Search results for "Isoprenaline"

showing 10 items of 48 documents

Adrenoceptor- and cholinoceptor-mediated mechanisms in the regulation of 5-hydroxytryptamine release from isolated tracheae of newborn rabbits

1996

Abstract 1. Isolated tracheae of newborn rabbits were incubated in vitro and the outflow of 5-hydroxytryptamine (5-HT) was determined by h.p.l.c. with electrochemical detection. Evidence has previously been provided that this 5-HT outflow derives from neuroendocrine epithelial (NEE) cells of the airway mucosa. 2. Phenylephrine (1, 10 and 30 microM) enhanced the outflow of 5-HT by 80, 290 and 205%, respectively. 5-HT outflow evoked by 10 microM phenylephrine was not affected by the presence of the neurotoxin tetrodotoxin (1 microM). 3. Rauwolscine, ARC 239 (an alpha(2B)-adrenoceptor preferring antagonist), yohimbine and prazosin antagonized the effect of 10 microM phenylephrine in a concentr…

MaleSerotoninmedicine.medical_specialtyRauwolscineIn Vitro TechniquesMuscarinic Agonistschemistry.chemical_compoundIsoprenalineInternal medicineReceptors Adrenergic betaCyclic AMPmedicinePrazosinAnimalsReceptors CholinergicPhenylephrinePharmacologyForskolinMuscarineHydroxyindoleacetic AcidReceptors AdrenergicYohimbineTracheaEndocrinologyAnimals NewbornchemistryFemaleHexamethoniumRabbitsResearch Articlemedicine.drugBritish Journal of Pharmacology
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Effects of beta-adrenoceptor drug stimulation on various models of gastric ulcer in rats.

1982

Abstract 1. Experiments were designed to evaluate the effect of the pharmacological activation of beta-adrenoceptors on various models of gastric ulcer in the rat. 2. Pretreatment with the beta-adrenoceptor stimulant drugs, isoprenaline or salbutamol, significantly inhibited stress-induced gastric ulcers. This anti-ulcer effect was abolished by propranolol but not by atenolol, suggesting that beta 2-adrenoceptors mediate this response. 3. In the pylorus-ligation model, salbutamol inhibited lesion formation and reduced the intragastric content of hydrogen ions, histamine and pepsin although the latter was only affected with the higher dose of salbutamol. 4. Salbutamol also prevented the ulce…

MaleTime FactorsStimulationPropranololPharmacologychemistry.chemical_compoundIsoprenalinemedicineGastric mucosaAnimalsHumansAlbuterolStomach UlcerPylorusPharmacologybusiness.industryIsoproterenolRats Inbred StrainsReserpineAdrenergic beta-AgonistsAtenololdigestive system diseasesRatsmedicine.anatomical_structurechemistryGastric MucosaAnesthesiaSalbutamolbusinessHistamineStress Psychologicalmedicine.drugResearch ArticleBritish journal of pharmacology
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Reduced basal and stimulated (isoprenaline, Gpp(NH)p, forskolin) adenylate cyclase activity in Alzheimer's disease correlated with histopathological …

1991

Cyclic adenosine monophosphate (cAMP) is an adenylate cyclase borne second messenger involved in basic metabolic events. The beta-adrenoceptor sensitive adenylate cyclase was studied in post-mortem hippocampi of controls and Alzheimer patients. Virtually identical subsets of each hippocampus homogenate were stimulated by 100 mumol isoprenaline, Gpp(NH)p and forskolin, respectively, in presence of an ATP-regenerating system. The determination of cAMP formed was carried out by means of a radioassay. The observed significant 50% reduction in basal as well as in stimulated adenylate cyclase activity in Alzheimer's disease is negatively correlated with semiquantitative evaluations of amyloid pla…

Malemedicine.medical_specialtyAdenylate kinaseCyclasechemistry.chemical_compoundAlzheimer DiseaseReference ValuesInternal medicineIsoprenalinemedicineHumansCyclic adenosine monophosphateSenile plaquesMolecular BiologyAgedAged 80 and overGuanylyl ImidodiphosphateForskolinChemistryGeneral NeuroscienceColforsinIsoproterenolBrainKineticsEndocrinologyPostmortem ChangesSecond messenger systemFemaleNeurology (clinical)Cyclase activityAdenylyl CyclasesDevelopmental Biologymedicine.drugBrain Research
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Pronounced cholinergic but only moderate purinergic effects in isolated atrial and ventricular heart muscle from cats.

1989

1 The effects of cholinergic and purinergic stimulation on action potential, force of contraction and 86Rb efflux were investigated in cat atrial and/or ventricular heart muscle. 2 Acetylcholine and carbachol exerted a concentration-dependent negative inotropic effect in cat atrial heart muscle. Carbachol 10 μmol l−1 completely abolished the force of contraction and increased the rate constant of 86Rb efflux 2–3 fold, whereas the action potential duration was shortened to about 1/10 of its length under control conditions. 3 The effects of acetylcholine and carbachol in cat atrial heart muscle were mimicked, qualitatively, by adenosine and its analogues 5′-(N-ethyl)-carboxamido-adenosine (NE…

Malemedicine.medical_specialtyCarbacholAdenosineAction PotentialsStimulationAdenosine-5'-(N-ethylcarboxamide)BiologyIn Vitro TechniquesPurinergic AgonistsInternal medicineIsoprenalinemedicineAnimalsVentricular FunctionReceptors CholinergicPharmacologyPurinergic receptorReceptors PurinergicHeartDipyridamoleAtrial FunctionAdenosineMyocardial ContractionAcetylcholineEndocrinologyCatsPhenylisopropyladenosineCholinergicCarbacholFemaleRubidium RadioisotopesAcetylcholinemedicine.drugResearch Article
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β-Adrenoceptors differentially regulate vascular tone and angiogenesis of rat aorta via ERK1/2 and p38

2014

β-Adrenoceptors (β-ARs) modulate ERK1/2 and p38 in different cells, but little is known about the contribution of these signaling pathways to the function of β-ARs in vascular tissue. Immunoblotting analysis of rat aortic rings, primary endothelial (ECs) and smooth muscle cells (SMCs) isolated from aorta showed that β-AR stimulation with isoprenaline activated p38 in aortic rings and in both cultured cell types, whereas it had a dual effect on ERK1/2 phosphorylation, decreasing it in ECs while increasing it in SMCs. These effects were reversed by propranolol, which by itself increased p-ERK1/2 in ECs. Isoprenaline β-AR mediated vasodilation of aortic rings was potentiated by the ERK1/2 inhi…

Malemedicine.medical_specialtyEndotheliumPhysiologyAngiogenesisVasodilator AgentsAdrenergic beta-AntagonistsMyocytes Smooth MuscleNeovascularization PhysiologicAorta ThoracicStimulationVasodilationFibroblast growth factorp38 Mitogen-Activated Protein KinasesMuscle Smooth VascularInternal medicineIsoprenalinemedicine.arteryReceptors Adrenergic betamedicineAnimalsHumansRats WistarMitogen-Activated Protein Kinase 1PharmacologyMatrigelAortaMitogen-Activated Protein Kinase 3business.industryAdrenergic beta-AgonistsPropranololRatsVasodilationHEK293 Cellsmedicine.anatomical_structureEndocrinologycardiovascular systemMolecular MedicineEndothelium Vascularbusinessmedicine.drugVascular Pharmacology
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Contribution of both ?- and ?-adrenoceptors to the inotropic effects of catecholamines in the rabbit heart

1992

The functional role of alpha-adrenoceptors was investigated in different parts of the rabbit heart. Phenylephrine (PE) caused a marked increase in force of contraction (Fc) and a prolongation of the action potential (AP) in preparations from the left atrium and the right ventricle. The response was less pronounced in the right atrium and in the left ventricle, whereas APs of spontaneously beating sinoatrial preparations remained completely unchanged. Phentolamine as well as the diesters phorbol 12,13 dibutyrate (PDBu) or 12-O-tetradecanoyl-phorbol-13-acetate (TPA) eliminated the effects of PE. The contribution of alpha-adrenoceptors to the effects of adrenaline (Adr) and noradrenaline (NA) …

Malemedicine.medical_specialtyEpinephrineAction PotentialsStimulationPropranololNorepinephrinechemistry.chemical_compoundPhentolamineInternal medicineIsoprenalinePhorbol EstersReceptors Adrenergic betamedicineAnimalsDrug InteractionsPhentolaminePhenylephrinePharmacologyChemistryMyocardiumHeartGeneral MedicineReceptors Adrenergic alphaMyocardial ContractionPropranololEndocrinologymedicine.anatomical_structureVentricleCirculatory systemPhorbolFemaleRabbitsmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Modulation of cytosolic free calcium concentration by ?1-adrenoceptors in rat atrial cells

1992

The effects of α1-adrenoceptor stimulation by phenylephrine (PE) and β-adrenoceptor stimulation by isoprenaline (ISO) on Ca2+ current (ICa) and free intracellular Ca2+ concentration ([Ca2+]i) were studied in isolated atrial myocytes from rat hearts. PE did not significantly affect the magnitude of ICa, whereas large increases of peak ICa were observed in response to ISO. In electrically driven cells, PE evoked a concentration-dependent, gradual increase in diastolic [Ca2+]i and, initially, an increase in the height of peak [Ca2+]i transients. When the diastolic [Ca2+]i was increased to a greater extent, the amplitude of [Ca2+]i transients was decreased. Simultaneous measurements of [Ca2+]i …

Malemedicine.medical_specialtyFura-2chemistry.chemical_elementStimulationIn Vitro TechniquesCalciumPhenylephrinechemistry.chemical_compoundInternal medicineIsoprenalinemedicineAnimalsHeart AtriaPhenylephrinePharmacologyMembrane potentialIsoproterenolRats Inbred StrainsDepolarizationGeneral MedicineReceptors Adrenergic alphaRatsElectrophysiologyElectrophysiologyEndocrinologychemistryCalciumAdrenergic alpha-Agonistsmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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The effects of phorbol 12,13-diacetate on responses of guinea-pig isolated trachea to methylxanthines, isoprenaline and ryanodine

1994

1. Using guinea-pig isolated trachea, we have studied how phorbol 12,13-diacetate (PDA) modulates mechanical responses of the tissue to methylxanthines, isoprenaline and ryanodine. 2. Caffeine (10 microM-5 mM), theophylline (10 microM-5 mM) and isoprenaline (1 nM-1 microM), each inhibited the spontaneous tone of the trachea. Pretreatment with PDA (0.1-10 microM) converted relaxant responses to high concentrations of the methylxanthines into contractions. PDA produced no equivalent effect against isoprenaline. Pretreatment with verapamil (1 or 10 microM), nifedipine (0.1 microM) or incubation with Ca(2+)-free, EGTA (0.1 mM)-containing physiological salt solution (PSS) suppressed the contract…

Malemedicine.medical_specialtyMuscle RelaxationGuinea PigsMepyramineIn Vitro TechniquesCalcium Chloridechemistry.chemical_compoundTheophyllineCaffeineIsoprenalineInternal medicinePhorbol EstersmedicineAnimalsDrug InteractionsTheophyllinePharmacologyRyanodineRyanodine receptorIsoproterenolMuscle SmoothCold TemperatureTracheaEndocrinologyMuscle relaxationVerapamilchemistryMuscle SpasticityXanthinesPotassiumTrachealis muscleVerapamilFemaleCaffeineResearch ArticleHistamineMuscle Contractionmedicine.drugBritish Journal of Pharmacology
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Beta-adrenoceptors mediate inhibition of [3H]-acetylcholine release from the isolated rat and guinea-pig trachea: role of the airway mucosa and prost…

1994

1. Rat or guinea pig isolated tracheae were labelled with [3H]-choline to measure evoked tritium outflow, which reflects neuronal release of [3H]-acetylcholine. Tritium outflow was evoked either by electrical stimulation of the extrinsic vagal nerve (rat tracheae) or by 27 mM potassium (guinea pig tracheae). 2. In rat tracheae isoprenaline (0.01, 0.1 microM) inhibited evoked [3H]-acetylcholine release, whereas beta 2-adrenoceptor-selective agonists (fenoterol, formoterol, salbutamol) were ineffective. 3. The inhibitory effect of isoprenaline was abolished under the following conditions: (i) presence of propranolol (1 microM) or of the beta 1-selective antagonist CGP 20712 A (0.1 microM); (i…

Malemedicine.medical_specialtyNeuroeffectorAdrenergic beta-AntagonistsGuinea PigsIndomethacinProstaglandinStimulationPropranololIn Vitro TechniquesCholineGuinea pigRats Sprague-Dawleychemistry.chemical_compoundIsoprenalineInternal medicineReceptors Adrenergic betamedicineAnimalsPharmacologyArachidonic AcidMucous MembranebiologyChemistryIsoproterenolMuscle Smoothrespiratory systemAdrenergic beta-AgonistsAcetylcholineRatsTracheaEndocrinologybiology.proteinProstaglandinsFemaleCyclooxygenaseAcetylcholinemedicine.drugResearch ArticleBritish journal of pharmacology
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Cardiac effects of isoliquiritigenin

1997

The effects of isoliquiritigenin on force of contraction (Fc), L-type Ca2+ current (I(Ca)) and intracellular Ca2+ concentration ([Ca2+]i) were investigated in rat ventricular heart muscle. Isoliquiritigenin increased Fc and I(Ca) and, after longer exposure times, resting tension and [Ca2+]i. The effect of isoliquiritigenin (100 microM) on I(Ca) was diminished by Rp-cAMPS (30 microM). 1H-[1,2,4]oxa- diazolo[4,3-a]quinoxalin-1-one (50 microM) did not influence the effects of isoliquiritigenin on Fc and I(Ca). The positive inotropic effects of isoprenaline and forskolin, but not of 3-isobutyl-1-methylxanthine, were potentiated by isoliquiritigenin (100 microM). In the presence of milrinone (10…

Malemedicine.medical_specialtyPatch-Clamp TechniquesFura-2In Vitro TechniquesMembrane PotentialsRats Sprague-Dawleychemistry.chemical_compoundChalconeChalconesAldehyde ReductaseInternal medicineIsoprenalinemedicineAnimalsDrug InteractionsEnzyme InhibitorsCyclic GMPPharmacologyPlants MedicinalForskolinMyocardiumPhosphodiesteraseHeartCyclic AMP-Dependent Protein KinasesMyocardial ContractionRatsElectrophysiologyEndocrinologychemistryGuanylate CyclaseMilrinoneCalciumFemalemedicine.symptomSoluble guanylyl cyclaseIsoliquiritigeninMuscle contractionmedicine.drugEuropean Journal of Pharmacology
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