Search results for "Insulin"

showing 10 items of 1360 documents

Insulin resistance in patients with familial combined hyperlipidemia and coronary artery disease.

1997

The minimum model modified by the administration of insulin provides an objective and relatively easily measured index of peripheral sensitivity to insulin which was significantly lower (p <0.02) in familial combined hyperlipidemia (FCH) with ischemic heart disease (IHD) than in FCH without IHD and in control subjects (1.2 +/- 0.6, 1.9 +/- 1.0, 2.9 +/- 1.2 x 10(-4) mU/L/ min, respectively). In patients with FCH, insulin resistance explains, at least in part, their metabolic alterations (hypertension, abnormal glucose tolerance, hyperinsulinemia) and elevated IHD.

Blood GlucoseMalemedicine.medical_specialtymedicine.medical_treatmentLipoproteinsHyperlipidemia Familial CombinedCoronary DiseaseDiseaseCoronary artery diseaseInsulin resistanceRisk FactorsInternal medicineHyperinsulinemiaMedicineHumansInsulinIn patientcardiovascular diseasesbusiness.industryInsulinGlucose Tolerance TestMiddle Agedmedicine.diseasePeripheralPedigreeEndocrinologyPhenotypeCardiologyInsulin ResistanceCardiology and Cardiovascular MedicinebusinessLipoproteinThe American journal of cardiology
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Anti-diabetic effects of mildronate alone or in combination with metformin in obese Zucker rats

2010

Abstract Mildronate is a cardioprotective drug, the mechanism of action of which is based on the regulation of l -carnitine concentration. We studied the metabolic effects of treatment with mildronate, metformin and a combination of the two in the Zucker rat model of obesity and impaired glucose tolerance. Zucker rats were p.o. treated daily with mildronate (200 mg/kg), metformin (300 mg/kg), and a combination of both drugs for 4 weeks. Weight gain and plasma metabolites reflecting glucose metabolism were measured. The expression of peroxisome proliferator-activated receptor (PPAR)-α and PPAR-γ and target genes was measured in rat heart and liver tissues. Each treatment decreased the blood …

Blood GlucoseMalemedicine.medical_specialtymedicine.medical_treatmentPeroxisome proliferator-activated receptorCarbohydrate metabolismImpaired glucose toleranceEatingInternal medicinemedicineAnimalsHypoglycemic AgentsInsulinPPAR alphaLactic AcidObesityRNA MessengerCarnitineCell NucleusPharmacologychemistry.chemical_classificationbusiness.industryMyocardiumInsulinBody WeightLipid Metabolismmedicine.diseaseMetforminRatsRats ZuckerMetforminPPAR gammaDrug CombinationsEndocrinologyGene Expression RegulationMechanism of actionchemistryCarnitine biosynthesismedicine.symptombusinessMethylhydrazinesmedicine.drugEuropean Journal of Pharmacology
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Effects of Two Months of Very Low Carbohydrate Ketogenic Diet on Body Composition, Muscle Strength, Muscle Area, and Blood Parameters in Competitive …

2021

Background: Ketogenic diet (KD) is a nutritional approach that restricts daily carbohydrates, replacing most of the reduced energy with fat, while maintaining an adequate quantity of protein. Despite the widespread use of KD in weight loss in athletes, there are still many concerns about its use in sports requiring muscle mass accrual. Thus, the present study sought to investigate the influence of a KD in competitive natural body builders. Methods: Nineteen volunteers (27.4 &plusmn

Blood GlucoseMalemedicine.medical_treatmentDiet Carbohydrate-Restricted0302 clinical medicineNeurotrophic factorsWeight lossInsulinTestosteroneHealth Ketogenic diet Resistance training StrengthInsulin-Like Growth Factor ITestosteroneNutrition and DieteticsMuscleshealthAdipose Tissueketogenic dietBody CompositionCytokinesmedicine.symptomDiet Ketogenicstrengthlcsh:Nutrition. Foods and food supplySettore M-EDF/01 - Metodi E Didattiche Delle Attivita' MotorieAdultmedicine.medical_specialtyAdolescentCarbohydrates030209 endocrinology & metabolismlcsh:TX341-641ArticleProinflammatory cytokineHealth; Ketogenic diet; Resistance training; Strength03 medical and health sciencesYoung AdultInternal medicineWeight LossmedicineHumansMuscle StrengthTriglyceridesbusiness.industryInsulinBrain-Derived Neurotrophic Factor030229 sport sciencesEndocrinologyGlucoseAthletesDiet WesternBasal metabolic rateLean body massBasal Metabolismresistance trainingbusinessFood ScienceKetogenic dietNutrients
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Phenotyping of type 2 diabetes mellitus at onset on the basis of fasting incretin tone: Results of a two-step cluster analysis.

2015

Aims/Introduction According to some authors, in type 2 diabetes there is a reduced postprandial action of glucagon-like peptide-1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP). However, little is known about the role of fasting incretins in glucose homeostasis. Our aim was to evaluate, through a two-step cluster analysis, the possibility of phenotyping patients with type 2 diabetes at onset on the basis of fasting GLP-1, GIP and ghrelin. Materials and Methods A total of 96 patients with type 2 diabetes within 6 months of onset (mean age 62.40 ± 6.36 years) were cross-sectionally studied. Clinical, anthropometric and metabolic parameters were evaluated. At fasting the follow…

Blood GlucoseMalemedicine.medical_treatmentEndocrinology Diabetes and MetabolismSettore MED/13 - Endocrinologiachemistry.chemical_compound0302 clinical medicineGlucagon-Like Peptide 1Glucose homeostasisCluster AnalysisHomeostasis030212 general & internal medicineGeneral MedicineArticlesMiddle AgedGlucagon-like peptide-1GhrelinGlucagon‐like peptide‐1PhenotypeClinical Science and CareFemaleOriginal Articlehormones hormone substitutes and hormone antagonistsGlucagon-like peptide-1medicine.medical_specialtyGlucose-dependent insulinotropic polypeptideIncretin030209 endocrinology & metabolismGastric Inhibitory PolypeptideGhrelin; Glucagon-like peptide-1; Glucose-dependent insulinotropic polypeptide; Endocrinology Diabetes and Metabolism; Internal MedicineIncretins03 medical and health sciencesInsulin resistanceGlucose‐dependent insulinotropic polypeptideInternal medicineDiabetes mellitusmedicineInternal MedicineHumansAgedAdiponectinbusiness.industryInsulinmedicine.diseaseEndocrinologyGlucosechemistryDiabetes Mellitus Type 2Glycated hemoglobinInsulin ResistancebusinessJournal of diabetes investigation
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The sodium-glucose co-transporter 2 inhibitor empagliflozin improves diabetes-induced vascular dysfunction in the streptozotocin diabetes rat model b…

2014

Objective In diabetes, vascular dysfunction is characterized by impaired endothelial function due to increased oxidative stress. Empagliflozin, as a selective sodium-glucose co-transporter 2 inhibitor (SGLT2i), offers a novel approach for the treatment of type 2 diabetes by enhancing urinary glucose excretion. The aim of the present study was to test whether treatment with empagliflozin improves endothelial dysfunction in type I diabetic rats via reduction of glucotoxicity and associated vascular oxidative stress. Methods Type I diabetes in Wistar rats was induced by an intravenous injection of streptozotocin (60 mg/kg). One week after injection empagliflozin (10 and 30 mg/kg/d) was adminis…

Blood GlucoseMalemedicine.medical_treatmentReceptor for Advanced Glycation End Productslcsh:MedicineGene ExpressionType 2 diabetesmedicine.disease_causeVascular MedicineGlucosidesMedicine and Health SciencesMedicineInsulinEndothelial dysfunctionReceptors Immunologiclcsh:ScienceMultidisciplinaryType 1 DiabetesCytokinesInflammation Mediatorsmedicine.drugSignal TransductionResearch Articlemedicine.medical_specialtyCardiologyBlood sugarStreptozocinCardiovascular PharmacologyDiabetes Mellitus ExperimentalDiabetes ComplicationsInternal medicineDiabetes mellitusEmpagliflozinDiabetes MellitusAnimalsRNA MessengerVascular DiseasesBenzhydryl CompoundsSodium-Glucose Transporter 2 InhibitorsPharmacologybusiness.industryInsulinlcsh:RHemodynamicsStreptozotocinmedicine.diseaseRatsOxidative StressEndocrinologyGlucoseMetabolic Disorderslcsh:QbusinessOxidative stressDiabetic AngiopathiesPloS one
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Effects of red wine polyphenols and alcohol on glucose metabolism and the lipid profile: a randomized clinical trial.

2013

Summary Background & aims Epidemiological data suggest that moderate red wine consumption reduces cardiovascular mortality and the incidence of diabetes. However, whether these effects are due to ethanol or to non-alcoholic components of red wine still remains unknown. The aim of the present study was to compare the effects of moderate consumption of red wine, dealcoholized red wine, and gin on glucose metabolism and the lipid profile. Methods Sixty-seven men at high cardiovascular risk were randomized in a crossover trial. After a run-in period, all received each of red wine (30 g alcohol/d), the equivalent amount of dealcoholized red wine, and gin (30 g alcohol/d) for 4 week periods, in a…

Blood GlucoseMalemedicine.medical_treatmentWineCarbohydrate metabolismCritical Care and Intensive Care Medicinechemistry.chemical_compoundInsulin resistanceFolic AcidAdipokinesRisk FactorsDiabetes mellitusmedicineHomeostasisHumansInsulinFood scienceHomocysteineTriglyceridesAgedWineNutrition and DieteticsCross-Over Studiesmedicine.diagnostic_testEthanolCholesterolbusiness.industryInsulindigestive oral and skin physiologyfood and beveragesPolyphenolsFastingMiddle Agedmedicine.diseaseDietVitamin B 12ApolipoproteinsCholesterolGlucosechemistryCardiovascular DiseasesInsulin ResistancebusinessLipid profileLipoproteinClinical nutrition (Edinburgh, Scotland)
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Obesity interferes with the orosensory detection of long-chain fatty acids in humans

2014

Background: The association between the orosensory detection of lipids, preference for fatty foods, and body mass index (BMI; in kg/m 2 ) is controversial in humans. Objective: We explored the oral lipid-sensing system and the orosensory-induced autonomic reflex system in lean and obese subjects. Design: Lean (BMI: 19 to ,25; n = 30) and obese (BMI .30; n = 29) age-matched men were enrolled. Their oral threshold sensitivity to linoleic acid (LA) was determined by using a 3-alternative forced-choice ascending procedure, and their eating habits were established by the analysis of 4 consecutive 24-h food-consumption diaries. The effect of brief oral lipid stimulations on plasma triglyceride [(…

Blood GlucoseMaleobesityChemical PhenomenaMESH : Insulin[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionCholesterol VLDLMedicine (miscellaneous)MESH: Food HabitsStimulationMESH: Energy IntakeMESH : TasteMESH : FastingBody Mass Indexchemistry.chemical_compoundMESH: Cholesterol VLDLMESH: Diet RecordsInsulinMESH: ObesityMESH : Physicochemical PhenomenaCompletely randomized designMESH : Food HabitsCross-Over StudiesMESH: Middle AgedMESH : Food PreferencesNutrition and DieteticsFastingMiddle AgedDiet RecordsMESH : TriglyceridesSensory ThresholdsTasteMESH : ObesityMESH: Triglyceridesmedicine.medical_specialtyMESH : MaleLinoleic acidMESH: FastingMESH: InsulinMESH: Cross-Over StudiesMESH: Body Mass IndexLinoleic AcidMESH : Diet RecordsFood PreferencesBMIMESH : Linoleic AcidInternal medicineAutonomic reflexmedicineHumansMESH : Middle AgedhumanMESH: Food PreferencesTriglyceridesMESH : Cholesterol VLDLMESH: Linoleic AcidMESH: HumansCholesterolbusiness.industryMESH : HumansMESH : Energy IntakeFeeding BehaviorMESH : Blood GlucoseMESH : Cross-Over Studiesmedicine.diseaseCrossover studyObesityMESH: Malefat taste[SDV.AEN] Life Sciences [q-bio]/Food and NutritionMESH: Physicochemical PhenomenaMESH : Body Mass IndexEndocrinologychemistryMESH: Blood GlucoseMESH: Tastefood preferenceMESH: Sensory ThresholdsEnergy Intakebusiness[SDV.AEN]Life Sciences [q-bio]/Food and NutritionBody mass indexMESH : Sensory ThresholdsThe American Journal of Clinical Nutrition
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The neuropeptide 26RFa (QRFP) is a key regulator of glucose homeostasis and its activity is markedly altered in obese/hyperglycemic mice

2019

International audience; Recent studies have shown that the hypothalamic neuropeptide 26RFa regulates glucose homeostasis by acting as an incretin, and increasing insulin sensitivity. In this study, we further characterized the role of the 26RFa/GPR103 peptidergic system in the global regulation of glucose homeostasis using a 26RFa receptor antagonist, and also assessed whether a dysfunction of the 26RFa/GPR103 system occurs in obese hyperglycemic mice. Firstly, we demonstrate that administration of the GPR103 antagonist reduces the global glucose-induced incretin effect and insulin sensitivity whereas, conversely, administration of exogenous 26RFa attenuates glucose-induced hyperglycemia. U…

Blood GlucoseMaleobesityPhysiologyEndocrinology Diabetes and Metabolism[SDV]Life Sciences [q-bio]RegulatorMice Obese26RFaMice0302 clinical medicineGlucose homeostasisHomeostasisInsulinglucoseComputingMilieux_MISCELLANEOUSCells Cultured0303 health sciencesdiabetesChemistryincretin[SDV] Life Sciences [q-bio]obésitéAlimentation et NutritionCarbohydrate Metabolismdiabètemedicine.medical_specialtyNeuropeptideIncretin030209 endocrinology & metabolism03 medical and health sciencesPhysiology (medical)Diabetes mellitusInternal medicinemedicineAnimalsHumansglucose homeostasisFood and Nutrition030304 developmental biologyhoméostasieNeuropeptidesIncreasing insulinQRFPNeurosciencesGlucose Tolerance Testmedicine.diseaseMice Inbred C57BLEndocrinologyHyperglycemiaNeurons and Cognitionincretin;glucose homeostasis;26RFa;diabetes;obesity
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Cardiorespiratory Fitness, Physical Activity, and Insulin Resistance in Children

2020

Purpose Few studies have investigated the independent and joint associations of cardiorespiratory fitness (CRF) and body fat percentage (BF%) with insulin resistance in children. We investigated the independent and combined associations of CRF and BF% with fasting glycemia and insulin resistance and their interactions with physical activity (PA) and sedentary time among 452 children age 6 to 8 yr. Methods We assessed CRF with a maximal cycle ergometer exercise test and used allometrically scaled maximal power output (Wmax) for lean body mass (LM) and body mass (BM) as measures of CRF. The BF% and LM were measured by dual-energy X-ray absorptiometry, fasting glycemia by fasting plasma glucos…

Blood GlucoseMaleobesitymedicine.medical_treatmentliikuntaBody fat percentage0302 clinical medicineHeart RateBody Fat DistributionHomeostasisInsulinOrthopedics and Sports MedicineChildyouthdiabetesexerciseCardiorespiratory FitnessHomeostatic model assessmentFemaleperformancefyysinen aktiivisuusinsulinmedicine.medical_specialtylapset (ikäryhmät)Physical Therapy Sports Therapy and RehabilitationArticle03 medical and health sciencesInsulin resistanceInternal medicineDiabetes mellitusmedicineinsulin sensitivityHumansExercisekehonkoostumusbusiness.industryInsulinCardiometabolic Risk FactorsCardiorespiratory fitness030229 sport sciencesinsuliiniresistenssimedicine.diseaseObesityEndocrinologyLean body massExercise TestInsulin ResistanceSedentary BehaviorbusinessEnergy Metabolism
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Impact on Glucose Homeostasis: Is Food Biofortified with Molybdenum a Workable Solution? A Two-Arm Study

2022

Diabetes is expected to increase up to 700 million people worldwide with type 2 diabetes being the most frequent. The use of nutritional interventions is one of the most natural approaches for managing the disease. Minerals are of paramount importance in order to preserve and obtain good health and among them molybdenum is an essential component. There are no studies about the consumption of biofortified food with molybdenum on glucose homeostasis but recent studies in humans suggest that molybdenum could exert hypoglycemic effects. The present study aims to assess if consumption of lettuce biofortified with molybdenum influences glucose homeostasis and whether the effects would be due to c…

Blood GlucoseMolybdenumGIPMineralsNutrition and DieteticsPYYdigestive oral and skin physiologyHuman healthGastric Inhibitory PolypeptideLettucebiofortification; lettuce; human heath; minerals; gut peptides; GIP; PYYGlucoseDiabetes Mellitus Type 2Glucagon-Like Peptide 1Food FortifiedGlucagon-Like Peptide 2Gut peptidesHomeostasisHumansInsulinPeptide YYInsulin ResistanceBiofortificationhormones hormone substitutes and hormone antagonistsFood ScienceNutrients; Volume 14; Issue 7; Pages: 1351
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