Search results for "Inflammation."

showing 10 items of 2627 documents

Pre-treatment with mesenchymal stem cells reduces ventilator-induced lung injury

2012

ckground information: Bone marrow-derived mesenchymal stem cells (MSCs) reduce acute lung injury in animals challenged by bleomycin or bacterial lipopolysaccaride. It is not known, however, whether MSCs protect from ventilator- induced lung injury (VILI). Question of the study: Whether MSCs have a potential role in preventing or modulating VILI in healthy rats subjected to high-volume ventilation. Materials and methods: 24 Sprague-Dawley rats (250-300 g) were subjected to high- volume mechanical ventilation (25 ml/kg). MSCs (5x106) were intravenously or intratracheally administered (N=8 each) 30 min before starting over-ventilation and 8 rats were MSC-untreated. Spontaneously breathing anes…

MalePulmonary and Respiratory MedicinePathologymedicine.medical_specialtyAdministration TopicalVentilator-Induced Lung Injurymedicine.medical_treatmentVascular Cell Adhesion Molecule-1Pulmonary EdemaSettore MED/10 - Malattie Dell'Apparato RespiratorioLung injuryMesenchymal Stem Cell TransplantationBleomycinRats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAcute lung injury cell therapy injurious ventilation lung edema lung inflammation mechanical ventilationAnimalsMedicineMacrophageCell adhesionLung030304 developmental biologyMechanical ventilation0303 health sciencesmedicine.diagnostic_testbusiness.industryMesenchymal stem cellrespiratory systemRatsrespiratory tract diseasesDisease Models AnimalBronchoalveolar lavage030228 respiratory systemchemistryBreathingAdministration IntravenousInflammation MediatorsbusinessBronchoalveolar Lavage FluidEuropean Respiratory Journal
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Aprotinin inhibits leukocyte–endothelial cell interactions after hemorrhage and reperfusion

2003

Background. The serine protease inhibitor aprotinin has been successfully used to reduce blood loss in patients undergoing cardiac operations. We studied aprotinin for its ability to modulate leukocyte– endothelial cell interactions after ischemia and reperfusion. Methods. The effects of aprotinin on leukocyte– endothelial cell interactions were observed by intravital microscopy in the rat mesenteric microcirculation and immunohistochemical analysis. The inflammatory cascade (leukocyte rolling, firm adherence, and transmigration) was studied after thrombin stimulation and after hemorrhage and reperfusion. Results. Intravenous bolus administration of aprotinin treatment (20,000 U/kg) signifi…

MalePulmonary and Respiratory MedicineSerine Proteinase InhibitorsEndotheliumHemorrhageInflammationLeukocyte RollingCell CommunicationPharmacologyMicrocirculationRats Sprague-DawleyAprotininThrombinLeukocytesmedicineAnimalsMesenteryAprotininbusiness.industryMicrocirculationThrombinImmunohistochemistryRatsEndothelial stem cellP-Selectinmedicine.anatomical_structureReperfusionImmunologySurgeryEndothelium Vascularmedicine.symptomCardiology and Cardiovascular Medicinebusinesshormones hormone substitutes and hormone antagonistsIntravital microscopymedicine.drugThe Annals of Thoracic Surgery
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Effect of Haemodynamic Changes on Epithelium-related Intestinal Injury in Off-pump Coronary Surgery

2014

Intestinal injury is thought to play a central role in the occurrence of multiorgan dysfunction after on-pump coronary surgery. Clinical benefits of off-pump revascularisation remain, however, controversial.Hepatic enzymes and plasmatic IL-6, IL-8 and intestinal-type fatty acid binding protein (I-FABP) were determined in 20 patients (age 65-75) undergoing either on-pump (n = 10) or off-pump (n = 10) coronary surgery. Haemodynamic and biochemical parameters, catecholamine and volume therapy were monitored.Central venous pressure (CVP) was significantly higher in the off-pump group during and 12h after operation (9.5 ± 1.35 vs. 6.21 ± 0.63 mmH2O, p = 0.012). Higher GGT and GLDH levels occurre…

MalePulmonary and Respiratory Medicinemedicine.medical_specialtyEnterocyteMedizinCoronary Artery Bypass Off-PumpHemodynamicsInflammationFatty Acid-Binding ProteinsGastroenterologyIntestinal mucosaInternal medicinemedicineHumansIntestinal MucosaGamma-glutamyltransferaseInterleukin 6AgedbiologyInterleukin-6business.industryInterleukin-8HemodynamicsCentral venous pressuregamma-GlutamyltransferaseIntestinal Diseasesmedicine.anatomical_structureAnesthesiaCatecholaminebiology.proteinFemalemedicine.symptomCardiology and Cardiovascular Medicinebusinessmedicine.drugHeart, Lung and Circulation
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Adipose tissue in obesity and obstructive sleep apnoea

2011

A European Respiratory Society research seminar on ‘‘Metabolic alterations in obstructive sleep apnoea (OSA)’’ was jointly organised in October 2009 together with two EU COST actions (Cardiovascular risk in the obstructive sleep apnoea syndrome, action B26, and Adipose tissue and the metabolic syndrome, action BM0602) in order to discuss the interactions between obesity and OSA. Such interactions can be particularly significant in the pathogenesis of metabolic abnormalities and in increased cardiovascular risk in OSA patients. However, studying the respective role of OSA and obesity is difficult in patients, making it necessary to refer to animal models or in vitro systems. Since most OSA p…

MalePulmonary and Respiratory Medicinemedicine.medical_specialtyLipoxygenaseAdipose tissueMiceInsulin resistancestomatognathic systemAnimalsHumansMedicineObesityHypoxiaIntensive care medicineDyslipidemiasInflammationSleep Apnea Obstructivebusiness.industryAdipocyte dyslipidaemia hypoxia liver dysfunction obesitySleep apneaHypoxia (medical)medicine.diseaseObesitySleep in non-human animalsRatsnervous system diseasesrespiratory tract diseasesOxidative StressAdipose TissuePhysical therapyFemaleInsulin ResistanceLiver dysfunctionMetabolic syndromemedicine.symptombusinessEuropean Respiratory Journal
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Patterns of Inflammatory Responses in Large and Small Airways in Smokers with and without Chronic Obstructive Pulmonary Disease

2010

<i>Background:</i> Chronic obstructive pulmonary disease (COPD) is characterised by progressive and irreversible airway obstruction. Smoking causes persistent inflammation in lung tissue. However, differences in inflammatory responses between the large and small airways have not been systematically explored among smokers with and without COPD. <i>Objectives:</i> The aim of our research was to characterise the expression and localisation of NF-ĸBp65 and histone deacetylase 2 (HDAC2) as well as inflammatory cell (macrophages, lymphocytes, neutrophils) distribution in large and small airways, in nonsmokers and in smokers with and without COPD. <i>Methods:</i&gt…

MalePulmonary and Respiratory Medicinemedicine.medical_specialtyNeutrophilsHistone Deacetylase 2Pulmonary diseaseBronchiCell CountInflammationCD8-Positive T-Lymphocytesurologic and male genital diseasesPersistent inflammationPulmonary Disease Chronic ObstructiveRisk FactorsCarcinoma Non-Small-Cell LungInternal medicineMacrophages AlveolarCarcinomamedicineHumansLungAgedCOPDSmall airwaysbusiness.industrySmokingRespiratory diseaseTranscription Factor RelAMiddle Agedrespiratory systemAirway obstructionmedicine.diseaseImmunohistochemistryrespiratory tract diseasesImmunologyDisease ProgressionCardiologyFemalemedicine.symptombusinessRespiration
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In vitro anti-inflammatory effects of AZD8999, a novel bifunctional muscarinic acetylcholine receptor antagonist /β2-adrenoceptor agonist (MABA) comp…

2019

Recent evidence indicates that AZD8999 (LAS190792), a novel muscarinic acetylcholine receptor antagonist and β2-adrenoceptor agonist (MABA) in development for chronic respiratory diseases, induces potent and sustained relaxant effects in human bronchi by adressing both muscarinic acetylcholine receptors and β2-adrenoceptor. However, the anti-inflammatory effects of the AZD8999 monotherapy or in combination with corticosteroids are unknown. This study investigates the anti-inflammatory effects of AZD8999 in monotherapy and combined with fluticasone propionate in neutrophils from healthy and chronic obstructive pulmonary disease (COPD) patients. Peripheral blood neutrophils from healthy and C…

MalePulmonologyNeutrophilsPhysiologyAnti-Inflammatory AgentsPharmacologyPathology and Laboratory MedicineBiochemistryPulmonary Disease Chronic ObstructiveWhite Blood CellsGlucocorticoid receptorAnimal CellsMuscarinic acetylcholine receptorMedicine and Health SciencesPost-Translational ModificationPhosphorylationReceptorImmune ResponseMultidisciplinaryPharmaceuticsQRDrug SynergismMiddle AgedReceptors MuscarinicHealthy VolunteersBody FluidsChemistryBloodPhysical SciencesQuinolinesMedicineDrug Therapy CombinationFemalemedicine.symptomCellular TypesAnatomymedicine.drugResearch ArticleSignal TransductionAgonistTransmembrane Receptorsmedicine.drug_classp38 mitogen-activated protein kinasesChronic Obstructive Pulmonary DiseaseImmune CellsScienceImmunologyInflammationMuscarinic AntagonistsThiophenesFluticasone propionateSigns and SymptomsDrug TherapyCyclohexanesDiagnostic MedicinemedicineHumansAdrenergic beta-2 Receptor AgonistsAgedInflammationBlood CellsDose-Response Relationship Drugbusiness.industryAntagonistChemical CompoundsBiology and Life SciencesProteinsCell BiologyAcetylcholine ReceptorsFluticasoneMuscarinic Acetylcholine ReceptorsReceptors Adrenergic beta-2PropionatesbusinessReceptor Antagonist TherapyPLoS ONE
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Mechanical strain causes adaptive change in bronchial fibroblasts enhancing profibrotic and inflammatory responses

2016

Asthma is characterized by periodic episodes of bronchoconstriction and reversible airway obstruction; these symptoms are attributable to a number of factors including increased mass and reactivity of bronchial smooth muscle and extracellular matrix (ECM) in asthmatic airways. Literature has suggested changes in cell responses and signaling can be elicited via modulation of mechanical stress acting upon them, potentially affecting the microenvironment of the cell. In this study, we hypothesized that mechanical strain directly affects the (myo)fibroblast phenotype in asthma. Therefore, we characterized responses of bronchial fibroblasts, from 6 normal and 11 asthmatic non-smoking volunteers,…

MalePulmonologyPulmonary FibrosisAdult; Asthma; Biomechanical Phenomena; Bronchi; Case-Control Studies; Female; Fibroblasts; Humans; Male; Pneumonia; Pulmonary Fibrosis; Stress Mechanical; Medicine (all); Biochemistry Genetics and Molecular Biology (all); Agricultural and Biological Sciences (all)Glycobiologylcsh:MedicinePathology and Laboratory MedicineBiochemistryAnimal CellsMedicine and Health Scienceslcsh:ScienceImmune ResponseMusculoskeletal SystemConnective Tissue CellsSmooth MusclesMusclesMedicine (all)Extracellular MatrixBiomechanical PhenomenaConnective TissueFibroblastProteoglycansFemaleCellular TypesAnatomyCellular Structures and OrganellesCase-Control StudieResearch ArticleHumanAdultPulmonary FibrosiImmunologyBronchiSigns and SymptomsExtraction techniquesDiagnostic MedicineHumansInflammationBiochemistry Genetics and Molecular Biology (all)Settore BIO/16 - Anatomia Umanalcsh:RBiology and Life SciencesProteinsCell BiologyPneumoniaFibroblastsRNA extractionAsthmaResearch and analysis methodsBiological TissueAgricultural and Biological Sciences (all)Case-Control Studieslcsh:QStress MechanicalCollagens
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Involvement of the P2X7 purinergic receptor in colonic motor dysfunction associated with bowel inflammation in rats

2014

Background and Purpose Recent evidence indicates an involvement of P2X7 purinergic receptor (P2X7R) in the fine tuning of immune functions, as well as in driving enteric neuron apoptosis under intestinal inflammation. However, the participation of this receptor in the regulation of enteric neuromuscular functions remains undetermined. This study was aimed at investigating the role of P2X7Rs in the control of colonic motility in experimental colitis. Experimental Approach Colitis was induced in rats by 2,4-dinitrobenzenesulfonic acid. P2X7R distribution was examined by immunofluorescence analysis. The effects of A804598 (selective P2X7R antagonist) and BzATP (P2X7R agonist) were tested on co…

MalePurinergic P2X Receptor AntagonistsInflammatory Diseaseslcsh:MedicineInflammationGastroenterology and HepatologyBiologyInflammatory bowel diseaseSettore BIO/09 - FisiologiaGuanidinesPurinergic P2X Receptor AgonistsRats Sprague-DawleyImmune systemAdenosine TriphosphatemedicineMedicine and Health SciencesAnimalsP2X7 purinergic receptorColitisReceptorlcsh:SciencePurinergic P2X Receptor AgonistsPharmacologyNeuronsMultidisciplinaryPurinergic receptorlcsh:RBenzenesulfonatesMuscle Smoothmedicine.diseaseColitisRatsDisease Models AnimalImmunologyQuinolineslcsh:QP2X7 Purinergic Receptor Bowel Inflammation colonReceptors Purinergic P2X7medicine.symptomPurinergic P2X Receptor AntagonistsResearch Article
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Indicaxanthin from cactus pear fruit exerts anti-inflammatory effects in carrageenin-induced rat pleurisy.

2014

Nutritional research has shifted recently from alleviating nutrient deficiencies to chronic disease prevention. We investigated the activity of indicaxanthin, a bioavailable phytochemical of the betalain class from the edible fruit of Opuntia ficus-indica (L. Miller) in a rat model of acute inflammation. Rat pleurisy was achieved by injection of 0.2 mL of l-carrageenin in the pleural cavity, and rats were killed 4, 24, and 48 h later; exudates were collected to analyze inflammatory parameters, such as nitric oxide (NO), prostaglandin E2 (PGE2), interleukin-1b (IL-1b), and tumor necrosis factor-a (TNF-a); cells recruited in pleura were analyzed for cyclooxygenase-2 (COX-2), inducible nitric …

MalePyridinesmedicine.medical_treatmentPyridineAnti-Inflammatory AgentsMedicine (miscellaneous)PharmacologyCarrageenanchemistry.chemical_compoundLeukocytesInflammation MediatorPleural CavityNutrition and DieteticsbiologyOpuntiaBetaxanthinsNitric oxide synthaseAnti-Inflammatory AgentTumor necrosis factor alphamedicine.symptomInflammation MediatorsIndicaxanthinProstaglandin Emedicine.drug_classInflammationAnti-inflammatoryNitric oxidePlant ExtractmedicineAnimalsRNA MessengerRats WistarPleurisyInflammationIndicaxanthin phytochemicals inflammation pleurisy redox state.business.industryAnimalPlant ExtractsLeukocyteCarrageenanRatsDisease Models AnimalchemistryBetaxanthinFruitImmunologybiology.proteinRatbusinessPhytotherapyThe Journal of nutrition
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Association between the polymorphism of CCR5 and Alzheimer's disease: results of a study performed on male and female patients from Northern Italy.

2007

Alzheimer's disease (AD) is the most common cause of dementia in Western society. The prevalence of AD is greater in women than in men, largely due to longevity and survival differences favoring women. However, some studies suggest that incidence rates may really be increased in women. One possible factor influencing AD incidence in women is the loss of ovarian estrogens production after menopause, which might be involved in AD pathogenesis. Estrogens seem to influence some neuronal functions. Many of these actions appear beneficial (i.e., neuroprotective action against a variety of insults, as oxidative stress, and reduction of beta-amyloid plaques formation). Furthermore, several studies …

MaleReceptors CCR5DiseaseBiologyGeneral Biochemistry Genetics and Molecular BiologyCohort StudiesHistory and Philosophy of ScienceGene FrequencyPolymorphism (computer science)Alzheimer DiseaseGenotypemedicineDementiaHumansSex RatioAlleleAllele frequencyAllelesAgedSequence DeletionAged 80 and overInflammationPolymorphism GeneticGeneral NeuroscienceIncidence (epidemiology)Middle Agedmedicine.diseaseMenopauseItalyCase-Control StudiesImmunologyFemaleAnnals of the New York Academy of Sciences
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