Search results for "Kidney"

showing 10 items of 1514 documents

The obsolescent renal glomerulus ? Collapse, sclerosis, hyalinosis, fibrosis

1977

By light and electron microscopical examination it is shown that four structural components can contribute to obsolescent glomeruli: capillary basement membranes, enriched mesangium matrix, “vascular” hyalin and collagen fibers. Each of these components can bring about glomerular damage alone. One non-reactive form — a glomerular collapse with only basement membrane remnants — can be separated from three reactive forms: the accumulation of mesangium matrix (sclerosis or matrix-sclerosis), deposition of vascular hyalin (hyalinosis in the narrow sense), and fiber development within the former urinary space (fibrosis or fibro-sclerosis). The use of the term “fibrinoid” in place of the descript…

AdultHyalinPathologymedicine.medical_specialtyHistologyRenal glomerulusKidney GlomerulusBasement MembranePathology and Forensic MedicineFibrosismedicineHumansChildMolecular BiologyCollapse (medical)HyalineBasement membraneMesangium matrixSclerosisUrinary spaceGlomerulosclerosis Focal SegmentalChemistryBiopsy NeedleGlomerulosclerosisCell BiologyGeneral MedicineAnatomymedicine.diseaseMicroscopy Electronmedicine.anatomical_structureCollagenAnatomymedicine.symptomVirchows Archiv A Pathological Anatomy and Histology
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Reduced secretion of proinflammatory cytokines of monosodium urate crystal‐stimulated monocytes in chronic renal failure: an explanation for infreque…

2000

In gouty arthritis, monosodium urate (MSU) crystals interact with monocytes and neutrophils to produce inflammatory reactions associated with acute synovitis. In patients with end-stage renal disease (ESRD), gouty arthritis is a rare condition despite often severe hyperuricaemia. We wondered whether differences in the secretion of proinflammatory cytokines by MSU crystal-stimulated monocytes might be one explanation for the low incidence of gouty arthritis in patients with ESRD compared with healthy controls.Thirteen patients with ESRD on intermittent haemodialysis treatment, six patients with chronic renal failure not yet on dialysis, and 15 age- and sex-matched healthy controls were exami…

AdultLipopolysaccharidesMalemusculoskeletal diseasescongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyGoutmedicine.medical_treatmentCytological TechniquesInflammationurologic and male genital diseasesProinflammatory cytokinechemistry.chemical_compoundInternal medicinemedicineHumansAgedAged 80 and overTransplantationInterleukin-6Tumor Necrosis Factor-alphabusiness.industryIncidencenutritional and metabolic diseasesMiddle Agedmedicine.diseasePathophysiologyUric AcidGoutEndocrinologyCytokinechemistryNephrologyAcute SynovitisImmunologyCytokinesKidney Failure ChronicUric acidFemaleInflammation Mediatorsmedicine.symptomCrystallizationbusinessInterleukin-1Kidney diseaseNephrology Dialysis Transplantation
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Heparin induces an accumulation of atherogenic lipoproteins during hemodialysis in normolipidemic end-stage renal disease patients.

2014

Dyslipidemias may account for the excess of cardiovascular mortality in end-stage renal disease (ESRD). Lipoprotein studies in ESRD patients are usually relative to prehemodialysis samples even if significative changes may occur after dialysis. In this study, we aimed to investigate the effects of ESRD on triglyceride-rich lipoproteins (TRL) subpopulations distribution and acute change following hemodialytic procedures, including the relative contribution of heparin administration. We selected a group of normolipidemic male middle-aged ESRD patients free of any concomitant disease affecting lipoprotein remnant metabolism compared with controls. We separated TRL subfractions according to den…

AdultLipoproteins LDLMaleSettore MED/09 - Medicina InternaHeparinRenal DialysisLipoproteinsHumansKidney Failure ChronicLipid metabolism drug and dialysis cardiovascularLipoproteins HDLTriglycerides
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Alterations in circadian rhythm of serum thyrotropin in critically ill patients.

1992

To evaluate the 24-h pattern of serum thyrotropin (TSH) in critically ill patients, we measured serum concentrations of TSH in blood samples collected every 2 h for 24 h from nine patients (six with malignancy, two with liver cirrhosis, one with chronic renal failure), who had subnormal levels of both triiodothyronine (T3) and thyroxine (T4), in the absence of history, symptoms or signs of thyroid disease. Analysis of the data, performed using a second-order inferential statistical methodology for rhythmometry (cosinor method), demonstrated that critically ill patients still had daily oscillations of serum TSH which significantly adapted to the function approximating the circadian rhythms (…

AdultLiver CirrhosisMaleendocrine systemmedicine.medical_specialtyCirrhosisEndocrinology Diabetes and MetabolismCritical IllnessRadioimmunoassayThyrotropinMalignancylaw.inventionEndocrinologyRhythmlawInternal medicineNeoplasmsmedicineHumansCircadian rhythmAgedTriiodothyroninebusiness.industryCritically illThyroid diseaseGeneral MedicineMiddle Agedmedicine.diseaseIntensive care unitCircadian RhythmThyroxineEndocrinologyKidney Failure ChronicTriiodothyronineFemalebusinessActa endocrinologica
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Predictive factors of transarterial chemoembolisation toxicity in unresectable hepatocellular carcinoma

2013

Abstract Background Transarterial chemoembolisation (TACE) is an effective treatment for unresectable hepatocellular carcinoma (HCC), but can cause severe toxicity. Aim To identify predictive factors of severe TACE-related toxicity in patients with unresectable HCC. Methods All HCC patients who underwent TACE at the Dijon University Hospital between 2008 and 2011 were included in this retrospective study. Severe TACE-related toxicity was defined as the occurrence of any adverse event grade ≥4, or any adverse event that caused a prolongation of hospitalisation of >8 days, or any additional hospitalisation within 1 month after TACE. Factors predicting toxicity were identified using a logistic…

AdultLiver CirrhosisMalemedicine.medical_specialtyCarcinoma HepatocellularMultivariate analysisLogistic regressionGastroenterologyCohort StudiesHepatitis B ChronicLiver Cirrhosis AlcoholicRisk FactorsInternal medicinemedicineHumansIn patientAspartate AminotransferasesChemoembolization TherapeuticAdverse effectAgedRetrospective StudiesAged 80 and overHepatologybusiness.industryLiver NeoplasmsGastroenterologyRetrospective cohort studyAcute Kidney InjuryHepatitis C ChronicLiver Failure AcuteMiddle Agedmedicine.diseaseUniversity hospitalTumor BurdenSurgeryLogistic ModelsTreatment OutcomeDoxorubicinHepatic EncephalopathyHepatocellular carcinomaMultivariate AnalysisToxicityFemaleChemical and Drug Induced Liver InjuryIdarubicinbusinessDigestive and Liver Disease
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Congenital hepatic fibrosis: CT findings in 18 adults.

2004

To evaluate the computed tomographic (CT) findings in adult patients with pathologically proved congenital hepatic fibrosis.This was a retrospective review of congenital hepatic fibrosis cases identified at two institutions over the course of 8 years. Eight men and 10 women with an age range of 22-72 years (mean age, 39 years) were included. Contrast material-enhanced and unenhanced CT scans were obtained through the liver in all patients. Two radiologists evaluated size of and morphologic findings (atrophy or hypertrophy localized according to hepatic segments) in the liver; increased diameter or number of hepatic arteries at the hilum; presence of hepatic nodules, varices, spontaneous spl…

AdultLiver CirrhosisMalemedicine.medical_specialtyCommon Bile Duct Diseasesmedicine.medical_treatmentSplenectomyHilum (biology)Hepatic VeinsKidneyGastroenterologyHepatic ArteryAtrophyFibrosisInternal medicineHypertension PortalHumansMedicineRadiology Nuclear Medicine and imagingAgedRetrospective StudiesPortal Veinbusiness.industryHypertrophyMiddle Agedmedicine.diseaseDuctal Plate MalformationLiverPortal hypertensionCongenital hepatic fibrosisFemaleRadiologyTomography X-Ray ComputedbusinessVaricesDilatation PathologicFollow-Up Studies
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RIFLE classification as predictive factor of mortality in patients with cirrhosis admitted to intensive care unit.

2009

Background and Aim: To evaluate the association of the Risk, Injury, Failure, Loss and End-stage renal failure (RIFLE) score on mortality in patients with decompensated cirrho- sis admitted to intensive care unit (ICU). Methods: A cohort of 412 patients with cirrhosis consecutively admitted to ICU was classified according to the RIFLE score. Multivariable logistic regression analysis was used to evaluate the factors associated with mortality. Liver-specific, Acute Physiology and Chronic Health Evaluation (APACHE) II, Sequential Organ Failure Assessment (SOFA) and RIFLE scores on admission, were compared by receiver-operator characteristic curves. Results: The overall mortality during ICU st…

AdultLiver CirrhosisMalemedicine.medical_specialtyTime FactorsRisk AssessmentSensitivity and SpecificitySeverity of Illness Indexlaw.inventionHepatorenal syndromelawPredictive Value of TestsRisk FactorsInternal medicineCause of DeathEpidemiologymedicineHealth Status IndicatorsHumansRifleAPACHERetrospective StudiesHepatologybusiness.industryGastroenterologyAcute kidney injuryReproducibility of ResultsOdds ratioAPACHE cirrhosis intensive care unit MELD renal failure RIFLE SOFA.Acute Kidney InjuryMiddle Agedmedicine.diseasePrognosisIntensive care unitSurgeryIntensive Care UnitsLogistic ModelsROC CurveCohortFemalebusinessKidney disease
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Risk of Second Primary Cancers in Multiple Myeloma Survivors in German and Swedish Cancer Registries

2016

AbstractWe aimed at investigating the distribution and risk of second primary cancers (SPCs) in multiple myeloma (MM) survivors in Germany and Sweden to provide etiological understanding of SPCs and insight into their incidence rates and recording practices. MM patients diagnosed in 1997–2010 at age ≥15 years were selected from the Swedish (nationwide) and 12 German cancer registries. Standardized incidence ratios (SIRs) were used to assess risk of a specific SPC compared to risk of the same first cancer in the corresponding background population. Among 18,735 survivors of first MM in Germany and 7,560 in Sweden, overall 752 and 349 SPCs were recorded, respectively. Significantly elevated S…

AdultMale0301 basic medicineOncologyPediatricsmedicine.medical_specialtyTime FactorsMyeloidAdolescentNervous System NeoplasmsPopulationAntineoplastic AgentsArticle03 medical and health sciences0302 clinical medicineRisk FactorsGermanyInternal medicinemedicineHumansRegistriesSurvivorseducationMultiple myelomaAgedSwedeneducation.field_of_studyMultidisciplinarybusiness.industryIncidence (epidemiology)CancerNeoplasms Second PrimaryMiddle Agedmedicine.diseaseKidney NeoplasmsLeukemia Myeloid AcuteLeukemiaEarly Diagnosis030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisEpidemiology ; Cancer epidemiology ; MyelomaEtiologyFemaleMultiple MyelomabusinessKidney cancerScientific Reports
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Second line therapy with axitinib after only prior sunitinib in metastatic renal cell cancer: Italian multicenter real world SAX study final results

2019

Abstract Background This multi-institutional retrospective real life study was conducted in 22 Italian Oncology Centers and evaluated the role of Axitinib in second line treatment in not selected mRCC patients. Methods 148 mRCC patients were evaluated. According to Heng score 15.5%, 60.1% and 24.4% of patients were at poor risk, intermediate and favorable risk, respectively. Results PFS, OS, DCR and ORR were 7.14 months, 15.5 months, 70.6% and 16.6%, respectively. The duration of prior sunitinib treatment correlated with a longer significant mPFS, 8.8 vs 6.3 months, respectively. Axitinib therapy was safe, without grade 4 adverse events. The most frequent toxicities of all grades were: fati…

AdultMale0301 basic medicineOncologymedicine.medical_specialtyMultivariate analysisAxitinibAxitinib; Metastatic; Renal cancer; Sunitinib; Treatmentmedicine.medical_treatmentPopulationlcsh:MedicineKaplan-Meier EstimateDisease-Free SurvivalGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineInternal medicineSunitinibHumansMedicineNeoplasm MetastasiseducationAdverse effectMetastatic renal cell cancerCarcinoma Renal CellAgedAged 80 and overSecond-line therapyeducation.field_of_studybusiness.industrySunitinibResearchlcsh:RGeneral MedicineMiddle AgedKidney NeoplasmsNephrectomyAxitinibTreatment030104 developmental biologyRenal cancer030220 oncology & carcinogenesisMultivariate AnalysisMetastaticFemalebusinessmedicine.drugJournal of Translational Medicine
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A constitutive BCL2 down-regulation aggravates the phenotype of PKD1-mutant-induced polycystic kidney disease

2017

IF 5.340; International audience; The main identified function of BCL2 protein is to prevent cell death by apoptosis. Mice knock-out for Bcl2 demonstrate growth retardation, severe polycystic kidney disease (PKD), gray hair and lymphopenia, and die prematurely after birth. Here, we report a 40-year-old male referred to for abdominal and thoracic aortic dissection with associated aortic root aneurysm, PKD, lymphocytopenia with a history of T cell lymphoblastic lymphoma, white hair since the age of 20, and learning difficulties. PKD, which was also detected in the father and sister, was related to an inherited PKD1 mutation. The combination of PKD with gray hair and lymphocytopenia was also r…

AdultMale0301 basic medicineTRPP Cation Channelsphenotypebcl2 geneBiologymicro rnaMice03 medical and health sciencesdown-regulationsymptom aggravating factorshemic and lymphatic diseasest-lymphocyteGene expressionGeneticsmedicinePolycystic kidney diseaseAnimalsHumansGenetic Predisposition to Disease[ SDV.GEN.GH ] Life Sciences [q-bio]/Genetics/Human geneticsgenesMolecular BiologyGeneGenetics (clinical)Exome sequencingMice KnockoutPKD1apoptosisExonsGeneral MedicinePolycystic Kidney Autosomal Dominantmedicine.diseasePhenotypePedigreeUp-Regulation3. Good healthMicroRNAs030104 developmental biologyMRNA SequencingProto-Oncogene Proteins c-bcl-2[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsImmunologyCancer researchLymphocytopeniapolycystic kidney diseasesbcl-2 proteinHuman Molecular Genetics
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