Search results for "Intestin"

showing 10 items of 2215 documents

Novel GPR120 agonist TUG891 modulates fat taste perception and preference and activates tongue-brain-gut axis in mice

2020

GPR120 is implicated as a lipid receptor in the oro-sensory detection of dietary fatty acids. However, the effects of GPR120 activation on dietary fat intake or obesity are not clearly understood. We investigated to determine whether the binding of TUG891, a novel GPR120 agonist, to lingual GPR120 modulates fat preference in mice. We explored the effects of TUG891 on obesity-related hormones and conducted behavioral choice tests on mice to better understand the physiologic relevance of the action of TUG891. In cultured mouse and human taste bud cells (TBCs), TUG891 induced a rapid increase in Ca2+ by acting on GPR120. A long-chain dietary fatty acid, linoleic acid (LA), also recruited Ca2+ …

Male0301 basic medicineAgonistlinoleic acidmedicine.medical_specialtyTasteextracellular signal-regulated kinase 1/2obesitymedicine.drug_classLinoleic acidQD415-436030204 cardiovascular system & hematologyBiochemistryReceptors G-Protein-CoupledMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyTongueInternal medicinemedicineAnimalsHumansReceptorCells CulturedResearch ArticlesCholecystokininchemistry.chemical_classificationPhenylpropionatesBiphenyl CompoundsBrainTaste PerceptionFatty acidGPR120Cell BiologyTaste BudsGastrointestinal MicrobiomeMice Inbred C57BL030104 developmental biologyEndocrinologychemistryglucagon-like peptide-1Hormone
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Tuft cell‐derived IL‐25 activates and maintains ILC2

2016

Parasitic helminths and allergens induce a type 2 immune response leading to profound changes in tissue physiology, including hyperplasia of mucus-secreting goblet cells1 and smooth muscle hypercontractility2. This response, known as ‘weep and sweep’, requires interleukin (IL)-13 production by tissue-resident group 2 innate lymphoid cells (ILC2s) and recruited type 2 helper T cells (TH2 cells)3. Experiments in mice and humans have demonstrated requirements for the epithelial cytokines IL-33, thymic stromal lymphopoietin (TSLP) and IL-25 in the activation of ILC2s4–11, but the sources and regulation of these signals remain poorly defined. In the small intestine, the epithelium consists of at…

Male0301 basic medicineAllergyImmunologyInnate immunologyBiologyArticle03 medical and health sciences0302 clinical medicineImmune systemIntestinal mucosaImmunitymedicineAnimalsImmunology and AllergyLymphocytesIntestinal MucosaImmunity MucosalInterleukin-17Cell Biologymedicine.diseaseImmunity Innate030104 developmental biologyMucosal immunologyImmunologyFemaleInterleukin 17Tuft cell030215 immunologyImmunology & Cell Biology
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Interleukin-17 Inhibition in Spondyloarthritis Is Associated With Subclinical Gut Microbiome Perturbations and a Distinctive Interleukin-25-Driven In…

2020

Objective To characterize the ecological effects of biologic therapies on the gut bacterial and fungal microbiome in psoriatic arthritis (PsA)/spondyloarthritis (SpA) patients. Methods Fecal samples from PsA/SpA patients pre- and posttreatment with tumor necrosis factor inhibitors (TNFi; n = 15) or an anti-interleukin-17A monoclonal antibody inhibitor (IL-17i; n = 14) underwent sequencing (16S ribosomal RNA, internal transcribed spacer and shotgun metagenomics) and computational microbiome analysis. Fecal levels of fatty acid metabolites and cytokines/proteins implicated in PsA/SpA pathogenesis or intestinal inflammation were correlated with sequence data. Additionally, ileal biopsies obtai…

Male0301 basic medicineArthritisPsoriatic0302 clinical medicineInterleukin 25Monoclonal2.1 Biological and endogenous factorsImmunology and AllergyMedicineAetiologyIntestinal MucosaCandida albicansHumanizedSubclinical infectionbiologyInterleukin-17Innate lymphoid cellMiddle AgedIntestinesPublic Health and Health ServicesFemaleTumor necrosis factor alphaInterleukin 17Clinical SciencesImmunologyAntibodies Monoclonal HumanizedAutoimmune DiseaseAntibodiesArticle03 medical and health sciencesRheumatologyClinical ResearchSpondylarthritisHumansMicrobiomeInflammation030203 arthritis & rheumatologybusiness.industryArthritisInflammatory and immune systemArthritis Psoriaticbiology.organism_classificationmedicine.diseaseArthritis & RheumatologyGastrointestinal Microbiome030104 developmental biologyImmunologyTumor Necrosis Factor InhibitorsDigestive Diseasesbusiness
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Nutriosomes: Prebiotic delivery systems combining phospholipids, a soluble dextrin and curcumin to counteract intestinal oxidative stress and inflamm…

2018

Nutriosomes, new phospholipid nanovesicles specifically designed for intestinal protection were developed by simultaneously loading a water-soluble dextrin (Nutriose® FM06) and a natural antioxidant (curcumin). Nutriosomes were easily fabricated in a one-step, organic solvent-free procedure. The stability and delivery performances of the vesicles were improved by adding hydroxypropyl methylcellulose. All the vesicles were small in size (mean diameter ∼168 nm), negatively charged (zeta potential ∼-38 mV, irrespective of their composition), and self-assembled predominantly in unilamellar vesicles stabilized by the presence of Nutriose®, which was located in both the inter-lamellar and inter-v…

Male0301 basic medicineBiodistributionAntioxidantCurcuminEstrès oxidatiumedicine.medical_treatmentPhospholipidBiological AvailabilityCurcumin analogues02 engineering and technologyAntioxidants03 medical and health scienceschemistry.chemical_compoundCryoprotective AgentsDrug Delivery SystemsCurcumaMicroscopy Electron TransmissionX-Ray DiffractionDextrinsScattering Small AnglemedicineZeta potentialAnimalsHumansTissue DistributionGeneral Materials ScienceRats WistarPhospholipidsInflammationchemistry.chemical_classificationVesicle021001 nanoscience & nanotechnologyRats3. Good healthBioavailabilityIntestinesOxidative StressFreeze DryingPrebiotics030104 developmental biologychemistryCurcuminBiophysicsDextrinCaco-2 Cells0210 nano-technology
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Melatonin protects rats from radiotherapy-induced small intestine toxicity

2017

Radiotherapy-induced gut toxicity is among the most prevalent dose-limiting toxicities following radiotherapy. Prevention of radiation enteropathy requires protection of the small intestine. However, despite the prevalence and burden of this pathology, there are currently no effective treatments for radiotherapy-induced gut toxicity, and this pathology remains unclear. The present study aimed to investigate the changes induced in the rat small intestine after external irradiation of the tongue, and to explore the potential radio-protective effects of melatonin gel. Male Wistar rats were subjected to irradiation of their tongues with an X-Ray YXLON Y.Tu 320-D03 irradiator, receiving a dose o…

Male0301 basic medicineCancer TreatmentDrug Evaluation Preclinicallcsh:MedicineExpressionApoptosisToxicologyPathology and Laboratory Medicinemedicine.disease_causeBiochemistryOxidative Phosphorylation0302 clinical medicineIntestinal mucosaGastrointestinal tractIntestine SmallMedicine and Health SciencesRadiation-injuryIntestinal Mucosalcsh:ScienceEnergy-Producing OrganellesMelatoninCancerMultidisciplinaryNF-kappa BInflammasomeLipid-peroxidationGlutathioneMitochondriaRadiation therapyRadiation Injuries Experimentalmedicine.anatomical_structureOncology030220 oncology & carcinogenesisCancer treatmentToxicityInflammasome activationSmall IntestineExperimental pathologyAnatomyCellular Structures and OrganellesResearch Articlemedicine.drugClinical OncologyMucositismedicine.medical_specialtyRadiation TherapyRadiation-Protective AgentsBioenergeticsBiologyRadiation enteropathyMelatonin03 medical and health sciencesTongueInternal medicineSepsisNLR Family Pyrin Domain-Containing 3 ProteinmedicineAnimalsRats WistarMouthToxicitylcsh:RBiology and Life SciencesCell BiologySmall intestinemedicine.diseaseHormonesSmall intestinePathobiologyGastrointestinal TractOxidative Stress030104 developmental biologyEndocrinologylcsh:QClinical MedicineDigestive SystemGelsOxidative stress
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Intraperitoneal administration of the anti-IL-23 antibody prevents the establishment of intestinal nematodes in mice

2018

AbstractPrevious studies have established that an increased Th-9 response creates a hostile environment for nematode parasites. Given that IL-23, a cytokine required for maintenance of the IL-17–secreting phenotype, has inhibitory effects on IL-9 production, we hypothesized that reducing circulating IL-23 by treatment with anti-IL-23 antibodies would reduce the establishment and development of parasitic intestinal nematodes. In this study, we show that animals treated with anti-IL-23 monoclonal antibodies showed a drastic reduction in the number of mouse pinworms (Aspiculuris tetraptera) recovered from the intestine (p < 0.001) at 23 days post-infection compared to the untreated animals.…

Male0301 basic medicineChemokinemedicine.drug_classmedicine.medical_treatmentlcsh:MedicineMonoclonal antibodyInterleukin-23Inflammatory bowel diseaseArticleParasite LoadMice03 medical and health sciencesInterleukin 23medicineAnimalsNematode Infectionslcsh:ScienceMice Inbred ICRMultidisciplinarybiologyInterleukinslcsh:RAntibodies Monoclonalmedicine.diseasebiology.organism_classificationIntestinesCCL20030104 developmental biologyNematodeCytokineImmunologybiology.proteinlcsh:QAntibodyInjections Intraperitoneal
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Diminished Vitamin D Receptor Protein Levels in Crohn´s Disease Fibroblasts: Effects of Vitamin D

2020

Vitamin D (VD) deficiency has been associated to Crohn&acute

Male0301 basic medicineCrohn’s diseaselcsh:TX341-641vitamin DCalcitriol receptorArticlePathogenesis03 medical and health sciences0302 clinical medicineCrohn DiseaseCell MovementFibrosisfibroblastsVitamin D and neurologyAnimalsHumansMedicineFibroblastWound HealingCrohn's diseaseNutrition and Dieteticsbusiness.industryfibrosisEpithelial CellsVitamin D Deficiencymedicine.diseasedigestive system diseasesIntestinesMice Inbred C57BLDisease Models Animal030104 developmental biologymedicine.anatomical_structureGene Expression Regulationvitamin D receptor (VDR)Cancer researchReceptors CalcitriolFemale030211 gastroenterology & hepatologyWound healing assaybusinesslcsh:Nutrition. Foods and food supplyFood ScienceHormoneCrohn´s diseaseNutrients
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Impact of Donor Human Milk in the Preterm Very Low Birth Weight Gut Transcriptome Profile by Use of Exfoliated Intestinal Cells

2019

[Background] Own mother’s milk (OMM) is the optimal nutrition for preterm infants. However, pasteurized donor human milk (DHM) is a valid alternative. We explored the differences of the transcriptome in exfoliated epithelial intestinal cells (EEIC) of preterm infants receiving full feed with OMM or DHM.

Male0301 basic medicineDonor milkGene Expressionintestinal cellsmedicine.disease_causeTranscriptome0302 clinical medicinemother’s milkGene expressionInfant Very Low Birth Weightoxidative stressgeneticsProspective StudiesIntestinal Mucosa2. Zero hungerPrincipal Component AnalysisNutrition and DieteticsCaseinsIntestinal cells3. Good healthdonor milkGestationFemalemedicine.symptomPrematuritylcsh:Nutrition. Foods and food supplyInfant PrematureGestational Agelcsh:TX341-641InflammationBiologyArticleAndrology03 medical and health sciences030225 pediatricsMother’s milkGeneticsmedicineHumansGeneInflammationMilk HumanprematurityInfant NewbornNADPH OxidasesEpithelial CellsNeutrophil cytosolic factor 1Low birth weight030104 developmental biologyMilk BanksOxidative stressinflammationCyclooxygenase 1LactalbuminTranscriptomeOxidative stressFood ScienceNutrients
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Sepsis in preterm infants causes alterations in mucosal gene expression and microbiota profiles compared to non-septic twins

2016

Sepsis is a life-threatening condition in preterm infants. Neonatal microbiota plays a pivotal role in the immune system maturation. Changes in gut microbiota have been associated to inflammatory disorders; however, a link with sepsis in the neonatal period has not yet been established. We aimed to analyze gut microbiota and mucosal gene expression using non-invasively obtained samples to provide with an integrative perspective of host-microbe interactions in neonatal sepsis. For this purpose, a prospective observational case-control study was conducted in septic preterm dizygotic twins and their non-septic twin controls. Fecal samples were used for both microbiota analysis and host genome-…

Male0301 basic medicineGene ExpressionInflammationGut floraModels Biologicaldigestive systemArticleTranscriptomesComputational biologySepsis03 medical and health sciencesfluids and secretions0302 clinical medicineImmune systemSepsis030225 pediatricsmedicineHumansGastrointestinal microbiomePrematureBifidobacteriumMucous MembraneMultidisciplinarybiologyNeonatal sepsisGene Expression ProfilingMicrobiotaInfant NewbornInfantMolecular Sequence AnnotationNewbornbiology.organism_classificationmedicine.diseaseBiological marker030104 developmental biologyGene Expression RegulationImmunologyMetagenomeFemaleMetagenomicsAnaerobic bacteriamedicine.symptomTranscriptomeDysbiosisBiomarkersInfant PrematureSignal TransductionScientific Reports
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Physiological relevance of the neuronal isoform of inositol-1,4,5-trisphosphate 3-kinases in mice

2020

Inositol-1,4,5-trisphosphate 3-kinase-A (ITPKA) is the neuronal isoform of ITPKs and exhibits both actin bundling and InsP3kinase activity. In addition to neurons, ITPKA is ectopically expressed in tumor cells, where its oncogenic activity increases tumor cell malignancy. In order to analyze the physiological relevance of ITPKA, here we performed a broad phenotypic screening of itpka deficient mice. Our data show that among the neurobehavioral tests analyzed, itpka deficient mice reacted faster to a hotplate, prepulse inhibition was impaired and the accelerating rotarod test showed decreased latency of itpka deficient mice to fall. These data indicate that ITPKA is involved in the regulatio…

Male0301 basic medicineGene isoformCentral nervous systemMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicinegenetics [Phosphotransferases (Alcohol Group Acceptor)]medicinephysiology [Prepulse Inhibition]AnimalsHumansdeficiency [Phosphotransferases (Alcohol Group Acceptor)]Inositolddc:610Prepulse inhibitionActinMice KnockoutNeuronsenzymology [Neurons]Prepulse InhibitionChemistryKinaseGeneral Neurosciencedeficiency [Isoenzymes]Small intestineCell biologyIsoenzymesPhosphotransferases (Alcohol Group Acceptor)030104 developmental biologymedicine.anatomical_structureCell cultureFemaleCaco-2 Cellsgenetics [Isoenzymes]030217 neurology & neurosurgeryNeuroscience Letters
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