Search results for "Interferon"

showing 10 items of 963 documents

Management of anemia induced by triple therapy in patients with chronic hepatitis C: Challenges, opportunities and recommendations

2013

SummaryThe addition of protease inhibitors, boceprevir or telaprevir, to peginterferon+ribavirin (PegIFN/RBV) increases the frequency as well as the severity, and hence, clinical relevance of anemia, which has now become one of the major complications associated with triple therapy. Most significant factors associated with anemia in patients receiving triple therapy include older age, lower body mass index (BMI), advanced fibrosis, and lower baseline hemoglobin. The variability in inosine triphosphate pyrophosphatase (ITPA) gene, which encodes a protein that hydrolyses inosine triphosphate (ITP), has been identified as an essential genetic factor for anemia both in dual and triple therapy. …

Hemolytic anemiamedicine.medical_specialtyAnemiaGastroenterologyTelaprevirTelaprevirchemistry.chemical_compoundhemic and lymphatic diseasesInternal medicineBoceprevirRibavirinMedicineBoceprevirHepatologyHepatitis C virusbusiness.industryRibavirinAnemiamedicine.diseaseTransplantationProtease inhibitorchemistryErythropoietinImmunologyITPAbusinessPegylated interferonEpoietinmedicine.drugJournal of Hepatology
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Hepatitis C virus and the controversial role of the interferon sensitivity determining region in the response to interferon treatment

2008

The degree of variability of the interferon sensitivity determining region (ISDR) in the hepatitis C virus (HCV) genome has been postulated to predict the response to interferon therapy, mainly in patients infected with subtype 1b, although this prediction has been the subject of a long controversy. This prediction has been tested by analyzing a cohort of 67 Spanish patients infected with HCV genotype 1, 23 of which were infected with subtype 1a and 44 with subtype 1b. A sample previous to therapy with α-interferon plus ribavirin was obtained and several clones (between 25 and 96) including the ISDR were sequenced from each patient. A significant correlation between mutations at the ISDR an…

Hepacivirusmedicine.medical_treatmentHepatitis C virusMolecular Sequence DataGenome ViralHepacivirusmedicine.disease_causeAntiviral AgentsViruschemistry.chemical_compoundFlaviviridaeInterferonVirologyDrug Resistance ViralRibavirinmedicineHumansAmino Acid SequencebiologyRibavirinSequence Analysis DNAbiology.organism_classificationVirologyHepatitis CInfectious DiseasesCytokinechemistryAmino Acid SubstitutionSpainImmunologyCohortMutationInterferonsmedicine.drug
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Overexpression of STAT-1 by adenoviral gene transfer does not inhibit hepatitis B virus replication.

2006

Objectives Interferons are known to inhibit the replication of hepatitis B viruses (HBV) in several animal models in vitro and in vivo as well in humans. The STAT-1 protein plays a central role in the biological activity of both type I and type II interferons. The lack of functional STAT-1 renders cells and organisms susceptible to bacterial and viral infectious agents. We analysed whether the overexpression of STAT-1 protein enhances the biological interferon response and whether it elicits antiviral acitivity against HBV in vitro. Methods To achieve an efficient STAT-1 overexpression in primary liver cells and hepatoma cells, we generated a recombinant, replication-deficient adenovirus ex…

Hepatitis B virusCarcinoma HepatocellularBlotting WesternGenetic Vectorsmedicine.disease_causeTransfectionVirus ReplicationVirusHepatitis B virus PRE betaAdenoviridaeOrthohepadnavirusInterferonmedicineTumor Cells CulturedAnimalsHumansCells CulturedHepatitis B virusHepatologybiologyLiver NeoplasmsGastroenterologyvirus diseasesHepatitis Bmedicine.diseasebiology.organism_classificationVirologyMolecular biologydigestive system diseasesIn vitroDucksSTAT1 Transcription FactorHepadnaviridaeGene Expression RegulationDNA ViralHepatocytesmedicine.drugEuropean journal of gastroenterologyhepatology
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Recent progress and new trends in the treatment of hepatitis B

2002

The annual rate of progression to cirrhosis in patients with chronic HBV is 0.4 to 14.2% and that of death 4 to 10%. HCC risk increases in parallel with the severity and duration of infection, with an annual incidence less than 0.5% in carriers and 6% in patients with cirrhosis. The main aim of antiviral therapy for chronic "wild-type" HBV infection is to suppress viral replication before cirrhosis and HCC develop. Two drugs are approved: IFN alpha and lamivudine. IFN alpha is costly, has a narrow range of efficacy, safety, and tolerability. Lamivudine is active, cheaper, and better tolerated but has limited efficacy, being associated with increasing resistance and loss of clinical response…

Hepatitis B virusCirrhosisbusiness.industryLamivudineAlpha interferonHepatitis Bmedicine.disease_causemedicine.diseaseChronic liver diseaseVirologydigestive system diseasesTransplantationInfectious DiseasesTolerabilityVirologymedicinebusinessmedicine.drugJournal of Medical Virology
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Mixed cryoglobulinemia type II in chronic hepatitis B associated with HBe-minus HBV mutant: Cellular immune reactions and response to interferon trea…

1994

The case of a young female patient with chronic active hepatitis B, vasculitic purpura, edema, and circulating immune complexes due to mixed Cryoglobulinemia is described. Serum transami-nases were elevated. Serological assays showed hepatitis B surface antigen (HBsAg), antibody to hepatitis B e antigen (anti-HBe), and antibody to hepatitis B core antigen (anti-HBc) antibodies but no antibody to hepatitis C virus (anti-HCV) or antibody to hepatitis delta virus (anti-HDV) antibodies. Using hepatitis B virus-polymerase chain reaction (HBV-PCR) and direct sequencing a precore/core (preC/C) mutant unable to synthesize HBeAg was detected in serum. HBV antigens were demonstrated in the circulatin…

Hepatitis B virusHBsAgAdolescentT-Lymphocytesmedicine.disease_causeAntigenVirologymedicineHumansHepatitis B e AntigensHepatitis ChronicHepatitis B virusImmunity Cellularbiologybusiness.industryInterferon-alphavirus diseasesHepatitis BHepatitis Bmedicine.diseasebiology.organism_classificationVirologydigestive system diseasesHBcAgInfectious DiseasesCryoglobulinemiaHBeAgHepadnaviridaeMutationImmunologyLeukocytes MononuclearFemalebusinessImmune complex diseaseFollow-Up StudiesJournal of Medical Virology
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Treatment options in HBV.

2005

The available evidence on interferon-alpha (IFN) treatment for chronic hepatitis B is sufficient to conclude that in patients with HBeAg positive chronic hepatitis, standard IFN therapy significantly improves clearance of HBeAg (number needed to treat [NNT] = 4), loss of HBV-DNA (NNT = 4) and clearance of HBsAg (NNT = 18). HBeAg positive patients with normal or slightly raised ALT should be treated only if there is histological evidence of progressive disease. In patients with HBeAg negative chronic hepatitis, less than 20% of subjects who have achieved an end-of-treatment virological response after a course of standard IFN maintain a sustained virological response in the long-term. IFN tre…

Hepatitis B virusHBsAgmedicine.medical_specialtyAdefovirmedicine.disease_causeAntiviral AgentsGastroenterologyHepatitis B AntigensLiver diseaseInternal medicineAdefovirHumansMedicineHepatitis B virusHepatologybusiness.industryvirus diseasesLamivudineHIVHepatitis Bmedicine.diseaseHepatitis Bdigestive system diseasesTreatmentTreatment OutcomeHBeAgLamivudineDNA ViralImmunologyNumber needed to treatInterferonbusinessLiver diseasemedicine.drug
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Pre-core mutants of hepatitis B virus in patients receiving immunosuppressive treatment after orthotopic liver transplantation.

1996

Orthotopic liver transplantation (OLT) is a possible treatment for acute or chronic liver failure due to hepatitis B virus (HBV) infection, but reinfection of the graft can be a serious complication. The aim of this study was to monitor HBV markers, to analyse pre-core-/core-mutations as well as to identify the viral population causing reinfection after OLT, and to investigate the emergence or disappearance of these mutants in patients receiving immunosuppressive treatment. Fifty-four pre-and posttransplant serum samples of 17 patients were analysed. All patients underwent OLT for HBV-related liver disease and had HBV-DNA before and after OLT. Total DNA was extracted from all sera and a 240…

Hepatitis B virusTime Factorsmedicine.medical_treatmentPopulationLiver transplantationInterferon alpha-2medicine.disease_causeAntiviral AgentsLiver diseaseVirologyMedicineHumansHepatitis B e AntigensProtein PrecursorseducationHepatitis B viruseducation.field_of_studyHepatitis B Surface Antigensbiologybusiness.industryInterferon-alphaImmunosuppressionSequence Analysis DNAHepatitis Bbiology.organism_classificationmedicine.diseaseHepatitis BVirologyHepatitis B Core AntigensRecombinant ProteinsLiver TransplantationTransplantationsurgical procedures operativeInfectious DiseasesHepadnaviridaeDNA ViralbusinessImmunosuppressive AgentsFollow-Up StudiesJournal of medical virology
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Selection of hepatitis B virus variants with aminoacid substitutions inside the core antigen during interferon-? therapy

2000

The hepatitis B virus (HBV) core antigen carries many epitopes relevant for B and T cell response that show aminoacid variation during viral infection. In a longitudinal analysis, sequential serum samples of 15 patients that suffered from chronic HBV infection were collected before, during, and after high-dose IFN-α treatment. The HBV preCore/Core (preC/C) sequence of the selected samples in each patient was determined and analysed for sequence variations compared to the pretreatment sample. The positions of HBV core aminoacid substitutions were assigned to immunodominant B, CD4+ and CD8+ cell epitopes. Seventy-five percent of all aminoacid substitutions were found within immunodominant T a…

Hepatitis B virusbiologyAlpha interferonmedicine.disease_causebiology.organism_classificationVirologyEpitopeVirusInfectious DiseasesAntigenOrthohepadnavirusHepadnaviridaeVirologymedicineAntigenic variationJournal of Medical Virology
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Interferon alfa for chronic hepatitis B infection: Increased efficacy of prolonged treatment

1999

Interferon alfa (IFN-alpha) is the primary treatment for chronic hepatitis B. The standard duration of IFN-alpha therapy is considered 16 weeks; however, the optimal treatment length is still poorly defined. We evaluated the efficacy and acceptability of prolonged IFN-alpha treatment in patients with chronic hepatitis B. To investigate whether treatment prolongation could enhance the rate of hepatitis B e antigen (HBeAg) seroconversion, we conducted a prospective, controlled, multicenter trial in which all patients were treated with a standard regimen of 10 million units IFN-alpha 3 times per week over 16 weeks. Patients who were still HBeAg-positive after 16 weeks of therapy were randomize…

Hepatitis B virusmedicine.medical_specialtyHepatologybusiness.industryAlpha interferonHepatitis Bmedicine.diseasemedicine.disease_causeGastroenterologySurgerylaw.inventionRegimenRandomized controlled trialHBeAglawInternal medicineMulticenter trialmedicinebusinessInterferon alfamedicine.drugHepatology
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Refined analysis of genetic variability parameters in hepatitis C virus and the ability to predict antiviral treatment response.

2008

Summary.  Hepatitis C virus (HCV) infects approximately 3% of the world population. The chronicity of hepatitis C seems to depend on the level of genetic variability. We have recently (Torres-Puente et al., J Viral Hepat, 2008; 15: 188) reported genetic variability estimates from a large-scale sequence analysis of 67 patients infected with HCV subtypes 1a (23 patients) and 1b (44 patients) and related them to response, or lack of, to alpha-interferon plus ribavirin treatment.. Two HCV genome regions were analysed in samples prior to antiviral therapy, one compressing the three hypervariable regions of the E2 glycoprotein and another one including the interferon sensitive determining region …

Hepatitis C virusMutation MissenseAlpha interferonHepacivirusBiologyViral Nonstructural Proteinsmedicine.disease_causeAntiviral AgentsNucleotide diversityViral Envelope ProteinsVirologyDrug Resistance ViralRibavirinmedicineHumansGenetic variabilityNS5AGeneticsHepatologyHaplotypeGenetic VariationHepatitis CHepatitis C Chronicmedicine.diseaseVirologyHypervariable regionInfectious DiseasesTreatment OutcomeHaplotypesInterferonsJournal of viral hepatitis
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