Search results for "LIGAND"

showing 10 items of 2559 documents

ICOS and CD28 reversely regulate IL-10 on re-activation of human effector T cells with mature dendritic cells

2002

With newly generated ICOS-ligand (ICOS-L)-specific monoclonal antibodies we determined that human Langerhans cells in situ express similar levels of ICOS-L, CD80, and CD86, compared to immature dendritic cells (DC) derived from monocytes in vitro. Maturation of DC strongly up-regulated CD80 and CD86 but did not significantly change ICOS-L levels. On coculture of "naive"CD4(+) T cells with mature DC in the presence of superantigen, ICOS was highly up-regulated on T cells, but played only a secondary role in the CD28-dominated release of TNF-alpha and IFN-gamma, and did not participate in the induction of IL-2. Cocultures of "effector" CD4(+) T cells with mature DC revealed CD28 as the drivin…

CD86EffectorImmunologyCD28chemical and pharmacologic phenomenahemic and immune systemsBiologyCell biologyICOS LIGANDInterleukin 10CTLA-4Immunology and AllergySecretionCD80European Journal of Immunology
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Comparing molecular dynamics-derived pharmacophore models with docking: A study on CDK-2 inhibitors

2020

Abstract We compared the performance of molecular dynamics (MD)-derived pharmacophore modeling approaches, Common Hit Approach (CHA), and the Molecular dYnamics SHAred PharmacophorE (MYSHAPE) approach, with semi-flexible constrained/unconstrained docking. The aim of this work is to enrich the hit-list of a virtual screening on CDK-2 known inhibitors as a case study. Cyclin-dependent kinases (CDKs) deregulation is associated with cancer growth. CDKs are an attractive target for anticancer agents. MD-derived pharmacophore models have been obtained with LigandScout 4.2.1. Docking analysis has been performed through Glide 7.6. The results highlighted the MYSHAPE approach has a better performanc…

CDK2Virtual screeningbiologyPharmacophore010405 organic chemistryChemistryGeneral ChemistryComputational biologyMolecular dynamics010402 general chemistry01 natural sciencesDynamic pharmacophoreLigandScout0104 chemical sciencesDockingMolecular dynamicsCyclin-dependent kinaseDocking (molecular)biology.proteinPharmacophore
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Cobalt Electrolyte/Dye Interactions in Dye-Sensitized Solar Cells: A Combined Computational and Experimental Study

2012

We report a combined experimental and computational investigation to understand the nature of the interactions between cobalt redox mediators and TiO2 surfaces sensitized by :ruthenium and organic dyes, and their impact on. the performance of the corresponding dye-sensitized solar cells (DSSCs). We : focus: on different ruthenium dyes and fully organic dyes, to understand the dramatic loss of efficiency observed for the prototype Ru(II) N719 dye in conjunction with :Cobalt: electrolytes. Both N719- and Z907-based DSSCs showed an increased lifetime in iodine-based electrolyte compared to the cobalt-based redox-shuttle; While the organic D21L6 and D25L6 cycles endowed.With long alkoxy chains,…

COLLOIDAL TIO2 FILMSinorganic chemicalsLOW QUANTUM YIELDSInorganic chemistrychemistry.chemical_element02 engineering and technologyElectrolyte010402 general chemistryPhotochemistry01 natural sciencesBiochemistryRedoxREDOX COUPLECatalysisEFFECTIVE CORE POTENTIALSDENSITY-FUNCTIONAL THEORYColloid and Surface ChemistryDENSITY-FUNCTIONAL THEORY; EFFECTIVE CORE POTENTIALS; INTRAMOLECULAR ELECTRON-TRANSFER; TRANSITION-METAL-COMPLEXES; COLLOIDAL TIO2 FILMS; LOW QUANTUM YIELDS; MOLECULAR CALCULATIONS; REDOX COUPLE; MAGNETIC-PROPERTIES; PHOTOVOLTAIC CELLSMAGNETIC-PROPERTIESPHOTOVOLTAIC CELLSLigandGeneral Chemistry021001 nanoscience & nanotechnologyMOLECULAR CALCULATIONSTRANSITION-METAL-COMPLEXES0104 chemical sciencesMarcus theoryRutheniumDye-sensitized solar cellchemistryAlkoxy groupINTRAMOLECULAR ELECTRON-TRANSFER0210 nano-technologyCobalt
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Spectroscopic, crystal structural, theoretical and biological studies of phenylacetohydrazide Schiff base derivatives and their copper complexes

2020

Two phenylacetohydrazide Schiff base derivatives: N’-(1-(2-hydroxyphenyl)ethylidene)-2-phenylacetohydrazide, HL1, and N’-((1-hydroxynaphthalen-2-yl)methylene)-2-phenylacetohydrazide, HL2, were synthesized. HL1 dimerizes in presence of HCl, probably via radical mechanism to give (2,2’-((1E)-hydrazine-1,2-diylidenebis(ethan-1-yl-1-ylidene))diphenol (DIM). Thermal reactions of Cu(II) ions with the two Schiff base ligands resulted in formation of the binuclear complexes [(CuL1)2] and [(CuL2)2]. The stoichiometry and structures of the reported compounds were investigated by several spectroscopic and analytical techniques. The structure of the HL1 ligand and its complex [(CuL1)2] as well as the D…

CT-DNA bindingantioxidant activitychemistry.chemical_elementkupari010402 general chemistry01 natural sciencesAnalytical ChemistryInorganic Chemistrykemialliset sidoksetchemistry.chemical_compoundDFT studiesReactivity (chemistry)copper complexesMethyleneSpectroscopyantioksidantitSchiff base010405 organic chemistryLigandtiheysfunktionaaliteoriaOrganic Chemistrymolecular dockingkompleksiyhdisteetCopper0104 chemical sciencesCrystallographyMolecular geometrychemistrySingle crystalröntgenkristallografiaStoichiometryX-ray analysisJournal of Molecular Structure
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Conformationally regulated fluorescent sensors. Study of the selectivity in Zn 2+ versus Cd 2+ sensing

2004

Abstract The Zn 2+ and Cd 2+ complexing properties of four ligands containing a 4,4′-substituted biphenyl moiety are described. Ligands 1 and 3 , containing only one 1-aza-18-crown-6 cavity, lead to selective complexation of Cd 2+ versus Zn 2+ . Ligand 4 , with two crown cavities linked to a tetramethylbenzidine unit, is able to form 1:1 complexes with Zn 2+ and Cd 2+ , showing a higher complexing constant with Zn 2+ than with Cd 2+ , probably due to enthalpic factors. Several complementary experiments suggest that the 1:1 complexes formed by ligand 4 involve both crown cavities acting together to give rise to clamp structures. The formation of this type of zinc complex gives rise to red sh…

CadmiumQuenching (fluorescence)ChemistryStereochemistryLigandOrganic Chemistrychemistry.chemical_elementZincBiochemistryFluorescenceCrystallographyDrug DiscoveryNitroMoietySelectivityTetrahedron
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Whole-body biodistribution of the cannabinoid type 1 receptor ligand [ 18 F]MK-9470 in the rat

2017

The endocannabinoid system participates in many processes in the body, including memory, reward, pain, motor activity, food intake, energy metabolism, and gastrointestinal functions. [18F]MK-9470 is a positron emission tomography (PET) ligand that binds with high affinity and selectivity to the cannabinoid type 1 receptor. In order to fully characterize ligand behavior, tracer uptake measured using in vivo microPET was compared with results from ex vivo tissue dissection. Twelve male Sprague-Dawley rats were divided into three subgroups and scanned over time periods of 10min, 30min and 90min using PET. Afterwards, a number of the animals' organs were dissected. Uptake of radioactivity was e…

Cancer ResearchBiodistributionChemistrymedicine.medical_treatmentMetaboliteMK-9470PharmacologyLigand (biochemistry)030218 nuclear medicine & medical imaging03 medical and health scienceschemistry.chemical_compound0302 clinical medicineIn vivomedicineMolecular MedicineRadiology Nuclear Medicine and imagingCannabinoidReceptor030217 neurology & neurosurgeryEx vivoNuclear Medicine and Biology
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SAHA induces apoptosis in hepatoma cells and synergistically interacts with the proteasome inhibitor Bortezomib.

2007

Histone deacetylase (HDAC) inhibitors represent a promising group of anticancer agents. This paper shows that the HDAC inhibitor suberoylanilide hydroxamic acid (SAHA) stimulated at 5-10 microM apoptosis in human hepatoma HepG2 and Huh6 cells, but was ineffective in primary human hepatocytes (PHH). In HepG2 cells SAHA induced the extrinsic apoptotic pathway, increasing the expression of both FasL and FasL receptor and causing the activation of caspase-8. Moreover, SAHA enhanced the level of Bim proteins, stimulated alternative splicing of the Bcl-X transcript with the expression of the proapoptotic Bcl-Xs isoform, induced degradation of Bid into the apoptotic factor t-Bid and dephosphorylat…

Cancer ResearchCarcinoma HepatocellularFas Ligand ProteinClinical BiochemistryPharmaceutical ScienceApoptosisHydroxamic AcidsFas ligandHistone DeacetylasesBortezomibCell Line TumormedicineHumansProtease InhibitorsProtein kinase BVorinostatHDAC inhibitors . HepG2 cells . PHH . Extrinsic and intrinsic apoptotic pathwaysbcl-2-Associated X ProteinPharmacologyMembrane Potential MitochondrialCaspase 8VorinostatbiologyChemistryBortezomibCytochrome cBiochemistry (medical)Cell BiologyBoronic AcidsHistone Deacetylase InhibitorsProteasomeApoptosisPyrazinesProteasome inhibitorbiology.proteinCancer researchApoptosis Regulatory ProteinsProteasome Inhibitorsmedicine.drug
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Membrane vesicles shed by oligodendroglioma cells induce neuronal apoptosis.

2006

In order to investigate the mechanism by which oligodendrogliomas cause neuronal damage, media conditioned by G26/24 oligodendroglioma cells, were fractionated into shed vesicles and vesicle-free supernatants, and added to primary cultures of rat fetal cortical neurons. After one night treatment with vesicles, a reproducible, dose-dependent, inhibitory effect on neurite outgrowth was already induced and, after 48-72 h of incubation, neuronal apoptosis was evident. Vesicle-free supernatants and vesicles shed by NIH-3T3 cells had no inhibitory effects on neurons. Western blot analyses showed that treated neurons expressed a decreased amount of neurofilament (NF), growth-associated protein (GA…

Cancer ResearchCell signalingProgrammed cell deathPathologymedicine.medical_specialtyNeurofilamentFas Ligand ProteinNeuriteCellOligodendrogliomaApoptosisCell CommunicationBiologyRats Sprague-DawleyMiceWestern blotmedicineAnimalsMyelin SheathCerebral CortexNeuronsmedicine.diagnostic_testVesicleCytoplasmic Vesiclesoligodendroglioma membrane vesicles neuronal apoptosis Fas-L Nogo.Cell biologyRatsmedicine.anatomical_structureOncologyNIH 3T3 CellsNeuronInternational journal of oncology
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Intracellular coexpression of CXC- and CC– chemokine receptors and their ligands in human melanoma cell lines and dynamic variations after xenotransp…

2014

Abstract Background Chemokines have been implicated in tumor progression and metastasis. In melanoma, chemokine receptors have been implicated in organ selective metastasis by regulating processes such as chemoattraction, adhesion and survival. Methods In this study we have analyzed, using flow cytometry, the systems formed by the chemokine receptors CXCR3, CXCR4, CXCR7, CCR7 and CCR10 and their ligands in thirteen human melanoma cell lines (five established from primary tumors and eight established from metastasis from different tissues). WM-115 and WM-266.4 melanoma cell lines (obtained from a primary and a metastatic melanoma respectively) were xenografted in nude mice and the tumors and…

Cancer ResearchChemokine receptorIntracellular SpaceBiologyCCL7LigandsChemokine receptorMiceReceptors CCRCell Line TumorGeneticsAnimalsHumansCCR10CXC chemokine receptorsCCL13MelanomaReceptors CXCRChemotaxisCell MembraneImmunohistochemistry3. Good healthCXCL2Disease Models AnimalOncologyChemokineCancer researchHeterograftsXenotransplantationCC chemokine receptorsCell lineCCL21Research ArticleBMC Cancer
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Ultraviolet light-induced DNA damage triggers apoptosis in nucleotide excision repair-deficient cells via Bcl-2 decline and caspase-3/-8 activation.

2001

Ultraviolet (UV) light is a potent mutagenic and genotoxic agent. Whereas DNA damage induced by UV light is known to be responsible for UV-induced genotoxicity, its role in triggering apoptosis is still unclear. We addressed this issue by comparing nucleotide excision repair (NER) deficient 27-1 and 43-3B Chinese hamster (CHO) cells with the corresponding wild-type and ERCC-1 complemented cells. It is shown that NER deficient cells are dramatically hypersensitive to UV-C induced apoptosis, indicating that DNA damage is the major stimulus for the apoptotic response. Apoptosis triggered by UV-C induced DNA damage is related to caspase- and proteosome-dependent degradation of Bcl-2 protein. Th…

Cancer ResearchDNA RepairDNA repairDNA damageUltraviolet RaysPoly ADP ribose polymeraseFas-Associated Death Domain ProteinApoptosisCHO CellsBiologyCysteine Proteinase InhibitorsCaspase 8TransfectionFas ligandMembrane PotentialsCricetinaeGeneticsUltraviolet lightAnimalsRNA MessengerMolecular BiologyAdaptor Proteins Signal TransducingCaspase 8Caspase 3Fas receptorMolecular biologyCaspase InhibitorsCaspase 9MitochondriaEnzyme ActivationProto-Oncogene Proteins c-bcl-2CaspasesPoly(ADP-ribose) PolymerasesCarrier ProteinsNucleotide excision repairDNA DamageOncogene
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