Search results for "Innate"

showing 10 items of 638 documents

Inflammation, Longevity, and Cardiovascular Diseases: Role of Polymorphisms of TLR4

2006

The total burden of infection at various sites may affect the progression of atherosclerosis, the risk being modulated by host genotype. The role of lipopolysaccaride receptor TLR4 is paradigmatic. It initiates the innate immune response against gram-negative bacteria; and TLR4 polymorphisms, as ASP299GLY, suggested to attenuate receptor signaling, have been described. We demonstrated that TLR4 ASP299GLY polymorphism shows a significantly lower frequency in patients affected by myocardial infarction compared to controls, whereas centenarians show a higher frequency. Thus, people genetically predisposed to developing weak inflammatory activity, seem to have fewer chances of developing cardio…

AdultLipopolysaccharidesMaleHeterozygoteTime Factorsmedia_common.quotation_subjectmedicine.medical_treatmentLongevityMyocardial InfarctionEnzyme-Linked Immunosorbent AssayInflammationBiologyGeneral Biochemistry Genetics and Molecular BiologyAMIHistory and Philosophy of SciencemedicineHumansGenetic Predisposition to DiseaseTLR4Interleukin 6media_commonInflammationPolymorphism GeneticInnate immune systemInterleukin-6General NeuroscienceLongevityInterleukinHeterozygote advantageMiddle AgedToll-Like Receptor 4CytokineAcute DiseaseMutationImmunologyTLR4biology.proteinFemalemedicine.symptomAnnals of the New York Academy of Sciences
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Proinflammatory CX3CR1+CD59+Tumor Necrosis Factor–Like Molecule 1A+Interleukin‐23+ Monocytes Are Expanded in Patients With Ankylosing Spondylitis and…

2018

Objective: Gut-derived innate lymphoid cell 3 (ILC3) has been shown to participate in the pathogenesis of ankylosing spondylitis (AS). CX3CR1+ mononuclear phagocytes (MNPs) have been demonstrated to modulate ILC3 function in the gut. This study was undertaken to investigate the role of proinflammatory CX3CR1+CD59+ MNPs in modulating ILC3 function in AS patients. Methods: MNP subsets in the blood of AS patients and controls were analyzed by flow cytometry. The presence of CX3CR1+CD59+ cells in tissue was confirmed by confocal microscopy. Expression of the proinflammatory chemokines CX3CL1 and CCL2 and decoy receptor 6 (DcR-6) was analyzed. Peripheral CX3CR1+CD59+ cells were cocultured with I…

AdultMale0301 basic medicineChemokineImmunologyPopulationCX3C Chemokine Receptor 1CD11cCD59 Antigenschemical and pharmacologic phenomenaCCL2Interleukin-23MonocytesProinflammatory cytokineFlow cytometry03 medical and health sciences0302 clinical medicineRheumatologymedicineHumansImmunology and AllergySpondylitis AnkylosingLymphocytesCX3CL1educationMononuclear Phagocyte System030203 arthritis & rheumatologyeducation.field_of_studybiologymedicine.diagnostic_testChemistryInnate lymphoid cellMiddle AgedImmunity Innate030104 developmental biologyReceptors Tumor Necrosis Factor Type ICase-Control Studiesbiology.proteinCancer researchFemaleArthritis & Rheumatology
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Characterization of γδ T cells in intestinal mucosa from patients with early onset or long standing inflammatory bowel disease and their correlation …

2019

Abstract Background and Aims Inflammatory bowel disease [IBD] is a complex chronic inflammatory disease of the human gut with no clear aetiology. Traditionally, dysregulated adaptive immune responses play an important role even though accumulating evidence suggests a role also for innate immunity. Because of the well-known plasticity of γδ T cells, we investigated their percentage occurrence, phenotypic features and effector functions in the intestinal mucosa of early-onset and long-standing IBD patients, as compared to healthy subjects. Methods Fresh biopsies from 30 Crohn’s disease and ulcerative colitis patients were obtained and digested, and cells were analysed by flow cytometry. Resul…

AdultMale0301 basic medicineNecrosisAdolescentBiopsyT-LymphocytesInflammatory bowel disease03 medical and health sciences0302 clinical medicineImmune systemIntestinal mucosainflammatory bowel diseasemedicineHumansgamma delta T cellsIntestinal MucosaAgedAged 80 and overInnate immune systembusiness.industryGastroenterologyGeneral MedicineMiddle AgedFlow CytometryInflammatory Bowel Diseasesmedicine.diseaseUlcerative colitisPhenotype030104 developmental biologyImmunologyFemaleTumor necrosis factor alphaInterleukin 17medicine.symptombusinessBiomarkers030215 immunology
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Genetic variations of the bitter taste receptor TAS2R38 are associated with obesity and impact on single immune traits

2015

Scope: Changes in genetic variations affecting the taste receptor, type 2, member 38 (TAS2R38) may identify the interacting mechanism leading to obesity and potential associations with proteins partaking in innate immunity, such as surfactant protein D (SPD) and mannan-binding lectin (MBL). Methods and results: We evaluated haplotypes of the bitter-taste receptor TAS2R38 in an identification sample of 210 women in different weight conditions, including anorexia nervosa and obesity. The association with SPD and MBL was tested in an independent sample picturing general population (n = 534). The relationship with obesity was validated in an extended final sample of 1319 participants. In the sa…

AdultMale0301 basic medicinemedicine.medical_specialtyAdolescentPopulation030209 endocrinology & metabolismSingle-nucleotide polymorphismBiologyMannose-Binding LectinPolymorphism Single NucleotideBody Mass IndexReceptors G-Protein-CoupledCohort StudiesYoung Adult03 medical and health sciences0302 clinical medicineInternal medicinemental disordersGenetic variationmedicineHumansObesityeducationAgedGeneticseducation.field_of_study030109 nutrition & dieteticsBody WeightSmokingHaplotypeGPR120Middle AgedPulmonary Surfactant-Associated Protein Dmedicine.diseaseObesityImmunity InnateTAS2R38EndocrinologyHaplotypesAnorexia nervosa (differential diagnoses)Case-Control StudiesTasteFemaleFood ScienceBiotechnologyMolecular Nutrition & Food Research
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Granulocyte and natural killer activity in the elderly

1999

The deterioration of the immune system in ageing, 'immunosenescence', is thought to contribute to increased morbidity and mortality from infections and possibly autoimmune diseases and cancer. The most profound changes involve effector and immunoregulatory T-cell functions. Immunosenescence appears also to be related to changes in non specific immunity as well. In the present study we have assessed superoxide production, chemotaxis and the expression of the apoptosis-related molecule APO1/Fas (CD95) on neutrophils (PMN) from young and old subjects. Furthermore, we have measured the basal natural killer (NK) activity of young and elderly subjects and we have compared the number of CD16+ cell…

AdultMaleAgingmedicine.medical_specialtyNeutrophilschemical and pharmacologic phenomenaBiologyCD16Natural killer cellImmune systemInternal medicinemedicineHumansAgedAged 80 and overInnate immune systemEffectorChemotaxisImmunosenescenceMiddle AgedFas receptorKiller Cells Naturalmedicine.anatomical_structureEndocrinologyImmunologyFemaleDevelopmental BiologyMechanisms of Ageing and Development
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Association Between Individual and Combined SNPs in Genes Related to Innate Immunity and Incidence of CMV Infection in Seropositive Kidney Transplant…

2014

In this study, we assessed the association between single-nucleotide polymorphisms (SNPs) in seven candidate genes involved in orchestrating the immune response against cytomegalovirus (CMV) and the 12-month incidence of CMV infection in 315 CMV-seropositive kidney transplant (KT) recipients. Patients were managed either by antiviral prophylaxis or preemptive therapy. CMV infection occurred in 140 patients (44.4%), including 13 episodes of disease. After adjusting for various clinical covariates, patients harboring T-allele genotypes of interleukin-28B (IL28B) (rs12979860) SNP had lower incidence of CMV infection (adjusted hazard ratio [aHR]: 0.66; 95% confidence interval [CI]: 0.46-0.96; p…

AdultMaleCandidate geneGenotypeReceptors Cell SurfaceSingle-nucleotide polymorphismDiseasePolymorphism Single NucleotideImmune systemGenotypeOdds RatioHumansImmunology and AllergyMedicineSNPLectins C-TypePharmacology (medical)Prospective StudiesAllelesAgedProportional Hazards ModelsTransplantationbusiness.industryIncidenceInterleukinsHazard ratioTLR9Middle AgedKidney TransplantationImmunity InnateTransplant RecipientsCytomegalovirus InfectionsImmunologyKidney Failure ChronicFemaleInterferonsbusinessCell Adhesion MoleculesAmerican Journal of Transplantation
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In vitroresponse toCandida albicansin cultures of whole human blood from young and aged donors

2007

Invasive infections with opportunistic fungi, such as Candida albicans, have become an increasing problem in aged adults in recent years. This work investigates the influence of human ageing on C. albicans recognition by toll-like receptors (TLRs), essential components of the innate immune system, using a cohort of 96 young (15-42 years) and aged (70 years) human volunteers. No significant differences between aged and young donors were observed on (1) cell surface TLR2, TLR6 and TLR4 expression on lymphocytes, monocytes and granulocytes, (2) production of cytokines [IL-8, IL-1beta, IL-6, IL-10, tumour necrosis factor (TNF)-alpha and IL-12p70] and prostaglandin E(2) (PGE(2)) by whole human b…

AdultMaleMicrobiology (medical)Adolescentmedicine.medical_treatmentImmunologyBiologyMicrobiologyDinoprostoneMonocytesCandida albicansmedicineHumansImmunology and AllergyBlood cultureLymphocytesCandida albicansAntibodies FungalAgedWhole bloodAged 80 and overMicrobial ViabilityInnate immune systemmedicine.diagnostic_testAge FactorsGeneral Medicinebiology.organism_classificationToll-Like Receptor 2Corpus albicansToll-Like Receptor 4TLR2BloodToll-Like Receptor 6Infectious DiseasesCytokineImmunologybiology.proteinCytokinesFemaleAntibodyGranulocytesFEMS Immunology & Medical Microbiology
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Over-expression of paneth cell-derived anti-microbial peptides in the gut of patients with ankylosing spondylitis and subclinical intestinal inflamma…

2010

OBJECTIVES: Subclinical gut inflammation has been demonstrated in patients with AS. Altered expression of paneth cell (PC) anti-microbial peptides have been reported in the inflamed ileum of patients with Crohn's disease (CD). Here, we investigated the expression of PC-derived peptides in subclinical gut inflammation in AS. METHODS: Multiple adjacent mucosal biopsies from terminal ileum were obtained from 25 patients with AS, 30 CD and 15 healthy controls (HCs). Expression of human α-defensin 5 (HD-5), phospholipase A2 (PLA2), lysozyme and SOX-9 molecules was assessed by quantitative Taqman RT-PCR on mucosal samples. Immunohistochemistry with anti-human HD-5 antibody and genotyping of relev…

AdultMalePaneth CellsPathologymedicine.medical_specialtyGene ExpressionInflammationIleumdigestive systemRheumatologyNOD2ankylosing spondylitismedicineHumansSpondylitis AnkylosingPharmacology (medical)IleitisPrecordial catch syndromeSubclinical infectionPaneth cellInnate immune systembusiness.industryMiddle Agedmedicine.diseaseGastroenteritisPaneth cells alpha-defensin ankylosing spondylitismedicine.anatomical_structureCase-Control StudiesPaneth cellImmunologyalpha-defensinFemalemedicine.symptombusinessAntimicrobial Cationic PeptidesRheumatology
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Type 3 innate lymphoid cells producing IL-17 and IL-22 are expanded in the gut, in the peripheral blood, synovial fluid and bone marrow of patients w…

2015

Background The aim of the study was to better characterise the immunological origin and the behaviour of interleukin (IL)-23-responsive innate lymphoid cells (ILCs) in the gut, synovial fluid (SF) and bone marrow (BM) of patients with ankylosing spondylitis (AS).Methods ILC1, ILC2 and ILC3 cells were determined and characterised by confocal microscopy and flow cytometry in ileal and BM biopsies, in peripheral blood (PB) and SF mononuclear cells obtained from patients with AS and controls. Mucosal vascular addressin cell adhesion molecule 1 (MADCAM-1), IL-7, IL-15 and aggregates of lymphoid tissue inducer cells (LTi) were evaluated by immunohistochemistry. The in vitro ability of epithelial …

AdultMalePathologymedicine.medical_specialtyAdolescentImmunologyHigh endothelial venulesImmunoglobulinsPeripheral blood mononuclear cellGeneral Biochemistry Genetics and Molecular BiologyInterleukin 22Young AdultMucoproteinsAnkylosing Spondylitis; Cytokines; InflammationRheumatologyBone MarrowIleumSynovial FluidAddressinImmunology and AllergyMedicineSynovial fluidHumansSpondylitis AnkylosingLymphocytesIntestinal MucosaCytokineAgedInterleukin-15InflammationMicroscopy ConfocalAnkylosing SpondylitibiologyNatural Cytotoxicity Triggering Receptor 2business.industryInterleukin-7InterleukinsInnate lymphoid cellInterleukin-17Middle AgedSettore MED/16 - Reumatologiamedicine.anatomical_structureLymphatic systemCase-Control Studiesbiology.proteinFemaleBone marrowbusinessCell Adhesion Molecules
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Increased expression of interleukin-32 in the inflamed ileum of ankylosing spondylitis patients

2012

Objective. To study the mRNA expression and protein tissue distribution of IL-32 in ileal biopsy specimens from patients with AS. Methods. Quantitative gene expression analysis, by real-time PCR, of IL-32, IL-1b, IL-10, TNF-a and IFN-g was performed on ileal biopsies of 15 AS and 15 Crohn’s disease (CD) patients and 10 healthy subjects (HSs). IL-32 tissue distribution was evaluated by immunohistochemistry. The effect of IL-32 on the production of IL-10 by intestinal epithelial cell lines was also evaluated. Results. In the ileal specimens of patients with AS and intestinal chronic inflammation, significant up-regulation of IL-32 at both the mRNA and protein levels was found as compared with…

AdultMalePathologymedicine.medical_specialtyAdolescentmedicine.medical_treatmentInterleukin-1betaInflammationInterferon-gammaYoung AdultCrohn DiseaseRheumatologyIleumBiopsyintestinal inflammationmedicineHumansSpondylitis AnkylosingPharmacology (medical)IleitisRNA MessengerCrohn's diseasemedicine.diagnostic_testTumor Necrosis Factor-alphabusiness.industryInterleukinsMacrophagesIL-32 ankylosing spondylitis IL-10 intestinal inflammationInterleukinEpithelial CellsIleitisMiddle AgedHCT116 Cellsmedicine.diseaseImmunity InnateInterleukin-10Settore MED/16 - ReumatologiaInterleukin 10Interleukin 32ankylosing spondylitiCytokineCase-Control StudiesImmunologyIL-32IL-10Femalemedicine.symptombusiness
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