Search results for "Insufficiency"

showing 10 items of 506 documents

Agenesis of the renal segment of inferior vena cava associated with venous stasis.

2010

Congenital anomalies of the inferior vena cava is an extremely rare vascular anomaly with controversial pathogenesis. Anomalies of the inferior vena cava (IVC) are present in 0.3% to 0.5% of otherwise healthy individual and in 0.6% to 2% of patients with other cardiovascular defects. The phenomenon of absence of the IVC has been described in a variety of ways such as absence, agenesis, anomalous, and interruption of a particular segment ([infra] hepatic, pre-renal, renal, or infrarenal) of the IVC. We describe a 42-year-old man with chronic venous insufficiency, without DVT, caused by congenital absence of renal segment of the IVC

AdultMaleSettore MED/09 - Medicina InternaCardiovascular AgentsVena Cava InferiorPhlebographySettore MED/11 - Malattie Dell'Apparato CardiovascolareCombined Modality TherapyExercise TherapyVenous InsufficiencyChronic DiseaseDiosminHumansAGENESIS OF THE RENAL SEGMENT OF INFERIOR VENA CAVA ASSOCIATED WITH VENOUS STASIS.Ultrasonography Doppler ColorTomography X-Ray ComputedStockings CompressionInternational angiology : a journal of the International Union of Angiology
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Alternatives to Endotracheal Intubation for Patients with Neuromuscular Diseases

2005

To evaluate the usefulness of continuous noninvasive mechanical ventilation and mechanical coughing aids to avoid endotracheal intubation and tracheostomy during episodes of acute respiratory failure in patients with neuromuscular disease.We conducted a prospective cohort study at the respiratory medicine ward of a university hospital to study the success rate of the use of continuous noninvasive mechanical ventilation and manually and mechanically (CoughAssist) assisted coughing to avert endotracheal intubation in 24 consecutive episodes of acute respiratory failure for 17 patients with neuromuscular disease. The noninvasive mechanical ventilation and coughing aids were used to reverse dec…

AdultMaleTime Factorsmedicine.medical_treatmentMEDLINEPhysical Therapy Sports Therapy and RehabilitationEndotracheal intubationIntermittent Positive-Pressure VentilationCohort StudiesConfidence IntervalsOdds RatiomedicineHumansProspective StudiesProspective cohort studyAgedMechanical ventilationAcademic Medical CentersRespiratory Distress Syndromebusiness.industryRehabilitationNeuromuscular DiseasesOdds ratioMiddle AgedConfidence intervalCoughAnesthesiaFemaleRespiratory InsufficiencybusinessCohort studyAmerican Journal of Physical Medicine & Rehabilitation
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Early auditory evoked potentials (EAEP) in vertebral basilar insufficiency

1979

Stimulation with a short tone pip elicits an acoustic nerve compound action potential (I) and different waves (II--VII) in the inital 10 ms. Seven waves have been studied in 40 control subjects and five waves in 12 patients with vertebral-basilar insufficiency. Abnormalities of the different waves were observed at levels such as cochlea and/or acoustic nerve, medulla, caudal pons, rostral pons, and midbrain. The recording of early auditory evoked potentials (EAEP) is a noninvasive method of confirming impairment of the auditory pathway caused by a reduced vascular supply of vertebral and basilar arteries.

AdultMaleVertebral basilar insufficiencyStimulationMidbrainVertebrobasilar InsufficiencymedicineHumansPharmacology (medical)Vertebrobasilar insufficiencyBiological PsychiatryMedullaCochleaAgedbusiness.industryGeneral MedicineAnatomyMiddle Agedmedicine.diseasePonsCompound muscle action potentialPsychiatry and Mental healthAcoustic StimulationEvoked Potentials AuditoryFemalesense organsbusinessArchiv f�r Psychiatrie und Nervenkrankheiten
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MFAP5 Loss-of-Function Mutations Underscore the Involvement of Matrix Alteration in the Pathogenesis of Familial Thoracic Aortic Aneurysms and Dissec…

2014

Thoracic aortic aneurysm and dissection (TAAD) is an autosomal-dominant disorder with major life-threatening complications. The disease displays great genetic heterogeneity with some forms allelic to Marfan and Loeys-Dietz syndrome, and an important number of cases still remain unexplained at the molecular level. Through whole-exome sequencing of affected members in a large TAAD-affected family, we identified the c.472CT (p.Arg158(∗)) nonsense mutation in MFAP5 encoding the extracellular matrix component MAGP-2. This protein interacts with elastin fibers and the microfibrillar network. Mutation screening of 403 additional probands identified an additional missense mutation of MFAP5 (c.62GT …

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesAdolescentExtracellular matrix componentNonsense mutationHaploinsufficiencyThoracic aortic aneurysmPathogenesisContractile ProteinsReportGeneticsmedicineHumansMissense mutationGenetics(clinical)ExomeChildGenetics (clinical)AgedGlycoproteinsAged 80 and overGeneticsAortic Aneurysm ThoracicbiologyGenetic heterogeneitySequence Analysis DNAFibroblastsMiddle Agedmedicine.diseasePedigree3. Good healthAortic DissectionAmino Acid SubstitutionCodon Nonsensebiology.proteinIntercellular Signaling Peptides and ProteinsFemaleHaploinsufficiencyElastinThe American Journal of Human Genetics
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Ten novel mutations found in Aniridia.

1998

Aniridia (AN) is a sight-threatening congenital ocular disorder characterized by iris hypoplasia, corneal pannus, foveal and optic nerve hypoplasia, cataract formation, and glaucoma. In two-thirds of the patients, AN is inherited in an autosomal dominant fashion with almost complete penetrance but variable expression. The remaining cases are sporadic. Aniridia has been shown to be associated with mutations in the PAX6 gene, located on chromosome 11p13, telomeric to the Wilms' tumor predisposition gene (WT1). This paper describes 14 mutations in the PAX6 gene in patients with AN. Among these 14 mutations, 10 have been unpublished until now. They result most probably in haploinsufficiency and…

AdultMalegenetic structuresAdolescentPAX6 Transcription FactorDNA Mutational AnalysisMolecular Sequence DataBiologyPolymerase Chain ReactionVariable ExpressionGeneticsmedicineHumansPaired Box Transcription FactorsAmino Acid SequenceChildEye ProteinsGeneAniridiaGenetics (clinical)Polymorphism Single-Stranded ConformationalGeneticsHomeodomain ProteinsOptic nerve hypoplasiaInfantMiddle Agedmedicine.diseasePenetranceeye diseasesDNA-Binding ProteinsRepressor ProteinsAniridiaChild PreschoolMutationHomeoboxFemalesense organsPAX6HaploinsufficiencyTranscription FactorsHuman mutation
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Analysis of Internal Jugular Vein Insufficiency—A Comparison of Two Ultrasound Methods

2006

Abstract Jugular venous valve insufficiency is a contributing factor to different pathologic conditions. For assessment of insufficiency, two ultrasound techniques have been developed, that are based on very different methodology (direct Doppler assessment versus air contrast ultrasound venography [ACUV]). This study was conducted to compare these two methods to improve comparability of existing studies and diagnostic accuracy in future studies. The function of 40 valves was determined in 20 individuals during a Valsalva maneuver. For direct Doppler assessment, valvular competence was assessed on basis of Doppler recordings, following recently established criteria. Valvular insufficiency in…

AdultMalemedicine.medical_specialtyAcoustics and Ultrasonicsmedicine.medical_treatmentBiophysicsVenographysymbols.namesakemedicineValsalva maneuverHumansRadiology Nuclear Medicine and imagingInternal jugular veinAgedMicrobubblesRadiological and Ultrasound Technologymedicine.diagnostic_testbusiness.industryUltrasoundUltrasonography DopplerPhlebographyMiddle Agedmedicine.diseaseValvular insufficiencyHypoplasiaVenous Insufficiencycardiovascular systemMicrobubblessymbolsFemaleRadiologyJugular VeinsbusinessDoppler effectUltrasound in Medicine & Biology
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Cardiac disease in patients with mucopolysaccharidosis: presentation, diagnosis and management

2011

The mucopolysaccharidoses (MPSs) are inherited lysosomal storage disorders caused by the absence of functional enzymes that contribute to the degradation of glycosaminoglycans (GAGs). The progressive systemic deposition of GAGs results in multi-organ system dysfunction that varies with the particular GAG deposited and the specific enzyme mutation(s) present. Cardiac involvement has been reported in all MPS syndromes and is a common and early feature, particularly for those with MPS I, II, and VI. Cardiac valve thickening, dysfunction (more severe for left-sided than for right-sided valves), and hypertrophy are commonly present; conduction abnormalities, coronary artery and other vascular in…

AdultMalemedicine.medical_specialtyAdolescentMucopolysaccharidosisClinical SciencesHeart Valve DiseasesReviewComorbidityCoronary Artery DiseaseDiseaseMuscle hypertrophyCoronary artery diseaseElectrocardiographyVentricular hypertrophyTachycardiaInternal medicineGeneticsmedicineHumansGenetics(clinical)Age of OnsetSinusChildPreschoolGenetics (clinical)GlycosaminoglycansGenetics & Hereditymedicine.diagnostic_testbusiness.industryMitral Valve InsufficiencyHypertrophyAortic Valve StenosisEnzyme replacement therapyMucopolysaccharidosesMiddle Agedmedicine.diseaseLeft VentricularCausalityTachycardia SinusEchocardiographyChild PreschoolAortic valve stenosisCardiologyHypertrophy Left VentricularFemalebusinessElectrocardiographyJournal of Inherited Metabolic Disease
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Traumatic lesion of the extracranial vertebral artery--a note-worthy potentially lethal injury.

1994

The autopsy findings from routine neuropathological investigations of the cervical spine after any history of trauma emphasized the vulnerability of the extracranial vertebral arteries. In 21 cases with trauma to the head and neck, normal autopsy procedures did not succeed in revealing an obvious cause of death. Traumatic lesions of the spinal cord such as contusion or neurorrhexis were seen in 10 cases. In 15 cases we observed different degrees and stages of traumatic lesions of the extracranial vertebral arteries. Sudden death due to acute brain stem ischemia might be considered as an explanation in some of these cases. Six case reports with traumatic vertebral artery (VA) lesions after s…

AdultMalemedicine.medical_specialtyAdolescentVertebral arteryPoison controlAutopsyHemorrhageWounds NonpenetratingSudden deathPathology and Forensic MedicineBrain IschemiaDeath SuddenFatal Outcomemedicine.arteryCause of DeathmedicineCraniocerebral TraumaHumansVertebrobasilar insufficiencySpinal Cord InjuriesVertebral ArteryAgedAged 80 and overbusiness.industryThrombosisMiddle AgedSpinal cordmedicine.diseaseThrombosisSurgerymedicine.anatomical_structureBlunt traumaCerebrovascular CirculationCervical VertebraeFemaleAutopsybusinessBrain StemInternational journal of legal medicine
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Intragenic KANSL1 mutations and chromosome 17q21.31 deletions: broadening the clinical spectrum and genotype-phenotype correlations in a large cohort…

2015

Background The 17q21.31 deletion syndrome phenotype can be caused by either chromosome deletions or point mutations in the KANSL1 gene. To date, about 60 subjects with chromosome deletion and 4 subjects with point mutation in KANSL1 have been reported. Prevalence of chromosome deletions compared with point mutations, genotype–phenotype correlations and phenotypic variability have yet to be fully clarified. Methods We report genotype–phenotype correlations in 27 novel subjects with 17q21.31 deletion and in 5 subjects with KANSL1 point mutation , 3 of whom were not previously reported. Results The prevalence of chromosome deletion and KANSL1 mutation was 83% and 17%, respectively. All patient…

AdultMalemedicine.medical_specialtyAdolescentgenotype-phenotype correlationsKoolen De Vries syndromeKANSL1 mutationHaploinsufficiencyBiologySettore MED/03 - GENETICA MEDICASeverity of Illness IndexCraniofacial AbnormalitiesYoung AdultSeizuresMolecular geneticsGeneticsmedicineHumansAbnormalities MultipleLanguage Development DisordersChildGenetics (clinical)Genetic Association StudiesGeneticsOptic nerve hypoplasiaFetal Growth RetardationPoint mutationMacrocephalyInfantNuclear ProteinsSyndromeclinical heterogeneitySmith–Magenis syndromemedicine.diseaseChild PreschoolSpeech delayFemalemedicine.symptomChromosome DeletionSmith-Magenis SyndromeHaploinsufficiencyChromosomes Human Pair 1717q21.31 deletion
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Prevalence and clinical characteristics of patients with true resistant hypertension in central and Eastern Europe

2013

Objective Scanty information is available on the clinical characteristics of resistant hypertension in Central and East European countries. The Blood Pressure (BP) control rate and CArdiovascular Risk profilE (BP-CARE) study allowed us to assess the prevalence and the main clinical features of resistant hypertension in this population. Design and method The study was carried out in 1312 treated hypertensive patients living in nine Central and East European countries. Results Four hundred and twenty-three patients had apparent resistant hypertension, of whom 168 had pseudo-resistant hypertension (noncompliant/white-coat) and 255 were true treatment-resistant hypertension patients (TRH). Clin…

AdultMalemedicine.medical_specialtyAmbulatory blood pressurePhysiologyCross-sectional studyPopulationDrug ResistanceRenal functionBlood PressureBody Mass IndexRisk FactorsInternal medicinePrevalenceInternal MedicinemedicineHumansEurope EasternObesityRenal InsufficiencyeducationAntihypertensive AgentsAgededucation.field_of_studybusiness.industryresistant hypertensionBlood Pressure DeterminationMiddle Agedmedicine.diseaseObesityCross-Sectional StudiesTreatment OutcomeEndocrinologyBlood pressureCardiovascular DiseasesHeart failureHypertensionFemaleCardiology and Cardiovascular MedicinebusinessBody mass indexWhite Coat HypertensionGlomerular Filtration RateJournal of Hypertension
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