Search results for "Interleukins"

showing 10 items of 137 documents

Increased expression of IL-19 in the epithelium of patients with chronic rhinosinusitis and nasal polyps.

2012

Background : Chronic rhinosinusitis (CRS) is an inflammation of the nose and of the paranasal sinuses. The involvement of the respiratory epithelium in the mechanisms of CRS is poorly understood. Aims : Among proteins expressed by nasal epithelial cells in CRS, IL-19 may have key functions. We here aimed to determine the expression and regulation of IL-19. Methods : Nasal biopsies from normal subjects (n = 12), subjects with CRS but without nasal polyps (NP) (CRSsNP, n = 12) and with CRS with NP (CRSwNP, n = 15) were collected. Human Asthma Gene Array and real-time PCR were used to evaluate gene expression, western blot analysis and immunohistochemistry for protein expression. Results for I…

AdultMalePathologymedicine.medical_specialtyAdolescentImmunologyGene ExpressionMucous membrane of noseYoung AdultInterleukin 20Nasal Polypsotorhinolaryngologic diseasesImmunology and AllergyMedicineHumansNasal polypsRNA MessengerSinusitisSinusitisAsthma inflammation nasal polyps rhinitisRhinitisbusiness.industryGene Expression ProfilingInterleukinsEpithelial CellsMiddle Agedmedicine.diseaseEpitheliumGene expression profilingNasal Mucosamedicine.anatomical_structureParanasal sinusesKi-67 AntigenImmunologyChronic DiseaseRespiratory epitheliumFemalebusinessAllergy
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Type 3 innate lymphoid cells producing IL-17 and IL-22 are expanded in the gut, in the peripheral blood, synovial fluid and bone marrow of patients w…

2015

Background The aim of the study was to better characterise the immunological origin and the behaviour of interleukin (IL)-23-responsive innate lymphoid cells (ILCs) in the gut, synovial fluid (SF) and bone marrow (BM) of patients with ankylosing spondylitis (AS).Methods ILC1, ILC2 and ILC3 cells were determined and characterised by confocal microscopy and flow cytometry in ileal and BM biopsies, in peripheral blood (PB) and SF mononuclear cells obtained from patients with AS and controls. Mucosal vascular addressin cell adhesion molecule 1 (MADCAM-1), IL-7, IL-15 and aggregates of lymphoid tissue inducer cells (LTi) were evaluated by immunohistochemistry. The in vitro ability of epithelial …

AdultMalePathologymedicine.medical_specialtyAdolescentImmunologyHigh endothelial venulesImmunoglobulinsPeripheral blood mononuclear cellGeneral Biochemistry Genetics and Molecular BiologyInterleukin 22Young AdultMucoproteinsAnkylosing Spondylitis; Cytokines; InflammationRheumatologyBone MarrowIleumSynovial FluidAddressinImmunology and AllergyMedicineSynovial fluidHumansSpondylitis AnkylosingLymphocytesIntestinal MucosaCytokineAgedInterleukin-15InflammationMicroscopy ConfocalAnkylosing SpondylitibiologyNatural Cytotoxicity Triggering Receptor 2business.industryInterleukin-7InterleukinsInnate lymphoid cellInterleukin-17Middle AgedSettore MED/16 - Reumatologiamedicine.anatomical_structureLymphatic systemCase-Control Studiesbiology.proteinFemaleBone marrowbusinessCell Adhesion Molecules
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Increased expression of interleukin-32 in the inflamed ileum of ankylosing spondylitis patients

2012

Objective. To study the mRNA expression and protein tissue distribution of IL-32 in ileal biopsy specimens from patients with AS. Methods. Quantitative gene expression analysis, by real-time PCR, of IL-32, IL-1b, IL-10, TNF-a and IFN-g was performed on ileal biopsies of 15 AS and 15 Crohn’s disease (CD) patients and 10 healthy subjects (HSs). IL-32 tissue distribution was evaluated by immunohistochemistry. The effect of IL-32 on the production of IL-10 by intestinal epithelial cell lines was also evaluated. Results. In the ileal specimens of patients with AS and intestinal chronic inflammation, significant up-regulation of IL-32 at both the mRNA and protein levels was found as compared with…

AdultMalePathologymedicine.medical_specialtyAdolescentmedicine.medical_treatmentInterleukin-1betaInflammationInterferon-gammaYoung AdultCrohn DiseaseRheumatologyIleumBiopsyintestinal inflammationmedicineHumansSpondylitis AnkylosingPharmacology (medical)IleitisRNA MessengerCrohn's diseasemedicine.diagnostic_testTumor Necrosis Factor-alphabusiness.industryInterleukinsMacrophagesIL-32 ankylosing spondylitis IL-10 intestinal inflammationInterleukinEpithelial CellsIleitisMiddle AgedHCT116 Cellsmedicine.diseaseImmunity InnateInterleukin-10Settore MED/16 - ReumatologiaInterleukin 10Interleukin 32ankylosing spondylitiCytokineCase-Control StudiesImmunologyIL-32IL-10Femalemedicine.symptombusiness
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IL-34 is overexpressed in the inflamed salivary glands of patients with Sjogren's syndrome and is associated with the local expansion of pro-inflamma…

2013

Objectives To investigate the expression of IL-34 in labial salivary glands (LSGs) of patients with primary SS (p-SS) and its role in inducing a pro-inflammatory monocyte phenotype. Methods LSG biopsies were obtained from 20 patients with p-SS and 10 patients with non-Sjogren's sicca syndrome (n-SS). The expression of IL-34, IL-1β, TNF-α, IL-17 and IL-23 was assessed by real-time PCR. IL-34 expression was also investigated in LSGs by immunohistochemistry. The frequencies of subpopulations of CD14(+) monocytes were evaluated by flow cytometry among isolated mononuclear cells from peripheral blood and salivary glands from both patients and controls. The role of recombinant IL-34 on isolated p…

AdultMalePathologymedicine.medical_specialtyCD14Interleukin-1betaLipopolysaccharide ReceptorsInflammationCD16Interleukin-23Peripheral blood mononuclear cellMonocytesSalivary GlandsRheumatologySicca syndromemedicineHumansPharmacology (medical)interleukin-34 Sjögren’s syndrome monocytesAgedInflammationSalivary glandTumor Necrosis Factor-alphabusiness.industryInterleukinsMonocyteInterleukin-17Receptors IgGMiddle AgedFlow CytometrySettore MED/16 - ReumatologiaSjogren's Syndromemedicine.anatomical_structureImmunologyInterleukin 34Femalemedicine.symptombusinessRheumatology
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Interleukin-22 and interleukin-22-producing NKp44+ natural killer cells in subclinical gut inflammation in ankylosing spondylitis

2012

Objective The intestinal inflammation observed in patients with ankylosing spondylitis (AS) is characterized by an overexpression of interleukin-23 (IL-23). IL-23 is known to regulate IL-22 production through lamina propria NKp44+ natural killer (NK) cells, which are thought to be involved in protective mucosal mechanisms. This study was undertaken to evaluate the frequency of NKp44+ NK cells and the expression of IL-22 in the ileum of AS patients. Methods Tissue NKp44+ NK cells, NKp46+ NK cells, and IL-22–producing cells were analyzed by flow cytometry. Quantitative gene expression analysis of IL-22, IL-23, IL-17, STAT-3, and mucin 1 (MUC-1) was performed by reverse transcriptase–polymeras…

AdultMaleSTAT3 Transcription FactorImmunologyIleumBiologyInterleukin-23Peripheral blood mononuclear cellFlow cytometryAnkylosing spondylitis IL-22 intestinal inflammation intestinal inflammationInterleukin 22Interleukin 21RheumatologyIleumintestinal inflammationIL-22medicineHumansImmunology and AllergySpondylitis AnkylosingPharmacology (medical)Intestinal MucosaInflammationLamina propriaNatural Cytotoxicity Triggering Receptor 2medicine.diagnostic_testInterleukinsMucin-1MucinMiddle AgedKiller Cells NaturalAnkylosing spondylitimedicine.anatomical_structureImmunologyImmunohistochemistryFemaleArthritis & Rheumatism
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Rituximab modulates IL-17 expression in the salivary glands of patients with primary Sjögren's syndrome.

2014

OBJECTIVE: The aim of this study was to evaluate the role of rituximab (RTX) in modulating the expression of the IL-17/IL-23 pathway in the salivary glands (SGs) of patients with primary SS (pSS). METHODS: Consecutive SG biopsies were obtained from 15 patients with pSS before and after 1 year of RTX therapy. The SG expression of IL-17, IL-23p19 and p-STAT3 was evaluated by immunohistochemistry at baseline and after RTX therapy. The role of mast cells in pSS patients in modulating the Th17 response and the immunologic effect of RTX on mast cells were also studied in in vitro experiments. RESULTS: IL-17 was overexpressed in the SGs of patients with pSS mainly by infiltrating T cells and mast …

AdultMaleSTAT3 Transcription FactorSjogren SyndromeApoptosisIn Vitro TechniquesInterleukin-23Peripheral blood mononuclear cellSalivary GlandsAntibodies Monoclonal Murine-DerivedRheumatologystomatognathic systemSettore BIO/13 - Biologia ApplicataBiopsyHumansMedicinePharmacology (medical)Mast CellsAgedmedicine.diagnostic_testbusiness.industryInterleukinsInterleukin-17IL17Middle AgedMast cellIn vitroSettore MED/16 - ReumatologiaSjogren's SyndromeTreatment Outcomemedicine.anatomical_structureApoptosisAntirheumatic AgentsImmunologyTh17 CellsImmunohistochemistryFemaleRituximabInterferonsInterleukin 17businessRituximabSignal Transductionmedicine.drug
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Vitamin D levels and IL28B polymorphisms are related to rapid virological response to standard of care in genotype 1 chronic hepatitis C.

2011

Background Genotype 1 (G1) chronic hepatitis C (CHC) patients achieving a rapid virological response (RVR) on pegylated interferon (PEG-IFN) plus ribavirin have a high chance of sustained virological response (SVR), influenced by IL28B status, viral load, fibrosis and insulin resistance. We assessed whether 25-hydroxyvitamin D (25[OH]D) serum levels are linked to RVR and can be used together with IL28B to construct a pretreatment model to predict RVR. Methods A total of 117 consecutive patients with G1 CHC were evaluated by biopsy and anthropometric and metabolic measurements. 25(OH)D serum levels were measured by HPLC. IL28B rs12979860 and rs8099917 polymorphisms were also evaluated. All p…

AdultMaleStandard of careGenotypeHepacivirusAntiviral AgentsPolymorphism Single NucleotidePolyethylene GlycolsVirological responsechemistry.chemical_compoundChronic hepatitisPegylated interferonRisk FactorsGenotypeVitamin D and neurologyMedicineHumansPharmacology (medical)Vitamin DPharmacologybusiness.industryRibavirinInterleukinsInterferon-alphaStandard of CareHepatitis C ChronicMiddle AgedViral LoadRecombinant ProteinsInfectious DiseasesTreatment OutcomechemistryImmunologyFemaleInterferonsbusinessmedicine.drugAntiviral therapy
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Effects of IL28B rs12979860 CC Genotype on Metabolic Profile and Sustained Virologic Response in Patients With Genotype 1 Chronic Hepatitis C

2013

Patients with genotype 1 chronic hepatitis C (G1 CHC) frequently develop steatosis and insulin resistance (IR), caused by metabolic and viral factors. These accelerate the progression of liver disease and reduce the response to therapy. A sustained virologic response (SVR) to therapy in patients with G1 CHC is associated strongly with polymorphisms near the interleukin-28B (IL28B) gene, but the interaction between IL28B genotype and IR, and their combined effects on SVR, have not been defined. We tested the association between the IL28B rs12979860 single-nucleotide polymorphism and metabolic features, including IR, and evaluated their effects on SVR.We performed genotype analysis of IL28B r…

AdultMalemedicine.medical_specialtyGenotypeHepacivirusHepatitis C virusSingle-nucleotide polymorphismHepacivirusmedicine.disease_causeAntiviral AgentsPolymorphism Single NucleotideGastroenterologyCohort StudiesLiver diseaseInsulin resistanceInternal medicineGenotypemedicineHumansAgedinsulin resistance steatosis interleukin-28B sustained virologic responseHepatologybiologybusiness.industryInterleukinsGastroenterologyHepatitis C ChronicMiddle AgedViral Loadmedicine.diseasebiology.organism_classificationTreatment OutcomeInterleukin 28BImmunologyMetabolomeRNA ViralFemaleInterferonsInsulin ResistanceSteatosisbusinessClinical Gastroenterology and Hepatology
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The exposure of healthy volunteers to 200 ppm 1,1,1-trichloroethane increases the concentration of proinflammatory cytokines in nasal secretions

1999

Objectives: Irritating effects of organic solvents have usually been measured by means of questionnaires. The aim of the present study was to evaluate the sensitivity of different methods of detecting subclinical irritating effects. Methods: Twelve healthy, non-smoking students were exposed to 200 ppm and to 20 ppm 1,1,1-trichloroethane in an exposure chamber, using a crossover design. The amounts of interleukins (IL)-1β, IL-6 and IL-8 and prostaglandin E2 (PGE2) in nasal secretions were measured. Mucociliary transport time was determined with the saccharine test. Ciliary beat frequency of nasal epithelial cells was measured with video-interference contrast microscopy. Subjective symptoms w…

AdultMalemedicine.medical_specialtyMucous membrane of nosemedicine.disease_causeDinoprostoneStatistics NonparametricProinflammatory cytokineInternal medicinemedicineHumansTrichloroethanesProstaglandin E2Subclinical infectionCross-Over StudiesInhalationInterleukin-6ChemistryInterleukinsInterleukin-8Public Health Environmental and Occupational HealthCrossover studyNasal MucosaEndocrinologyMucociliary ClearanceImmunologyToxicitySolventsIrritationInterleukin-1medicine.drugInternational Archives of Occupational and Environmental Health
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TNF-α, IL-17, and IL-22 production in the rectal mucosa of nonceliac wheat sensitivity patients: role of adaptive immunity.

2020

In recent years, a new gluten- or wheat-related disease has emerged, a condition labeled "nonceliac gluten sensitivity" (NCGS) or "nonceliac wheat sensitivity" (NCWS). NCWS pathogenesis is still uncertain and attributed to very different mechanisms. We aimed to study the different T-lymphocyte subsets in the rectal mucosa of NCWS patients to demonstrate the possible contribution of adaptative immune response. Twelve patients (11 women, 1 man, age range 23-61 yr, median 32 yr) with a definitive diagnosis of NCWS were recruited at random for the present study. They underwent rectal endoscopy with multiple mucosal biopsies at the end of a double-blind placebo-controlled (DBPC) wheat challenge …

AdultMalemedicine.medical_specialtyNecrosisSettore MED/09 - Medicina InternaPhysiologynon-celiac wheat sensitivityBiopsyTNFWheat HypersensitivityAdaptive ImmunityGastroenterologyInterleukin 22PathogenesisYoung AdultImmune systemDouble-Blind MethodAntigens CDPhysiology (medical)Internal medicinemedicineIL-22HumansMucous MembraneHepatologybusiness.industryTumor Necrosis Factor-alphaInterleukinsInterleukin-17GastroenterologyRectumColonoscopyMiddle AgedAcquired immune systemLymphocyte SubsetsIL-17Tumor necrosis factor alphaFemaleInterleukin 17medicine.symptombusinessCD8American journal of physiology. Gastrointestinal and liver physiology
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