Search results for "Intraepithelial lymphocyte"

showing 5 items of 25 documents

Confocal Endomicroscopy Shows Food-Associated Changes in the Intestinal Mucosa of Patients With Irritable Bowel Syndrome

2014

Background & Aims We investigated suspected food intolerances in patients with irritable bowel syndrome (IBS) using confocal laser endomicroscopy (CLE) for real-time visualization of structural/functional changes in the intestinal mucosa after food challenge. Patients with functional changes after food challenge (CLE+) were placed on personalized exclusion diets and followed up for long-term symptom relief. Methods Thirty-six IBS patients with suspected food intolerance and 10 patients with Barrett's esophagus (controls) without IBS symptoms were examined by CLE at University Hospital Schleswig-Holstein (Kiel, Germany). Diluted food antigens were administered directly to the duodenal mucosa…

chemistry.chemical_classificationmedicine.medical_specialtyPathologyHepatologybusiness.industryfungiGastroenterologyIntervillous spacemedicine.diseaseGastroenterologyFood intoleranceIntestinal mucosaIntoleranceschemistryFood allergyInternal medicinemedicineIntraepithelial lymphocyteFODMAPbusinessIrritable bowel syndromeGastroenterology
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Human γδ T-Cells: From Surface Receptors to the Therapy of High-Risk Leukemias

2018

γδ T lymphocytes are potent effector cells, capable of efficiently killing tumor and leukemia cells. Their activation is mediated by γδ T-cell receptor (TCR) and by activating receptors shared with NK cells (e.g., NKG2D and DNAM-1). γδ T-cell triggering occurs upon interaction with specific ligands, including phosphoantigens (for Vγ9Vδ2 TCR), MICA-B and UL16 binding protein (for NKG2D), and PVR and Nectin-2 (for DNAM-1). They also respond to cytokines undergoing proliferation and release of cytokines/chemokines. Although at the genomic level γδ T-cells have the potential of an extraordinary TCR diversification, in tissues they display a restricted repertoire. Recent studies have identified …

lcsh:Immunologic diseases. Allergy0301 basic medicineαβ T-cellChemokineB-cell depletion; hematopoietic stem cells; HLA-haploidentical transplantation; receptors; αβ T-cell; γδ T-cellsReceptors Antigen T-Cell alpha-betaMini ReviewHLA-haploidentical transplantationImmunologyGenes MHC Class Ichemical and pharmacologic phenomenaMajor histocompatibility complexCD19Mice03 medical and health sciencesγδ T-cellsAntigenReceptorsMHC class ImedicineAnimalsHumansImmunology and AllergyIntraepithelial LymphocytesB-LymphocytesLeukemiaB-cell depletionbiologyT-cell receptorHematopoietic Stem Cell Transplantationmedicine.diseaseNKG2DKiller Cells NaturalLeukemia030104 developmental biologySettore MED/38 - PEDIATRIA GENERALE E SPECIALISTICACytomegalovirus InfectionsImmunologybiology.proteinlcsh:RC581-607Hematopoietic stem cellsFrontiers in Immunology
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IL-12 Expands and Differentiates Human Vγ2Vδ2 T Effector Cells Producing Antimicrobial Cytokines and Inhibiting Intracellular Mycobacterial Growth

2019

While IL-12 plays a key role in differentiation of protective CD4+ Th1 response, little is known about mechanisms whereby IL-12 differentiates other T-cell populations. Published studies suggest that predominant Vγ2Vδ2 T cells in humans/nonhuman primates (NHP) are a fast-acting T-cell subset, with capacities to rapidly expand and produce Th1 and cytotoxic cytokines in response to phosphoantigen (E)-4-hydroxy-3-methyl-but-2-enyl pyrophosphate (HMBPP) produced by Mycobacterium tuberculosis (Mtb) or others. However, whether IL-12 signaling pathway mediates fast-acting and Th1 or anti-microbial features of Vγ2Vδ2 T cells remains poorly defined. Here, we show that IL-12, but not other IL-12 fami…

lcsh:Immunologic diseases. AllergyCells1.1 Normal biological development and functioningproliferationImmunologyLymphocyte ActivationV gamma 2V delta 2 T cellsVaccine Related03 medical and health sciencesPhosphatidylinositol 3-Kinases0302 clinical medicineRare DiseasesUnderpinning researchT-Lymphocyte SubsetsImmunology and AllergyTuberculosis2.1 Biological and endogenous factorsHumansAetiologyIntraepithelial LymphocytesCells Cultured030304 developmental biologyOriginal Researchanti-tuberculosis0303 health sciencesCulturedVγ2Vδ2 T cellsTumor Necrosis Factor-alphaInflammatory and immune systemCorrectionCell DifferentiationMycobacterium tuberculosisdifferentiationSTAT4 Transcription FactorTh1 CellsInterleukin-12Organophosphates3. Good healthInfectious DiseasesGood Health and Well BeingMedical MicrobiologyIL-12Infectionlcsh:RC581-607Proto-Oncogene Proteins c-akt030215 immunologySignal TransductionFrontiers in Immunology
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Minimal Lesions of the Small Intestinal Mucosa: More than Morphology

2020

Minimal lesions of the small bowel are mucosal changes characterized by an increased number of intraepithelial lymphocytes (with or without crypt hyperplasia) and normal villous architecture. Such changes are associated with a wide spectrum of conditions, ranging from food intolerances to infections, and from drugs to immune diseases, with different clinical profiles and manifestations, which complicates the formulation of a differential diagnosis. Patient history, symptom evaluation, and histopathology are the diagnostic features needed to establish a correct diagnosis. Physicians should assist pathologists in formulating a precise morphological evaluation by taking well-oriented small int…

medicine.medical_specialtyPathologySettore MED/09 - Medicina InternaPhysiologyBiopsyDiseaseWheat HypersensitivityMucosal enteropathiesNONon-celiac gluten/wheat sensitivityDiagnosis Differential03 medical and health sciencesIntra-epithelial lymphocytes0302 clinical medicineIntolerancesPredictive Value of TestsRisk FactorsInternal medicineIntestine SmallmedicineHumansCeliac diseaseMedical historyIntestinal MucosaIntraepithelial LymphocytesImmunoglobulin A-tranglutaminase 2 depositiHyperplasiaImmunoglobulin A-tranglutaminase 2 depositisbusiness.industryGastroenterologyPotential celiac diseaseMucosal enteropathieHepatologySmall intestineIntra-epithelial lymphocytemedicine.anatomical_structureCeliac disease Immunoglobulin A-tranglutaminase 2 depositis Intra-epithelial lymphocytes Mucosal enteropathies Non-celiac gluten/wheat sensitivity Potential celiac disease030220 oncology & carcinogenesisIntraepithelial lymphocyte030211 gastroenterology & hepatologyHistopathologyDifferential diagnosisbusiness
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The production of the oral mucosa of antiendomysial and anti-tissue-transglutaminase antibodies in patients with celiac disease: a review.

2010

Celiac disease (CD) is a lifelong, T cell—mediated enteropathy, triggered by the ingestion of gluten and related prolamins in genetically susceptible subjects, resulting in minor intestinal mucosal injury, including villous atrophy with crypt hyperplasia and intraepithelial lymphocytosis, and subsequent nutrient malabsorption. Although serological tests for antiendomysial (EMA) and anti—tissue transglutaminase (anti-tTG) autoantibodies are used to screen and follow up on patients with CD, diagnostic confirmation is still based on the histological examination of the small intestinal mucosa. Although the small intestinal mucosa is the main site of the gut involved in CD, other mucosal surface…

medicine.medical_specialtySettore MED/09 - Medicina InternaMalabsorptionGlutensTissue transglutaminaseBiopsyantiendomysial antibodieslcsh:Medicineoral biopsyReview Articlelcsh:TechnologyGastroenterologySensitivity and SpecificityGeneral Biochemistry Genetics and Molecular BiologySettore MED/28 - Malattie Odontostomatologicheanti–tissue transglutaminase antibodiesInternal medicineBiopsymedicineHumansEnteropathyOral mucosalcsh:ScienceGeneral Environmental ScienceAutoantibodiesSettore MED/12 - GastroenterologiaGastrointestinal tractTransglutaminasesbiologymedicine.diagnostic_testoral mucosalcsh:Tbusiness.industrylcsh:RMouth MucosaMuscle SmoothGeneral Medicinemedicine.diseaseCeliac Diseasemedicine.anatomical_structureceliac disease oral mucosa anti–tissue transglutaminase antibodies antiendomysial antibodies oral biopsy.Immunologybiology.proteinIntraepithelial lymphocytelcsh:QGliadinbusinessTheScientificWorldJournal
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