccharidesLipopolysaccharideDocosahexaenoic Acidsmedicine.drug_classBiophysicsAnti-Inflammatory AgentsInflammationPharmacologymedicine.disease_causeBiochemistryAnti-inflammatoryAntioxidantsCell Linechemistry.chemical_compoundImmune systemmedicineHumansOmega 3 fatty acidMolecular BiologyInflammationChemistryMacrophagesSmokingfood and beveragesEpithelial CellsHeme oxygenaseBiochemistryCyclooxygenase 2Leukocytes Mononuclearlipids (amino acids peptides and proteins)Tumor necrosis factor alphamedicine.symptomOxidative stressBiochimica et biophysica acta
researchProduct

TNFα Primes Polymorphonuclear Leukocytes for an Enhanced Respiratory Burst to a Similar Extent As Bacterial Lipopolysaccharide

1990

We examined whether preincubating polymorphonuclear leukocytes (PMN) with TNF alpha would result in an enhanced respiratory burst upon subsequent stimulation by various agents. Bacterial lipopolysaccharide (LPS), a known primer of PMN, was used as control. We found that both LPS (0.01 to 10.0 microgram/ml) and recombinant TNF alpha (0.001 to 1.0 microgram/ml) act as direct stimulants of PMN as measured by chemiluminescence. Sixty minutes of preincubation of PMN with 1 microgram/ml TNF alpha or 10 micrograms/ml LPS resulted in similar priming for the respiratory burst elicited by opsonized zymosan, phorbol myristate acetate, zymosan, zymosan-activated serum, aggregated immunoglobulin, and f-…

LipopolysaccharidesLipopolysaccharideNeutrophilsPriming (immunology)StimulationDermatologyPharmacologyBiochemistryAntibodieschemistry.chemical_compoundOxygen ConsumptionHumansReceptors ImmunologicReceptorOpsoninMolecular BiologyTumor Necrosis Factor-alphaZymosanhemic and immune systemsCell BiologyReceptors Formyl PeptideRespiratory burstN-Formylmethionine Leucyl-PhenylalaninechemistryImmunologyTumor necrosis factor alphaJournal of Investigative Dermatology
researchProduct

Immune Cell Toll-like Receptor 4 Mediates the Development of Obesity- and Endotoxemia-Associated Adipose Tissue Fibrosis

2014

International audience; Adipose tissue fibrosis development blocks adipocyte hypertrophy and favors ectopic lipid accumulation. Here, we show that adipose tissue fibrosis is associated with obesity and insulin resistance in humans and mice. Kinetic studies in C3H mice fed a high-fat diet show activation of macrophages and progression of fibrosis along with adipocyte metabolic dysfunction and death. Adipose tissue fibrosis is attenuated by macrophage depletion. Impairment of Toll-like receptor 4 signaling protects mice from obesity-induced fibrosis. The presence of a functional Toll-like receptor 4 on adipose tissue hematopoietic cells is necessary for the initiation of adipose tissue fibros…

LipopolysaccharidesMESH: Signal TransductionMESH: InflammationMESH : Toll-Like Receptor 4Adipose tissueMESH : AdipocytesMESH : LipopolysaccharidesMicechemistry.chemical_compoundFibrosisAdipocyteAdipocytes[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyMESH: ObesityMESH: Animalslcsh:QH301-705.5Mice Inbred C3HToll-like receptorMESH : Diet High-FatMESH: Toll-Like Receptor 43. Good healthMESH: Insulin ResistanceAdipose TissueMESH: FibrosisMESH : Fibrosis[SDV.IMM]Life Sciences [q-bio]/ImmunologyMESH : ObesityMESH : Insulin ResistanceMESH: Adipose TissueSignal Transductionmedicine.medical_specialty[SDV.IMM] Life Sciences [q-bio]/ImmunologyAdipose tissue macrophagesBiologyDiet High-FatMESH : Adipose TissueGeneral Biochemistry Genetics and Molecular BiologyImmune systemMESH : Mice Inbred C3HInternal medicineMESH : MicemedicineAnimalsHumansObesityMESH: Mice Inbred C3HMESH: MiceMESH: AdipocytesInflammationMESH : Signal TransductionMESH : InflammationMESH: HumansMESH : EndotoxemiaMESH : Humans3T3-L1medicine.diseaseFibrosisMESH : Disease Models AnimalEndotoxemiaToll-Like Receptor 4Disease Models AnimalMESH: Diet High-FatEndocrinologylcsh:Biology (General)chemistryMESH: EndotoxemiaMESH : AnimalsInsulin ResistanceMESH: Disease Models AnimalMESH: LipopolysaccharidesAdipocyte hypertrophyCell Reports
researchProduct

Assessing the Cost of Mounting an Immune Response

2003

International audience; The evolution of parasite resistance has often been assumed to be governed by antagonistic selection pressures. Defense against pathogens, by mounting an immune response, confers evident benefits but may also incur costs, so that the optimal level of defense is expected to depend on the balance between benefits and costs. Although the benefits of immune surveillance are well known, estimates of costs are still equivocal. Here we studied the behavioral and physiological modifications associated with exposure to a onreplicating antigen (lipopolysaccharide [LPS] of Escherichia coli) in a passerine species, the house sparrow (Passer domesticus).We further investigated wh…

LipopolysaccharidesMale0106 biological sciencesLPSLitter SizeEcoimmunologyEnzyme-Linked Immunosorbent AssayTrade-off010603 evolutionary biology01 natural sciencesHost-Parasite InteractionsNesting BehaviorLife history theorySongbirds03 medical and health sciencesImmune systemAntigenbiology.animalEscherichia coliAnimalsEcology Evolution Behavior and Systematicstrade-off030304 developmental biologyparental effort[ SDE.BE ] Environmental Sciences/Biodiversity and Ecology0303 health sciencesbiologyReproductive successReproductionBody WeightFeeding BehaviorAntibodies BacterialBiological EvolutionBroodPasserinelife-history traitsparasite resistance[SDE.BE] Environmental Sciences/Biodiversity and Ecologyreproductive successImmunoglobulin Gtrade-off.ImmunologyFemale[SDE.BE]Environmental Sciences/Biodiversity and EcologyThe American Naturalist
researchProduct

Parental experience of a risky environment leads to improved offspring growth rate.

2014

Abstract Parasites (or diseases) are major selective force for the evolution of life history traits and parasite-host evolution. Mothers can show a variety of responses to parasites during pregnancy with different consequences for them or their offspring. However, whether information in the maternal environment before pregnancy can cause a change in the phenotype of the offspring is unknown. To avoid the confounding effect of pathogens and to reduce the risk of direct effect of mother's immune activation, we injected female laboratory mice with lipopolysaccharides (LPS) before mating. In order to provide a constant information on the potential infectious risk of the environment, females wer…

LipopolysaccharidesMale0106 biological sciencesPhysiologyOffspringPhysiologyAquatic ScienceBiology010603 evolutionary biology01 natural sciencesLife history theoryMice03 medical and health sciencesMaternal effectPregnancyFetal programmingmedicine[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisAnimalsBody SizeSexual maturityMolecular BiologyThrifty gene hypothesisEcology Evolution Behavior and Systematics030304 developmental biologyInflammation0303 health sciencesPregnancy[ SDE.BE ] Environmental Sciences/Biodiversity and EcologyRodentReproductive successMaternal effectmedicine.diseasePaternal ExposureMaternal ExposureImmune SystemInsect SciencePaternal ExposureImmunologyFemaleAnimal Science and Zoology[SDE.BE]Environmental Sciences/Biodiversity and EcologyParent–offspring conflict[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/Symbiosis
researchProduct

An experimental test of the dose-dependent effect of carotenoids and immune activation on sexual signals and antioxidant activity.

2004

Carotenoid‐based sexual traits are thought to be reliable indicators of male quality because they might be scarce and therefore might indicate the ability of males to gather high‐quality food and because they are involved in important physiological functions (as immune enhancers and antioxidants). We performed an experiment where male and female zebra finches (Taeniopygia guttata) were provided with increasing carotenoid doses in the drinking water during 4 weeks (bill color of this species is a carotenoid‐based sexual signal). Simultaneously, birds were split into two groups: one receiving weekly injections of Escherichia coli lipopolysaccharide in order to activate the immune system, the …

LipopolysaccharidesMaleAntioxidantLipopolysaccharidemedicine.medical_treatmentAnimal Communication; Animals; Antioxidants/physiology; Beak/anatomy & histology; Body Size; Carotenoids/blood; Carotenoids/pharmacology; Color; Cues; Escherichia coli/metabolism; Female; Finches/anatomy & histology; Finches/immunology; Immunity Innate/drug effects; Lipopolysaccharides/pharmacology; Male; Sexual Behavior AnimalColormacromolecular substancesPharmacologymedicine.disease_causeAntioxidantschemistry.chemical_compoundSexual Behavior AnimalImmune systemImmunitypolycyclic compoundsmedicineEscherichia coliAnimalsBody SizeCarotenoidZebra finchEcology Evolution Behavior and Systematicschemistry.chemical_classificationbiologyorganic chemicalsBeakfood and beveragesbiology.organism_classificationCarotenoidsbiological factorsImmunity InnateAnimal CommunicationchemistryImmunologyFemaleFinchesCuesTaeniopygiaOxidative stressThe American naturalist
researchProduct

Phase I study of OM-174, a lipid A analogue, with assessment of immunological response, in patients with refractory solid tumors.

2013

International audience; BACKGROUND: Lipids A, the lipophilic partial structure of lipopolysaccharides, induce regression of several tumor types in animal models. Rather than exerting direct cytotoxic effect, these compounds trigger the immune system which in turn stimulates secretion of cytokines, and activates the inducible nitric oxide synthase, as well as immune cell infiltration of tumors. OM-174 is an analogue of lipid A with dual action on toll-like receptors 2 and 4. In an experimental model of peritoneal carcinomatosis induced in BDIX rats by intraperitoneal injection of syngeneic PROb colon cancer cells, it induced a complete regression of tumors. The present phase I trial was cond…

LipopolysaccharidesMaleCancer Researchmedicine.medical_treatmentPharmacologyRefractory solid tumors[ SDV.CAN ] Life Sciences [q-bio]/CancerOM-1740302 clinical medicineNeoplasmsLipid A analogue0303 health sciencesMiddle Aged3. Good healthKiller Cells NaturalTreatment OutcomeCytokineOncology030220 oncology & carcinogenesisVomitingCytokinesFemaleChillsmedicine.symptomResearch ArticleAdultMaximum Tolerated DoseDoseIntraperitoneal injectionAntineoplastic Agents[SDV.CAN]Life Sciences [q-bio]/CancerDrug Administration Schedule03 medical and health sciencesImmune systemPhase IPharmacokinetics[SDV.CAN] Life Sciences [q-bio]/CancerCell Line TumormedicineGeneticsAnimalsHumansImmune responseAged030304 developmental biologyChemotherapyPolymorphism Geneticbusiness.industryRatsToll-Like Receptor 4Disease Models Animalbusiness
researchProduct

Neuronal activity triggers uptake of hematopoietic extracellular vesicles in vivo

2019

Communication with the hematopoietic system is a vital component of regulating brain function in health and disease. Traditionally, the major routes considered for this neuroimmune communication are by individual molecules such as cytokines carried by blood, by neural transmission, or, in more severe pathologies, by the entry of peripheral immune cells into the brain. In addition, functional mRNA from peripheral blood can be directly transferred to neurons via extracellular vesicles (EVs), but the parameters that determine their uptake are unknown. Using varied animal models that stimulate neuronal activity by peripheral inflammation, optogenetics, and selective proteasome inhibition of dop…

LipopolysaccharidesMaleGene ExpressionStimulationHippocampusBiochemistryStereotaxic Techniques0302 clinical medicineShort ReportsAnimal CellsMedicine and Health SciencesPremovement neuronal activityBiology (General)Routes of AdministrationNeurons0303 health sciencesBrain MappingKainic AcidBrainAnimal ModelsPeripheralCell biologyHaematopoiesisBioassays and Physiological AnalysisExperimental Organism SystemsHippocampus ; Yellow flourescent protein ; Intravenous injections ; Marker genes ; Gene expression ; Neurons ; Microglial cells ; OptogeneticsFemaleCellular TypesSignal TransductionProteasome Endopeptidase ComplexQH301-705.5Yellow Fluorescent ProteinMice TransgenicGlial CellsMouse ModelsStimulus (physiology)BiologyResearch and Analysis Methods03 medical and health sciencesExtracellular VesiclesImmune systemModel OrganismsIn vivoIntravenous InjectionsGeneticsAnimalsddc:610Molecular Biology TechniquesMolecular BiologyMicroglial Cells030304 developmental biologyInflammationPharmacologyMessenger RNABlood CellsUbiquitinDopaminergic NeuronsBiology and Life SciencesProteinsMarker GenesCell BiologyNeurophysiological AnalysisOptogeneticsLuminescent ProteinsCellular NeuroscienceAnimal Studies030217 neurology & neurosurgeryNeuroscience
researchProduct

CD11c+ Alveolar Macrophages are a Source of IL-23 During Lipopolysaccharide-Induced Acute Lung Injury

2013

Acute lung injury (ALI) is a severe pulmonary disease causing high numbers of fatalities worldwide. Innate immune responses are an integral part of the pathophysiologic events during ALI. Interleukin 23 (IL-23) is a proinflammatory mediator known to direct the inflammatory responses in various settings of infection, autoimmunity, and cancer. Interleukin 23 has been associated with proliferation and effector functions in T(H)17 cells. Surprisingly, little is known about production of IL-23 during ALI. In this study, we found expression of mRNA for IL-23p19 to be 10-fold elevated in lung homogenates of C57BL/6 mice after lipopolysaccharide (LPS)-induced ALI. Likewise, concentrations of IL-23 …

LipopolysaccharidesMaleLipopolysaccharideAcute Lung InjuryCD11cBiologyLung injuryCritical Care and Intensive Care MedicineInterleukin-23ArticleProinflammatory cytokineMicechemistry.chemical_compoundMacrophages AlveolarmedicineAnimalsInnate immune systemmedicine.diagnostic_testrespiratory systemCD11c Antigenrespiratory tract diseasesBronchoalveolar lavagechemistryImmunologyEmergency MedicineAlveolar macrophageInterleukin 17Shock
researchProduct

HSP60 and CpG-DNA-oligonucleotides differentially regulate LPS-tolerance of hepatic Kupffer cells

2004

Background/aims: Hepatic Kupffer cells (KC) are major regulators of the immune response to gut-derived bacterial products; uncontrolled activation of KC by bacterial components is of pathogenic relevance in alcoholic hepatitis and septic shock. Methods: We examined the role of bacterial lipopolysaccharide (LPS), bacterial and autologous HSP60 and bacterial DNA, which are recognized by innate Toll-like receptors, during activation of murine KC. Results: In cultivated KC, autologous HSP60 induced a state of LPS-hyporesponsiveness; bacterial DNA did not mitigate the response to subsequent LPS-challenge in vitro; in contrast, pre-treatment of mice with bacterial DNA even significantly increased…

LipopolysaccharidesMaleLipopolysaccharideKupffer CellsImmunologyGene ExpressionGalactosamineReceptors Cell SurfaceCell LineMicrobiologyMicechemistry.chemical_compoundImmune systemImmunityHeat shock proteinAnimalsImmunology and AllergyInterleukin 6Cells CulturedbiologyInterleukin-6Reverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaAlanine TransaminaseChaperonin 60Macrophage ActivationToll-Like Receptor 9DNA-Binding ProteinsToll-Like Receptor 4LiverOligodeoxyribonucleotideschemistryToll-Like Receptor 9Immunologybiology.proteinFemaleHSP60Tumor necrosis factor alphaLiver FailureImmunology Letters
researchProduct