Search results for "Inflammation."

showing 10 items of 2627 documents

Computational Analysis of Clinical and Molecular Markers and New Theranostic Possibilities in Primary Open-Angle Glaucoma

2020

Primary open-angle glaucoma (POAG) is a paramount cause of irreversible visual disability worldwide. We focus on identifying clinical and molecular facts that may help elucidating the pathogenic mechanisms of the disease. By using ophthalmological approaches (biomicroscopy, ocular fundus, optical coherence tomography, and perimetry) and experimental tests (enzyme-linked immunosorbent assay (ELISA), high performance liquid chromatography (HPLC), and Western blot/immunoblotting) directed to evaluate the oxidative stress, inflammation, apoptosis, and neurodegeneration processes, we gather information to build a network of data to perform a computational bioinformatics analysis. Our results sho…

Posterior Eye Segmentmedicine.medical_specialtytheranosticsprimary open-angle glaucomaOpen angle glaucomagenetic structuresGlaucomalcsh:MedicineVisual disabilityDiseaseArticlePathogenesis03 medical and health sciences0302 clinical medicineOphthalmologymedicineoxidative stressComputational analysisbusiness.industrypathogenesisNeurodegenerationlcsh:RapoptosisneurodegenerationGeneral Medicinemedicine.diseaseeye diseasesinflammation030221 ophthalmology & optometrysense organsbusiness030217 neurology & neurosurgeryJournal of Clinical Medicine
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A new method to isolate microglia from adult mice and culture them for an extended period of time

2009

As the major immuno-competent cells of the brain, microglia are highly implicated in neuro-protection as well as in neurodegeneration. Therefore, they are of key interest for research on numerous CNS diseases. Currently, to model inflammation in the brain, microglial cell lines or primary microglia prepared from embryonic or neo-natal rodents are widely used. However, these in vitro microglial models are not suitable for research in the field of neuro-degenerative diseases where aging is a crucial parameter. Only a few in vitro studies on aged microglia have been published so far, most of which use ex vivo microglia which cannot be kept in culture for prolonged periods of time. In the prese…

Potassium Channelsmedicine.medical_treatmentPopulationFluorescent Antibody TechniqueInflammationCell SeparationBiologyNitric OxideCell LineMicePhagocytosismedicineAnimalseducationCells Culturededucation.field_of_studyMicrogliaGeneral NeuroscienceCell MembraneNeurodegenerationFlow Cytometrymedicine.diseaseEmbryonic stem cellIn vitroElectrophysiologyMice Inbred C57BLCytokinemedicine.anatomical_structureAnimals NewbornCytokinesFemaleMicrogliamedicine.symptomNeuroscienceEx vivoJournal of Neuroscience Methods
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Anti-Aging Physiological Roles of Aryl Hydrocarbon Receptor and Its Dietary Regulators

2021

The vast majority of the literature on the aryl hydrocarbon receptor is concerned with its functions in xenobiotic detoxification. However, in the course of evolution, this receptor had to have physiological (rather than toxicological) functions. Our aim was to review the aryl hydrocarbon receptor’s role in the physiological functions involved in aging. This study was performed by searching the MEDLINE and Google Academic databases. A total of 34 articles were selected that focused specifically on the aryl hydrocarbon receptor and aging, the aryl hydrocarbon receptor and physiological functions, and the combination of both. This receptor’s main physiological functions (mediated by the modul…

Premature agingAgingCellInflammationReviewphysiological functionCatalysisXenobioticslcsh:ChemistryInorganic Chemistrychemistry.chemical_compoundDetoxificationmedicineAnimalsHumansPhysical and Theoretical ChemistryReceptorlcsh:QH301-705.5Molecular BiologySpectroscopybiologyaryl hydrocarbon receptorChemistryOrganic ChemistryAging PrematureGeneral MedicineAryl hydrocarbon receptorComputer Science ApplicationsCell biologymedicine.anatomical_structurelcsh:Biology (General)lcsh:QD1-999Receptors Aryl Hydrocarbonbiology.proteinmedicine.symptomSignal transductionXenobioticSignal TransductionInternational Journal of Molecular Sciences
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P80Cannabinoid receptor CB2 prevents development of heart failure in a murine model of pressure overload

2014

Purpose: Cardiac adaptation to pressure overload is associated with inflammatory reaction, which untreated leads to myocardial fibrosis and heart failure. We have recently demonstrated that endogenous cannabinoids and the cannabinoid receptor 2 (CB2) are activated and associated with persistent inflammation in hypertrophic myocardium of patients with aortic valve stenosis. Therefore, we investigated the role of the CB2 in a mouse model of pressure overload. Methods: Transverse aortic constriction was performed in CB2-/--mice and their wildtype littermates (CB2+/+; n=8-12/group). Taqman® RT-qPCR analysis was performed after 3 and 7 days. After M-mode echocardiography and Millar® pressure-vol…

Pressure overloadmedicine.medical_specialtyPhysiologybusiness.industryInflammationmedicine.diseaseMuscle hypertrophyInterleukin 10EndocrinologyPhysiology (medical)Heart failureInternal medicineAortic valve stenosismedicineCardiologylipids (amino acids peptides and proteins)Myocardial fibrosismedicine.symptomCardiology and Cardiovascular MedicinebusinessReceptorCardiovascular Research
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Compromised central tolerance of ICA69 induces multiple organ autoimmunity

2014

For reasons not fully understood, patients with an organ-specific autoimmune disease have increased risks of developing autoimmune responses against other organs/tissues. We identified ICA69, a known β-cell autoantigen in Type 1 diabetes, as a potential common target in multi-organ autoimmunity. NOD mice immunized with ICA69 polypeptides exhibited exacerbated inflammation not only in the islets, but also in the salivary glands. To further investigate ICA69 autoimmunity, two genetically modified mouse lines were generated to modulate thymic ICA69 expression: the heterozygous ICA69(del/wt) line and the thymic medullary epithelial cell-specific deletion Aire-ΔICA69 line. Suboptimal central neg…

Primary Sjogren's syndromeGenetically modified mouseImmunologyThyroid GlandAutoimmune diabeteMice TransgenicThymus GlandBiologymedicine.disease_causeAutoantigensArticleSalivary GlandsSettore MED/13 - EndocrinologiaAutoimmune DiseasesAutoimmunityImmune toleranceAutoimmune thyroiditisIslets of LangerhansMiceICA69Mice Inbred NODImmune TolerancemedicineAnimalsImmunology and AllergyThymuAutoimmune thyroiditiNOD miceInflammationAutoimmune diseaseStomachmedicine.diseaseGene Expression RegulationAutoimmune polyendocrine syndromeImmunologyAutoimmune polyendocrine syndromeCentral toleranceJournal of Autoimmunity
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Incretin-Based Therapies Role in COVID-19 Era: Evolving Insights

2020

The current coronavirus disease 2019 (COVID-19) pandemic has led the scientific community to breach new frontiers in the understanding of human physiology and disease pathogenesis. It has been hypothesized that the human dipeptidyl peptidase 4 (DPP4) enzyme receptor may be a functional target for the spike proteins of severe acute respiratory syndrome-coronavirus-2 (SARS-CoV-2). Since DPP4-inhibitors are currently used for the treatment of patients with type-2 diabetes (T2DM), there is currently high interest in the possibility that these agents, or incretin-based therapies (IBTs) in general, may be of benefit against the new coronavirus infection. Diabetes is associated with increased COV…

Prognostic variableCoronavirus disease 2019 (COVID-19)Pneumonia ViralIncretin030209 endocrinology & metabolism030204 cardiovascular system & hematologymedicine.disease_causeBioinformaticsIncretinsSeverity of Illness IndexGlucagon-Like Peptide-1 ReceptorBetacoronavirus03 medical and health sciences0302 clinical medicineDiabetes mellitusPandemicSeverity of illnessHumansHypoglycemic AgentsMedicinePharmacology (medical)PandemicsDipeptidyl peptidase-4CoronavirusPharmacologyDipeptidyl-Peptidase IV InhibitorsSARS-CoV-2business.industryCOVID-19medicine.diseaseDiabetes Mellitus Type 2Inflammation MediatorsCoronavirus InfectionsCardiology and Cardiovascular Medicinebusinessdiabetes DPP4 GLP1 incretins Betacoronavirus COVID-19 Coronavirus Infections Diabetes Mellitus Type 2 Dipeptidyl-Peptidase IV Inhibitors Glucagon-Like Peptide-1 Receptor Humans Hypoglycemic Agents Incretins Inflammation Mediators Pandemics Pneumonia Viral SARS-CoV-2 Severity of Illness Index
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Modulation of Nrf2/ARE pathway by food polyphenols: a nutritional neuroprotective strategy for cognitive and neurodegenerative disorders

2011

In recent years, there has been a growing interest, supported by a large number of experimental and epidemi-ological studies, for the beneficial effects of some phenolic substances, contained in commonly used spices and herbs, in preventing various age-related pathologic conditions, ranging from cancer to neurodegenerative diseases. Although the exact mechanisms by which polyphenols promote these effects remain to be elucidated, several reports have shown their ability to stimulate a general xenobiotic response in the target cells, activating multiple defense genes. Data from our and other laboratories have previously demonstrated that curcumin, the yellow pigment of curry, strongly induces…

Programmed cell deathAntioxidantCurcuminNF-E2-Related Factor 2medicine.medical_treatmentCentral nervous systemNeuroscience (miscellaneous)InflammationPharmacologyBiologyResponse ElementsHeterodimers of NF-E2-related factors 2(Nrf2) Antioxidant responsive element (ARE) Heme oxygenase 1 (HO-1) Neurodegenerative disorders Alzheimer’s disease Polyphenols Curcumin (-)- epigallocatechin-3- gallate (EGCG) Brain ageingNeuroprotectionAntioxidantsCatechinArticleCellular and Molecular Neurosciencechemistry.chemical_compoundmedicineAnimalsHumansCognitive declineCaffeic acid phenethyl esterSettore MED/04 - Patologia GeneraleMolecular StructurePolyphenolsNeurodegenerative DiseasesDietmedicine.anatomical_structureNeuroprotective AgentsNeurologyBiochemistrychemistryFoodCurcuminmedicine.symptomCognition DisordersHeme Oxygenase-1
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Acute organ failure following the loss of anti-apoptotic cellular FLICE-inhibitory protein involves activation of innate immune receptors

2014

Apoptosis signaling is involved in both physiological tissue homeostasis and acute and chronic diseases. The role of regulatory apoptosis signaling molecules and their organ-specific functions are less defined. Therefore, we investigated the loss of the anti-apoptotic cellular FLICE-inhibitory protein (cFLIP) and the mechanisms of the resulting lethal organ failure in vivo using inducible knockout mice. These were generated by crossing floxed cFLIP mice to a tamoxifen inducible Rosa26-creERT2 mouse strain. Death following global loss of cFLIP resulted from liver failure, accumulation of M1-polarized macrophages and accompanying hepatic cell death and inflammation. Apoptosis was also promine…

Programmed cell deathCASP8 and FADD-Like Apoptosis Regulating ProteinMice TransgenicInflammationBiologyMiceImmune systemmedicineAnimalsMolecular BiologyTissue homeostasisOriginal PaperInnate immune systemMacrophagesMembrane ProteinsCell BiologyLiver Failure AcuteImmunity InnateCell biologyToll-Like Receptor 4TransplantationApoptosisToll-Like Receptor 9Stimulator of interferon genesHepatocytesmedicine.symptomCell Death & Differentiation
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Biological activities of the LXRα and β agonist, 4β-hydroxycholesterol, and of its isomer, 4α-hydroxycholesterol, on oligodendrocytes: effects on cel…

2013

The biochemical and biological properties of 4β-hydroxycholesterol and of its isomer, 4α-hydroxycholesterol, are not well known. So, we determined the ability of 4α- and 4β-hydroxycholesterol to react with LXRα and LXRβ, and we characterized the activities of these oxysterols on oligodendrocytes which are myelin synthesizing cells. The effects of 4α- and 4β-hydroxycholesterol were studied on 158N murine oligodendrocytes to assess their activities on cell growth and viability, oxidative and inflammatory status. To this end different parameters were used: cell counting with trypan blue; identification of dead cells and cell cycle analysis with propidium iodide; evaluation of mitochondrial dep…

Programmed cell deathCell SurvivalBiologyBiochemistrychemistry.chemical_compoundMiceIsomerismpolycyclic compoundsmedicineAnimalsPropidium iodideProtein Structure QuaternaryCell ProliferationLiver X ReceptorsInflammationSuperoxideCell growthAcridine orangeDepolarizationGeneral MedicineOrphan Nuclear ReceptorsOligodendrocyteActinsHydroxycholesterolsCell biologyMitochondriaOligodendrogliamedicine.anatomical_structurechemistryCytokineslipids (amino acids peptides and proteins)Trypan blueProtein MultimerizationLysosomesReactive Oxygen SpeciesOxidation-ReductionBiochimie
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Natural Products as Modulators of Apoptosis and their Role in Inflammation

2006

ABSTRACT: Modifications in apoptosis, a programmed form of cell death that participates in a wide variety of physiological processes, can contribute to many diseases, principally those associated with cell accumulation, such as inflammation. One hallmark of inflammation is the infiltration of leukocytes, which are programmed to undergo apoptosis at inflamed tissues. Resolution of inflammation thus involves the elimination of excess inflammatory cells by physiological cell death and the subsequent removal of apoptotic cells by phagocytes. Controlling this process may therefore be useful in the treatment of pathologies, which include an inflammatory component. Interestingly, while in some cas…

Programmed cell deathCellInflammationDiseaseBiologymedicine.diseasemedicine.anatomical_structureApoptosisImmunologymedicineCancer researchEczematous dermatitismedicine.symptomInfiltration (medical)Glucocorticoidmedicine.drug
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