Search results for "Inflammation."

showing 10 items of 2627 documents

Time-restricted eating effects on performance, immune function, and body composition in elite cyclists: a randomized controlled trial.

2020

Abstract Background Although there is substantial interest in intermittent fasting as a dietary approach in active individuals, information regarding its effects in elite endurance athletes is currently unavailable. The present parallel randomized trial investigated the effects of a particular intermittent fasting approach, called time-restricted eating (TRE), during 4 weeks of high-level endurance training. Methods Sixteen elite under-23 cyclists were randomly assigned either to a TRE group or a control group (ND). The TRE group consumed 100% of its estimated daily energy needs in an 8-h time window (from 10:00 a.m. to 6:00 p.m.) whilst energy intake in the ND group was distributed in 3 me…

MaleTime FactorsElite cyclists Endurance Immune system Inflammation Intermittent fastingPhysiologyIntermittent fastinglaw.inventionEnduranceLeukocyte Count0302 clinical medicineRandomized controlled trialWeight losslawIntermittent fastingElectric ImpedanceTestosteroneInsulin-Like Growth Factor INutrition and DieteticsFastingCreatinineBody Compositionmedicine.symptomlcsh:RC1200-1245lcsh:Nutrition. Foods and food supplySettore M-EDF/01 - Metodi E Didattiche Delle Attivita' MotorieBioelectrical impedance analysisResearch ArticleElite cyclistslcsh:TX341-641030209 endocrinology & metabolismClinical nutritionAthletic Performance03 medical and health sciencesYoung AdultEndurance trainingWeight LossmedicineHumansLymphocyte Countlcsh:Sports medicineInflammationbusiness.industryInterleukin-6Elite cyclists; Endurance; Immune system; Inflammation; Intermittent fasting030229 sport sciencesTransforming Growth Factor alphaGas analyzerBicyclingDietSports Nutritional Physiological PhenomenaImmune systemAthletesBasal metabolic rateBasal MetabolismbusinessEnergy IntakeFood ScienceJournal of the International Society of Sports Nutrition
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Effects of exercise training on airway responsiveness and airway cells in healthy subjects.

2010

J Appl Physiol. 2010 Aug;109(2):288-94. Epub 2010 Jun 10. Effects of exercise training on airway responsiveness and airway cells in healthy subjects. Scichilone N, Morici G, Zangla D, Chimenti L, Davì E, Reitano S, Paternò A, Santagata R, Togias A, Bellia V, Bonsignore MR. SourceDept. of Internal Medicine, Div. of Pulmonology (DIBIMIS Univ. of Palermo, "Villa Sofia-Cervello" Hospital, Via Trabucco 180, 90146 Palermo, Italy. n.scichilone@libero.it Abstract Airway responsiveness to methacholine (Mch) in the absence of deep inspirations (DIs) is lower in athletes compared with sedentary individuals. In this prospective study, we tested the hypothesis that a training exercise program reduces th…

MaleTime FactorsFunctional Residual CapacityPhysiologyVital CapacitySettore BIO/09 - FisiologiaBronchoconstrictor Agentsneutrophilsrowing; methacoline; neutrophilsForced Expiratory VolumeUteroglobinProspective StudiesRespiratory systemProspective cohort studyLungMethacholine Chloriderowingbiologyrespiratory systemResidual Volumemedicine.anatomical_structureInhalationAnesthesiaBronchial HyperreactivityInflammation MediatorsAirway responsivenessmedicine.drugAdultmedicine.medical_specialtyBronchoconstrictionPhysical exercisemethacolineSettore MED/10 - Malattie Dell'Apparato RespiratorioBronchial Provocation TestsYoung AdultPhysiology (medical)medicineHumansMuscle StrengthExerciseAthletesbusiness.industryInterleukin-8Total Lung CapacitySputumbiology.organism_classificationSpirometryPhysical therapyMethacholineSedentary BehaviorbusinessAirwayRespiratory tractJournal of applied physiology (Bethesda, Md. : 1985)
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A mouse model of in vivo chemical inhibition of retinal calcium-independent phospholipase A2 (iPLA2).

2013

International audience; Numerous studies have reported the implication of calcium-independent phospholipase A2 (iPLA2) in various biological mechanisms. Most of these works have used in vitro models and only a few have been carried out in vivo on iPLA2(-/-) mice. The functions of iPLA2 have been investigated in vivo in the heart, brain, pancreatic islets, and liver, but not in the retina despite its very high content in phospholipids. Phospholipids in the retina are known to be involved in several various key mechanisms such as visual transduction, inflammation or apoptosis. In order to investigate the implication of iPLA2 in these processes, this work was aimed to build an in vivo model of…

MaleTime Factors[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionDrug Evaluation PreclinicalInflammationBiochemistryRetinaGroup VI Phospholipases A2Mice03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePhospholipase A2In vivomedicineAnimalsHomeostasisEnzyme Inhibitors030304 developmental biology0303 health sciencesRetinaDose-Response Relationship DrugbiologyPancreatic isletsRetinalGeneral MedicineCell biologyMice Inbred C57BLmedicine.anatomical_structureBiochemistrychemistryApoptosisModels Animalbiology.proteinmedicine.symptom[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryVisual phototransduction
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Correlation of virus replication, cytokine (TNF-? and IL-1) producing cells, neuronal necrosis and inflammation after intranasal infection of mice wi…

1995

The number of TNF-alpha and IL-1 beta producing cells was investigated during the acute replication phase of herpes simplex virus (HSV) in trigeminal ganglia after intranasal infection with strains of different virulence. The highly virulent strain WAL replicated strongly and induced many cytokine producing cells early in the ganglia. The low virulent strain HFEM replicated less, only few cytokine producing cells were detected late. The thymidine-kinase negative (TK-) virus 1301 did not replicate but produced some lymphocytic inflammation. The higher the virulence of strains of HSV-1 or -2 was, the stronger was the extent of histopathological lesions; moreover, a dissociation in time betwee…

MaleTime Factorsmedicine.medical_treatmentVirulenceInflammationBiologyVirus Replicationmedicine.disease_causeHerpesviridaeVirusMiceNecrosisT-Lymphocyte SubsetsVirologymedicineAnimalsSimplexvirusAdministration IntranasalNeuronsMice Inbred BALB CTumor Necrosis Factor-alphaHerpes SimplexGeneral MedicineVirologyCytokineHerpes simplex virusTrigeminal GanglionViral replicationmedicine.symptomCD8Interleukin-1Archives of Virology
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Xanthine oxidase-induced oxidative stress causes activation of NF-κB and inflammation in the liver of type I diabetic rats

2009

We previously showed that xanthine oxidase activity increases in type I diabetic animals and that this is a significant cause of the oxidative stress which occurs in the disease. The aim of this work was to search for molecular links between xanthine oxidase-induced oxidative stress and inflammation in Type I diabetes and to assess the ability of allopurinol, a drug widely used in clinical practice, to prevent both processes. 3-month-old male Wistar rats were made diabetic by injection (i.p.) of either streptozotocin or alloxan. Allopurinol (32 mg/Kg) was administered (i.p) to diabetic rats after they had shown clear signs of diabetes such as glucosuria and polyuria. Hepatic phospho-IKKbeta…

MaleTranscriptional ActivationXanthine Oxidasemedicine.medical_specialtyNeutrophilsAllopurinolAllopurinolInterleukin 6Free radicalsInflammationmedicine.disease_causeBiochemistryStreptozocinDiabetes Mellitus ExperimentalDiabetic complicationsProinflammatory cytokineInterleukin 1βchemistry.chemical_compoundCell MovementPhysiology (medical)Internal medicineDiabetes mellitusAlloxanmedicineAnimalsRats WistarXanthine oxidasePolyuriabusiness.industryAllopurinol; Interleukin 1β; Interleukin 6; Diabetic complications; Free radicalsNF-kappa BXanthineStreptozotocinmedicine.diseaseRatsOxidative StressEndocrinologyLiverchemistryCytokinesInflammation Mediatorsmedicine.symptombusinessOxidative stressmedicine.drugFree Radical Biology and Medicine
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T helper type 17-related cytokine expression is increased in the bronchial mucosa of stable chronic obstructive pulmonary disease patients.

2009

Summary There are increased numbers of activated T lymphocytes in the bronchial mucosa of stable chronic obstructive pulmonary disease (COPD) patients. T helper type 17 (Th17) cells release interleukin (IL)-17 as their effector cytokine under the control of IL-22 and IL-23. Furthermore, Th17 numbers are increased in some chronic inflammatory conditions. To investigate the expression of interleukin (IL)-17A, IL-17F, IL-21, IL-22 and IL-23 and of retinoic orphan receptor RORC2, a marker of Th17 cells, in bronchial biopsies from patients with stable COPD of different severity compared with age-matched control subjects. The expression of IL-17A, IL-17F, IL-21, IL-22, IL-23 and RORC2 was measure…

MaleTranslational StudiesReceptors Retinoic Acidmedicine.medical_treatmentImmunologyautoimmunity bronchial biopsies emphysema neutrophilsInflammationBronchiInterleukin-23Polymerase Chain ReactionStatistics NonparametricPulmonary Disease Chronic ObstructiveAutoimmunity bronchial biopsies emphysema neutrophils pathologymedicineInterleukin 23Immunology and AllergyHumansRNA MessengerAgedDNA PrimersCOPDAnalysis of VarianceMucous MembraneReceptors Thyroid Hormonebusiness.industryInterleukinsRespiratory diseaseInterleukin-17SmokingInterleukinT-Lymphocytes Helper-InducerMiddle AgedNuclear Receptor Subfamily 1 Group F Member 3medicine.diseaseImmunohistochemistryrespiratory tract diseasesRespiratory Function TestsCytokineCase-Control StudiesImmunologyFemaleInterleukin 17medicine.symptombusinessCD8
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The antioxidative, non-psychoactive tricyclic phenothiazine reduces brain damage after experimental traumatic brain injury in mice.

2014

Abstract Oxidative stress due to free radical formation is an important mechanism of secondary brain damage following traumatic brain injury (TBI). Phenothiazine has been found to be a strong antioxidant in eukaryotic cells in vitro and in invertebrates in vivo. The present study was designed to determine the neuroprotective potency of unsubstituted phenothiazine in a paradigm of acute brain injury. Thirty minutes after pneumatic, controlled cortical impact (CCI) injury, C57BI6 mice were randomly assigned to “low dose” (3 mg/kg, LD) or “high dose” (30 mg/kg, HD) s.c. phenothiazine or vehicle treatment. Brain lesion, neurofunctional impairment, body weight, and markers of cerebral inflammati…

MaleTraumatic brain injuryGene ExpressionInflammationCell CountBrain damagePharmacologymedicine.disease_causeNeuroprotectionAntioxidantsRandom AllocationIn vivoPhenothiazinesMedicineAnimalschemistry.chemical_classificationInflammationDose-Response Relationship Drugbusiness.industryGeneral NeuroscienceCalcium-Binding ProteinsMicrofilament ProteinsBrainmedicine.diseaseMice Inbred C57BLchemistryAnesthesiaBrain InjuriesTumor necrosis factor alphamedicine.symptombusinessNeurogliaOxidative stressTricyclicNeuroscience letters
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Potential role of heme oxygenase-1 in the progression of rat adjuvant arthritis

2004

Rat adjuvant arthritis is an experimental model widely used to evaluate etiopathogenetic mechanisms in chronic inflammation. We have examined the participation of heme oxygenase-1 (HO-1) in this experimental arthritis. In this study, an increased nitric oxide (NO) production in the paw preceded the upregulation of HO-1, whereas selective inhibition of inducible NO synthase (iNOS) after the onset of arthritis decreased HO-1 expression, suggesting that the induction of this enzyme may depend on NO produced by iNOS. Therapeutic administration of the HO-1 inhibitor tin protoporphyrin IX was able to control the symptoms of arthritis. This agent significantly decreased leukocyte infiltration, hyp…

MaleVascular Endothelial Growth Factor AOsteolysisAngiogenesisNitric Oxide Synthase Type IIProtoporphyrinsArthritisInflammationPharmacologyNitric OxidePathology and Forensic MedicineSynovitismedicineAnimalsEnzyme InhibitorsMolecular BiologyHeat-Shock ProteinsbiologyTumor Necrosis Factor-alphabusiness.industryLysineCell Biologymedicine.diseaseArthritis ExperimentalHindlimbRatsUp-RegulationNitric oxide synthaseHeme oxygenaseDisease Models AnimalCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesRats Inbred LewHeme Oxygenase (Decyclizing)ImmunologyOxygenasesbiology.proteinNitric Oxide Synthasemedicine.symptomVascular endothelial growth factor productionbusinessLaboratory Investigation
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Endothelial Dysfunction in Tristetraprolin-deficient Mice Is Not Caused by Enhanced Tumor Necrosis Factor-α Expression

2014

Cardiovascular events are important co-morbidities in patients with chronic inflammatory diseases like rheumatoid arthritis. Tristetraprolin (TTP) regulates pro-inflammatory processes through mRNA destabilization and therefore TTP-deficient mice (TTP(-/-) mice) develop a chronic inflammation resembling human rheumatoid arthritis. We used this mouse model to evaluate molecular signaling pathways contributing to the enhanced atherosclerotic risk in chronic inflammatory diseases. In the aorta of TTP(-/-) mice we observed elevated mRNA expression of known TTP targets like tumor necrosis factor-α (TNF-α) and macrophage inflammatory protein-1α, as well as of other pro-atherosclerotic mediators, l…

MaleVasculitismedicine.medical_specialtyMRNA destabilizationRNA StabilityTristetraprolinInflammationBiochemistryNitric oxideMicechemistry.chemical_compoundOrgan Culture TechniquesTristetraprolinhemic and lymphatic diseasesInternal medicinemedicineAnimalsEndothelial dysfunctionMolecular BiologyAortaReactive nitrogen speciesMice KnockoutMembrane GlycoproteinsNADPH oxidasebiologyTumor Necrosis Factor-alphaEndothelial CellsNADPH OxidasesMolecular Bases of DiseaseCell Biologyrespiratory systemAtherosclerosismedicine.diseaseReactive Nitrogen SpeciesMice Inbred C57BLOxidative StressCholesterolEndocrinologychemistryMice Inbred DBAChronic DiseaseNADPH Oxidase 2biology.proteinFemaleTumor necrosis factor alphamedicine.symptomReactive Oxygen SpeciesJournal of Biological Chemistry
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Associations of black carbon with lung function and airway inflammation in schoolchildren

2019

Background: Few studies have investigated the 24-hour respiratory health effects of personal black carbon (BC) and ultrafine particles (UFP) exposure in schoolchildren. The objective of this study was to investigate these associations with the lung function in children 10-years old with and without persistent respiratory symptoms. Methods: We conducted a cross-sectional study in 305 children (147 and 158 with and without persistent respiratory symptoms, respectively) from three European birth-cohorts: PARIS (France) and INMA Sabadell and Valencia (Spain). Personal 24-hour measurements of exposure concentrations to BC and UFP were performed by portable devices, before lung function testing. …

MaleVital capacity010504 meteorology & atmospheric sciencesVital Capacity010501 environmental sciences[SDV.MHEP.PSR]Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tract01 natural sciencesLung function parameters (FVC and FEV1)Forced Expiratory VolumeRespiratory systemChildLunglcsh:Environmental sciencesLung functionGeneral Environmental Sciencelcsh:GE1-350Airway inflammationrespiratory systemRespiratory Function Tests3. Good healthChild PreschoolBreathingFemaleFrancemedicine.medical_specialtyLung function parameters (FVC and FEV(1))SchoolchildrenNitric OxideFEV1/FVC ratioBlack carbonExposure concentrationsSootInternal medicineLung function parameters (FVC and FEV1medicineHumansFeNOBronchitis0105 earth and related environmental sciencesInflammation[SDV.EE.SANT]Life Sciences [q-bio]/Ecology environment/Health[SDV.MHEP.PED]Life Sciences [q-bio]/Human health and pathology/Pediatricsbusiness.industryInfant NewbornInfantConfidence intervalrespiratory tract diseasesCross-Sectional Studies) FeNOUltrafine particlesSpainExhaled nitric oxideParticulate Matter[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologiebusiness
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