Search results for "Keratin-18"

showing 7 items of 7 documents

Usefulness of the index of NASH - ION for the diagnosis of steatohepatitis in patients with non-alcoholic fatty liver: An external validation study

2018

Background & Aims The non-invasive identification of steatohepatitis (NASH) in patients with Non-Alcoholic Fatty Liver Disease is an unmet need in clinical practice. Index of NASH (ION) is a new tool for the prediction of NASH. We aimed to externally validate ION and to compare it with CK-18. Since necroinflammation precedes fibrosis, we also tested ION in combination with non-invasive tools for fibrosis. Methods We analysed data from 292 Italian patients (169 Southern cohort, and 123 Northern cohort) with an histological diagnosis of NAFLD. The ION, FIB-4 and NFS scores were calculated according to published algorithms. Serum cytokeratin18-Aspartate396 levels and liver stiffness (LS) by Fi…

AdultLiver CirrhosisMale0301 basic medicinemedicine.medical_specialtysteatohepatitisSeverity of Illness IndexGastroenterology03 medical and health sciencesLiver disease0302 clinical medicineNon-alcoholic Fatty Liver DiseaseFibrosisInternal medicineHumansMedicineIn patientProspective StudiesKeratin-18Hepatologymedicine.diagnostic_testbusiness.industryFatty liverExternal validationReproducibility of ResultsMiddle Agedmedicine.disease3. Good health030104 developmental biologyItalyLiverROC Curvenon-invasive markerLiver biopsyCohortElasticity Imaging TechniquesFemale030211 gastroenterology & hepatologySteatohepatitisfibrosibusinessAlgorithmsBiomarkersLiver International
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Cytokeratin-18 fragments predict treatment response and overall survival in gastric cancer in a randomized controlled trial

2018

Background:Gastric cancer is common malignancy and exhibits a poor prognosis. At the time of diagnosis, the majority of patients present with metastatic disease which precludes curative treatment. Non-invasive biomarkers which discriminate early from advanced stages or predict the response to treatment are urgently required. This study explored the cytokeratin-18 fragment M30 and full-length cytokeratin-18 M65 in predicting treatment response and survival in a randomized, placebo-controlled trial of advanced gastric cancer.Methods:Patients enrolled in the SUN-CASE study received sunitinib or placebo as an adjunct to standard therapy with leucovorin (Ca-folinate), 5-fluorouracil, and irinote…

AdultMale0301 basic medicineOncologymedicine.medical_specialtyIndolesmedicine.medical_treatmentLeucovorinAntineoplastic AgentsPlaceboDisease-Free SurvivalMetastasislaw.inventionPlacebos03 medical and health sciences0302 clinical medicineRandomized controlled trialStomach NeoplasmslawInternal medicineAntineoplastic Combined Chemotherapy ProtocolsBiomarkers TumorSunitinibHumansMedicinePyrrolesProgression-free survivalRC254-282AgedAged 80 and overChemotherapyKeratin-18business.industrySunitinibCancerNeoplasms. Tumors. Oncology. Including cancer and carcinogensGeneral MedicineMiddle Agedmedicine.diseasePeptide FragmentsIrinotecan030104 developmental biology030220 oncology & carcinogenesisCamptothecinFemaleFluorouracilbusinessmedicine.drugTumor Biology
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cDNA sequences of the authentic keratins 8 and 18 in zebrafish

2003

From the zebrafish Danio rerio, we have cDNA cloned and sequenced a novel type II and a novel type I keratin, termed DreK8 and DreK18, respectively. We identified DreK8/18 as the true orthologs of the human keratin pair K8/18 as follows: (i) MALDI-MS assignment to the biochemically identified K8 and K18 candidates that are co-expressed in simple epithelia and absent in epidermal keratinocytes; (ii) multiple sequence alignments and phylogenetic tree analysis, showing that DreK8, within the phylogenetic tree of type II keratins, forms a highly bootstrap-supported branch together with K8 from goldfish and rainbow trout, whereas DreK18, within the phylogenetic tree of type I keratins, groups wi…

Cancer Researchanimal structuresType I keratinMolecular Sequence DataDaniomacromolecular substancesBiologyType II keratinComplementary DNAKeratinAnimalsHumansTissue DistributionAmino Acid SequenceCloning MolecularMolecular BiologyZebrafishPhylogenyZebrafishGeneticschemistry.chemical_classificationKeratin-18integumentary systemPhylogenetic treeKeratin-8Nucleic acid sequenceCell BiologyZebrafish Proteinsbiology.organism_classificationchemistryKeratinsSequence AlignmentDevelopmental BiologyDifferentiation
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Onset of heart failure determines the hepatic cell death pattern.

2011

Background and rationale. Acute and chronic heart failure (HF) may affect the liver, but the underlying mechanisms that lead to progressive liver damage are poorly understood. The hepatic cytokeratin-18 (CK18) epitopes M65 and M30 have been reported to distinguish between overall (necrotic) and apoptotic cell death, respectively. We aimed to evaluate the predominant hepatic cell death pattern in acute vs. chronic heart failure and examined if these assays predict the course of the disease. Main results. In a prospective study comprising 21 patients with acute HF (AHF) and 18 patients with chronic HF (CHF) serum levels of M65 and M30 were assessed. Compared with CHF, M65 levels were signific…

Cell deathMalemedicine.medical_specialtyProgrammed cell deathMedizinM65Specialties of internal medicineApoptosisLiver injuryEpitopesPredictive Value of TestsInternal medicinemedicineHumansProspective StudiesProspective cohort studySurvival analysisHeart FailureLiver injuryKeratin-18HepatologyCytokeratin 18business.industryM30Liver DiseasesGeneral MedicineMiddle AgedPrognosismedicine.diseaseSurvival AnalysisRC581-951LiverApoptosisPredictive value of testsHeart failureAcute DiseaseChronic DiseaseHepatic stellate cellCardiologyFemalebusinessBiomarkers
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Fatal neuroinvasion and SARS-CoV-2 tropism in K18-hACE2 mice is partially independent on hACE2 expression

2022

ABSTRACTAnimal models recapitulating distinctive features of severe COVID-19 are critical to enhance our understanding of SARS-CoV-2 pathogenesis. Transgenic mice expressing human angiotensin-converting enzyme 2 (hACE2) under the cytokeratin 18 promoter (K18-hACE2) represent a lethal model of SARS-CoV-2 infection. The precise mechanisms of lethality in this mouse model remain unclear. Here, we evaluated the spatiotemporal dynamics of SARS-CoV-2 infection for up to 14 days post-infection. Despite infection and moderate pneumonia, rapid clinical decline or death of mice was invariably associated with viral neuroinvasion and direct neuronal injury (including brain and spinal neurons). Neuroinv…

Genetically modified mousevirusesMice TransgenicViremiaBiologyArticleVirusPathogenesisMiceVirologymedicineAnimalsHumansMelphalanTropismKeratin-18SARS-CoV-2COVID-19medicine.diseaseOlfactory bulbViral TropismInfectious DiseasesViral replicationtranslational animal model; comparative pathology; immunohistochemistry; in situ hybridization; viral pathogenesis; transmission electron microscopy; in vivo imagingImmunologyNeuropathogenesisAngiotensin-Converting Enzyme 2gamma-Globulins
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The Impact of Liver Cell Injury on Health-Related Quality of Life in Patients with Chronic Liver Disease.

2015

Background Patients with chronic liver disease often suffer from unspecific symptoms and report severe impairment in the quality of life. The underlying mechanisms are multifactorial and include disease-specific but also liver related causes. The current analysis evaluated the association of hepatocellular apoptosis in non-viral chronic liver disease and health-related quality of life (HRQL). Furthermore we examined factors, which influence patient's physical and mental well-being. Methods A total of 150 patients with non-infectious chronic liver disease were included between January 2014 and June 2015. The German version of the Chronic Liver Disease Questionnaire (CLDQ-D), a liver disease …

Liver CirrhosisMaleCirrhosisPhysiologylcsh:MedicineApoptosisChronic liver diseaseGastroenterologyChronic Liver DiseaseBody Mass Index0302 clinical medicineQuality of lifeFibrosisMedicine and Health Scienceslcsh:ScienceMultidisciplinaryLiver cellLiver DiseasesFatty liverMiddle AgedLiverCirrhosisPhysiological Parameters030220 oncology & carcinogenesis030211 gastroenterology & hepatologyFemaleResearch ArticleAdultmedicine.medical_specialtyGastroenterology and Hepatology03 medical and health sciencesInternal medicinemedicineHumansObesityAgedKeratin-18business.industrylcsh:RBody WeightBiology and Life Sciencesmedicine.diseaseObesityFibrosisFatty LiverHealth CareChronic DiseaseQuality of Lifelcsh:QbusinessBody mass indexDevelopmental BiologyPloS one
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Focal adhesions are hotspots for keratin filament precursor formation

2006

Recent studies showed that keratin filament (KF) formation originates primarily from sites close to the actin-rich cell cortex. To further characterize these sites, we performed multicolor fluorescence imaging of living cells and found drastically increased KF assembly in regions of elevated actin turnover, i.e., in lamellipodia. Abundant KF precursors (KFPs) appeared within these areas at the distal tips of actin stress fibers, moving alongside the stress fibers until their integration into the peripheral KF network. The earliest KFPs were detected next to actin-anchoring focal adhesions (FAs) and were only seen after the establishment of FAs in emerging lamellipodia. Tight spatiotemporal …

TalinKeratin 14Intermediate Filamentsmacromolecular substancesBiologyTransfectionKeratin 18Cell LineFocal adhesionMiceReportStress FibersCell cortexMetalloproteinsAnimalsHumansRNA AntisensePseudopodiaCytoskeletonActinResearch ArticlesCell Line TransformedFocal AdhesionsKeratin FilamentKeratin-18Keratin-14Cell BiologyBridged Bicyclo Compounds HeterocyclicActinsZyxinCell biologyProtein TransportThiazolesBiochemistryEpidermolysis Bullosa SimplexMutationKeratinsThiazolidinesMarine ToxinsLamellipodiumPaxillinThe Journal of Cell Biology
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