Search results for "Isoenzyme"

showing 10 items of 247 documents

Clinical manifestations of Fabry disease in children: data from the Fabry Outcome Survey.

2006

Background Fabry disease is a rare X-linked disorder caused by deficient activity of the enzyme alpha-galactosidase A. This produces progressive lysosomal accumulation of globotriaosylceramide throughout the body, leading to organ failure and premature death. Aim Here, we present the clinical manifestations of Fabry disease in children enrolled in FOS--the Fabry Outcome Survey--a European database of the natural history of Fabry disease and the effects of enzyme replacement therapy with agalsidase alfa (Replagal). Methods Currently, there are 545 patients in FOS, from 11 European countries. We analysed the baseline demographic and clinical characteristics of 82 of these patients (40 boys, 4…

Malemedicine.medical_specialtyAbdominal painPediatricsHeterozygoteAdolescentDNA Mutational AnalysisGlobotriaosylceramidechemistry.chemical_compoundOutcome Assessment Health CaremedicineHumansAge of OnsetChildStrokebusiness.industryVascular diseaseGeneral MedicineEnzyme replacement therapymedicine.diseaseFabry diseaseRecombinant ProteinsSurgeryAngiokeratomaIsoenzymeschemistryChild Preschoolalpha-GalactosidasePediatrics Perinatology and Child HealthFabry DiseaseFemaleAge of onsetmedicine.symptombusinessActa paediatrica (Oslo, Norway : 1992)
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Prognostic information of glycogen phosphorylase isoenzyme BB in patients with suspected acute coronary syndrome.

2012

Early and adequate risk stratification is essential in patients with suspected acute coronary syndrome (ACS). The aim of the present study was to investigate whether glycogen phosphorylase BB (GPBB) could add prognostic information in the context of contemporary sensitive troponin I determination and B-type natriuretic peptide (BNP). Patients with suspected ACS were consecutively enrolled at 3 German study centers from January 2007 through December 2008. Troponin I, GPBB, and BNP were determined at admission. Follow-up information on the combined end point of death, myocardial infarction, revascularization, and hospitalization owing to a cardiovascular cause was obtained 6 months after enro…

Malemedicine.medical_specialtyAcute coronary syndromeChest PainMyocardial InfarctionKaplan-Meier EstimateGlycogen phosphorylase isoenzyme BBChest painRisk AssessmentSensitivity and SpecificitySeverity of Illness IndexCohort StudiesTroponin TGlycogen Phosphorylase Brain FormPredictive Value of TestsInternal medicineTroponin INatriuretic Peptide BrainMedicineHumansMyocardial infarctionAngina UnstableAcute Coronary SyndromeAgedTroponin Tbusiness.industryUnstable anginaHazard ratioMiddle Agedmedicine.diseasePrognosisSurvival AnalysisCase-Control StudiesCardiologyFemalemedicine.symptomCardiology and Cardiovascular MedicinebusinessBiomarkersThe American journal of cardiology
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Upregulation of Phospholipase D Expression and Activation in Ventricular Pressure-Overload Hypertrophy

2005

Evidence for a role of phospholipase D (PLD) in cellular proliferation and differentiation is accumulating. We studied PLD activity and expression in normal and hypertrophic rat and human hearts. In rat heart, abdominal aortic banding (constriction to 50% of original lumen) caused hypertrophy in the left ventricle (as shown by weight index and ANP expression) by about 15% after 30 days without histological evidence of fibrosis or signs of decompensation and in the right ventricle after 100 days. The hypertrophy was accompanied by small increases of basal PLD activity and strong potentiation of stimulated PLD activity caused by 4β-phorbol-12β,13α-dibutyrate (PDB) and by phenylephrine. The mR…

Malemedicine.medical_specialtyBlotting WesternCardiomegalyBiologyGene Expression Regulation EnzymologicMuscle hypertrophyRats Sprague-DawleyDownregulation and upregulationInternal medicinePhospholipase DVentricular PressuremedicineAnimalsHumansRNA MessengerPhenylephrineProtein Kinase CProtein kinase CPharmacologyReverse Transcriptase Polymerase Chain ReactionPhospholipase DPLD2lcsh:RM1-950Body WeightRatsReceptors AdrenergicUp-RegulationEnzyme ActivationIsoenzymeslcsh:Therapeutics. Pharmacologymedicine.anatomical_structureEndocrinologyVentricleVentricular pressureMolecular Medicinelipids (amino acids peptides and proteins)Signal Transductionmedicine.drugJournal of Pharmacological Sciences
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Captopril does not affect plasma endothelin-1 during thrombolysis and reperfusion.

1995

Studies showed that endothelin-1 (ET-1) was increased in the acute myocardial infarction (AMI). Experimental studies reported that captopril was able to reduce ET-1 secretion, and that ET-1 was increased during reperfusion. This study was aimed to verify if captopril was able to reduce plasma ET-1 during thrombolysis in AMI. Seventy-three patients, hospitalized for suspected AMI within 4 h from the onset of symptoms suitable for thrombolysis (1st episode), Killip class 1-2, were randomized (double blind) into two groups: group 1 (37 pts), 8 F/29 M, received captopril, 6.25 mg, orally 15 min before thrombolysis. Group 2: (36 pts) 8 F/28 M, received placebo before thrombolysis. All patients m…

Malemedicine.medical_specialtyCaptoprilTime Factorsmedicine.medical_treatmentMyocardial InfarctionAdministration OralAngiotensin-Converting Enzyme InhibitorsBlood PressureMyocardial ReperfusionPlaceboAnginaPlacebosElectrocardiographyDouble-Blind MethodHeart RateInternal medicineFibrinolysismedicineHumansThrombolytic TherapyMyocardial infarctionAngina UnstableCreatine KinaseKillip classbusiness.industryUnstable anginaEndothelinsCaptoprilThrombolysismedicine.diseaseRecombinant ProteinsSurgeryIsoenzymesTissue Plasminogen ActivatorCardiologyFemaleCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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Differential stabilization of cytochrome P-450 isoenzymes in primary cultures of adult rat liver parenchymal cells.

1991

Cytochrome P-450 dependent hydroxylation of testosterone was measured in 7-day-old cultures of primary rat liver parenchymal cells. Determinations were carried out in monocultures of parenchymal cells and co-cultures of parenchymal cells with rat liver nonparenchymal epithelial cells, or mouse embryo fibroblasts. In the monoculture system, testosterone metabolism was drastically reduced and hardly measurable after 7 days in culture. In the co-culture systems, individual P-450 isoenzymes were stabilized on different levels. P-450s p and presumably c were well preserved, P-450 a was reduced but clearly measurable, P-450 h was totally lost whereas P-450s b and e were not measurable after 7 day…

Malemedicine.medical_specialtyClinical BiochemistryHydroxylationHydroxylationchemistry.chemical_compoundMiceCytochrome P-450 Enzyme SystemInternal medicineParenchymaEnzyme StabilitymedicineAnimalsTestosteroneFibroblastCells CulturedMice Inbred C3HbiologyCytochrome P450Rats Inbred StrainsCell BiologyGeneral MedicineFibroblastsEmbryo MammalianRatsIsoenzymesmedicine.anatomical_structureEndocrinologychemistryLiverCell cultureHepatocyteCollagenasebiology.proteinStem cellDevelopmental Biologymedicine.drugIn vitro cellulardevelopmental biology : journal of the Tissue Culture Association
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The effect of postsurgical administration of a selective cyclo-oxygenase-2 inhibitor on the healing of intrabony defects following treatment with ena…

2003

Regenerative treatment with enamel matrix proteins (EMD) has been shown to promote regeneration in intrabony periodontal defects. However, up to now various postoperative regimens such as the routine administration of nonsteroidal anti-inflammatory drugs (NSAIDs) were often used in combination with enamel matrix proteins. Therefore, it cannot be excluded that the results might have been influenced by the effect of the postoperative medication. The aim of this randomized, controlled, blinded, clinical investigation was to determine the effect of postsurgical administration of a selective cyclo-oxygenase-2 inhibitor on the healing of intrabony periodontal defects following regenerative period…

Malemedicine.medical_specialtyGingival and periodontal pocketBleeding on probingUrologyAlveolar Bone LossDentistryLactonesDental Enamel ProteinsOral administrationPeriodontal Attachment LossmedicineHumansPeriodontal PocketCyclooxygenase InhibitorsGingival RecessionSingle-Blind MethodSulfonesGeneral DentistryGingival recessionRofecoxibWound HealingCyclooxygenase 2 Inhibitorsbusiness.industryAnti-Inflammatory Agents Non-SteroidalDental Plaque IndexMembrane ProteinsIsoenzymesRegimenClinical attachment lossCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesSystemic administrationGuided Tissue Regeneration PeriodontalFemalemedicine.symptomPeriodontal Indexbusinessmedicine.drugClinical oral investigations
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Selective induction of bilirbuin UDP-glucuronosyl-transferase by perfluorodecanoic acid

1991

Differential effects of perfluorodecanoic acid (PFDA) on rat liver UDP-glucuronosyltransferase isoenzymes have been observed after a single i.p. administration of the compound to young male Sprague-Dawley rats. (1) Bilirubin glucuronidation was induced 2-fold. The induced state was stable for at least 3 weeks. (2) Glucuronidation of 1-naphthol, morphine and testosterone was decreased to half of the control values. These decreases were maximal after 12 days but all three activities returned to normal levels after 3 weeks. (3) Immunoblotting experiments indicated that the differential effects of PFDA on UDP-glucuronosyltransferase activities were due to modulation of enzyme protein concentrat…

Malemedicine.medical_specialtyGlucuronosyltransferaseBilirubinImmunoblottingGlucuronidationToxicologyIsozymechemistry.chemical_compoundInternal medicinemedicineAnimalsInducerGlucuronosyltransferaseEnzyme inducerTestosteroneFluorocarbonsDose-Response Relationship DrugbiologyRats Inbred StrainsGeneral MedicineRatsIsoenzymesEndocrinologychemistryEnzyme InductionToxicitybiology.proteinDecanoic AcidsChemico-Biological Interactions
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New bundle branch block after coronary artery bypass grafting— evaluation by CK-MB isoenzyme analysis and transoesophageal echocardiography

1990

Twelve patients with a new complete bundle branch block after coronary artery bypass grafting underwent transoesophageal echocardiography (TEE). The results of TEE were compared with the pre-operative ventriculography, CK-MB isoenzyme time-release curves and clinical course. In eight patients with transient right bundle branch block or bifascicular block, low CK-MB activities and an uncomplicated postoperative course, transoesophageal echocardiography showed no new segmental wall motion abnormalities apart from a paradoxical septal movement in five. A persistent right or left bundle branch block was associated with either elevated isoenzyme activities, transoesophageal echocardiographic evi…

Malemedicine.medical_specialtyHeart VentriclesBundle-Branch BlockBifascicular blockElectrocardiographyCoronary artery bypass surgeryInternal medicinemedicineHumansCoronary Artery BypassCreatine KinaseAgedBundle branch blockmedicine.diagnostic_testbusiness.industryLeft bundle branch blockMiddle AgedRight bundle branch blockmedicine.diseaseMyocardial ContractionElectrocardiographic FindingIsoenzymesmedicine.anatomical_structureEchocardiographyCardiologyFemaleCardiology and Cardiovascular MedicinebusinessElectrocardiographyFollow-Up StudiesArteryEuropean Heart Journal
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Differential effect of hypophysectomy and growth hormone treatment on hepatic glucuronosyltransferases in male rats: Evidence for an action at a pret…

1997

International audience; The influence of growth hormone (GH) on 4-nitrophenol, bilirubin, testosterone, androsterone and estrone glucuronidation activities was studied in fully activated male rat hepatic microsomes. Sham-operated and hypophysectomized animals were injected with two different dosages of GH, mimicking either the male or female GH secretion pattern. Half the animals received thyroxine and cortisol in concentrations chosen to compensate for the lack of thyroid hormones and glucocorticoids in hypophysectomized rats. GH induced a decrease in several glucuronidation activities: bilirubin glucuronidation in both sham-operated and cortisol/ thyroxine-treated hypophysectomized rats i…

Malemedicine.medical_specialtyHypophysectomyGlucuronosyltransferaseHydrocortisoneBilirubin[SDV]Life Sciences [q-bio]medicine.medical_treatmentImmunoblottingGlucuronidationEstronePolymerase Chain ReactionBiochemistryRats Sprague-DawleyRat Biochimie03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicineAnimalsRNA MessengerGlucuronosyltransferaseObjet d'étude : rattus rattus ; foie Composé chimique Facteur du milieu : transférase ; hormone de croissance Dispositif technique et méthode d'étude : médicament ; hypophysectomie Phénomène processus et fonction : pulsatilitéTestosteroneHypophysectomy030304 developmental biologyPharmacology0303 health sciencesAndrosteronebiologyDiscriminant AnalysisBilirubinGrowth hormone secretionRats[SDV] Life Sciences [q-bio]IsoenzymesThyroxineEndocrinologychemistryGrowth HormoneProtein Biosynthesis030220 oncology & carcinogenesisMicrosomes Liverbiology.proteinBiochemical Pharmacology
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Genetic deficiency of tartrate-resistant acid phosphatase associated with skeletal dysplasia, cerebral calcifications and autoimmunity

2010

Vertebral and metaphyseal dysplasia, spasticity with cerebral calcifications, and strong predisposition to autoimmune diseases are the hallmarks of the genetic disorder spondyloenchondrodysplasia. We mapped a locus in five consanguineous families to chromosome 19p13 and identified mutations in ACP5, which encodes tartrate-resistant phosphatase (TRAP), in 14 affected individuals and showed that these mutations abolish enzyme function in the serum and cells of affected individuals. Phosphorylated osteopontin, a protein involved in bone reabsorption and in immune regulation, accumulates in serum, urine and cells cultured from TRAP-deficient individuals. Case-derived dendritic cells exhibit an …

Malemedicine.medical_specialtyLymphocyteT cellAcid PhosphatasePhosphataseAutoimmunityOsteochondrodysplasiasmedicine.disease_causeBone and BonesAutoimmune DiseasesAutoimmunity03 medical and health sciences0302 clinical medicineInternal medicineGeneticsmedicineHumansGenetic Predisposition to DiseaseOsteopontinPhosphorylationChild030304 developmental biologyTartrate-resistant acid phosphatase030203 arthritis & rheumatologyBone Diseases Developmental0303 health sciencesbiologyTartrate-Resistant Acid PhosphataseHomozygoteBrainMetaphyseal dysplasiamedicine.disease3. Good healthIsoenzymesRadiographymedicine.anatomical_structureEndocrinologyDysplasiaMutationbiology.proteinCalciumOsteopontinNature Genetics
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