Search results for "LIGAND"

showing 10 items of 2559 documents

Prostasomes as zinc ligands in human seminal plasma

2004

Prostasomes are small vesicles, containing zinc, secreted by prostate in human seminal plasma and showing a physiological role on sperm properties. In this study, the possible correspondence between prostasomes and a prostatic high molecular weight protein complex, recently indicated as zinc ligand, has been investigated. Isolated prostasomes, examined by scanning electron microscopy, were dialysed to evaluate their zinc binding capacity. Furthermore, seminal plasma Sephadex G-75 elution was carried out before and after prostasome removal. Prostasome preparations, containing typical vesicles of 50-500 nm, showed a positive correlation between their zinc and protein levels. They were able to…

AdultMalemedicine.medical_specialtyUrologyEndocrinology Diabetes and Metabolismchemistry.chemical_elementSemenZincBiologyLigandsSemenProstateInternal medicinemedicineHumansLigandVesicleProstateSpermMolecular WeightZincmedicine.anatomical_structureEndocrinologyReproductive MedicinechemistryBiochemistrySephadexHuman Prostasomes Prostate Seminal plasma Zinc ligandsMicroscopy Electron ScanningProstasomesCarrier ProteinsSettore M-EDF/01 - Metodi E Didattiche Delle Attivita' MotorieInternational Journal of Andrology
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Polymorphisms in TRAIL receptor genes and risk of breast cancer in Spanish women

2007

TRAIL is a potent inducer of apoptosis in malignant but not in normal cells. TRAIL binds to the proapoptotic death receptor DR4 and DR5 as well as to the decoy receptors DcR1 and DcR2. To evaluate the involvement of TRAIL receptor genes in breast cancer, we carried out a case-control study of eight selected polymorphisms in a large sample of Spanish women. Three of the eight selected SNPs (626G/C and 1322G/A in DR4 and 2699A/G in DcR2) showed some evidence of different genotype distributions in a random selection of 535 cases and 480 controls and were therefore studied in our entire sample (1008 cases and 768 controls). For the two DR4 polymorphisms, no differences in genotype or haplotype …

AdultUntranslated regionCancer ResearchLinkage disequilibriumBreast NeoplasmsSingle-nucleotide polymorphismBiologyPolymerase Chain ReactionPolymorphism Single NucleotideBreast cancerGenotypeGeneticsmedicineHumansDecoy receptorsskin and connective tissue diseasesReceptorAgedHaplotypeAge FactorsGeneral MedicineMiddle Agedmedicine.diseaseReceptors TNF-Related Apoptosis-Inducing LigandOncologySpainImmunologyFemaleCancer Biomarkers
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Control of target cell survival in thyroid autoimmunity by T helper cytokines via regulation of apoptotic proteins

2000

After autoimmune inflammation, interactions between CD95 and its ligand (CD95L) mediate thyrocyte destruction in Hashimoto's thyroiditis (HT). Conversely, thyroid autoimmune processes that lead to Graves' disease (GD) result in autoantibody-mediated thyrotropin receptor stimulation without thyrocyte depletion. We found that GD thyrocytes expressed CD95 and CD95L in a similar manner to HT thyrocytes, but did not undergo CD95-induced apoptosis either in vivo or in vitro. This pattern was due to the differential production of TH1 and TH2 cytokines. Interferon gamma promoted caspase up-regulation and CD95-induced apoptosis in HT thyrocytes, whereas interleukin 4 and interleukin 10 protected GD …

Adultendocrine systemmedicine.medical_specialtyFas Ligand Proteinendocrine system diseasesCell SurvivalImmunologyCASP8 and FADD-Like Apoptosis Regulating ProteinThyroid Glandbcl-X ProteinApoptosisMice TransgenicIn Vitro TechniquesThyroiditisThyrotropin receptorMiceTh2 CellsSettore MED/04 - PATOLOGIA GENERALEInternal medicinemedicineImmunology and AllergyAnimalsHumansInterferon gammafas ReceptorInterleukin 4CaspaseMembrane GlycoproteinsbiologyThyroidIntracellular Signaling Peptides and ProteinsThyroiditis AutoimmuneT-Lymphocytes Helper-InducerMiddle AgedTh1 CellsFas receptormedicine.diseaseGraves DiseaseInterleukin 10medicine.anatomical_structureEndocrinologyProto-Oncogene Proteins c-bcl-2biology.proteinCytokinesCarrier Proteinsmedicine.drug
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Nickel(II) Complex of a Hexadentate Ligand with Two o-Iminosemiquinonato(1-) π-Radical Units and Its Monocation and Dication.

2016

Aerobic reaction of a hexadentate redox-active o-aminophenol-based ligand, H4L(3) = N,N'-bis(2-hydroxy-3,5-di-tert-butylphenyl)-2,2'-diamino(diphenyldithio)-ethane, in CH3OH with Ni(II)(O2CCH3)2·4H2O and Et3N afforded isolation of a reddish-brown crystalline solid [Ni(L(3))] 1. Cyclic voltammetry (CV) experiment exhibits two oxidative responses at E1/2 = 0.09 and 0.53 V vs SCE (saturated calomel electrode). Chemical oxidation of 1 in air by [Fe(III)(η(5)-C5H5)2][PF6] and AgBF4 in CH2Cl2 led to the isolation of one-electron oxidized species [1](1+) as purple [1][PF6]·CH2Cl2 and two-electron oxidized species [1](2+) as dark purple [1][BF4]2·CH2Cl2, respectively. X-ray crystallographic analysi…

Aerobic reaction010405 organic chemistryStereochemistryChemistryLigandchemistry.chemical_element010402 general chemistry01 natural sciences0104 chemical sciencesDicationInorganic ChemistryHexadentate ligandCrystallographyNickelSaturated calomel electrodePhysical and Theoretical ChemistryCyclic voltammetryInorganic chemistry
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CCR5 Receptor: Biologic and Genetic Implications in Age-Related Diseases

2007

The CC chemokine receptor 5 (CCR5) is a member of CC-chemokine receptor family. CCR5 has the characteristic structure of a seven transmembrane G protein-coupled receptor (GPCR), which regulates trafficking and effector functions of memory/effector Th1 cells, macrophages, NK cells, and immature dendritic cells. CCR5 and its ligands are important molecules in viral pathogenesis. CCR5 represents the co-receptor for macrophage (M) and dual (T cell and M)-tropic immunodeficiency viruses. Recent evidence has also demonstrated the role of CCR5 in a variety of human diseases, ranging from infectious and inflammatory diseases to cancer. In this article, we describe the involvement of CCR5 in two age…

AgingChemokineReceptors CCR5Chemokine receptor CCR5virusesT cellViral pathogenesisDiseaseLigandsModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyHistory and Philosophy of Sciencecardiovascular diseaseAlzheimer DiseasemedicineHumansMacrophageSettore MED/04 - Patologia GeneraleInflammationGenomebiologyEffectorMacrophagesGeneral Neurosciencevirus diseasesDendritic CellsAtherosclerosisKiller Cells Naturalmedicine.anatomical_structureCardiovascular DiseasesImmunologybiology.proteinMicrogliaCC chemokine receptorsAlzheimer’s diseaseCCR5Gene DeletionAnnals of the New York Academy of Sciences
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Nociceptin/orphanin FQ opioid receptor (NOP) selective ligand MCOPPB links anxiolytic and senolytic effects

2021

Accumulation of senescent cells may drive age-associated alterations and pathologies. Senolytics are promising therapeutics that can preferentially eliminate senescent cells. Here, we performed a high-throughput automatized screening (HTS) of the commercial LOPAC®Pfizer library on aphidicolin-induced senescent human fibroblasts, to identify novel senolytics. We discovered the nociceptin receptor FQ opioid receptor (NOP) selective ligand 1-[1-(1-methylcyclooctyl)-4-piperidinyl]-2-[(3R)-3-piperidinyl]-1H-benzimidazole (MCOPPB, a compound previously studied as potential anxiolytic) as the best scoring hit. The ability of MCOPPB to eliminate senescent cells in in vitro models was further tested…

Agingmedicine.drug_classNarcotic AntagonistsNOPMCOPPBSenescenceLigandsAnxiolyticMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePiperidinesSenotherapeuticsOpioid receptormedicineAnimalsHumansSenolyticCaenorhabditis elegansReceptorSenolyticCellular Senescence030304 developmental biology0303 health sciencesNOPSenolytic.ChemistryLigand (biochemistry)High-Throughput Screening Assays3. Good healthCell biologyAnalgesics OpioidNociceptin receptorAnti-Anxiety AgentsOpioid PeptidesReceptors OpioidOriginal ArticleGeriatrics and Gerontology030217 neurology & neurosurgeryGeroScience
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Opiate-induced dopamine release is modulated by severity of alcohol dependence: an [(18)F]fallypride positron emission tomography study.

2011

Background Preclinical data implicate the reinforcing effects of alcohol to be mediated by interaction between the opioid and dopamine systems of the brain. Specifically, alcohol-induced release of β-endorphins stimulates μ-opioid receptors (MORs), which is believed to cause dopamine release in the brain reward system. Individual differences in opioid or dopamine neurotransmission have been suggested to be responsible for enhanced liability to abuse alcohol. In the present study, a single dose of the MOR agonist remifentanil was administered in detoxified alcohol-dependent patients and healthy control subjects to mimic the β-endorphin-releasing properties of ethanol and to assess the effect…

AgonistAdultMaleFluorine RadioisotopesPyrrolidinesmedicine.drug_classDopamineReceptors Opioid muPharmacologySeverity of Illness IndexRemifentanilRadioligand AssayDopamine receptor D1PiperidinesDopamine receptor D3DopaminemedicineLimbic SystemHumansBiological PsychiatryReceptors Dopamine D2PutamenFunctional NeuroimagingVentral striatumAlcohol dependenceMiddle AgedAnalgesics OpioidBehavior AddictiveAlcoholismmedicine.anatomical_structurenervous systemFallypridePositron-Emission TomographyBenzamidesPsychologymedicine.drugBiological psychiatry
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Synthesis and Characterization of a Novel Series of Agonist Compounds as Potential Radiopharmaceuticals for Imaging Dopamine D-2/3 Receptors in Their…

2014

Imaging of dopamine D2/3 receptors (D2/3R) can shed light on the nature of several neuropsychiatric disorders in which dysregulation of D2/3R signaling is involved. Agonist D2/3 tracers for PET/SPECT imaging are considered to be superior to antagonists because they are more sensitive to dopamine concentrations and may selectively label the high-affinity receptor state. Carbon-11-labeled D2/3R agonists have been developed, but these short-lived tracers can be used only in centers with a cyclotron. Here, we report the development of a series of novel D2R agonist compounds based on the 2-aminomethylchromane (AMC) scaffold that provides ample opportunities for the introduction of longer-lived […

AgonistD-3 RECEPTORPHARMACOPHOREChemistrymedicine.drug_classDERIVATIVESPharmacologyIN-VIVO ACTIVITYHUMAN BRAINRadioligand AssayANTERIOR-PITUITARYPOSITRON-EMISSION-TOMOGRAPHYENDOGENOUS DOPAMINEIn vivoDopamineSpect imagingDopamine receptor D2Drug DiscoveryLIGAND-BINDINGmedicineMolecular MedicineReceptorAGENTSEndogenous agonistmedicine.drugJournal of Medicinal Chemistry
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Human oxytocin receptors in cholesterol-rich vs. cholesterol-poor microdomains of the plasma membrane

2000

We analyzed the properties of a G protein-coupled receptor localized in cholesterol-poor vs. cholesterol-rich microdomains of the plasma membrane. For this purpose, the human oxytocin receptor, which is very sensitive against alterations of the membrane cholesterol level, was stably expressed in HEK293 cells. To calculate the total number of receptors independent of ligand binding studies, the oxytocin receptor was tagged with an enhanced green fluorescent protein (EGFP) which did not change the functional properties of the receptor. Only 1% of the oxytocin receptors were present in cholesterol-rich detergent-insoluble domains. In contrast, employing a detergent-free fractionation scheme th…

AgonistGTP'medicine.drug_classPlasma protein bindingBiologyBiochemistryOxytocin receptorReceptor–ligand kineticsMembraneBiochemistryCaveolaeBiophysicsmedicineReceptorEuropean Journal of Biochemistry
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Na+ ions binding to the bradykinin B2 receptor suppress agonist-independent receptor activation.

1996

Control of the balance between receptor activation and inactivation is a prerequisite for seven transmembrane domain (7TM) receptor function. We asked for a mechanism to stabilize the inactive receptor conformation which prevents agonist-independent receptor activation. Na+ ions have reciprocal effects on agonist versus antagonist interaction with various 7TM receptors. To investigate the Na+ dependence of receptor activation we chose the bradykinin B2 receptor as a prototypic 7TM receptor. Decrease of the intracellular Na+ content from 40 mM to 10 mM of COS-1 cells transiently expressing rat B2 receptors activated the B2 receptor in the absence of agonist as shown by a 3-fold increase in t…

AgonistIntracellular FluidIntrinsic activityReceptor Bradykinin B2medicine.drug_classInositol PhosphatesBradykininIn Vitro TechniquesBradykininLigandsBiochemistryCell Linechemistry.chemical_compoundmedicineAnimalsHumansPoint MutationBradykinin receptorPhosphorylationReceptorG protein-coupled receptorReceptors BradykininSodiumRatschemistryCOS CellsBiophysicsMutagenesis Site-DirectedAlpha-4 beta-2 nicotinic receptorIntracellularBiochemistry
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