Search results for "Insulin"

showing 10 items of 1360 documents

Glucose lowering and anti-atherogenic effects of incretin-based therapies: GLP-1 analogues and DPP-4-inhibitors

2009

Type 2 diabetes is a chronic, progressive disease with a multi-faceted pathophysiology. Beyond the known defects of insulin resistance and beta-cell insufficiency, derangement of incretin hormones normally produced from the gut wall in response to food intake play an important role. In recent years, the 'incretin-based' therapies (IBTs) have been developed to address hyperglycemia through either mimicking the action of the endogenous incretin glucagon-like polypeptide (GLP-1) (GLP-1 receptor agonists) or by inhibiting the activity of the enzyme that degrades GLP-1 (the dipeptyl peptidase-4 inhibitors).We reviewed available evidence on the glucose lowering and anti-atherogenic effects of IBT…

endocrine systemmedicine.medical_specialtyDipeptidyl Peptidase 410265 Clinic for Endocrinology and DiabetologyIncretin610 Medicine & healthType 2 diabetesCarbohydrate metabolismIncretinsInsulin resistancecardiovascular risk diabetes DPP-4 inhibitors GLP-1 analoguesGlucagon-Like Peptide 1Risk FactorsInternal medicineDiabetes mellitusmedicineAnimalsHumans2736 Pharmacology (medical)Pharmacology (medical)Dipeptidyl peptidase-4PharmacologyClinical Trials as TopicDipeptidyl-Peptidase IV Inhibitorsbusiness.industrydigestive oral and skin physiologyGeneral MedicineAtherosclerosismedicine.diseaseGlucose3004 PharmacologyEndocrinologyPostprandialDiabetes Mellitus Type 2aterosclerosibusinesshormones hormone substitutes and hormone antagonistsHormoneExpert Opinion on Investigational Drugs
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GLP-2 as Beneficial Factor in the Glucose Homeostasis in Mice Fed a High Fat Diet

2015

Glucagon like peptide-2 (GLP-2) is a gastrointestinal hormone released in response to dietary nutrients, which acts through a specific receptor, the GLP-2 receptor (GLP-2R). The physiological effects of GLP-2 are multiple, involving also the intestinal adaptation to high fat diet (HFD). In consideration of the well-known relationship between chronic HFD and impaired glucose metabolism, in the present study we examined if the blocking of the GLP-2 signaling by chronic treatment with the GLP-2R antagonist, GLP-2 (3-33), leads to functional consequences in the regulation of glucose metabolism in HFD-fed mice. Compared with animals fed standard diet (STD), mice at the 10th week of HFD showed hy…

endocrine systemmedicine.medical_specialtyPhysiologyPancreatic isletsInsulinmedicine.medical_treatmentdigestive oral and skin physiologyClinical BiochemistryCell BiologyBiologyCarbohydrate metabolismmedicine.diseaseGlucagonEndocrinologymedicine.anatomical_structureInsulin resistanceGlucose Metabolism DisorderInternal medicinemedicineGlucose homeostasisBeta cellhormones hormone substitutes and hormone antagonistsJournal of Cellular Physiology
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Acute selective ablation of rat insulin promoter-expressing (RIP HER ) neurons defines their orexigenic nature

2012

Rat insulin promoter (RIP)-expressing neurons in the hypothalamus control body weight and energy homeostasis. However, genetic approaches to study the role of these neurons have been limited by the fact that RIP expression is predominantly found in pancreatic β-cells, which impedes selective targeting of neurons. To define the function of hypothalamic RIP-expressing neurons, we set out to acutely and selectively eliminate them via diphtheria toxin-mediated ablation. Therefore, the diphtheria toxin receptor transgene was specifically expressed upon RIP-specific Cre recombination using a RIP-Cre line first described by Herrera (RIP HER -Cre) [Herrera PL (2000) Development 127:2317–2322]. Usi…

endocrine systemmedicine.medical_specialtyPituitary glandBiologyReal-Time Polymerase Chain ReactionEnergy homeostasisMiceArcuate nucleusOrexigenicInternal medicineWeight LossmedicineAnimalsInsulinPromoter Regions GeneticDorsomedial hypothalamic nucleusNeuronsDiphtheria toxinMultidisciplinarydigestive oral and skin physiologyArcuate Nucleus of HypothalamusFeeding BehaviorBiological SciencesGlucose Tolerance TestRatsEndocrinologymedicine.anatomical_structurenervous systemHypothalamusNucleushormones hormone substitutes and hormone antagonistsParaventricular Hypothalamic Nucleusmedicine.drugProceedings of the National Academy of Sciences
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Recovery of Endogenous β-Cell Function in Nonhuman Primates After Chemical Diabetes Induction and Islet Transplantation

2008

OBJECTIVE—To describe the ability of nonhuman primate endocrine pancreata to reestablish endogenous insulin production after chemical β-cell destruction. RESEARCH DESIGN AND METHODS—Eleven monkeys (Macaca fascicularis) were rendered diabetic with streptozotocin. Eight diabetic monkeys received intraportal porcine islet transplantation. RESULTS—Two monkeys transplanted after 75 days of type 1 diabetes showed recovery of endogenous C-peptide production a few weeks after transplantation, concomitant with graft failure. Histological analysis of the pancreas of these monkeys showed insulin-positive cells, single or in small aggregates, scattered in the pancreas and adjacent to ducts. Interesting…

endocrine systemmedicine.medical_specialtySwineEndocrinology Diabetes and Metabolismmedicine.medical_treatmentIslets of Langerhans TransplantationBiologyStreptozocinDiabetes Mellitus ExperimentalInsulin-Secreting CellsInternal medicineDiabetes mellitusInternal MedicinemedicineAnimalsInsulinProinsulingeographyType 1 diabetesgeography.geographical_feature_categoryInsulinHaplorhiniStreptozotocinmedicine.diseaseIsletTransplantationmedicine.anatomical_structureEndocrinologyIslet StudiesPancreasmedicine.drugDiabetes
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Endokrinologische Vorhersage der Therapieansprechbarkeit depressiver Patienten auf Lofepramin

1979

In a pilot study of 15 depressive patients of the neurotic and endogenous type we could show that some neuroendocrinological parameters are apt to predict the thymoleptic efficacy of lofepramine. These parameters, which were measured with a simple global stimulation test (insulin hypoglycaemia combined with injection of TRH and LHRH), were as follows: high basal blood glucose; high hypoglycaemic blood glucose; high decrease of blood glucose in comparison to the basal level; low basal TSH; low increase of HGH and low increase of cortisol after hypoglycemia. A synopsis of these parameters allowed a correct classification of 14 out of 15 patients according to therapy response and therapy resis…

endocrine systemmedicine.medical_specialtybusiness.industryStimulationEndogenyGeneral MedicineHypoglycemiamedicine.diseasePsychiatry and Mental healthInsulin hypoglycaemiaEndocrinologyBasal (medicine)Internal medicinemedicinePharmacology (medical)businesshormones hormone substitutes and hormone antagonistsBiological PsychiatryLofepramineHydrocortisonemedicine.drugEndocrine glandArchiv f�r Psychiatrie und Nervenkrankheiten
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Inhibition of TSH/IGF-1 Receptor Crosstalk by Teprotumumab as a Treatment Modality of Thyroid Eye Disease

2021

Abstract Context We previously presented evidence that TSH receptor (TSHR)-stimulating autoantibodies (TSAbs) bind to and activate TSHRs but do not bind to IGF1 receptors (IGF1Rs). Nevertheless, we showed that IGF1Rs were involved in thyroid eye disease (TED) pathogenesis because TSAbs activated crosstalk between TSHR and IGF1R. Teprotumumab, originally generated to inhibit IGF1 binding to IGF1R, was recently approved for the treatment of TED (Tepezza). Objective To investigate the role of TSHR/IGF1R crosstalk in teprotumumab treatment of TED. Design We used orbital fibroblasts from patients with TED (TEDOFs) and measured stimulated hyaluronan (HA) secretion as a measure of orbital fibrobla…

endocrine systemmedicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and MetabolismClinical BiochemistryThyrotropinStimulationContext (language use)Antibodies Monoclonal HumanizedBiochemistryReceptor IGF Type 1EndocrinologyInternal medicinemedicineHumansHyaluronic AcidOnline Only ArticlesReceptorFibroblastInsulin-like growth factor 1 receptorGene knockdownTeprotumumabChemistryBiochemistry (medical)Receptors Thyrotropineye diseasesGraves Ophthalmopathybody regionsCrosstalk (biology)Endocrinologymedicine.anatomical_structuremedicine.drugThe Journal of Clinical Endocrinology & Metabolism
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Ovarian size and blood flow in women with polycystic ovary syndrome (PCOS) and their correlations with some endocrine parameters

2005

Objective: To determine how common polycystic ovarian morphology may be in women given the clinical diagnosis of polycystic ovary syndrome (PCOS) based on chronic anovulation and hyperandrogenism and whether certain hormonal factors correlate with ovarian morphology and blood flow. Design: Prospective study. Setting: Two academic endocrinology centers in Italy. Patient(s): Three hundred twenty-six women with PCOS and 50 age-matched and weight-matched ovulatory women. Intervention(s): Ultrasound assessment of ovarian morphology in patients and controls and ovarian blood flow and fasting hormone levels in a subset of 50 patients and matched controls. Main Outcome Measure(s): Ovarian morpholog…

endocrine systemmedicine.medical_specialtyendocrine system diseasesmedicine.drug_classPolycystic ovary syndrome ovarian size blood flow insulin testosterone inhibin Bmedicine.medical_treatmentHemodynamicsOvaryBiologyStatistics NonparametricAnovulationInsulin resistanceEndocrine GlandsInternal medicinemedicineHumansProspective StudiesGonadal Steroid Hormonesendocrine parametersAnalysis of VarianceInsulinOvaryHyperandrogenismObstetrics and GynecologyOrgan Sizemedicine.diseasePolycystic ovaryfemale genital diseases and pregnancy complicationsmedicine.anatomical_structureEndocrinologyReproductive MedicineRegional Blood FlowEstrogenpolycystic ovary syndromeFemaleHyperandrogenismpolycystic ovary syndrome; endocrine parametersAnovulation
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Effects of combined hormone replacement therapy or its effective agents on the IGF-1 pathway in skeletal muscle.

2010

Objectives To investigate the effects of combined hormone replacement therapy (HRT) and its effective agents on the IGF-1 signaling pathway. Design and methods To examine the effects of HRT on skeletal muscle in vivo, we utilized pre- and post-intervention samples from a randomized double blinded trial with 50–57-year-old women. The intervention included the year-long use of either HRT preparation (2 mg 17β-estradiol, E2; 1 mg norethisterone acetate, NETA, n = 10) or placebo (CO, n = 9). Microarray technology and quantitative PCR (qPCR) were used to study the expression of insulin-like growth factor I (IGF-1) and its splice variants as well as IGF-1 receptor, Akt1, mTOR, FOXO1, FOXO3, atrog…

estradioliTranscription GeneticEndocrinology Diabetes and MetabolismMuscle Fibers SkeletalEstrogen receptorpostmenopausal womenMuscle ProteinsFOXO1Receptor IGF Type 10302 clinical medicineEndocrinologyProtein IsoformsTestosteroneInsulin-Like Growth Factor IReceptorRandomized Controlled Trials as Topic0303 health sciencesEstradiolMyogenesisForkhead Box Protein O1TOR Serine-Threonine KinasesEstrogen Replacement TherapyForkhead Box Protein O3Forkhead Transcription FactorsMiddle Agedmedicine.anatomical_structureReceptors EstrogenReceptors AndrogenFemalemedicine.medical_specialtynorethisterone acetate030209 endocrinology & metabolismBiologypostmenopausaalinen nainen03 medical and health sciencesInternal medicinemedicineHumansnoretisteroniasetaattiluurankolihasskeletal muscleMuscle SkeletalProtein kinase BPI3K/AKT/mTOR pathway030304 developmental biologyhormonikorvaushoitoSKP Cullin F-Box Protein LigasesSkeletal muscleAndrogen receptorNorethindrone AcetateEndocrinologyHormone replacement therapyIGF-1 signalointiNorethindroneIGF-1 signalingProto-Oncogene Proteins c-aktGrowth hormoneIGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society
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Sobrecarga oral con grasa insaturada y estrés oxidativo en sujetos con obesidad abdominal

2015

Introducción: la enfermedad cardiovascular (ECV) constituye la primera causa de morbimortalidad en los países desarrollados. La base patogénica de la ECV es la arteriosclerosis. La obesidad abdominal (OA) es una de las principales causas para el desarrollo de alteraciones metabólicas como insulinorresistencia (IR), DM-2, HTA, y dislipemia mixta y por tanto del riesgo de desarrollar arteriosclerosis y ECV. La OA y la IR generan estrés oxidativo, mecanismo implicado en el desarrollo de la arteriosclerosis. Los sujetos con OA e IR presenta un aumento de estrés oxidativo tanto en estado basal como postprandial. Desde varias décadas se considera la lipemia postprandial (LPP) como un factor de ri…

estrés oxidativo:CIENCIAS MÉDICAS ::Medicina interna::Endocrinología [UNESCO]lipemia postprandialgrasa insaturadaobesidad abdominalenfermedad cardiovascularUNESCO::CIENCIAS MÉDICAS ::Medicina interna::Endocrinologíasobrecarga oral grasainsulinorresistencia
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The clinical impact of an extra virgin olive oil enriched mediterranean diet on metabolic syndrome: Lights and shadows of a nutraceutical approach

2022

For years it has been established that the only truly effective treatment of metabolic syndrome (MS) is lifestyle modification to prevent its cardiovascular (e.g., coronary artery disease and atherosclerosis), metabolic (e.g., diabetes mellitus), and hepatic (e.g., steatosis and non-alcoholic steatohepatitis) complications. The focal points of this approach are to increase physical activity and intake of a diet characterized by high quantities of fruits, vegetables, grains, fish, and low-fat dairy products, the so called mediterranean diet (MD); however, the added value of MD is the presence of extra virgin olive oil (EVOO), a healthy food with a high content of monounsaturated fatty acids,…

functional foodSettore MED/09 - Medicina InternaNutrition and Dieteticsnutraceuticals.cardiovascular diseaseinsulin resistanceEndocrinology Diabetes and Metabolismextra virgin olive oil (EVOO)metabolic syndromeFood ScienceFrontiers in Nutrition
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