Search results for "Inflammation"

showing 10 items of 2662 documents

Benefits of immune protection versus immunopathology costs: a synthesis from cytokine KO models.

2017

5 pages; International audience; The inflammatory response can produce damage to host tissues and in several infectious diseases the most severe symptoms are due to immunopathology rather than a direct effect of pathogen multiplication. One hypothesis for the persistence of inflammatory damage posits that the benefits of protection towards infection outweigh the costs. We used data on knocked-out (KO) cytokine models [and the corresponding wild-type (WT) controls] to test this hypothesis. We computed differences in pathogen load and host survival between WT and KO and divided them by the WT values. Using this ratio provides an internal control for variation in pathogen species, host strain,…

0106 biological sciences0301 basic medicineMicrobiology (medical)medicine.medical_treatmentVirulenceInflammationImmunopathologyBiologyCommunicable Diseases010603 evolutionary biology01 natural sciencesMicrobiologyPersistence (computer science)Gene Knockout Techniques03 medical and health sciencesImmunopathologyGeneticsmedicine[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisAnimalsHumans[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyMolecular BiologyPathogenCytokineEcology Evolution Behavior and SystematicsInflammationVirulenceHost (biology)Immune protectionHost survivalComputational BiologyPathogen loadDisease Models Animal030104 developmental biologyInfectious DiseasesCytokineHost-Pathogen InteractionsImmunologyCytokinesmedicine.symptom
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Early Plasmodium-induced inflammation does not accelerate aging in mice

2019

10 pages; International audience; Aging is associated with a decline of performance leading to reduced reproductive output and survival. While the antagonistic pleiotropy theory of aging has attracted considerable attention, the molecular/physiological functions underlying the early-life benefits/late-life costs paradigm remain elusive. We tested the hypothesis that while early activation of the inflammatory response confers benefits in terms of protection against infection, it also incurs costs in terms of reduced reproductive output at old age and shortened longevity. We infected mice with the malaria parasite Plasmodium yoelii and increased the inflammatory response using an anti-IL-10 r…

0106 biological sciences0301 basic medicineSenescencesenescencemedia_common.quotation_subjectlcsh:EvolutionInflammationBiologysourisantagonistic pleiotropy010603 evolutionary biology01 natural sciencessurvival03 medical and health sciencesPlasmodium malariaePleiotropyBiologie animaleGeneticsmedicinelcsh:QH359-425Survival rateEcology Evolution Behavior and Systematicsmedia_commonAnimal biology[SDV.BA]Life Sciences [q-bio]/Animal biologyLongevityAntagonistic pleiotropy hypothesisPlasmodium yoeliimedicine.diseasebiology.organism_classificationinfection3. Good healthsurvie030104 developmental biologyinflammationImmunology[SDV.IMM]Life Sciences [q-bio]/ImmunologyAntagonistic pleiotropyantagonistic pleiotropy;inflammation;Plasmodium yoelii;senescence;survivalmedicine.symptom[SDE.BE]Environmental Sciences/Biodiversity and EcologyGeneral Agricultural and Biological SciencesMalariaPlasmodium yoelii
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Male health status, signalled by courtship display, reveals ejaculate quality and hatching success in a lekking species.

2010

8 pages; International audience; Summary 1. The information content of secondary sexual traits and the benefits gathered by choosy females are at the heart of sexual selection theory. Indicator models of sexual selection assume that secondary sexual traits reflect the phenotypic/genetic quality of their bearers and that females gather benefits from choosing these high-quality males. 2. Here, we tested the idea that courtship display reflects the health status in a bird species with a lek-based mating system, the houbara bustard (Chlamydotis undulata undulata). A group of males was treated with the lipopolysaccharide (LPS) of the cell wall of the bacterium Escherichia coli during the seasona…

0106 biological sciencesLipopolysaccharidesMale[ SDV.BDLR.RS ] Life Sciences [q-bio]/Reproductive Biology/Sexual reproductionmedia_common.quotation_subjectHealth StatusZoologyEmbryonic Development[SDV.BID]Life Sciences [q-bio]/BiodiversityBiology010603 evolutionary biology01 natural sciences[SDV.BDLR.RS]Life Sciences [q-bio]/Reproductive Biology/Sexual reproductionCourtshipBirds03 medical and health sciencesSemen qualityLek matingembryo mortalityEscherichia colisexual selectionAnimalsEcology Evolution Behavior and Systematics030304 developmental biologymedia_common[ SDV.BID ] Life Sciences [q-bio]/Biodiversity[ SDE.BE ] Environmental Sciences/Biodiversity and Ecology0303 health sciencesSex CharacteristicsCourtship displayReproductive successEcologyReproductionCourtshipMating systemSpermSpermatozoainflammationSexual selectionFertilizationAnimal Science and ZoologyFemaleGenetic Fitness[SDE.BE]Environmental Sciences/Biodiversity and EcologyThe Journal of animal ecology
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Correlational selection on pro- and anti-inflammatory effectors.

2012

9 pages; International audience; Parasites impose a permanent threat for hosts. As a consequence, immune defenses are important for host fitness. However, the immune response can also produce self-damage and impair host fitness if not properly regulated. Effectors that up- and downregulate the immune response should, therefore, evolve in concert, and be under the action of correlational selection. To address this issue, we assessed the shape of the selection operating on pro- and anti-inflammatory effectors following an inflammatory challenge in laboratory mice. We found that selection acts on the combination of these two traits as individuals that produced large amount of pro-inflammatory …

0106 biological sciencesLipopolysaccharidesMale[ SDV.IMM.IA ] Life Sciences [q-bio]/Immunology/Adaptive immunology01 natural sciencesMiceImmunopathologyDarwinian medicine[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisimmunopathologyGenetics0303 health sciencesEffectorFlow CytometryInterleukin-10Interleukin 10[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologyIL-10CytokinesRegression AnalysisFemalemedicine.symptomGeneral Agricultural and Biological Sciences[ SDV.MP.PAR ] Life Sciences [q-bio]/Microbiology and Parasitology/Parasitologymedicine.drug_classInflammationBiology010603 evolutionary biologysurvivalAnti-inflammatory03 medical and health sciencesImmune systemImmunityGeneticsmedicineEscherichia coliAnimals[SDV.MP.PAR]Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologySelection GeneticEcology Evolution Behavior and SystematicsSelection (genetic algorithm)030304 developmental biologyIL-6[ SDE.BE ] Environmental Sciences/Biodiversity and Ecologyevolutionary immunologyInterleukin-6inflammationImmunology[SDE.BE]Environmental Sciences/Biodiversity and Ecology[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/SymbiosisEvolution; international journal of organic evolution
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Dynamics of PHA-induced immune response and plasma carotenoids in birds: should we have a closer look?

2009

SUMMARY Allocation trade-offs of limited resources are thought to ensure the honesty of sexual signals and are often studied using controlled immune challenges. One such trade-off between immunity and ornaments is that involving carotenoids. Phytohemagglutinin (PHA)-induced immune response is a widely used immune challenge, yet more details on the underlying physiological mechanisms and potential costs are needed. We investigated the temporal dynamics of PHA-induced immune response and associated changes in blood carotenoids, body mass and a carotenoid-based coloured signal. We found variation in individual response patterns to PHA after peak swelling was reached, with birds showing either …

0106 biological sciencesMalePhysiology[ SDV.IMM.IA ] Life Sciences [q-bio]/Immunology/Adaptive immunology01 natural sciencesSongbirds[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisBody Sizebill colourCarotenoidchemistry.chemical_classification0303 health sciencesimmunocompetenceBeakOrnamentsblackbird[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologymedicine.symptomImmunocompetencemedicine.medical_specialtycell-mediated immune responseInflammationchemical and pharmacologic phenomenaAquatic ScienceBiology010603 evolutionary biology03 medical and health sciencesImmune systemImmunityInternal medicinemedicineAnimalsPhytohemagglutininsMolecular BiologyEcology Evolution Behavior and Systematics030304 developmental biologyInflammationimmunocompetence.[ SDE.BE ] Environmental Sciences/Biodiversity and EcologyCell-mediated immune responseCarotenoidsEndocrinologychemistryInsect ScienceImmunologycarotenoid-based signalsAnimal Science and Zoology[SDE.BE]Environmental Sciences/Biodiversity and EcologyMitogensLimited resources[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/SymbiosisThe Journal of experimental biology
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2,3-Dihydrobenzofuran privileged structures as new bioinspired lead compounds for the design of mPGES-1 inhibitors

2016

International audience; 2,3-Dihydrobenzofurans are proposed as privileged structures and used as chemical platform to design small compound libraries. By combining molecular docking calculations and experimental verification of biochemical interference, we selected some potential inhibitors of microsomal prostaglandin E2 synthase (mPGES)-1. Starting from low affinity natural product 1, by our combined approach we identified the compounds 19 and 20 with biological activity in the low micromolar range. Our data suggest that the 2,3-dihydrobenzofuran derivatives might be suitable bioinspired lead compounds for development of new generation mPGES-1 inhibitors with increased affinity.

0301 basic medicine300323-Dihydrobenzofuran privileged structure; Cancer; Inflammation; Molecular docking; mPGES-1 inhibitors; Biochemistry; Clinical Biochemistry; Molecular Biology; Molecular Medicine; Organic Chemistry; Drug Discovery3003 Pharmaceutical Science; 3003Amino Acid MotifsClinical BiochemistryGene ExpressionPharmaceutical Science01 natural sciencesClinical biochemistryBiochemistry[ CHIM ] Chemical SciencesProtein Structure Secondary[ SDV.CAN ] Life Sciences [q-bio]/Cancerchemistry.chemical_compoundLow affinityDrug DiscoveryEnzyme Inhibitors23-Dihydrobenzofuran privileged structure; Molecular docking; mPGES-1 inhibitors; Cancer; InflammationProstaglandin-E SynthasesCancerAnti-Inflammatory Agents Non-SteroidalBiological activityProto-Oncogene Proteins c-metIntramolecular OxidoreductasesMolecular Docking SimulationMolecular dockingMolecular Medicinelipids (amino acids peptides and proteins)Cell SurvivalStereochemistryMolecular Sequence Data2Antineoplastic Agents[SDV.CAN]Life Sciences [q-bio]/Cancer3-Dihydrobenzofuran privileged structureInhibitory Concentration 50Structure-Activity Relationship03 medical and health sciencesCell Line TumorMicrosomesHumans[CHIM]Chemical SciencesMolecular BiologyBenzofuransInflammationNatural product010405 organic chemistryDrug Discovery3003 Pharmaceutical ScienceOrganic ChemistryEpithelial CellsmPGES-1 inhibitorsCombinatorial chemistryCombined approach0104 chemical sciences030104 developmental biologychemistryDrug DesignDrug Screening Assays Antitumor
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Indicaxanthin from Opuntia Ficus Indica (L. Mill) impairs melanoma cell proliferation, invasiveness, and tumor progression.

2018

Abstract Background: A strong, reciprocal crosstalk between inflammation and melanoma has rigorously been demonstrated in recent years, showing how crucial is a pro-inflammatory microenvironment to drive therapy resistance and metastasis. Purpose: We investigated on the effects of Indicaxanthin, a novel, anti-inflammatory and bioavailable phytochemical from Opuntia Ficus Indica fruits, against human melanoma both in vitro and in vivo. Study Design and Methods: The effects of indicaxanthin were evaluated against the proliferation of A375 human melanoma cell line and in a mice model of cutaneous melanoma. Cell proliferation was assessed by MTT assay, apoptosis by Annexin V-Fluorescein Isothio…

0301 basic medicine3003MaleSkin NeoplasmsPyridinesPyridinePhytochemicalsMelanoma ExperimentalPharmaceutical ScienceIndicaxanthinApoptosisBcl-2 B cell lymphoma gene-2 (Bcl-2)chemistry.chemical_compoundMice0302 clinical medicineOpuntia Ficus Indica (L.Mill)Settore BIO/10 - BiochimicaDrug DiscoveryCXCL1 chemokine (C-X-C motif) ligand 1MelanomaNF-κB nuclear factor kappa BMTT 3-[45-dimethyltiazol-2-yl]-25-diphenyl tetrazolium bromideMelanomaNF-kappa BOpuntiaComplementary and Alternative Medicine2708 DermatologyBetaxanthinsCXCL1030220 oncology & carcinogenesisMolecular MedicinePhC phytochemicalGrowth inhibitionIndicaxanthinHumanBiologyPhytochemicalNHEM normal human epidermal melanocyte03 medical and health sciencesc-FLIP FLICE-inhibitory proteinIn vivoCell Line TumormedicineAnimalsHumansNeoplasm InvasivenessSkin NeoplasmCell ProliferationNeoplasm InvasiveneInflammationPharmacologyCell growthAnimalDrug Discovery3003 Pharmaceutical ScienceApoptosimedicine.diseaseMice Inbred C57BL030104 developmental biologyComplementary and alternative medicinechemistryTumor progressionList of Abbrevations: AxV-FITC annexin V-fluorescein isothiocyanateBetaxanthinFruitCutaneous melanomaCancer researchPI propidium iodide PIPhytomedicine : international journal of phytotherapy and phytopharmacology
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Mechanics Insights of Alpha-Lipoic Acid against Cardiovascular Diseases during COVID-19 Infection

2021

Coronavirus disease 2019 (COVID-19) was first reported in Wuhan, China, in late December 2019. Since then, COVID-19 has spread rapidly worldwide and was declared a global pandemic on 20 March 2020. Cardiovascular complications are rapidly emerging as a major peril in COVID-19 in addition to respiratory disease. The mechanisms underlying the excessive effect of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection on patients with cardiovascular comorbidities remain only partly understood. SARS-CoV-2 infection is caused by binding of the viral surface spike (S) protein to the human angiotensin-converting enzyme 2 (ACE2), followed by the activation of the S protein by transme…

0301 basic medicineARDSEndotheliumQH301-705.5InflammationReviewmedicine.disease_causeCatalysisAntioxidantsProinflammatory cytokineInorganic Chemistry03 medical and health sciences0302 clinical medicinecardiovascular diseasemedicineoxidative stressAnimalsHumansPhysical and Theoretical ChemistryEndothelial dysfunctionBiology (General)Molecular BiologyQD1-999SpectroscopyThioctic Acidbusiness.industrySARS-CoV-2alpha-lipoic acidOrganic ChemistryRespiratory diseaseCOVID-19Endothelial CellsGeneral Medicinemedicine.diseaseComputer Science ApplicationsCOVID-19 Drug TreatmentChemistry030104 developmental biologymedicine.anatomical_structureinflammationCardiovascular Diseases030220 oncology & carcinogenesisImmunologyAngiotensin-Converting Enzyme 2medicine.symptombusinessCytokine stormCytokine Release SyndromeOxidative stressInternational Journal of Molecular Sciences
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COVID-19 severity associates with pulmonary redistribution of CD1c+ DCs and inflammatory transitional and nonclassical monocytes

2020

SARS-CoV-2 is responsible for the development of coronavirus disease 2019 (COVID-19) in infected individuals, who can either exhibit mild symptoms or progress toward a life-threatening acute respiratory distress syndrome (ARDS). Exacerbated inflammation and dysregulated immune responses involving T and myeloid cells occur in COVID-19 patients with severe clinical progression. However, the differential contribution of specific subsets of dendritic cells and monocytes to ARDS is still poorly understood. In addition, the role of CD8+ T cells present in the lung of COVID-19 patients and relevant for viral control has not been characterized. Here, we have studied the frequencies and activation p…

0301 basic medicineARDSLungMyeloidbusiness.industryT cellInflammationGeneral Medicinemedicine.diseasePathogenesis03 medical and health sciences030104 developmental biology0302 clinical medicinemedicine.anatomical_structureImmune system030220 oncology & carcinogenesisImmunologymedicinemedicine.symptombusinessCD8Journal of Clinical Investigation
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Assessment of tumor-infiltrating TCRV γ 9V δ 2 γδ lymphocyte abundance by deconvolution of human cancers microarrays

2017

Most human blood γδ cells are cytolytic TCRVγ9Vδ2+lymphocytes with antitumor activity. They are currently investigated in several clinical trials of cancer immunotherapy but so far, their tumor infiltration has not been systematically explored across human cancers. Novel algorithms allowing the deconvolution of bulk tumor transcriptomes to find the relative proportions of infiltrating leucocytes, such as CIBERSORT, should be appropriate for this aim but in practice they fail to accurately recognize γδ T lymphocytes. Here, by implementing machine learning from microarray data, we first improved the computational identification of blood-derived TCRVγ9Vδ2+γδ lymphocytes and then appl…

0301 basic medicineAcute promyelocytic leukemia[SDV.MHEP.HEM] Life Sciences [q-bio]/Human health and pathology/Hematologylcsh:Immunologic diseases. AllergyArtificial intelligenceMicroarrayLymphocytemedicine.medical_treatmentImmunologyInflammationchemical and pharmacologic phenomenagamma delta lymphocyteBiologydeconvolutionlcsh:RC254-28203 medical and health sciences0302 clinical medicineCancer immunotherapymedicineImmunology and AllergycancerOriginal ResearchTumor-infiltrating lymphocytesAntigen processingMyeloid leukemiahemic and immune systems[SDV.MHEP.HEM]Life Sciences [q-bio]/Human health and pathology/Hematologydata miningmedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens3. Good health030104 developmental biologymedicine.anatomical_structuremachine learningOncology030220 oncology & carcinogenesisImmunologymedicine.symptomlcsh:RC581-607microarraytranscriptome
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