Search results for "Immunity"

showing 10 items of 1537 documents

Autoantigens in the trabecular meshwork and glaucoma‐specific alterations in the natural autoantibody repertoire

2019

Abstract Objectives Primary open‐angle glaucoma (POAG) is a neurodegenerative disorder leading to a gradual vision loss caused by progressive damage to the optic nerve. Immunological processes are proposed to be involved in POAG pathogenesis. Altered serological autoantibody levels have been frequently reported, but complete analyses of the natural autoantibodies with respect to disease‐related alterations are scarce. Here, we provide an explorative analysis of pathways and biological processes that may involve naturally immunogenic proteins and highlight POAG‐specific alterations. Methods Mass spectrometry‐based antibody‐mediated identification of autoantigens (MS‐AMIDA) was carried out in…

lcsh:Immunologic diseases. Allergy0301 basic medicinegenetic structuresImmunologyGlaucomaPDGFRBBiologymedicine.disease_causeAutoimmunityPathogenesis03 medical and health sciences0302 clinical medicineAntigenmedicineImmunology and AllergyGeneral Nursingtrabecular meshworkAutoantibodymedicine.diseaseimmunoproteomicsautoantigeneye diseasesglaucoma030104 developmental biologymedicine.anatomical_structureImmunologybiomarkerBiomarker (medicine)Original Articlenatural autoantibodiessense organsTrabecular meshworklcsh:RC581-607030217 neurology & neurosurgeryClinical & Translational Immunology
researchProduct

Non-specific Effects of Vaccines Illustrated Through the BCG Example: From Observations to Demonstrations

2018

Epidemiological studies regarding many successful vaccines suggest that vaccination may lead to a reduction in child mortality and morbidity worldwide, on a grander scale than is attributable to protection against the specific target diseases of these vaccines. These non-specific effects (NSEs) of the Bacille Calmette-Guérin (BCG) vaccine, for instance, implicate adaptive and innate immune mechanisms, with recent evidence suggesting that trained immunity might be a key instrument at play. Collectively referring to the memory-like characteristics of innate immune cells, trained immunity stems from epigenetic reprogramming that these innate immune cells undergo following exposure to a primary…

lcsh:Immunologic diseases. Allergy0301 basic medicinemedicine.medical_treatmentImmunologyReviewImmunity HeterologousWorld Health OrganizationEpigenesis Genetictrained immunity03 medical and health sciences0302 clinical medicineImmunityHumansTuberculosisImmunology and AllergyMedicineBCG030212 general & internal medicineEpigeneticsImmunity CellularInnate immune systemepigeneticsbusiness.industryVaccinationvaccinesMycobacterium bovisImmunity InnateChild mortalityVaccination030104 developmental biologyCell metabolismCytokinenon-specific effectsImmunologyBCG Vaccineepidemiologylcsh:RC581-607businessImmunologic MemoryReprogrammingFrontiers in Immunology
researchProduct

Role of the IL-23/IL-17 Pathway in Rheumatic Diseases: An Overview

2021

Interleukin-23 (IL-23) is a pro-inflammatory cytokine composed of two subunits, IL-23A (p19) and IL-12/23B (p40), the latter shared with Interleukin-12 (IL-12). IL-23 is mainly produced by macrophages and dendritic cells, in response to exogenous or endogenous signals, and drives the differentiation and activation of T helper 17 (Th17) cells with subsequent production of IL-17A, IL-17F, IL-6, IL-22, and tumor necrosis factor α (TNF-α). Although IL-23 plays a pivotal role in the protective immune response to bacterial and fungal infections, its dysregulation has been shown to exacerbate chronic immune-mediated inflammation. Well-established experimental data support the concept that IL-23/IL…

lcsh:Immunologic diseases. Allergy0301 basic medicinemedicine.medical_treatmentImmunologyinflammatory diseasesInflammationautoimmune diseaseAutoimmunityReviewInflammatory bowel diseaseInterleukin-23Th17 CellRheumatic Disease03 medical and health sciencesPsoriatic arthritis0302 clinical medicineImmune systemIL-23PsoriasisRheumatic DiseasesInterleukin 23medicineAnimalsHumansImmunology and Allergyautoimmune diseasesMolecular Targeted TherapyIL-23/IL-17 axi030203 arthritis & rheumatologyInflammationbusiness.industryIL-23/IL-17 axisAnimalInterleukin-17medicine.diseaseinflammatory diseaseIL-17030104 developmental biologyCytokineImmunologyTh17 CellsInterleukin 17medicine.symptomlcsh:RC581-607businessHuman
researchProduct

Deciphering the Roles of Innate Lymphoid Cells in Cancer

2019

Cancer is a complex disease and the role played by innate lymphoid cells (ILCs) in cancer development has begun to be uncovered over recent years. We aim to provide an exhaustive summary of the knowledge acquired on the role of ILCs in cancer. ILCs are classified into 3 different categories, ILC1s, ILC2s, and ILC3s, each encompassing specific and unique functions. ILC1s exhibit NK cells characteristics and can exert anti-tumor functions, but surprisingly their IFNγ production is not associated with a better immune response. In response to TGF-β or IL-12, ILC1s were shown to exert pro-tumor functions and to favor tumor growth. ILC2s role in cancer immune response is dependent on cytokine con…

lcsh:Immunologic diseases. Allergy0301 basic medicinemedicine.medical_treatmentMDSCImmunologyinnate lymphoid cellsContext (language use)ReviewBiology03 medical and health sciences0302 clinical medicineImmune systemImmunityNeoplasmsEosinophil activationcytokinemedicinecancerAnimalsHumansImmunology and AllergyLymphocytesInnate lymphoid cellCancerImmunotherapymedicine.diseaseImmunity Innate030104 developmental biologyCytokineCancer researchimmunotherapylcsh:RC581-607030215 immunologyFrontiers in Immunology
researchProduct

Tolerance through Education: How Tolerogenic Dendritic Cells Shape Immunity

2017

Dendritic cells (DCs) are central players in the initiation and control of responses, regulating the balance between tolerance and immunity. Tolerogenic DCs are essential in the maintenance of central and peripheral tolerance by induction of clonal T cell deletion and T cell anergy, inhibition of memory and effector T cell responses, and generation and activation of regulatory T cells. Therefore, tolerogenic DCs are promising candidates for specific cellular therapy of allergic and autoimmune diseases and for treatment of transplant rejection. Studies performed in rodents have demonstrated the efficacy and feasibility of tolerogenic DCs for tolerance induction in various inflammatory diseas…

lcsh:Immunologic diseases. Allergy0301 basic medicinemedicine.medical_treatmentT cellImmunologyCellReviewregulatory T cellsCell therapy03 medical and health sciences0302 clinical medicineImmunitymedicineImmunology and Allergytolerancebusiness.industrytolerogenic dendritic cellsPeripheral toleranceImmunotherapymedicine.diseaseTransplant rejectionTolerance induction030104 developmental biologymedicine.anatomical_structureImmunologynanoparticlesimmunotherapylcsh:RC581-607business030215 immunologyFrontiers in Immunology
researchProduct

Hyaluronic Acid Present in the Tumor Microenvironment Can Negate the Pro-apototic Effect of a Recombinant Fragment of Human Surfactant Protein D on B…

2020

Copyright © 2020 Murugaiah, Agostinis, Varghese, Belmonte, Vieni, Alaql, Alrokayan, Khan, Kaur, Roberts, Madan, Bulla and Kishore. Human surfactant protein D (SP-D) belongs to the family of collectins that is composed of a characteristic amino-terminal collagenous region and a carboxy-terminal C-type lectin domain. Being present at the mucosal surfaces, SP-D acts as is a potent innate immune molecule and offers protection against non-self and altered self-such as pathogens, allergens, and tumour. Here, we examined the effect of a recombinant fragment of human SP-D (rfhSP-D) on a range of breast cancer lines. Breast cancer has four molecular subtypes characterised by varied expression of oes…

lcsh:Immunologic diseases. Allergy0301 basic medicinesurfactant protein DImmunologyCollectinApoptosisBreast Neoplasms03 medical and health sciencesbreast cancer0302 clinical medicineEpidermal growth factorCell Line Tumorhyaluronic acidTumor MicroenvironmentHumansImmunology and Allergyskin and connective tissue diseasesinnate immunityOriginal ResearchTumor microenvironmentChemistryimmune surveillanceIntrinsic apoptosisCell cyclePulmonary Surfactant-Associated Protein DRecombinant Proteins030104 developmental biologyApoptosisCell cultureSKBR3Cancer researchFemalelcsh:RC581-607030215 immunologyFrontiers in Immunology
researchProduct

TLR7 controls VSV replication in CD169(+) SCS macrophages and associated viral neuroinvasion

2019

Vesicular stomatitis virus (VSV) is an insect-transmitted rhabdovirus that is neurovirulent in mice. Upon peripheral VSV infection, CD169+ subcapsular sinus (SCS) macrophages capture VSV in the lymph, support viral replication, and prevent CNS neuroinvasion. To date, the precise mechanisms controlling VSV infection in SCS macrophages remain incompletely understood. Here, we show that Toll-like receptor-7 (TLR7), the main sensing receptor for VSV, is central in controlling lymph-borne VSV infection. Following VSV skin infection, TLR7−/− mice display significantly less VSV titers in the draining lymph nodes (dLN) and viral replication is attenuated in SCS macrophages. In contrast to effects o…

lcsh:Immunologic diseases. Allergy0301 basic medicinevirusesImmunologyMedizinDENDRITIC CELLSRIG-IACTIVATION03 medical and health sciences0302 clinical medicinesubcapsular sinus macrophagesSUBCAPSULAR SINUS MACROPHAGESImmunitySIMULIUM-VITTATUM DIPTERAINFECTIONImmunology and Allergyinnate immunityvirus replicationHost factorconditional knock-out miceInnate immune systemScience & TechnologyLYMPH-NODESbiologysubcutaneous infectionPattern recognition receptorpattern recognition receptorsvirus diseasesTLR7VESICULAR STOMATITIS-VIRUSbiology.organism_classificationVirologyddc:Toll-like receptor 7stomatognathic diseases030104 developmental biologyViral replicationVesicular stomatitis virusNEW-JERSEY SEROTYPEINNATE IMMUNITYvesicular stomatitis viruslcsh:RC581-607Viral loadLife Sciences & Biomedicine030215 immunology
researchProduct

Age and immunity

2006

Abstract Longitudinal studies are defining progressive alterations to the immune system associated with increased mortality in the very elderly. Many of these changes are exacerbated by or even caused by chronic T cell stimulation by persistent antigen, particularly from Cytomegalovirus. The composition of T cell subsets, their functional integrity and representation in the repertoire are all markedly influenced by age and by CMV. How these findings relate to epidemiological, functional, genetic, genomic and proteomic studies of human T cell immunosenescence was the subject of intense debate at an international conference held just before Christmas 2005 in the Black Forest.

lcsh:Immunologic diseases. AllergyAgingbiologyT cellRepertoireImmunologyShort ReportCongenital cytomegalovirus infectionImmunosenescencelcsh:Geriatricsmedicine.diseaseaged aging apoptosis article CD4+ CD25+ T lymphocytelcsh:RC952-954.6Immune systemmedicine.anatomical_structureAntigenImmunityImmunologybiology.proteinmedicineAntibodylcsh:RC581-607
researchProduct

Role of persistent CMV infection in configuring T cell immunity in the elderly

2007

Abstract Ageing is associated with declines in many physiological parameters, including multiple immune system functions. The rate of acceleration of the frequency of death due to cardiovascular disease or cancer seems to increase with age from middle age up to around 80 years, plateauing thereafter. Mortality due to infectious disease, however, does not plateau, but continues to accelerate indefinitely. The elderly commonly possess oligoclonal expansions of T cells, especially of CD8 cells, which, surprisingly, are often associated with cytomegalovirus (CMV) seropositivity. This in turn is associated with many of the same phenotypic and functional alterations to T cell immunity that have b…

lcsh:Immunologic diseases. AllergyAgingbiologybusiness.industryImmunologyMembrane raftReviewDiseaseImmunosenescencelcsh:GeriatricsBioinformaticsVaccinationlcsh:RC952-954.6aged aging antigen expression apoptosis cancer incidence CD4+ T lymphocyte CD8+ T lymphocyte cellular immunityAgeingImmune systemInfectious disease (medical specialty)ImmunityImmunologybiology.proteinMedicineAntibodybusinesslcsh:RC581-607Immunity & Ageing
researchProduct

INNATE IMMUNITY AND INFLAMMATION IN AGEING: A KEY FOR UNDERSTANDING AGE-RELATED DISEASES.

2005

Abstract The process of maintaining life for the individual is a constant struggle to preserve his/her integrity. This can come at a price when immunity is involved, namely systemic inflammation. Inflammation is not per se a negative phenomenon: it is the response of the immune system to the invasion of viruses or bacteria and other pathogens. During evolution the human organism was set to live 40 or 50 years; today, however, the immune system must remain active for much a longer time. This very long activity leads to a chronic inflammation that slowly but inexorably damages one or several organs: this is a typical phenomenon linked to ageing and it is considered the major risk factor for a…

lcsh:Immunologic diseases. AllergyAgingmedia_common.quotation_subjectLongevityImmunologyInflammationReviewDiseaselcsh:GeriatricsSystemic inflammationImmune systemImmunitymedicinePermissiveCytokinemedia_commonInflammationInnate immune systembusiness.industryLongevityInnate Immunitylcsh:RC952-954.6Immunologymedicine.symptomlcsh:RC581-607businessAge-related diseases
researchProduct