Search results for "Iontophoresi"

showing 10 items of 69 documents

Simultaneous controlled iontophoretic delivery of pramipexole and rasagiline in vitro and in vivo: Transdermal polypharmacy to treat Parkinson's dise…

2018

[EN] Effective treatment of Parkinson's disease (PD) involves administration of therapeutic agents with complementary mechanisms of action in order to replenish, sustain or substitute endogenous dopamine. The objective of this study was to investigate anodal co-iontophoresis of pramipexole (PRAM; dopamine agonist) and rasagiline (RAS; MAO-B inhibitor) in vitro and in vivo. Passive permeation of PRAM and RAS (20 mM each) across porcine skin after 6 h was 15.7 +/- 1.9 and 16.0 +/- 2.9 mu g/cm(2), respectively. Co-iontophoresis at 0.15, 0.3 and 0.5 mA/cm(2) resulted in statistically significant increases in delivery of PRAM and RAS; at 0.5 mA/cm(2), cumulative permeation of PRAM and RAS was 61…

MaleParkinson's diseaseSwineChemistry PharmaceuticalSkin AbsorptionPharmaceutical SciencePharmacologyAdministration Cutaneous030226 pharmacology & pharmacyDopamine agonistPermeability03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePramipexolePharmacokineticsIn vivomedicineAnimalsHumansBenzothiazolesMAO-B inhibitorRats WistarTransdermalSkinRasagilinePramipexoleIontophoresisDopamine agonistPatient complianceParkinson DiseaseGeneral MedicineIontophoresismedicine.diseaseRatschemistryIndansPolypharmacyElectroosmosisTransdermal030217 neurology & neurosurgeryBiotechnologymedicine.drugEuropean journal of pharmaceutics and biopharmaceutics : official journal of Arbeitsgemeinschaft fur Pharmazeutische Verfahrenstechnik e.V
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Modulation of in vivo GABA-evoked responses by nitric oxide-active compounds in the globus pallidus of rat.

2012

Nitric oxide (NO) is a gaseous molecule acting as a messenger in both the peripheral and the central nervous systems. NO affects synaptic activity by modulating neurotransmitter release and/or receptor function. We previously observed that NO-active compounds modify the bioelectric activity of basal ganglia (BG) units. In this study, we applied microiontophoresis to extracellular in vivo recordings to investigate the effect of NO-active compounds on GABA-evoked responses in the globus pallidus (GP) of anesthetized rats. The changes induced by NO-active drugs on the GABA-induced inhibition were used as indicators of NO modulation. The response to GABA release was tested on recorded GP neuron…

MalePharmacologyBiologyNeurotransmissionGlobus PallidusNitric OxideSettore BIO/09 - FisiologiaSynaptic Transmissiongamma-Aminobutyric acidNitric oxidechemistry.chemical_compoundIn vivomedicineAnimalsRats WistarNeurotransmitterEvoked PotentialsBiological Psychiatrygamma-Aminobutyric AcidNeuronsNitric oxide Basal ganglia Globus pallidus Microiontophoresis GABA transmissionIontophoresisRatsNitric oxide synthaseElectrophysiologyPsychiatry and Mental healthElectrophysiologyGlobus pallidusnervous systemNeurologychemistrybiology.proteinNeurology (clinical)Neurosciencemedicine.drugJournal of neural transmission (Vienna, Austria : 1996)
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Indicaxanthin from Opuntia ficus-indica Crosses the Blood–Brain Barrier and Modulates Neuronal Bioelectric Activity in Rat Hippocampus at Dietary-Con…

2015

Indicaxanthin is a bioactive and bioavailable betalain pigment from the Opuntia ficus-indica fruits. In this in vivo study, kinetic measurements showed that indicaxanthin is revealed in the rat brain within 1 h from oral administration of 2 μmol/ kg, an amount compatible with a dietary consumption of cactus pear fruits in humans. A peak (20 ± 2.4 ng of indicaxanthin per whole brain) was measured after 2.5 h; thereafter the molecule disappeared with first order kinetics within 4 h. The potential of indicaxanthin to affect neural activities was in vivo investigated by a microiontophoretic approach. Indicaxanthin, administered in a range between 0.085 ng and 0.34 ng per neuron, dose-dependentl…

MalePyridinesHippocampusPharmacologyBiologyHippocampal formationBlood–brain barrierInhibitory postsynaptic potentialHippocampuschemistry.chemical_compoundSettore BIO/10 - BiochimicamedicineAnimalsRats WistarNeuronsGlutamate receptorOpuntiaGeneral Chemistryindicaxanthin phytochemicals BBB electrophysiology hippocampus microiontophoresis molecular modelingBetaxanthinsElectrophysiologymedicine.anatomical_structureReceptors GlutamateBiochemistrychemistryBlood-Brain BarrierNMDA receptorNeuronGeneral Agricultural and Biological SciencesIndicaxanthinJournal of Agricultural and Food Chemistry
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Lateral habenula and hippocampal units: electrophysiological and iontophoretic study

1995

In previous works we studied, on cats, the effects of lateral habenula (LH) stimulation on hippocampal units. In particular, the results showed an excitation or an inhibition in relation to the stimulation frequency (0.5-3.0 Hz or 5.0-20 Hz, respectively). All the LH stimulation effects were antagonised by iontophoretic intrahippocampal application of methysergide (MS). In this series of experiments it was possible to demonstrate, on rats, that LH stimulation causes an excitatory effect in a major number of hippocampal units in relation to the frequency increase. The inhibitory effect by iontophoretic serotonine application and the reversible blockade of habenular modulation after iontophor…

MaleSerotoninN-MethylaspartateMethysergideStimulationHippocampal formationHippocampusMicromanipulationDorsal raphe nucleusThalamusmedicineAnimalsRats WistarNeuronsAnalysis of VarianceRapheMethysergideChemistryGeneral NeuroscienceIontophoresisElectric StimulationRatsHabenulaExcitatory postsynaptic potentialRaphe NucleiRaphe nucleiNeurosciencemedicine.drugBrain Research Bulletin
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Electrophysiological and Iontophoretic Aspects of the Habenular Influence on Hippocampal Neurones

1995

In previous experimental studies, carried out on cats, we demonstrated that electrical stimulation of lateral habenula (LH) at 0.5-3.0 Hz or 5-20 Hz had a double effect (low frequency-excitation; high frequency-inhibition) on the spontaneous firing rate of single hippocampal neurones. Our results, in agreement with similar case studies, allowed us to hypothesise that in the habenular modulation of the hippocampus the raphe nucleus is probably involved. In fact, all the effects of LH stimulation were antagonised by the iontophoretic intrahippocampal application of methysergide. In the present series of experiments, performed on rats, it was possible to demonstrate that LH stimulation at 1-10…

MaleSerotoninN-MethylaspartatePhysiologyMethysergideHippocampusStimulationHippocampal formationHippocampusPhysiology (medical)medicineAnimalsRats WistarNeuronsIontophoresisMethysergideChemistryGeneral MedicineIontophoresisElectric StimulationCochleaRatsElectrophysiologyElectrophysiologyRaphe NucleiNMDA receptorRaphe nucleiNeurosciencemedicine.drugArchives of Physiology and Biochemistry
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Nitric oxide- and cGMP-active compounds affect the discharge of substantia nigra pars reticulata neurons: in vivo evidences in the rat

2009

The nitric oxide (NO)-active drugs influence on the bioelectric activity of neurons of the pars reticulata of the substantia nigra was studied in urethane-anesthetized rats. A first group of animals was treated with 7-nitro-indazole (7-NI), a preferential inhibitor of neuronal NO synthase. In a second group of rats, electrophysiological recordings were coupled with microiontophoretic administration of Nomega-nitro-L-arginine methyl ester (L-NAME, a NO synthase inhibitor), 3-morpholino-sydnonimin-hydrocloride (SIN-1, a NO donor) and 8-Br-cGMP (a cell-permeable analogue of cGMP, the main second-messenger of NO neurotransmission). 7-NI and L-NAME caused a statistically significant decrease in …

MaleSubstantia nigra pars reticulataAction PotentialsDown-RegulationSubstantia nigraNitric Oxide Synthase Type INeurotransmissionPharmacologyBiologySettore BIO/09 - FisiologiaNitric oxidechemistry.chemical_compoundIn vivoAnimalsSingle unit electrophysiologyNitric Oxide DonorsEnzyme InhibitorsRats WistarCyclic GMPBiological PsychiatrySubstantia nigra pars reticulataNeuronsMicroiontophoresisNeural InhibitionNitric oxideIontophoresisRatsUp-RegulationSubstantia NigraPsychiatry and Mental healthElectrophysiologyNG-Nitroarginine Methyl EsterNeurologychemistryMolsidomineExcitatory postsynaptic potentialNeurology (clinical)Pars reticulataNeuroscienceSignal TransductionJournal of Neural Transmission
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Controlled Iontophoretic Delivery in Vitro and in Vivo of ARN14140—A Multitarget Compound for Alzheimer’s Disease

2019

ARN14140 is a galantamine-memantine conjugate that acts upon both cholinergic and glutamatergic pathways for better management of Alzheimer's disease. Poor oral bioavailability and pharmacokinetics meant that earlier preclinical in vivo studies employed intracerebroventricular injection to administer ARN14140 directly to the brain. The aim of the present study was to evaluate the feasibility of using constant current transdermal iontophoresis for the noninvasive systemic delivery of ARN14140 and to quantify the amounts present in the blood and the brain. Preliminary experiments in vitro were performed using porcine skin and validated with human skin. Cumulative ARN14140 permeation across th…

MaleSwineSkin Absorptionbrain deliveryBiological AvailabilityPharmaceutical ScienceHuman skin02 engineering and technologyPharmacologyAdministration Cutaneous030226 pharmacology & pharmacyPermeability03 medical and health sciences0302 clinical medicineDrug StabilityPharmacokineticsIn vivoDrug DiscoveryARN14140AnimalsBrain/metabolismHumansSkin/metabolismMedicineTissue DistributionRats WistarNootropic Agents/administration & dosage/pharmacokineticsTransdermalddc:615galantamine-memantine conjugateAlzheimer Disease/drug therapyIontophoresisbusiness.industryGalantamine/administration & dosage/pharmacokineticsiontophoresiIontophoresisMemantine/administration & dosage/pharmacokinetics021001 nanoscience & nanotechnologyIn vitroRatsBioavailabilityHeterocyclic Compounds 4 or More Rings/administration & dosage/pharmacologytransdermalFeasibility StudiesMolecular MedicineCholinergic0210 nano-technologybusinessMolecular Pharmaceutics
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Effects of nitric oxide-active drugs on the discharge of subthalamic neurons: microiontophoretic evidence in the rat.

2006

The presence of nitric oxide (NO) synthase and of soluble guanylyl cyclase, the main NO-activated metabolic pathway, has been demonstrated in many cells of the subthalamic nucleus. In this study, the effects induced on the firing of 96 subthalamic neurons by microiontophoretically administering drugs modifying NO neurotransmission were explored in anaesthetized rats. Recorded neurons were classified into regularly and irregularly discharging on the basis of their firing pattern. Nω-nitro-l-arginine methyl ester (L-NAME; a NO synthase inhibitor), 3-morpholino-sydnonimin-hydrocloride (SIN-1; a NO donor), S-nitroso-glutathione (SNOG; another NO donor) and 8-Br-cGMP (a cell-permeable analogue o…

MaleTime FactorsAction PotentialsNeurotransmissionInhibitory postsynaptic potentialNitric OxideSettore BIO/09 - FisiologiaNitric oxideS-Nitrosoglutathionechemistry.chemical_compoundSubthalamic NucleusAnimalsNitric Oxide DonorsEnzyme InhibitorsRats WistarCyclic GMPNeuronsAnalysis of VarianceIontophoresisDose-Response Relationship DrugChemistryGeneral Neuroscience8-Br-cGMP L-NAME SIN-1 SNOG subthalamic nucleusIontophoresisThionucleotidesRatsEnzyme ActivationSubthalamic nucleusNG-Nitroarginine Methyl EsterMolsidomineS-NitrosoglutathioneExcitatory postsynaptic potentialSoluble guanylyl cyclaseNeuroscience
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Transdermal iontophoresis of dexamethasone sodium phosphate in vitro and in vivo: effect of experimental parameters and skin type on drug stability a…

2010

The aim of this study was to investigate the cathodal iontophoresis of dexamethasone sodium phosphate (DEX-P) in vitro and in vivo and to determine the feasibility of delivering therapeutic amounts of the drug for the treatment of chemotherapy-induced emesis. Stability studies, performed to investigate the susceptibility of the phosphate ester linkage to hydrolysis, confirmed that conversion of DEX-P to dexamethasone (DEX) upon exposure to samples of human, porcine and rat dermis for 7 h was limited (82.2+/-0.4%, 72.5+/-4.8% and 78.6+/-6.0% remained intact) and did not point to any major inter-species differences. Iontophoretic transport of DEX-P across dermatomed porcine skin (0.75 mm thic…

MaleTime FactorsVomitingSwineSkin AbsorptionPharmaceutical ScienceAntineoplastic AgentsPharmacologyAdministration CutaneousHigh-performance liquid chromatographyDexamethasoneGlucocorticoids/administration & dosage/pharmacokineticsDexamethasone Sodium PhosphatePharmacokineticsDrug StabilitySpecies SpecificityIn vivoAnimalsHumansSkin/metabolismVomiting/chemically induced/prevention & controlRats WistarGlucocorticoidsTransdermalSkinddc:615IontophoresisDose-Response Relationship DrugChemistryHydrolysisGeneral MedicineAntineoplastic Agents/adverse effectsPermeationIontophoresisRatsDose–response relationshipDexamethasone/administration & dosage/analogs & derivatives/pharmacokineticsBiotechnologyNuclear chemistry
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Nitric oxide-induced inhibition on striatal cells and excitation on globus pallidus neurons: a microiontophoretic study in the rat.

2003

Single units were recorded in the striatum and in the globus pallidus (GP) of urethane-anesthetized rats under microiontophoretic administration of either Nomega-nitro-L-arginine methyl ester (L-NAME, inhibitor of nitric oxide synthase), or 3-morpholino-sydnonimin-hydrocloride (SIN-1, nitric oxide, NO donor). A steady baseline firing of sporadically discharging striatal neurons (basal firing rate <0.1 spikes/s) was evoked by a pulsed microiontophoretic ejection of glutamate. On striatal neurons, microiontophoretic application of SIN-1 induced a current-dependent inhibition (11/13), whereas L-NAME administration produced a clear excitation (9/9). On GP cells, the administration of SIN-1 had …

MaleVasodilator AgentsAction PotentialsGlutamic AcidStriatumBiologyPharmacologyGlobus PallidusNitric OxideNitric oxidechemistry.chemical_compoundBasal gangliaPremovement neuronal activityAnimalsEnzyme InhibitorsRats WistarDose-Response Relationship DrugGeneral NeuroscienceGlutamate receptorIontophoresisCorpus StriatumRatsNitric oxide synthaseGlobus pallidusNG-Nitroarginine Methyl Esternervous systemchemistryMolsidomineExcitatory postsynaptic potentialbiology.proteinNeuroscienceNeuroscience letters
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