Search results for "Inflammation."

showing 10 items of 2627 documents

The Anti-amyloid Compound DO1 Decreases Plaque Pathology and Neuroinflammation-Related Expression Changes in 5xFAD Transgenic Mice

2018

Self-propagating amyloid-β (Aβ) aggregates or seeds possibly drive pathogenesis of Alzheimer's disease (AD). Small molecules targeting such structures might act therapeutically in vivo. Here, a fluorescence polarization assay was established that enables the detection of compound effects on both seeded and spontaneous Aβ42 aggregation. In a focused screen of anti-amyloid compounds, we identified Disperse Orange 1 (DO1) ([4-((4-nitrophenyl)diazenyl)-N-phenylaniline]), a small molecule that potently delays both seeded and non-seeded Aβ42 polymerization at substoichiometric concentrations. Mechanistic studies revealed that DO1 disrupts preformed fibrillar assemblies of synthetic Aβ42 peptides …

MaleGenetically modified mouse1303 BiochemistryAmyloid10017 Institute of AnatomyClinical BiochemistryMice TransgenicPlaque Amyloid610 Medicine & healthBiologyProtein aggregation1308 Clinical Biochemistry01 natural sciencesBiochemistryPolymerizationPathogenesisMiceProtein AggregatesStructure-Activity RelationshipAlzheimer DiseaseGene expressionDrug Discovery1312 Molecular BiologyAnimalsColoring AgentsMolecular BiologyNeuroinflammationInflammationPharmacologyAmyloid beta-PeptidesDose-Response Relationship DrugMolecular Structure010405 organic chemistry3002 Drug DiscoveryBrainSmall moleculeMolecular medicine0104 chemical sciencesCell biologyMice Inbred C57BL3004 Pharmacology10036 Medical Clinic1313 Molecular Medicine570 Life sciences; biologyMolecular MedicineFemaleAzo Compounds
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Neuroprotective effect of Fn14 deficiency is associated with induction of the granulocyte-colony stimulating factor (G-CSF) pathway in experimental s…

2010

Using a transgenic mouse model of ischemic stroke we checked for a possible interaction of antiphospholipid antibodies (aPL) which often cause thromboses as well as central nervous system (CNS) involvement under non-thrombotic conditions and the TWEAK/Fn14 pathway known to be adversely involved in inflammatory and ischemic brain disease. After 7 days, infarct volumes were reduced in Fn14 deficient mice and were further decreased by aPL treatment. This was associated with strongest increase of the endogenous neuroprotective G-CSF/G-CSF receptor system. This unexpected beneficial action of aPL is an example for a non-thrombogenic action and the double-edged nature of aPL.

MaleGenetically modified mouseImmunologyMice TransgenicBiologyNeuroprotectionReceptors Tumor Necrosis FactorBrain IschemiaMiceRandom AllocationTissue factorimmune system diseasesAntiphospholipid syndromeGranulocyte Colony-Stimulating FactormedicineAnimalsHumansImmunology and AllergyneoplasmsStrokeLupus anticoagulantmedicine.diseaseMice Inbred C57BLDisease Models AnimalNeurologyTWEAK ReceptorReceptors Granulocyte Colony-Stimulating FactorImmunologyAntibodies AntiphospholipidTumor necrosis factor alphaNeurology (clinical)Inflammation MediatorsGranulocyte colony-stimulating factor receptorSignal TransductionJournal of Neuroimmunology
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Impact of pathogen burden in patients with coronary artery disease in relation to systemic inflammation and variation in genes encoding cytokines.

2003

The number of infectious pathogens to which an individual has been exposed (pathogen burden) has been linked to the development and the prognosis of coronary artery disease (CAD). The interaction among infection, genetic host susceptibility, and CAD remains unclear. This study was aimed at evaluating the modulation of the association between CAD and pathogen burden, by serum levels of inflammatory markers and polymorphisms of the interleukin (IL)-6 and tumor necrosis factor (TNF)-alpha genes. Immmunoglobulin (Ig) G or IgA antibodies to 8 pathogens were determined in 991 patients with CAD and 333 control subjects. Serum levels of high-sensitivity C-reactive protein, fibrinogen, IL-6, and TNF…

MaleGenotypemedicine.medical_treatmentInflammationCoronary Artery DiseaseSystemic inflammationInfectionsRisk FactorsmedicinePrevalenceHumansGenetic Predisposition to DiseasePathogenAgedInflammationChlamydiaPolymorphism Geneticbiologybusiness.industryInterleukin-6Tumor Necrosis Factor-alphaCholesterol HDLInterleukinFibrinogenEnvironmental ExposureHelicobacter pyloriMiddle Agedmedicine.diseasebiology.organism_classificationPrognosisImmunoglobulin ACytokineC-Reactive ProteinCase-Control StudiesImmunoglobulin GImmunologyCytokinesTumor necrosis factor alphaFemalemedicine.symptomCardiology and Cardiovascular MedicinebusinessBiomarkersThe American journal of cardiology
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Gender-Related Immune-Inflammatory Factors, Age-Related Diseases, and Longevity

2010

This review discusses the role of estrogens as pro- or antiinflammatory players in immune-inflammatory responses. In particular, their role in Alzheimer’s disease (AD), an example of immune-inflammatory disease, is discussed briefly. AD is a progressive neurodegenerative disease, which in Western societies accounts for the majority of cases of clinical senile dementia. However, sexual dimorphism of diseases may also depend on factors independent of sex hormones (i.e., a gender effect), as demonstrated by our data on differential longevity in females and males. In fact, differences in mortality between men and women are not only a question of sex that refers to biological differences, but ra…

MaleGerontologyAgingmedia_common.quotation_subjectLongevityDiseasegender inflammation age-related diseases longevityImmune systemAlzheimer DiseaseAnimalsHumansImmunologic FactorsSettore MED/05 - Patologia ClinicaDiseasemedia_commonSettore MED/04 - Patologia GeneraleSex CharacteristicsEstrogen Replacement TherapyLongevitySocial constructionismGender psychologySexual dimorphismImmune SystemFemaleInflammation MediatorsGeriatrics and GerontologyPsychologySex characteristicsHormoneClinical psychologyRejuvenation Research
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Immunogenetics, Gender, and Longevity

2007

In this article we discuss relevant data on aging, longevity, and gender with particular focus on inflammation gene polymorphisms which could affect an individual's chance to reach the extreme limit of human life. The present review is not an extensive revision of the literature, but rather an expert opinion based on selected data from the authors' laboratories. In 2000-2005 in the more developed regions, the life expectancy at birth is 71.9 years for men (78.3 in Japan) and 79.3 years for women (86.3 in Japan). Indeed, gender accounts for important differences in the prevalence of a variety of age-related diseases. Considering people of far-advanced age, demographic data document a clear-c…

MaleGerontologyAgingmedia_common.quotation_subjectLongevityPopulationDiseaseGeneral Biochemistry Genetics and Molecular BiologyHistory and Philosophy of ScienceHLA AntigensImmunogeneticsHumansMedicineSex Ratioeducationmedia_commonInflammationeducation.field_of_studySuccessful agingbusiness.industryGeneral NeuroscienceMortality rateAging Immune response Inflammation LongevityLongevityInfectious disease (medical specialty)Life expectancyFemalebusinessDeveloped countryAnnals of the New York Academy of Sciences
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The relevance of nutrition for the concept of cognitive frailty

2016

Purpose of review Physical and cognitive frailty are interrelated and synergistic syndromes more frequently seen in old age, which represent intermediate stages between aging successfully and disability. Poor nutrition is a fundamental determinant for both conditions, while various dietary components are proposed to prevent and/or improve them. This updated review discusses the possible influence of nutritional factors on cognitive frailty and its potential mediators. Recent findings Oxidative stress, low-grade systemic inflammation, neuroinflammation, and altered autophagy, all associated with obesity, metabolic syndrome and insulin resistance, are proposed mechanisms to explain the influe…

MaleGerontologySettore MED/09 - Medicina InternaMediterranean dietFrail ElderlyPsychological interventionNutritional StatusMedicine (miscellaneous)Affect (psychology)03 medical and health sciences0302 clinical medicineAutophagyHumansMental exerciseMedicineCognitive DysfunctionObesity030212 general & internal medicineOvereatingCognitive declineAgedAged 80 and overInflammationaging cognition frailty inflammation nutritionNutrition and DieteticsFrailtybusiness.industrymedicine.diseaseObesityMiddle ageOxidative StressCognitive AgingFemalebusiness030217 neurology & neurosurgeryCurrent Opinion in Clinical Nutrition & Metabolic Care
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Protein oxidation in chronic kidney disease.

2013

An imbalance between oxidative processes and antioxidant systems has been widely demonstrated in chronic kidney diseases (CKD). In this study we enrolled 26 healthy subjects, 27 patients with CKD on conservative treatment (CT-CKD) with various degrees of renal failure, and 31 CKD subjects in haemodialysis treatment (HD-CKD), evaluated before and after a standard haemodialysis session. In each group we measured protein carbonyl groups (PC) as an index of protein oxidation, lipid peroxidation (TBARS) and two plasma markers of leukocyte activation, elastase and myeloperoxidase (MPO). In CT-CKD subjects the PC level was significantly higher than in normal controls, and it was negatively correla…

MaleHEMODIALYSISmedicine.medical_specialtySettore MED/09 - Medicina InternaPhysiologyBIOMARKERSRenal functionurologic and male genital diseasesProtein oxidationThiobarbituric Acid Reactive SubstancesLipid peroxidationDiabetes Complicationschemistry.chemical_compoundCARBONYL STRESSMARKERSINFLAMMATIONGlycationRenal DialysisPhysiology (medical)Internal medicinemedicineTBARSHumansRenal Insufficiency ChronicPeroxidasebiologyPancreatic Elastasebusiness.industryNITRIC-OXIDE METABOLITESElastaseHematologyMiddle Agedmedicine.diseasefemale genital diseases and pregnancy complicationsOxidative StressEndocrinologychemistryMyeloperoxidaseNITRIC-OXIDE METABOLITES; CHRONIC-RENAL-FAILURE; CARBONYL STRESS; HEMODIALYSIS; BIOMARKERS; MARKERS; INFLAMMATIONImmunologybiology.proteinFemaleCHRONIC-RENAL-FAILURELipid PeroxidationCardiology and Cardiovascular MedicinebusinessOxidation-ReductionKidney diseaseClinical hemorheology and microcirculation
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Severe hypovitaminosis D correlates with increased inflammatory markers in HIV infected patients

2013

Abstract Background Even though it has been suggested that antiretroviral therapy has an impact on severe hypovitaminosis D (SHD) in HIV infected patients, it could be speculated that the different levels of residual inflammation on HAART (Highly Active Anti Retroviral Therapy) could contribute to SHD and aggravate bone catabolism in these patients. Methods A cross-sectional study was carried out in an unselected cohort of 263 HIV infected outpatients consulting during Spring 2010. Clinical examinations were performed and medical history, food habits, sun exposure and addictions were collected. Fasting blood samples were taken for immunological, virological, inflammation, endocrine and bone…

MaleHIV InfectionsGastroenterologyBone remodeling0302 clinical medicineRisk Factors[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesAntiretroviral Therapy Highly Active030212 general & internal medicineYoung adult0303 health sciencesUnivariate analysisHepatitis CMiddle Aged25-hydroxyvitamin D3. Good healthAntiretroviral therapy[ SDV.MHEP.MI ] Life Sciences [q-bio]/Human health and pathology/Infectious diseasesInfectious DiseasesCohort[SDV.MHEP.MI] Life Sciences [q-bio]/Human health and pathology/Infectious diseasesFemalemedicine.symptomInflammation MediatorsResearch ArticleAdultmedicine.medical_specialtyBone metabolismantiretroviral therapy;bone metabolism;HIV;inflammation;25-hydroxyvitamin DInflammationvitamin D deficiencyBone and Boneslcsh:Infectious and parasitic diseases03 medical and health sciencesYoung AdultInternal medicinemedicineHumansMedical historylcsh:RC109-216AgedInflammation030306 microbiologybusiness.industryHIVmedicine.diseaseVitamin D DeficiencyImmunologybusinessBiomarkersBMC Infectious Diseases
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Bimodal Oscillation Frequencies of Blood Flow in the Inflammatory Colon Microcirculation

2008

Rhythmic changes in blood flow direction have been described in the mucosal plexus of mice with acute colitis. In this report, we studied mice with acute colitis induced either by dextran sodium sulfate or by trinitrobenzenesulfonic acid. Both forms of colitis were associated with blood flow oscillations as documented by fluorescence intravital videomicroscopy. The complex oscillation patterns suggested more than one mechanism for these changes in blood flow. By tracking fluorescent nanoparticles in the inflamed mucosal plexus, we identified two forms of blood flow oscillations within the inflammatory mouse colon. Stable oscillations were associated with a base frequency of approximately 2 …

MaleHistologyColonInflammationBiologyArticleMicrocirculationMiceBiological ClocksmedicineAnimalsColitisEcology Evolution Behavior and SystematicsAcute colitisInflammationMice Inbred BALB CPlexusOscillationMicrocirculationBlood flowAnatomyColitismedicine.diseaseRegional Blood FlowBiophysicsAnatomymedicine.symptomBlood Flow VelocityIntravital microscopyBiotechnologyThe Anatomical Record: Advances in Integrative Anatomy and Evolutionary Biology
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Molecular adaptations of the blood–brain barrier promote stress resilience vs. depression

2020

Significance Thirty to fifty percent of depressed individuals are unresponsive to commonly prescribed antidepressant treatments, suggesting that biological mechanisms, such as stress-induced inflammation and blood vessel dysfunction, remain untreated. The blood–brain barrier is the ultimate frontier between the brain and harmful toxins or inflammatory signals circulating in the blood. Depression and vulnerability to chronic social stress are associated with loss of this barrier integrity; however, the mechanisms involved remain poorly understood. Identification of adaptations leading to resilience under stressful conditions could help develop novel treatments. Here we combined behavioral, p…

MaleHistone Deacetylase 1InflammationFOXO1Blood–brain barrierNucleus AccumbensEpigenesis GeneticProinflammatory cytokineMice03 medical and health sciences0302 clinical medicinevascularmedicineAnimalsHumansClaudin-5030304 developmental biologyInflammationSocial stressDepressive Disorder Major0303 health sciencesantidepressantMultidisciplinaryDepressionbusiness.industrySystems BiologyBiological Sciencesmedicine.diseasemood disordersAntidepressive Agents3. Good healthMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureMood disordersBlood-Brain BarrierMajor depressive disorderAntidepressantmedicine.symptombusinessNeuroscienceStress Psychologicalepigenetic030217 neurology & neurosurgerySignal TransductionProceedings of the National Academy of Sciences
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