Search results for "Ituri"

showing 10 items of 123 documents

Elevated advanced oxidation protein products (AOPPs) indicate metabolic risk in severely obese children.

2012

Abstract Background and aims The assessment of oxidative stress may aid in the identification of subsequent metabolic risk in obese children. The objective of this study was to determine whether the plasma level of advanced oxidation protein products, analyzed with a recently proposed modified assay that involves a delipidation step (mAOPPs), was related to metabolic risk factors (MRFs) in severely obese children. Methods and results The plasma levels of mAOPPs were determined by spectrophotometry in 54 severely obese and 44 healthy children. We also measured lipid peroxidation biomarkers (thiobarbituric acid-reactive substances, malondialdehyde, and 8-isoprotane F 2α ) and sulfhydryl group…

MaleAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentMedicine (miscellaneous)Protein oxidationDinoprostSeverity of Illness IndexLipid peroxidationchemistry.chemical_compoundRisk FactorsMalondialdehydeAge of OnsetChildMetabolic SyndromeNutrition and DieteticsMalondialdehydeLipidsUp-RegulationSpectrophotometryHypertensionFemaleCardiology and Cardiovascular MedicineOxidation-Reductionmedicine.medical_specialtyAdolescentRisk AssessmentThiobarbituric Acid Reactive SubstancesInsulin resistanceInternal medicinemedicineHumansObesitySulfhydryl CompoundsDyslipidemiasChi-Square Distributionbusiness.industryProteinsmedicine.diseaseOxidative StressEndocrinologychemistryAdvanced oxidation protein productsSpainLinear ModelsLipid PeroxidationInsulin ResistancebusinessBody mass indexDyslipidemiaBiomarkersNutrition, metabolism, and cardiovascular diseases : NMCD
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Protein oxidation in chronic kidney disease.

2013

An imbalance between oxidative processes and antioxidant systems has been widely demonstrated in chronic kidney diseases (CKD). In this study we enrolled 26 healthy subjects, 27 patients with CKD on conservative treatment (CT-CKD) with various degrees of renal failure, and 31 CKD subjects in haemodialysis treatment (HD-CKD), evaluated before and after a standard haemodialysis session. In each group we measured protein carbonyl groups (PC) as an index of protein oxidation, lipid peroxidation (TBARS) and two plasma markers of leukocyte activation, elastase and myeloperoxidase (MPO). In CT-CKD subjects the PC level was significantly higher than in normal controls, and it was negatively correla…

MaleHEMODIALYSISmedicine.medical_specialtySettore MED/09 - Medicina InternaPhysiologyBIOMARKERSRenal functionurologic and male genital diseasesProtein oxidationThiobarbituric Acid Reactive SubstancesLipid peroxidationDiabetes Complicationschemistry.chemical_compoundCARBONYL STRESSMARKERSINFLAMMATIONGlycationRenal DialysisPhysiology (medical)Internal medicinemedicineTBARSHumansRenal Insufficiency ChronicPeroxidasebiologyPancreatic Elastasebusiness.industryNITRIC-OXIDE METABOLITESElastaseHematologyMiddle Agedmedicine.diseasefemale genital diseases and pregnancy complicationsOxidative StressEndocrinologychemistryMyeloperoxidaseNITRIC-OXIDE METABOLITES; CHRONIC-RENAL-FAILURE; CARBONYL STRESS; HEMODIALYSIS; BIOMARKERS; MARKERS; INFLAMMATIONImmunologybiology.proteinFemaleCHRONIC-RENAL-FAILURELipid PeroxidationCardiology and Cardiovascular MedicinebusinessOxidation-ReductionKidney diseaseClinical hemorheology and microcirculation
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Effect of acidosis and anoxia on iron delocalization from brain homogenates.

1992

Abstract Cortical homogenates were prepared from rat brain in Krebs-Ringer phosphate media adjusted to pH 7, 6 or 5 and incubated for 1 hr under aerotic or anaerobic conditions in the presence of dipyridyl, an iron chelator. Low molecular weight species (LMWS) iron was measured spectrophotometrically after passing of the homogenates through a 10,000- M , ultrafiltration membrane. Following aerobic incubation, LMWS iron reached 1.24 μg/g tissue at pH 7, and increased 1.7-fold at pH 6 and 3.1-fold at pH 5. Anoxia enhanced significantly the amount of ultrafiltrable iron at the three pH values, the LMWS iron level being increased by 190% at pH 7, by 113% at pH 6, and by 77% at pH 5. Addition of…

MaleLipid PeroxidesThiobarbituric acidIronAscorbic AcidBiochemistryLipid peroxidationchemistry.chemical_compound22'-DipyridylmedicineAnimalsChelationFerrous CompoundsHypoxia BrainAcidosisPharmacologyBrain ChemistryRats Inbred StrainsHydrogen-Ion ConcentrationPhosphateRatsOxygenUltrafiltration (renal)chemistryBiochemistryLipid Peroxidationmedicine.symptomAcidosisAnaerobic exerciseIntracellularBiochemical pharmacology
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General oxidative stress during doxorubicin-induced cardiotoxicity in rats: Absence of cardioprotection and low antioxidant efficiency of alpha-lipoi…

2012

International audience; To evaluate the effects of alpha-lipoic acid (AL) in a model of doxorubicin (DOX)-induced cardiotoxicity, male Wistar rats were treated with DOX (1 mg/kg/d; 10 d) in combination or not with AL (50 mg/kg/d; 15 d). Plasma oxidative stress was determined by hydroperoxides (ROOH) and the ascorbyl radical/ascorbate ratio. One and two months later, the functional parameters of the hearts were determined in vivo by catheterization and cardiac oxidative stress was assessed by malonedialdehyde (MDA) and O₂*⁻ (dihydroethidium fluorescence) content in tissue. After two months, body weight was higher in the DOX-AL group than in DOX (+16%), but this was due to ascites. Histologic…

MaleMESH : Oxidative StressAntioxidantmedicine.medical_treatmentMESH : HematocritMESH : Thioctic AcidBiochemistryAntioxidants0302 clinical medicineSuperoxidesAscitic FluidMESH: AnimalsMESH : Body WeightComputingMilieux_MISCELLANEOUS0303 health sciencesThioctic AcidCumulative doseMESH: Heart DiseasesHeartGeneral Medicine3. Good healthMESH: Ascitic Fluid[SDV] Life Sciences [q-bio]030220 oncology & carcinogenesisMESH : Ascitic FluidMESH: Hydrogen PeroxideMESH : AntioxidantsMESH: Thioctic Acidmedicine.medical_specialtyCardiotonic AgentsCardiotoxinsMESH: Hematocrit03 medical and health sciencesMESH: Doxorubicin[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemIn vivoRats Wistar[ SDV ] Life Sciences [q-bio]MyocardiumMESH: AntioxidantsHydrogen PeroxideMESH: Cardiotonic AgentsMESH : Organ SizeMESH: Body WeightMESH: Heartcarbohydrates (lipids)EndocrinologyMESH: LiverMESH : SuperoxidesMESH: Organ Size[SDV]Life Sciences [q-bio]MESH : Cardiotonic AgentsAscorbic AcidMESH: Superoxidesmedicine.disease_causeMESH: EatingEatingpolycyclic compoundsMESH : MyocardiumMESH: Thiobarbituric Acid Reactive SubstancesMESH: Ascorbic AcidAntibiotics AntineoplasticMESH: Oxidative StressChemistryMESH : RatsOrgan SizeMESH : Antibiotics Antineoplastic[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemBiochemistryHematocritLiverMESH : Cardiotoxinsmedicine.drugMESH : EatingMESH: MyocardiumHeart DiseasesMESH: RatsMESH : MaleMESH : Thiobarbituric Acid Reactive SubstancesMESH : Rats WistarThiobarbituric Acid Reactive SubstancesContractilityMESH : HeartInternal medicinemedicineTBARSAnimalsMESH : DoxorubicinDoxorubicinMESH: Antibiotics AntineoplasticMESH : Ascorbic Acid030304 developmental biologyCardiotoxicityBody WeightMESH : LiverMESH : Heart DiseasesMESH: Rats WistarMESH: MaleRatsOxidative StressMESH: CardiotoxinsDoxorubicinMESH : AnimalsMESH : Hydrogen PeroxideOxidative stress
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Long-term moderate magnesium-deficient diet shows relationships between blood pressure, inflammation and oxidant stress defense in aging rats

2006

International audience; Epidemiological and experimental studies have indicated a relationship among aging, dietary Mg, inflammatory stress, and cardiovascular disease. Our aim in the present study was to investigate possible links between dietary Mg, oxidant stress parameters, and inflammatory status with aging in rats. We designed a long-term study in which rats were fed for 22 months with moderately deficient (150 mg/kg), standard (800 mg/kg), or supplemented (3200 mg/kg) Mg diets. Comparisons were made with young rats fed with the same diets for 1 month. Compared to the standard and supplemented diets, the Mg-deficient diet significantly increased blood pressure, plasma interleukin-6, f…

MaleMESH: Inflammationmedicine.medical_specialtyMESH: RatsThiobarbituric acid[SDV]Life Sciences [q-bio]Blood PressureInflammationMESH: Rats Sprague-Dawley030204 cardiovascular system & hematologymedicine.disease_causeFibrinogenBiochemistryRats Sprague-Dawley03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePhysiology (medical)Internal medicinemedicineTBARSAnimalsMESH: Animals030304 developmental biologyInflammation2. Zero hunger0303 health sciencesMESH: Oxidative StressMESH: Blood Pressuremedicine.diseaseMESH: MaleHemolysisRats3. Good healthOxidative StressLysophosphatidylcholineBlood pressureEndocrinologychemistryImmunologymedicine.symptomMagnesium DeficiencyMESH: Magnesium DeficiencyOxidative stressmedicine.drugFree Radical Biology and Medicine
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Effects of gender, diet, exogenous melatonin and subchronic PCB exposure on plasma immunoglobulin G in mink

2002

Abstract Effects of different fish-based diets (freshwater smelt, Baltic herring, marine herring/cod offal or their mixtures), gender, β-glucan supplement, exogenous melatonin, and PCB exposure (Aroclor 1242®, 1 mg per animal per day in feed) on plasma immunoglobulin G (IgG) in the mink (Mustela vison) were studied. The aims of the study were to find out whether plasma IgG of the mink is affected by the subchronic PCB exposure, and whether biological, nutritional and hormonal effects are large enough to mask the possible IgG response. The concentration of IgG was determined using enzyme-linked immunosorbent assay (ELISA). Sexual dimorphism was detected, the males having higher levels of pla…

MalePhysiologyHealth Toxicology and Mutagenesismedicine.medical_treatment010501 environmental sciencesToxicology01 natural sciencesBiochemistryImmunoglobulin Gchemistry.chemical_compoundHerringVitamin EMinkChromatography High Pressure LiquidMelatoninSex Characteristics0303 health sciencesbiologyFishesRetinolGeneral MedicinePolychlorinated BiphenylsLiverFemaleSeasonsmedicine.drugmedicine.medical_specialtyAnimals WildEnzyme-Linked Immunosorbent AssayThiobarbituric Acid Reactive SubstancesMelatonin03 medical and health sciencesFish OilsImmune systembiology.animalInternal medicineCytochrome P-450 CYP1A1medicineAnimals030304 developmental biology0105 earth and related environmental sciencesVitamin ECell BiologyAnimal FeedDietEndocrinologychemistryMinkImmunoglobulin Gbiology.proteinHormoneComparative Biochemistry and Physiology Part C: Toxicology & Pharmacology
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Retinal oxidation, apoptosis and age- and sex-differences in the mnd mutant mouse, a model of neuronal ceroid lipofuscinosis

2004

Retinal degeneration is an early and progressive event in many forms of neuronal ceroid lipofuscinoses (NCLs), a heterogeneous group of neurodegenerative disorders with unknown pathogenesis. We here used the mutant motor neuron degeneration (mnd) mouse, a late-infantile NCL variant, to investigate the retinal oxidative state and apoptotic cell death as a function of age and sex. Total superoxide dismutase (SOD) activities and thiobarbituric acid-reactive substance (TBARS) levels revealed progressive increases in retinal oxyradicals and lipid peroxides of mnd mice of both sexes. Female mnd retinas showed a higher oxidation rate and consistently exhibited the 4-hydroxy-2-nonenal (4-HNE)-adduc…

MaleRetinal degenerationPathologymedicine.medical_specialtyApoptosisBiologymedicine.disease_causeThiobarbituric Acid Reactive SubstancesRetinaMiceMice Neurologic Mutantschemistry.chemical_compoundSex FactorsNeuronal Ceroid-LipofuscinosesIn Situ Nick-End LabelingmedicineAnimalsOuter nuclear layerMolecular BiologyAldehydesRetinaTUNEL assayLipid peroxideCaspase 3Superoxide DismutaseGeneral NeuroscienceRetinal DegenerationRetinalmedicine.diseaseImmunohistochemistryEnzyme ActivationMice Inbred C57BLDisease Models AnimalOxidative Stressmedicine.anatomical_structureBiochemistrychemistryCaspasesFemaleNeuronal ceroid lipofuscinosisNeurology (clinical)Oxidation-ReductionOxidative stressDevelopmental BiologyBrain Research
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Effect of a two-year supplementation with low doses of antioxidant vitamins and/or minerals in elderly subjects on levels of nutrients and antioxidan…

1997

Eighty-one elderly hospitalized subjects (65 years) were recruited for a double-blind placebo-controlled study to examine low dose supplementation of antioxidant vitamins and minerals on biological and functional parameters of free radical metabolism. Subjects were randomly assigned to one of the four treatment groups, daily receiving for 2 years: placebo group; mineral group: 20 mg zinc, 100 micrograms selenium; vitamin group: 120 mg vitamin C (Vit C), 6 mg beta-carotene (beta CA), 15 mg vitamin E (Vit E); mineral and vitamin group: Zn 20 mg, Se 100 micrograms, Vit C 120 mg, beta CA 6 mg, Vit E 15 mg.Fifty-seven subjects completed the study. A large frequency of Vit C, Zn and Se deficienci…

MaleVitaminmedicine.medical_specialtyErythrocytesAntioxidantmedicine.medical_treatmentNutritional StatusMedicine (miscellaneous)chemistry.chemical_elementAscorbic AcidThiobarbituric Acid Reactive SubstancesAntioxidantsSeleniumchemistry.chemical_compoundOral administrationInternal medicinemedicineHumansVitamin ENutritional Physiological PhenomenaAgedAged 80 and overGlutathione PeroxidaseMineralsNutrition and DieteticsVitamin CSuperoxide DismutaseVitamin EVitaminsMetabolismbeta CaroteneAscorbic acidGlutathioneZincEndocrinologychemistryBiochemistryFemaleSeleniumJournal of the American College of Nutrition
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Iridoid extracts from Ajuga iva increase the antioxidant enzyme activities in red blood cells of rats fed a cholesterol-rich diet.

2010

The lyophilized aqueous extract of Ajuga iva (Ai) is able to reduce oxidative stress, which may prevent lipid peroxidation in hypercholesterolemic rats. Iridoids (I) were isolated from Ai. We hypothesized that the antioxidant defense status in red blood cells (RBC) and tissues in rats fed a cholesterol-rich diet and treated with Ai may be correlated to these compounds. Male Wistar rats (n = 32) weighing 120 +/- 5 g were fed a diet containing 1% cholesterol for 15 days. After this phase, hypercholesterolemic (HC) rats were divided into groups, fed the same diet, and received either the same or different doses (5, 10, or 15 mg/kg body weight by intraperitoneal injection) of I for 15 days. Com…

Malemedicine.medical_specialtyAntioxidantErythrocytesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentGlutathione reductaseHypercholesterolemiaAjugamedicine.disease_causeThiobarbituric Acid Reactive SubstancesAntioxidantsLipid peroxidationSuperoxide dismutaseCholesterol Dietarychemistry.chemical_compoundEndocrinologyInternal medicinemedicineTBARSAnimalsIridoidsRats Wistarchemistry.chemical_classificationNutrition and DieteticsbiologyDose-Response Relationship DrugPlant ExtractsGlutathione peroxidaseAnticholesteremic AgentsMusclesMyocardiumGlutathioneRatsOxidative StressEndocrinologychemistryLiverbiology.proteinOxidative stressPhytotherapyNutrition research (New York, N.Y.)
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Changes in antioxidant defense status in hypercholesterolemic rats treated with Ajuga iva.

2007

Abstract The aim of the study was to investigate the effect of aqueous extract of Ajuga iva ( Ai ) on serum and tissues lipid peroxidation as well as antioxidant enzymes activities in red blood cells (RBC) and tissues, in high hypercholesterolemic rats (HC). Male Wistar rats ( n =12) were fed on 1% cholesterol-enriched diet for 15 d. After this adaptation phase, hypercholesterolemic rats (total cholesterol=6.5±0.6 mol/l) were divided into two groups fed the same diet and treated or not with Ai for 15 d. Thiobarbituric acid reactive substances (TBARS) concentrations in serum, LDL-HDL 1 , HDL 2 and HDL 3 were respectively, 5-, 7.8-, 2.3- and 5-fold lower in Ai treated than untreated hyperchol…

Malemedicine.medical_specialtyAntioxidantErythrocytesThiobarbituric acidmedicine.medical_treatmentGlutathione reductaseHypercholesterolemiaPharmaceutical ScienceAdipose tissueAjugaNitric OxideAjugaAntioxidantsLipid peroxidationCholesterol Dietarychemistry.chemical_compoundEatingInternal medicineDrug DiscoverymedicineTBARSAnimalsRats WistarPharmacologychemistry.chemical_classificationbiologyPlant ExtractsGlutathione peroxidaseBody WeightVitaminsbiology.organism_classificationGlutathioneRatsEndocrinologyCholesterolComplementary and alternative medicinechemistryBiochemistryMolecular MedicineLipid PeroxidationPhytotherapyPhytomedicine : international journal of phytotherapy and phytopharmacology
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