Search results for "Immunotherapy."

showing 10 items of 819 documents

Regulatory T cells--the renaissance of the suppressor T cells.

2007

Immune reactions are stringently regulated and balanced by complex interactions of stimulating and suppressing mechanisms. Dysfunctions of this sophisticated immune regulatory network can lead to a variety of diseases such as autoimmunity, allergy, cancer, and pregnancy disorders. The rediscovery of suppressor T cells a decade ago--now designated as T regulatory cells--set off a huge avalanche of research activities leading to a multitude of preclinical and clinical studies. Herein, we give a comprehensive review about this research on T regulatory cells and the relevance of this suppressive T cell population for the development of innovative immune therapeutic strategies.

Encephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentT cellPopulationAutoimmunitymedicine.disease_causeInfectionsT-Lymphocytes RegulatoryAutoimmunitylaw.inventionMiceImmune systemlawPregnancyT-Lymphocyte SubsetsTransplantation ImmunologyNeoplasmsmedicineSuppressor Factors ImmunologicAnimalsHumanseducationeducation.field_of_studybusiness.industryModels ImmunologicalGeneral MedicineT lymphocyteImmunotherapyInflammatory Bowel DiseasesTransplantationDisease Models Animalmedicine.anatomical_structureDiabetes Mellitus Type 1ImmunologySuppressorFemaleImmunotherapybusinessAnnals of medicine
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The first European interdisciplinary ewing sarcoma research summit.

2012

This is an open-access article distributed under the terms of the Creative Commons Attribution Non Commercial License.-- et al.

EpigenomicsCancer ResearchAlternative medicineMedizinComputingMilieux_LEGALASPECTSOFCOMPUTINGReview ArticleBioinformatics[SDV.BBM.BM] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologydrug screen0302 clinical medicineDrug screenCancer genomicssignallingSarcomagenesis0303 health sciencessarcomagenesisSummitgeography.geographical_feature_categoryOpinion leadershipGenomicsLaboratory resultslcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensPrognosisanimal models3. Good healthAnimal modelsMetastatic Ewing SarcomaOncology030220 oncology & carcinogenesis[SDV.BBM.GTP] Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]EpigeneticsSarcomaImmunotherapyPrioritizationmedicine.medical_specialty[SDV.CAN]Life Sciences [q-bio]/Cancerlcsh:RC254-28203 medical and health sciences[SDV.CAN] Life Sciences [q-bio]/Cancer[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]medicinegenomics030304 developmental biologyMedical educationgeographyepigeneticsbusiness.industrybiomarkers[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologymedicine.diseaseClinical trialprognosisbusinessBiomarkersEwing sarcomaFrontiers in oncology
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A neoepitope generated by an FLT3 internal tandem duplication (FLT3-ITD) is recognized by leukemia-reactive autologous CD8+ T cells.

2006

Abstract The FLT3 receptor tyrosine kinase is expressed in more than 90% of acute myelogeneous leukemias (AMLs), up to 30% of which carry an internal tandem duplication (ITD) within the FLT3 gene. Although varying duplication sites exist, most FLT3-ITDs affect a single protein domain. We analyzed the FLT3-ITD of an AML patient for encoding HLA class I–restricted immunogenic peptides. One of the tested peptides (YVDFREYEYY) induced in vitro autologous T-cell responses restricted by HLA-A*0101 that were also detectable ex vivo. These peptide-reactive T cells recognized targets transfected with the patient's FLT3-ITD, but not wild-type FLT3, and recognized the patient's AML cells. Our results …

FLT3 Internal Tandem DuplicationMyeloidmedicine.medical_treatmentImmunologyAntigen presentationMolecular Sequence DataHuman leukocyte antigenBiologyCD8-Positive T-LymphocytesIn Vitro TechniquesTransfectionBiochemistryCell LineEpitopesfluids and secretionshemic and lymphatic diseasesCell Line TumorGene DuplicationGene duplicationmedicineCytotoxic T cellHumansAmino Acid SequenceRNA MessengerHLA-A1 AntigenAntigen PresentationHLA-A Antigenshemic and immune systemsCell BiologyHematologyImmunotherapymedicine.diseaseMolecular biologyLeukemiaLeukemia Myeloid Acutemedicine.anatomical_structurefms-Like Tyrosine Kinase 3embryonic structurespsychological phenomena and processesBlood
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Immunotherapy of Malignant Melanomas

1989

A large number of antigens on human melanoma cells have been identified by means of monoclonal antibody technology [18, 21]. The availability of monoclonal antibodies has rapidly led to clinical investigations. Experience with monoclonal antibodies in the treatment of patients with melanoma is limited [4, 9, 10, 12, 13, 17, 19] because only small groups of patients have been treated so far. According to these studies, treatment with ganglioside antibodies appears most promising.

Gangliosidebiologymedicine.drug_classbusiness.industryMelanomamedicine.medical_treatmentMelanoma antigenImmunotherapyMonoclonal antibodymedicine.diseaseAntigenmedicinebiology.proteinCancer researchHuman melanomaAntibodybusiness
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Preemptive CD8 T-Cell Immunotherapy of Acute Cytomegalovirus Infection Prevents Lethal Disease, Limits the Burden of Latent Viral Genomes, and Reduce…

1998

ABSTRACT In the immunocompetent host, primary cytomegalovirus (CMV) infection is resolved by the immune response without causing overt disease. The viral genome, however, is not cleared but is maintained in a latent state that entails a risk of virus recurrence and consequent organ disease. By using murine CMV as a model, we have shown previously that multiple organs harbor latent CMV and that reactivation occurs with an incidence that is determined by the viral DNA load in the respective organ (M. J. Reddehase, M. Balthesen, M. Rapp, S. Jonjic, I. Pavic, and U. H. Koszinowski. J. Exp. Med. 179:185–193, 1994). This predicts that a therapeutic intervention capable of limiting the load of lat…

Genes Viralmedicine.medical_treatmentImmunologyViral Pathogenesis and ImmunityGenome ViralCD8-Positive T-LymphocytesBiologymedicine.disease_causeMicrobiologyVirusMiceImmune systemRecurrenceRisk FactorsVirologyVirus latencymedicineAnimalsHumansCytotoxic T cellLungCells CulturedBone Marrow TransplantationMice Inbred BALB CCytomegalovirusImmunotherapyViral Loadmedicine.diseaseVirologyVirus LatencyDisease Models AnimalInsect ScienceAcute DiseaseCytomegalovirus InfectionsDNA ViralImmunologyFemaleImmunotherapyViral loadCD8Journal of Virology
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Immunotherapy in gastrointestinal cancer: Recent results, current studies and future perspectives

2016

The new therapeutic approach of using immune checkpoint inhibitors as anticancer agents is a landmark innovation. Early studies suggest that immune checkpoint inhibition might also be effective in patients with gastrointestinal cancer. To improve the efficacy of immunotherapy, different strategies are currently under evaluation. This review summarises the discussion during the European Organisation for Research and Treatment of Cancer Gastrointestinal Tract Cancer Translational Research Meeting in Mainz in November 2014 and provides an update on the most recent results of immune therapy in gastrointestinal cancers. Knowledge of potential relationships between tumour cells and their microenv…

Genetic Markers0301 basic medicineCancer Researchmedicine.medical_treatmentAntineoplastic AgentsTranslational researchContext (language use)Antibodies Monoclonal Humanized03 medical and health sciencesGastrointestinal cancer0302 clinical medicineImmune systemBiomarkers TumormedicineHumansMolecular Targeted TherapyGastrointestinal cancerGastrointestinal NeoplasmsOncolytic Virotherapybusiness.industryCancerCell Cycle CheckpointsImmunotherapymedicine.diseaseImmune checkpointOncolytic virusTreatment Outcome030104 developmental biologyOncology030220 oncology & carcinogenesisImmunologyCancer researchImmunotherapyEpidemiologic MethodsbusinessCheckpoint inhibitorsForecastingEuropean Journal of Cancer
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Gene transfer approaches for the treatment of inflammatory bowel disease.

2003

The pathogenesis of Crohn's disease and ulcerative colitis, the two major forms of inflammatory bowel disease, involves a complex interplay between certain genetic, environmental and immunological factors. Considerable research progress in the last decade defined key inflammatory pathways in the inflamed gut and identified new potential therapeutic targets. Since the current medical treatment with corticosteroids and anti-inflammatory drugs is often associated with undesired side effects and cannot completely cure IBD, these current advances in our understanding of intestinal pathology may now allow the development of new biologic treatment strategies including gene therapy. In this review,…

Genetic enhancementGenetic VectorsGene ExpressionGene transferDiseaseInflammatory bowel diseaseAdenoviridaePathogenesisCrohn DiseaseIntestinal inflammationGeneticsMedicineAnimalsHumansMolecular BiologyMedical treatmentbusiness.industryBacterial InfectionsGenetic Therapymedicine.diseaseInflammatory Bowel DiseasesUlcerative colitisIntestinesDisease Models AnimalImmunologyMolecular MedicineCytokinesColitis UlcerativeImmunotherapybusinessStem Cell TransplantationGene therapy
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A Potent Tumor-Reactive p53-Specific Single-Chain TCR without On- or Off-Target Autoimmunity In Vivo

2018

Genetic engineering of T cells with a T cell receptor (TCR) targeting tumor antigen is a promising strategy for cancer immunotherapy. Inefficient expression of the introduced TCR due to TCR mispairing may limit the efficacy and adversely affect the safety of TCR gene therapy. Here, we evaluated the safety and therapeutic efficiency of an optimized single-chain TCR (scTCR) specific for an HLA-A2.1-restricted (non-mutated) p53(264–272) peptide in adoptive T cell transfer (ACT) models using our unique transgenic mice expressing human p53 and HLA-A2.1 that closely mimic the human setting. Specifically, we showed that adoptive transfer of optimized scTCR-redirected T cells does not induce on-tar…

Genetically modified mouseAdoptive cell transfermedicine.medical_treatmentGenetic enhancementT-LymphocytesReceptors Antigen T-CellAutoimmunityBiology03 medical and health sciencesMice0302 clinical medicineAntigenCancer immunotherapyDrug DiscoveryHLA-A2 AntigenGeneticsmedicineAnimalsHumansMolecular Biology030304 developmental biologyPharmacology0303 health sciencesT-cell receptorImmunotherapyGenetic TherapyTumor antigen030220 oncology & carcinogenesisCancer researchMolecular MedicineOriginal ArticleTumor Suppressor Protein p53
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Strategies for tumor elimination by cytotoxic T lymphocytes.

1998

Despite differences in their tissue of origin, many tumors share high level expression of certain tumor-associated proteins. Our laboratory has focused on the possibility of utilizing antigenic components of these proteins as a focus for T-cell immunotherapy of cancer. The advantage of targeting such commonly expressed proteins is the fact that such therapy could be of value in eliminating many different types of tumors. A potential barrier in the identification of T-cell epitopes derived from these proteins and presented by tumor cells is the fact that these proteins are also expressed at low levels in some normal tissues, and therefore, self-tolerance may eliminate T cells that are capabl…

Genetically modified mousePolymers and Plasticsmedicine.medical_treatmentTransgeneHemagglutinin (influenza)ImmunotherapyBiologyEpitopeCell biologyMiceImmune systemAntigenAntigens NeoplasmNeoplasmsbiology.proteinmedicineImmune ToleranceCytotoxic T cellAnimalsHumansTumor Suppressor Protein p53General Environmental ScienceT-Lymphocytes Cytotoxic
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In the literature: October 2016

2016

A consortium on clinical and molecular stratification on oesophageal adenocarcinoma established in Britain has recently published in Nature Genetics , a whole-genomic sequencing analysis of more than 100 samples.1 Interestingly, they describe three distinct molecular subtypes with potential treatment relevance. This observation has also been verified in an independent validation cohort. Those three types are: (1) the ones showing homologous recombination and chromosome segregation pathways defects with enrichment of a BRCA signature. These tumours would be sensitive to DNA damaging agents, including neutron and photon irradiation with the addition of PARP inhibitors, (2) a group with high m…

GeneticsCancer ResearchChemotherapyMutationbiologymedicine.medical_treatmentliteratureImmunotherapyNewsmedicine.disease_causeGenomeOncologyGene duplicationmedicineCancer researchbiology.protein1506AntibodyHomologous recombinationCD8ESMO Open
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