Search results for "Insulin"

showing 10 items of 1360 documents

Insulin acts at different CNS sites to decrease acute sucrose intake and sucrose self-administration in rats.

2008

Findings from our laboratory and others have demonstrated that the hormone insulin has chronic effects within the CNS to regulate energy homeostasis and to decrease brain reward function. In this study, we compared the acute action of insulin to decrease intake of a palatable food in two different behavioral tasks—progressive ratios sucrose self-administration and mu opioid-stimulated sucrose feeding—when administered into several insulin-receptive sites of the CNS. We tested insulin efficacy within the medial hypothalamic arcuate (ARC) and paraventricular (PVN) nuclei, the nucleus accumbens, and the ventral tegmental area. Administration of insulin at a dose that has no chronic effect on …

Malemedicine.medical_specialtySucrosePhysiologymedicine.medical_treatmentReceptors Opioid muSelf AdministrationBiologyNucleus accumbensNucleus Accumbenschemistry.chemical_compoundRewardPhysiology (medical)Internal medicinemedicineAnimalsInsulinOpioid peptidePancreatic hormoneMotivationBehavior AnimalAppetite RegulationInsulinVentral Tegmental AreaArcuate Nucleus of HypothalamusBrainEnkephalin Ala(2)-MePhe(4)-Gly(5)-RatsVentral tegmental areaDAMGOmedicine.anatomical_structureEndocrinologychemistryCall for PapersBrain stimulation rewardSelf-administrationParaventricular Hypothalamic NucleusAmerican journal of physiology. Regulatory, integrative and comparative physiology
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Insulin resistance as common molecular denominator linking obesity to Alzheimer’s disease

2015

Alzheimer’s disease (AD) is an aging-related multi-factorial disorder to which metabolic factors contribute at what has canonically been considered a centrally mediated process. Although the exact underlying mechanisms are still unknown, obesity is recognized as a risk factor for AD and the condition of insulin resistance seems to be the link between the two pathologies. Using mice with high fat diet (HFD) obesity we dissected the molecular mechanisms shared by the two disorders. Brains of HFD fed mice showed elevated levels of APP and Aβ 40 /Aβ 42 together with BACE, GSK3β and Tau proteins involved in APP processing and Aβ accumulation. Immunofluorescence, Thioflavin T staining experiments…

Malemedicine.medical_specialtyTime FactorsAdipokineAmyloidogenic ProteinsInflammationBiologyDiet High-Fatmedicine.disease_causeAdipokines Alzheimer’s disease gene expression inflammation insulin resistance mitochondrial dysfunction obesity.Settore BIO/09 - FisiologiaGlycogen Synthase Kinase 3MiceInsulin resistanceAlzheimer DiseaseInternal medicinemedicineAnimalsInsulinObesityReceptorGSK3BGlycogen Synthase Kinase 3 betaSettore BIO/16 - Anatomia UmanaNeurodegenerationBrainmedicine.diseaseReceptor InsulinMice Inbred C57BLDisease Models AnimalOxidative StressInsulin receptorEndocrinologyGene Expression RegulationNeurologyCase-Control Studiesbiology.proteinCytokinesNeurology (clinical)Amyloid Precursor Protein SecretasesInsulin Resistancemedicine.symptomOxidative stressSignal Transduction
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Visual evoked potentials in insulin-dependent diabetics

1985

The latency of pattern-reversal VEPs was studied in type I insulin-dependent diabetics without retinal and extraocular involvement. One hundred eyes of 50 diabetics (mean age 19.8 +/- 7.2 years) formed the study group; the duration of the disease ranged between 1 month and 15 years. The latencies of VEPs were progressively delayed in relation to the duration of the disease, becoming more and more evident and stabilizing after about 6 years from the onset of diabetes. The VEP alterations probably indicate alteration of membrane balance or demyelinization.

Malemedicine.medical_specialtyTime FactorsAdolescentgenetic structuresEndocrinology Diabetes and MetabolismVisual evoked potentialsAudiologychemistry.chemical_compoundEndocrinologyDiabetes mellitusOphthalmologyElectroretinographyInternal MedicinemedicineHumansChildBalance (ability)business.industryMean ageRetinalGeneral Medicinemedicine.diseaseDiabetes Mellitus Type 1chemistryEvoked Potentials VisualFemalebusinessInsulin dependentActa Diabetologica Latina
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Janus kinase (JAK) 2 V617F mutation as the cause of primary thrombocythemia in acromegaly with severe visceromegaly and divergence between growth hor…

2012

OBJECTIVE: An increased prevalence of hematological abnormalities is reported in acromegaly, but to date no reports about the presence of the Janus Kinase (JAK) 2 mutation in acromegalic patients have been described. DESIGN: We report the complex clinical presentation of the unique case, never described, of acromegaly due to GH-secreting pituitary adenoma associated with JAK2 V617F mutation. RESULTS: The patient shows primary thrombocythemia and myelofibrosis, due to JAK2 V617F mutation, severe visceromegaly and a peculiar clinical course of the disease characterized by discrepant values of GH and IGF-1 during somatostatin analog (SA) treatment despite a significant reduction in pituitary a…

Malemedicine.medical_specialtyTime FactorsEndocrinology Diabetes and MetabolismGrowth hormone receptorAcromegaly HGH IGF-1GastroenterologySettore MED/13 - EndocrinologiaCohort StudiesEndocrinologyPituitary adenomahemic and lymphatic diseasesInternal medicineAcromegalymedicineAnimalsHumansPituitary NeoplasmsInsulin-Like Growth Factor IRadiometryMyelofibrosisAgedJanus kinase 2biologyHuman Growth HormonePlatelet Countbusiness.industryJanus Kinase 2medicine.diseaseAcromegaly Growth hormone Insulin-like growth factor-1 Janus kinase 2 Growth hormone receptorEndocrinologyAcromegalyMutationPegvisomantbiology.proteinJanus kinasebusinessVisceromegalyFollow-Up StudiesThrombocythemia Essentialmedicine.drug
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Human leukocyte/endothelial cell interactions and mitochondrial dysfunction in type 2 diabetic patients and their association with silent myocardial …

2013

OBJECTIVE Diabetes is associated with oxidative stress and increased mortality, but a possible correlation between leukocyte-endothelium interactions, oxidative stress, and silent myocardial ischemia (SMI) is yet to be confirmed. RESEARCH DESIGN AND METHODS Mitochondrial dysfunction and interactions between leukocytes and human umbilical vein endothelial cells were evaluated in 200 type 2 diabetic patients (25 with SMI) and 60 body composition– and age-matched control subjects. A possible correlation between these parameters and the onset of SMI was explored, and anthropometric and metabolic parameters were also analyzed. RESULTS Waist, levels of triglycerides, proinflammatory cytokines (i…

Malemedicine.medical_specialtyUmbilical VeinsCardiovascular and Metabolic RiskEndocrinology Diabetes and Metabolismmedicine.medical_treatmentMyocardial IschemiaVascular Cell Adhesion Molecule-1Type 2 diabetesmedicine.disease_causeProinflammatory cytokineInsulin resistanceDiabetes mellitusInternal medicineInternal MedicinemedicineLeukocytesHumansOriginal ResearchAdvanced and Specialized NursingbiologyGlutathione Disulfidebusiness.industryInsulinC-reactive proteinEndothelial CellsMiddle Agedmedicine.diseaseOxidative StressEndocrinologyC-Reactive ProteinDiabetes Mellitus Type 2biology.proteinFemalebusinessReactive Oxygen SpeciesHomeostasisOxidative stress
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Testosterone Levels in Males with Type 2 Diabetes and Their Relationship with Cardiovascular Risk Factors and Cardiovascular Disease

2010

ABSTRACT Introduction One of the factors involved in type 2 diabetes in males is a reduction in levels of testosterone, which has been shown to predict resistance to insulin and the development of cardiovascular diseases. Aim To assess the levels of testosterone in patients with type 2 diabetes and to evaluate their relationship with cardiovascular risk factors, peripheral arterial disease (PAD) and silent myocardial ischemia (SMI). Methods Total testosterone and sex hormone binding globulin were measured and free and bioavailable testosterones were calculated using Vermeulen's formula. Levels of total testosterone ≥12 nmol/L or free testosterone >225 pmol/L were considered normal. PAD was …

Malemedicine.medical_specialtyUrologyEndocrinology Diabetes and MetabolismMyocardial IschemiaArterial Occlusive DiseasesType 2 diabetesEndocrinologySex hormone-binding globulinInsulin resistanceRisk FactorsDiabetes mellitusInternal medicineHumansMedicineTestosteroneAgedFramingham Risk Scorebiologymedicine.diagnostic_testbusiness.industryTestosterone (patch)Middle Agedmedicine.diseasePsychiatry and Mental healthEndocrinologyDiabetes Mellitus Type 2Reproductive MedicineCardiovascular Diseasesbiology.proteinInsulin ResistanceMetabolic syndromebusinessLipid profileThe Journal of Sexual Medicine
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Relationship between erectile dysfunction and silent myocardial ischemia in type 2 diabetic patients with no known macrovascular complications.

2011

ABSTRACT Introduction Erectile dysfunction (ED) is associated with cardiovascular disease (CVD) because it is highly prevalent among those with cardiovascular risk factors (CVRFs). Moreover, it precedes the development of CVD and is considered a subrogate marker of subclinical CVD. Aim The aim of this study was to evaluate the presence of ED among patients with type 2 diabetes (DM2) without macroangiopathy, and to assess the association between ED and other CVRFs, chronic diabetes complications, silent myocardial ischemia (SMI), and peripheral arterial disease (PAD). Methods One hundred fifty‐four male patients with DM2 and without clinical evidence of CVD were included in the study. The pr…

Malemedicine.medical_specialtyUrologyEndocrinology Diabetes and MetabolismMyocardial IschemiaType 2 diabetesAsymptomaticNephropathyPeripheral Arterial DiseaseEndocrinologyInsulin resistanceErectile DysfunctionDiabetes mellitusInternal medicinemedicinePrevalenceHumansAnkle Brachial Indexcardiovascular diseasesmedicine.diagnostic_testbusiness.industryAge FactorsMiddle Agedmedicine.diseaseSurgeryPsychiatry and Mental healthErectile dysfunctionBlood pressureDiabetes Mellitus Type 1Reproductive MedicineHypertensionCardiologymedicine.symptomLipid profilebusinessThe journal of sexual medicine
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Treatment Response in Patients with Stones, and Low Urinary pH and Hypocitraturia Stratified by Body Mass Index.

2015

Obesity has been shown to be a risk factor for kidney stone formation. Obesity leads to insulin resistance which subsequently leads to low urinary pH. Low urinary pH is typically treated with potassium citrate. We determined if the response to potassium citrate for the treatment of low urinary pH and hypocitraturia varied when patients were stratified by body mass index.We retrospectively reviewed the records of patients with urolithiasis and concomitant hypocitraturia and low urinary pH as unique abnormalities upon metabolic evaluation treated exclusively with potassium citrate. Based on body mass index the cohort was divided into the 4 groups of normal weight, overweight, obese and morbid…

Malemedicine.medical_specialtyUrologyUrinary systemPotassium030232 urology & nephrologyUrologychemistry.chemical_elementOverweightCitric AcidBody Mass Index03 medical and health sciencesKidney Calculi0302 clinical medicineInsulin resistanceInternal medicinePotassium CitratemedicineHumansAgedRetrospective Studiesbusiness.industryHydrogen-Ion ConcentrationMiddle Agedmedicine.diseaseObesityEndocrinologyTreatment Outcomechemistry030220 oncology & carcinogenesisKidney stonesFemalemedicine.symptombusinessHypocitraturiaBody mass indexThe Journal of urology
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Revaluation of the clinical and metabolic behavior of children with isolated growth hormone deficiency during GH treatment according to newly propose…

2015

Purpose To evaluate the clinical and metabolic behavior of children with isolated growth hormone (GH)-deficiency (GHD), grouped according to the new AIFA criteria for the appropriateness of use and reimbursement of GH treatment in children. Methods The clinical and metabolic data of 310 prepubertal children (220 M, 90 F; mean age 10.8 years) grouped, according to new AIFA note 39, into group A (No 181 with a peak of GH 10 µg/L) were retrospectively analyzed. Group A and B, diagnosed as having GHD, were treated with GH for at least 24 months, while group C was analyzed only at baseline. Results At baseline, group A showed higher waist circumference than B (p=0.031) and C (p=0.041), while no …

Malemedicine.medical_specialtyWaistAdolescentEndocrinology Diabetes and Metabolismmedicine.medical_treatmentGroup AGroup BGrowth hormone deficiencySettore MED/13 - Endocrinologiachemistry.chemical_compoundGovernment AgenciesEndocrinologyInternal medicineOutcome Assessment Health CaremedicineHumansChildDwarfism PituitaryGrowth hormone childrenbusiness.industryCholesterolInsulinmedicine.diseaseEndocrinologyItalychemistryChild PreschoolGrowth HormoneConcomitantPractice Guidelines as TopicGh treatmentFemalebusinessFollow-Up Studies
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Added impact of obesity and insulin resistance in nocturnal blood pressure elevation in children and adolescents.

2008

The aim of the present study was to analyze the relationship between insulin resistance and the ambulatory blood pressure components in obese children and adolescents. Eighty-seven overweight and obese white children and adolescents of both sexes, of European origin from 6 to 18 years of age (mean age: 10.9±2.7 years), were selected. Obesity was defined on the basis of a threshold body mass index z score >2 (Cole’s least mean square method) and overweight with a body mass index from the 85th to 97th percentile. A validated oscillometric method was used to measure ambulatory BP (Spacelabs 90207) during 24 hours. Fasting glucose and insulin were measured, and the homeostasis model assessm…

Malemedicine.medical_specialtyWaistAmbulatory blood pressureAdolescentBlood PressureOverweightBody Mass IndexCohort StudiesInsulin resistanceHeart RateInternal medicineInternal MedicineHyperinsulinemiaMedicineHumansObesityChildbusiness.industryWaist-Hip Ratiomedicine.diseaseObesityCircadian RhythmBlood pressureEndocrinologyCross-Sectional StudiesCardiovascular DiseasesHypertensionFemaleKidney Diseasesmedicine.symptomInsulin ResistancebusinessBody mass indexHypertension (Dallas, Tex. : 1979)
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