Search results for "Immune system"

showing 10 items of 2885 documents

Modification of Peripheral Blood T-Lymphocyte Surface Receptors and Langerhans Cell Numbers in Hereditary Angioedema

1986

Hereditary angioedema (HAE) is a disease related to a complement disorder, namely a deficiency of C1 esterase inhibitor. Complement-split products are implicated in the regulation of the immune response, and we have compared some immunologic parameters between HAE and normal individuals. T-lymphocytes with receptors for IgG were increased in HAE, but no difference in T-cell suppressor activity for B-cells was detected. Furthermore, increased IgG receptor expression was not accompanied by any significant changes in the ratios of OKT4- and OKT8-defined antigens. Numbers of peripheral mononuclear cells (MNC) detected by alpha-naphthyl acetate esterase (ANAE) staining positivity were not signif…

AdultMaleLangerhans cellT-LymphocytesReceptor expressionReceptors Antigen T-CellFluorescent Antibody TechniqueCell CountT-Lymphocytes RegulatoryPeripheral blood mononuclear cellMonocytesLeukocyte CountImmune systemmedicineHumansAngioedemaReceptorSkinChemistryAntibodies MonoclonalGeneral MedicineMiddle Agedmedicine.diseaseComplement systemmedicine.anatomical_structureLangerhans CellsHereditary angioedemaImmunologyFemaleCell activationAmerican Journal of Clinical Pathology
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Vaccination with Mage-3a1 Peptide–Pulsed Mature, Monocyte-Derived Dendritic Cells Expands Specific Cytotoxic T Cells and Induces Regression of Some M…

1999

Dendritic cells (DCs) are considered to be promising adjuvants for inducing immunity to cancer. We used mature, monocyte-derived DCs to elicit resistance to malignant melanoma. The DCs were pulsed with Mage-3A1 tumor peptide and a recall antigen, tetanus toxoid or tuberculin. 11 far advanced stage IV melanoma patients, who were progressive despite standard chemotherapy, received five DC vaccinations at 14-d intervals. The first three vaccinations were administered into the skin, 3 × 106 DCs each subcutaneously and intradermally, followed by two intravenous injections of 6 × 106 and 12 × 106 DCs, respectively. Only minor (less than or equal to grade II) side effects were observed. Immunity t…

AdultMaleLung NeoplasmsImmunologyCD8-Positive T-LymphocytesTuberculincytotoxic T lymphocytesCancer VaccinesMonocytesLymphocytes Tumor-InfiltratingImmune systemAntigenAntigens NeoplasmTetanus ToxoidmelanomaHumansImmunology and AllergyMedicineCytotoxic T celldendritic cellsNeoplasm MetastasisLymph nodeImmunization ScheduleAgedNeoplasm Stagingactive immunotherapybusiness.industryMelanomaDendritic cellMiddle Agedvaccinationmedicine.diseaseTumor antigenNeoplasm Proteinsmedicine.anatomical_structureImmunologyFemaleOriginal ArticlebusinessCD8T-Lymphocytes CytotoxicJournal of Experimental Medicine
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HLA-B8,DR3 phenotype and lymphocyte responses to phytohaemagglutinin.

1990

Several reports have shown that HLA-B8,DR3 positive subjects may display some changes in immune parameters when compared with HLA-B8,DR3 negative ones and are prone to develop several immunological diseases. In the present study we have analysed the proliferative response to phytohaemagglutin (PHA) in HLA-typed healthy subjects. A twin method was also employed to assess the role of genetic and environmental factors in the regulation of the response to the mitogen. It was not possible to demonstrate any difference in proliferative response to optimal doses of PHA between groups of subjects carrying or not carrying the HLA-B8,DR3 phenotype. When suboptimal responses were studied, however, the…

AdultMaleLymphocyteImmunologyImmunogeneticsmedicine.disease_causeLymphocyte ActivationHLA-B8 AntigenImmune systemHLA-DR3 AntigenGeneticsmedicineTwins DizygoticHumansPhytohemagglutininsPhytohaemagglutininbiologyCell growthEnvironmental factorTwins MonozygoticPhenotypeProliferative responsemedicine.anatomical_structurePhenotypeImmunologybiology.proteinFemaleJournal of immunogenetics
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Unique Characteristics of the Intestinal Immune System as an Inductive Site after Antigen Reencounter

2004

Background Immunization prepares the body for a reencounter with the microbe. Information on the targeting of immune effector cells during secondary immune response--that is, lymphocyte homing--is scarce. In the present study, the homing potentials of lymphocytes are examined after antigen reencounter at mucosal versus nonmucosal sites. Methods Orally or parenterally immunized volunteers were reimmunized orally or parenterally with Salmonella typhi Ty21a, and the expression of the gut homing receptor (HR), alpha(4)beta(7), and of the peripheral lymph node HR, L-selectin, was investigated in circulating antigen-specific antibody-secreting cells (ASCs). Lymphocytes were sorted by HR expressio…

AdultMaleLymphocyteReceptors Lymphocyte HomingAdministration OralPriming (immunology)chemical and pharmacologic phenomenaLymphocyte migration into lymph nodeLymphocyte Activation03 medical and health sciences0302 clinical medicineImmune systemAntigenmedicineHumansImmunology and AllergyInfusions ParenteralAntibody-Producing CellsLymphocyte homing receptor030304 developmental biology0303 health sciencesbiologyDrug Administration RoutesTyphoid-Paratyphoid VaccinesSalmonella typhi3. Good healthIntestinesInfectious Diseasesmedicine.anatomical_structureImmune SystemImmunologybiology.proteinFemaleL-selectin030215 immunologyHoming (hematopoietic)The Journal of Infectious Diseases
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CD52‐negative T cells predict acute graft‐versus‐host disease after an alemtuzumab‐based conditioning regimen

2020

Allogeneic haematopoietic stem cell transplantation (HSCT) after a reduced-intensity conditioning (RIC) regimen with fludarabine, melphalan and alemtuzmab is an effective therapy for haematological malignancies. Alemtuzumab, a monoclonal antibody against CD52, a glycosylphosphatidylinositol-anchor-bound surface protein on lymphocytes, depletes T cells to prevent graft-versus-host disease (GVHD). Despite this, acute and chronic GVHD (a/cGVHD) remain life-threatening complications after HSCT. The aim of the present study was to identify parameters to predict GVHD. In 69 patients after HSCT, T-cell subsets were functionally analysed. Reconstitution of CD52neg T cells and CD52neg regulatory T c…

AdultMaleMelphalanReceptors CXCR3Transplantation ConditioningReceptors CCR5CD52Graft vs Host Diseasechemical and pharmacologic phenomenaCXCR3T-Lymphocytes Regulatory03 medical and health sciences0302 clinical medicineRisk Factorsimmune system diseaseshemic and lymphatic diseasesmedicineHumansAlemtuzumabAgedbusiness.industryHematopoietic Stem Cell TransplantationMembrane ProteinsHematologyMiddle AgedAllograftsFludarabineTransplantationHaematopoiesissurgical procedures operativeCD52 Antigen030220 oncology & carcinogenesisAcute DiseaseImmunologyAlemtuzumabFemaleStem cellbusiness030215 immunologymedicine.drugBritish Journal of Haematology
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In vitroresponse toCandida albicansin cultures of whole human blood from young and aged donors

2007

Invasive infections with opportunistic fungi, such as Candida albicans, have become an increasing problem in aged adults in recent years. This work investigates the influence of human ageing on C. albicans recognition by toll-like receptors (TLRs), essential components of the innate immune system, using a cohort of 96 young (15-42 years) and aged (70 years) human volunteers. No significant differences between aged and young donors were observed on (1) cell surface TLR2, TLR6 and TLR4 expression on lymphocytes, monocytes and granulocytes, (2) production of cytokines [IL-8, IL-1beta, IL-6, IL-10, tumour necrosis factor (TNF)-alpha and IL-12p70] and prostaglandin E(2) (PGE(2)) by whole human b…

AdultMaleMicrobiology (medical)Adolescentmedicine.medical_treatmentImmunologyBiologyMicrobiologyDinoprostoneMonocytesCandida albicansmedicineHumansImmunology and AllergyBlood cultureLymphocytesCandida albicansAntibodies FungalAgedWhole bloodAged 80 and overMicrobial ViabilityInnate immune systemmedicine.diagnostic_testAge FactorsGeneral Medicinebiology.organism_classificationToll-Like Receptor 2Corpus albicansToll-Like Receptor 4TLR2BloodToll-Like Receptor 6Infectious DiseasesCytokineImmunologybiology.proteinCytokinesFemaleAntibodyGranulocytesFEMS Immunology & Medical Microbiology
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Relevance of gamma interferon, tumor necrosis factor alpha, and interleukin-10 gene polymorphisms to susceptibility to Mediterranean spotted fever.

2009

Several studies have demonstrated that cellular immunity plays a critical role in the protective immune response against Rickettsia conorii. Immune CD4+ and CD8+ T cells are both involved in the control of rickettsial infection (38). Perivascular infiltrated CD4+ and CD8+ T lymphocytes, macrophages, and natural killer cells produce chemokines and cytokines that activate endothelial rickettsicidal activities. Infected human cells, including endothelial cells, hepatocytes, and macrophages, activated by gamma interferon (IFN-γ), tumor necrosis factor alpha (TNF-α), and interleukin-1β (IL-1β), kill intracellular rickettsiae by one or a combination of three mechanisms, involving nitric oxide syn…

AdultMaleMicrobiology (medical)Meningitidesgene polymorphismgene polymorphisms; mediterranean spotted fevermedicine.medical_treatmentClinical BiochemistryImmunologyBiologyBoutonneuse FeverPolymorphism Single NucleotideInterferon-gammaImmune systemInterferonmedicineHumansImmunology and AllergyCytotoxic T cellSicilyTumor Necrosis Factor-alphaMiddle AgedInterleukin-10mediterranean spotted feverInterleukin 10CytokineImmunologyFemaleTumor necrosis factor alphaDisease SusceptibilityMicrobial ImmunologyCD8medicine.drug
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TpF1 from Treponema pallidum Activates Inflammasome and Promotes the Development of Regulatory T Cells

2011

Abstract Human syphilis is a multistage disease, with diverse and wide-ranging manifestations caused by Treponema pallidum. Despite the fact that a cell-mediated immune response takes part in the course of syphilis, T. pallidum often manages to evade host immunity and, in untreated individuals, may trigger chronic infection. With this study, we demonstrate for the first time, to our knowledge, that Treponema pallidum induces a regulatory T (Treg) response in patients with secondary syphilis and we found that the miniferritin TpF1, produced by the bacterium, is able to expand this response and promote the production of TGF-β. Accordingly, TpF1 stimulates monocytes to release IL-10 and TGF-β,…

AdultMaleMultiprotein complexInflammasomesVirulence FactorsCellsT-LymphocytesImmunologyAdult; Antigens Helminth; Cell Differentiation; Cells Cultured; Down-Regulation; Female; Humans; Inflammasomes; Inflammation Mediators; Male; Middle Aged; Monocytes; Syphilis; T-Lymphocytes Regulatory; Transforming Growth Factor beta; Treponema pallidum; Virulence FactorsDown-RegulationBiologyT-Lymphocytes RegulatoryMonocytesMicrobiologyProinflammatory cytokineImmune systemAntigenTransforming Growth Factor betaHelminthmedicineHumansImmunology and AllergySyphilisTreponema pallidumAntigensCells CulturedCulturedTreponemaCell DifferentiationInflammasomeMiddle Agedbiology.organism_classificationmedicine.diseaseRegulatoryChronic infectionAntigens HelminthImmunologyFemaleSyphilisInflammation Mediatorsmedicine.drugThe Journal of Immunology
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High-resolution HLA matching in hematopoietic stem cell transplantation: a retrospective collaborative analysis.

2013

To validate current donor selection strategies based on previous international studies, we retrospectively analyzed 2646 transplantations performed for hematologic malignancies in 28 German transplant centers. Donors and recipients were high resolution typed for HLA-A, -B, -C, -DRB1, and -DQB1. The highest mortality in overall survival analysis was seen for HLA-A, -B, and DRB1 mismatches. HLA-DQB1 mismatched cases showed a trend toward higher mortality, mostly due to HLA-DQB1 antigen disparities. HLA incompatibilities at >1 locus showed additive detrimental effects. HLA mismatching had no significant effect on relapse incidence and primary graft failure. Graft source had no impact on surviv…

AdultMaleMultivariate analysisAdolescentmedicine.medical_treatmentImmunologyGraft vs Host DiseaseHematopoietic stem cell transplantationHuman leukocyte antigenHLA-C AntigensBiochemistry03 medical and health sciencesYoung Adult0302 clinical medicineimmune system diseasesTransplantation ImmunologyGermanymedicineHLA-DQ beta-ChainsHumansAlleleSurvival rateSurvival analysis030304 developmental biologyAgedRetrospective Studies0303 health sciencesHLA-A AntigensDonor selectionbusiness.industryHistocompatibility TestingHazard ratioHematopoietic Stem Cell TransplantationCell BiologyHematologyMiddle AgedSurvival AnalysisTissue Donors3. Good healthSurvival RateHLA-B AntigensHematologic NeoplasmsHistocompatibilityImmunologyMultivariate AnalysisFemalebusiness030215 immunologyHLA-DRB1 ChainsBlood
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Characterization of target antigens from anti-neutrophil cytoplasmic antibodies in autoimmune hepatitis type-I.

1997

The occurrence of anti-neutrophil cytoplasmic antibodies (ANCA) has been described in sera of patients with autoimmune hepatitis (AIH). The significance of this finding remains uncertain and the nature of the target antigen(s) has not yet been defined. We studied 32 sera from patients with AIH type-I and prepared extracts of human neutrophils to identify the target antigen(s). A 43 kDa dominant immunoreactive protein was found and identified as the cytoskeletal component actin. Initial studies to define the antigenic determinants identified three different actin domains.

AdultMaleNeutrophilsClinical BiochemistryBlotting WesternMolecular Sequence DataFluorescent Antibody TechniqueAutoimmune hepatitisBiologyBiochemistryAnalytical ChemistryAntibodies Antineutrophil CytoplasmicAutoimmune DiseasesHepatitisEpitopesAntigenimmune system diseasesmedicineTumor Cells CulturedAnimalsHumansAmino Acid SequenceAntigensCytoskeletonAnti-neutrophil cytoplasmic antibodyAgedAged 80 and overMiddle Agedmedicine.diseaseRatsBlotEpitope mappingCytoplasmImmunologybiology.proteinFemaleAntibodyElectrophoresis
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