Search results for "Interferon"

showing 10 items of 963 documents

Case Report: The JAK-Inhibitor Ruxolitinib Use in Aicardi-Goutieres Syndrome Due to ADAR1 Mutation

2021

Type I Interferonopathies comprise inherited inflammatory diseases associated with perturbation of the type I IFN response. Use of Janus kinase (JAK) inhibitors has been recently reported as possible tools for treating some of those rare diseases. We describe herein the clinical picture and treatment response to the JAK-inhibitor ruxolitinib in a 5-year-old girl affected by Aicardi-Goutières Syndrome type 6 (AGS6) due to ADAR1 mutation. The girl's interferon score (IS) was compared with that of her older brother, suffering from the same disorder, who was not treated. We observed a limited, but distinct neurological improvement (Gross Motor Function and Griffiths Mental Development Scales). …

Mental developmentRuxolitinibMutationTreatment responseAicardi-Goutières syndrome; JAK-inhibitor; interferonopathies; ruxolitinib; type I interferonbusiness.industryOlder brotherruxolitinibJAK-inhibitorCase ReportAicardi-Goutières syndromemedicine.diseasemedicine.disease_causePediatricsRJ1-570interferonopathiesInterferonImmunologyPediatrics Perinatology and Child HealthmedicineAicardi–Goutières syndrometype I interferonJanus kinasebusinessmedicine.drugFrontiers in Pediatrics
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Gamma-Interferon Regulates Secretion of G-CSF in Human Monocytes on the Transcriptional Level

1987

The production of colony stimulating factors (CSF) for granulocytes and monocytes is integrated into a network of communicating soluble messenger molecules resulting from T-cell/monocyte interactions. We assessed the capactiy of gamma-Interferon to modulate monocyte secretion of CSF by colony assays and Northern blot analysis to hybridize monocyte RNA with cDNA probes of different CSF-types. Whereas mRNA for GM-CSF was undetectable in untreated and gamma-IFN treated peripheral blood monocytes, the constitutive expression of mRNA for G-CSF and subsequent production of a CSF with biological activities similar to G-CSF could highly be enhanced by exposure of monocytes to gamma-IFN.

Messenger RNAmedicine.anatomical_structureChemistryComplementary DNAMonocyteGamma interferonmedicineRNASecretionNorthern blotColony-stimulating factorMolecular biology
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T-bet as a possible therapeutic target in autoimmune disease

2002

The prominent role of pro-inflammatory cytokines produced by T helper-1 (T(H1)) cells in regulating autoimmune responses in vitro and in vivo has been demonstrated. Recent observations of T cell polarisation by regulatory transcription factors--especially T-bet (T-box expressed in T cells)--raise the question of their influence in controlling autoimmune diseases. Here, the authors summarise recent observations of the role of T-bet in controlling chronic inflammatory and autoimmune diseases and discuss the implications of these findings for future therapeutic approaches.

Mice Inbred MRL lprTranscription GeneticTransgeneT cellCellular differentiationClinical BiochemistryMice TransgenicLymphocyte ActivationAutoimmune DiseasesInterferon-gammaMiceTh2 CellsCrohn DiseaseDrug DiscoverymedicineAnimalsLupus Erythematosus SystemicIL-2 receptorIntestinal MucosaMice KnockoutPharmacologyAutoimmune diseaseLupus erythematosusbusiness.industryZAP70Cell DifferentiationTh1 CellsColitisInflammatory Bowel Diseasesmedicine.diseaseCeliac DiseaseDisease Models Animalmedicine.anatomical_structureCTLA-4ImmunologyCytokinesMolecular MedicineT-Box Domain ProteinsbusinessTranscription FactorsExpert Opinion on Therapeutic Targets
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Correlation of renal tubular epithelial cell-derived interleukin-18 up-regulation with disease activity in MRL-Faslpr mice with autoimmune lupus neph…

2002

Objective MRL-Faslpr mice spontaneously develop an autoimmune disease that mimics systemic lupus erythematosus in humans. Infiltrating T cells expressing interferon-γ (IFNγ) are responsible for the autoimmune kidney destruction in MRL-Faslpr mice, and interleukin-18 (IL-18) released by mononuclear phagocytes stimulates T cells to produce the IFNγ. Since MRL-Faslpr T cells are characterized by an overexpression of the IL-18 receptor accessory chain, we sought to determine the impact of IL-18 on the progression of lupus nephritis in MRL-Faslpr mice. Methods IL-18 expression in sera and kidney tissues from MRL-Faslpr mice was determined by enzyme-linked immunosorbent assay (ELISA), reverse tra…

Mice Inbred MRL lprmedicine.medical_treatmentImmunologyBlotting WesternLupus nephritisEnzyme-Linked Immunosorbent AssayBiologymedicine.disease_causeAutoimmunityAutoimmune DiseasesMiceRheumatologyimmune system diseasesInterferonmedicineImmunology and AllergyMacrophageAnimalsPharmacology (medical)Interferon gammaskin and connective tissue diseasesLupus erythematosusCell adhesion moleculeReverse Transcriptase Polymerase Chain ReactionCaspase 1Interleukin-18Epithelial Cellsmedicine.diseaseMolecular biologyImmunohistochemistryLupus NephritisUp-RegulationCytokineKidney TubulesImmunologymedicine.drugArthritis and rheumatism
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Hepatocytes of double-transgenic mice expressing high levels of hepatitis B virus e antigen and interferon-gamma are not injured by HBeAg specific au…

2000

Seroconversion from HBeAg to alphaHBe of persons chronically infected by HBV is usually associated with a transient exacerbation of liver disease and subsequent normalization of liver histology. It is speculated that these clinico-pathological features may be due to the activation of cytodestructive mechanisms by alphaHBe antibodies. The aim of the present study was to investigate the pathogenic potential of alphaHBe antibodies in a transgenic mouse model. Therefore, alphaHBe autoantibodies were elicited in double-transgenic mice expressing high amounts of HBeAg and interferon-gamma in the liver. Interferon-gamma has reviously been shown to play an important role in the development of hepat…

Mice Transgenicmedicine.disease_causeTransfectionCell LineLiver diseaseInterferon-gammaMiceInterferonAntibody SpecificityVirologymedicineAnimalsInterferon gammaHepatitis B e AntigensSeroconversionHepatitis B AntibodiesProtein PrecursorsAutoantibodiesHepatitis B virusbiologyViral Core Proteinsvirus diseasesInterferon-alphaGeneral Medicinemedicine.diseasebiology.organism_classificationFlow CytometryHepatitis BVirologydigestive system diseasesHepadnaviridaeHBeAgLiverImmunologybiology.proteinAntibodymedicine.drugArchives of virology
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Cytokine-mediated regulation of monocyte/macrophage cytotoxicity in human immunodeficiency virus-1 infection.

1992

Monocyte/macrophage-mediated tumor cytotoxicity was studied in patients infected with human immunodeficiency virus-1 (HIV-1) at various stages [Center for disease control (CDC) classification] of the disease. using the P-815 tumor cell line as target cells, the results demonstrated reduced monocyte/macrophage cytotoxicity early in HIV-1-related disease (CDCIII, P0.01). This cellular dysfunction sustained during the progression of the disease. Evidence could be presented that neither exogenous application of macrophage-stimulating cytokines (e.g. interferons) nor their endogenous induction in vitro restored monocyte/macrophage cytotoxicity. However, enhanced tumor necrosis factor (TNF)-alpha…

Microbiology (medical)AdultCytotoxicity Immunologicmedicine.medical_treatmentImmunologyHIV InfectionsBiologyVirusMonocytesmedicineTumor Cells CulturedImmunology and AllergyMacrophageHumansProstaglandin E2CytotoxicityCells CulturedTumor Necrosis Factor-alphaMonocyteInterleukinsMacrophagesGeneral MedicineMiddle AgedIn vitroCytokinemedicine.anatomical_structureImmunologyHIV-1CytokinesTumor necrosis factor alphaInterferonsmedicine.drugMedical microbiology and immunology
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Performance of the QuantiFERON-cytomegalovirus (CMV) assay for detection and estimation of the magnitude and functionality of the CMV-specific gamma …

2012

ABSTRACTThe performance of the QuantiFERON-cytomegalovirus (CMV) assay was compared to that of a flow cytometry intracellular cytokine staining (ICS) method for the detection of CMV-specific gamma interferon (IFN-γ)-producing CD8+T-cell responses in allogeneic stem cell transplant (allo-SCT) recipients and for estimations of their magnitude and functionality. A total of 90 whole-blood specimens from 23 allo-SCT recipients was analyzed by both methods. Overall, the percentage of specimens that yielded concordant results by both methods was 68.8% (κ = 0.691; 95% confidence interval [CI], 0.548 to 0.835), and the sensitivity of the QuantiFERON-CMV assay for the detection of positive IFN-γ T-ce…

Microbiology (medical)AdultMaleClinical BiochemistryImmunologyCongenital cytomegalovirus infectionCytomegalovirusBiologyCD8-Positive T-LymphocytesSensitivity and SpecificityFlow cytometryQuantiFERONGamma interferonDiagnostic Laboratory ImmunologymedicineImmunology and AllergyCytotoxic T cellHumansAgedTransplantationmedicine.diagnostic_testMiddle Agedmedicine.diseaseConfidence intervalImmunologyCytomegalovirus InfectionsFemaleStem cellCD8Interferon-gamma Release TestsStem Cell TransplantationClinical and vaccine immunology : CVI
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Evolutionary dynamics of the E1-E2 viral populations during combination therapy in non-responder patients chronically infected with hepatitis C virus…

2012

Abstract Half of the patients chronically infected with hepatitis C virus (HCV) genotype 1 fail to respond to pegylated interferon alpha (PEG-IFN) and ribavirin (RBV) therapy. This study assesses the effects of treatment on the evolution of the E1–E2 viral region in non-responder patients infected with HCV-1b. Twenty-three HCV-1b chronically infected patients were studied retrospectively, including 19 non-responders to PEG-IFN/RBV therapy (11 null-responders and 8 relapsers) in the study group, and 4 untreated patients in the control group. Genetic and phylogenetic analyses of the E1–E2 viral populations were performed at baseline and at the time of treatment failure to assess changes in ge…

Microbiology (medical)AdultMaleCombination therapyHepatitis C virusAdaptation BiologicalHepacivirusBiologymedicine.disease_causeMicrobiologyAntiviral AgentsEvolution Molecularchemistry.chemical_compoundViral Envelope ProteinsPegylated interferonGenotypeGeneticsmedicineHumansGenetic variabilityTreatment FailureMolecular BiologyEcology Evolution Behavior and SystematicsPhylogenyAgedRetrospective StudiesGenetic diversityRibavirinGenetic VariationHepatitis C ChronicMiddle AgedViral LoadVirologyInfectious DiseaseschemistryAmino Acid SubstitutionViral evolutionImmunologyDrug Therapy CombinationFemalemedicine.drugInfection, genetics and evolution : journal of molecular epidemiology and evolutionary genetics in infectious diseases
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Normal interleukin-12 production in individuals with antibodies toHelicobacter pylori

1997

It is increasingly recognized that the inability of the immune system to clear H. pylori infection is caused by an inadequate immune response and is associated with chronic gastric inflammation. To further investigate the cellular immune response to H. pylori, we studied PBMC from 31 H. pylori antibody-negative and 16 H. pylori antibody-positive individuals for H. pylori-induced DNA synthesis, secretion of the Th1-type cytokine IFN-gamma and secretion of IL-12, a cytokine produced by bacteria-stimulated monocyte/macrophages and a potent inducer of antibacterial immune responses and Th1-type T cells. All experiments were performed using Y. enterocolitica 03 as control. Our results demonstrat…

Microbiology (medical)Cellular immunitymedicine.medical_treatmentInflammationHelicobacter InfectionsPathology and Forensic MedicineMicrobiologyInterferon-gammaImmune systemmedicineHumansImmunology and AllergyImmunity CellularHelicobacter pyloribiologyMonocyteGeneral MedicineMiddle AgedHelicobacter pyloribacterial infections and mycosesbiology.organism_classificationAntibodies BacterialInterleukin-12Cytokinemedicine.anatomical_structureImmunologyInterleukin 12biology.proteinmedicine.symptomAntibodyAPMIS
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Mode of selection and experimental evolution of antiviral drugs resistance in vesicular stomatitis virus

2004

Abstract The possession of an antiviral resistance mutation benefits a virus when the corresponding antiviral is present. But does the resistant virus pay a fitness cost when the antiviral is absent? Would an evolutionary history of association between a genotype and a resistance mutation overcome this cost by changes compensating the harmful side-effect of resistance mutations? Are combined therapies more effective against the rise of resistant viruses or against evolutionary compensations? To explore all these questions, we took an experimental evolution approach. After selecting vesicular stomatitis virus (VSV) populations able to replicate under increasing concentrations of ribavirin an…

Microbiology (medical)GenotypeBiologyVirus ReplicationAntiviral AgentsMicrobiologyVirusVesicular stomatitis Indiana virusEvolution Molecularchemistry.chemical_compoundGenotypeDrug Resistance ViralRibavirinGeneticsMolecular BiologyEcology Evolution Behavior and SystematicsGeneticsExperimental evolutionDose-Response Relationship DrugRibavirinAntiviral therapyInterferon-alphaDrug SynergismResistance mutationbiology.organism_classificationVirologyInfectious DiseaseschemistryVesicular stomatitis virusMutationFitness costInfection, Genetics and Evolution
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