Search results for "LIPIDS"

showing 10 items of 2228 documents

Membrane-Derived Phospholipids Control Synaptic Neurotransmission and Plasticity

2015

Synaptic communication is a dynamic process that is key to the regulation of neuronal excitability and information processing in the brain. To date, however, the molecular signals controlling synaptic dynamics have been poorly understood. Membrane-derived bioactive phospholipids are potential candidates to control short-term tuning of synaptic signaling, a plastic event essential for information processing at both the cellular and neuronal network levels in the brain. Here, we showed that phospholipids affect excitatory and inhibitory neurotransmission by different degrees, loci, and mechanisms of action. Signaling triggered by lysophosphatidic acid (LPA) evoked rapid and reversible depress…

MalePatch-Clamp TechniquesQH301-705.5NeurotransmissionBiologyInhibitory postsynaptic potentialSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyMicePregnancySynaptic augmentationMetaplasticityAnimalsRats WistarBiology (General)Motor Neuronsrho-Associated KinasesNeuronal PlasticityGeneral Immunology and MicrobiologyCalcineurinGeneral NeuroscienceReceptors GABA-ACell biologySynaptic fatigueBiochemistrySynapsesSynaptic plasticityExcitatory postsynaptic potentialFemalelipids (amino acids peptides and proteins)Synaptic signalingLysophospholipidsrhoA GTP-Binding ProteinGeneral Agricultural and Biological SciencesResearch Article
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Plasma lysosphingomyelin demonstrates great potential as a diagnostic biomarker for Niemann-Pick disease type C in a retrospective study.

2015

Niemann-Pick disease type C (NP-C) is a devastating, neurovisceral lysosomal storage disorder which is characterised by variable manifestation of visceral signs, progressive neuropsychiatric deterioration and premature death, caused by mutations in the NPC1 and NPC2 genes. Due to the complexity of diagnosis and the availability of an approved therapy in the EU, improved detection of NP-C may have a huge impact on future disease management. At the cellular level dysfunction or deficiency of either the NPC1 or NPC2 protein leads to a complex intracellular endosomal/lysosomal trafficking defect, and organ specific patterns of sphingolipid accumulation. Lysosphingolipids have been shown to be e…

MalePathologyEndocrinology Diabetes and Metabolismlcsh:MedicineDiseaseBiochemistryEndocrinologySphingosineTandem Mass Spectrometrylcsh:ScienceBlood Specimen CollectionMultidisciplinaryNiemann-Pick Disease Type CInherited Metabolic DisordersLipidsBiomarker (medicine)FemaleNiemann–Pick diseaseNiemann-Pick diseaseResearch ArticleAdultmedicine.medical_specialtyAdolescentPhosphorylcholineYoung AdultDiagnostic MedicineGeneticsmedicineHumansSphingolipidosisClinical geneticsMolecular BiologyEdetic AcidAgedRetrospective StudiesMedicine and health sciencesSphingolipidsNiemann–Pick disease type Cbusiness.industryHeparinlcsh:RCase-control studyPsychosineReproducibility of ResultsBiology and Life SciencesRetrospective cohort studymedicine.diseaseSphingolipidCase-Control StudiesAutosomal recessive diseasesMetabolic Disorderslcsh:QNPC1businessLysosphingomyelinBiomarkersPloS one
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Lipid composition of the human eye: are red blood cells a good mirror of retinal and optic nerve fatty acids?

2011

International audience; BACKGROUND: The assessment of blood lipids is very frequent in clinical research as it is assumed to reflect the lipid composition of peripheral tissues. Even well accepted such relationships have never been clearly established. This is particularly true in ophthalmology where the use of blood lipids has become very common following recent data linking lipid intake to ocular health and disease. In the present study, we wanted to determine in humans whether a lipidomic approach based on red blood cells could reveal associations between circulating and tissue lipid profiles. To check if the analytical sensitivity may be of importance in such analyses, we have used a do…

MalePathologyErythrocytes[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionBlood lipidsBiochemistryAnalytical Chemistrychemistry.chemical_compound0302 clinical medicineMolecular Cell BiologyPathologychemistry.chemical_classificationAged 80 and overLiquid Chromatography0303 health sciencesChromatographyMultidisciplinaryLipid ClassesQFatty AcidsRLipids3. Good healthChemistrymedicine.anatomical_structureDocosahexaenoic acidAlimentation et NutritionBlood ChemistryOptic nerveMedicineRetinal DisordersArachidonic acidFemalelipids (amino acids peptides and proteins)Fatty AlcoholsCellular TypesPolyunsaturated fatty acidResearch Articlemedicine.medical_specialtySpectrometry Mass Electrospray IonizationScienceGas Chromatography-Mass SpectrometryRetinaGlycerides03 medical and health sciencesDiagnostic MedicineLipidomicsLipid StructuremedicineFood and NutritionHumansBiology030304 developmental biologyAgedGas ChromatographyNutritionRetinaBlood CellsRetinalOptic NerveGlaucomaOphthalmologychemistryMacular Disorders030221 ophthalmology & optometry[SDV.AEN]Life Sciences [q-bio]/Food and NutritionBiomarkersGeneral Pathology
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Role of Natural Killer Activity in Development of Spontaneous Metastases in Murine Renal Cancer

1985

Abstract We have studied the role of natural killer activity during the growth and dissemination of a transplantable renal adenocarcinoma (Renca) of spontaneous origin in BALB/c mice. The pattern of growth of this tumor accurately mimics that of adult human renal cell carcinoma in terms of clinical stages I–IV, particularly with regard to spontaneous metastasis to lung and liver. Renca is moderately sensitive to lysis by natural killer cells from normal mice and is more efficiently lysed by natural killer cells from mice treated with the biological response modifier maleic anhydride divinyl ether, a pyran copolymer. Our studies demonstrate that selective depression of natural killer activit…

MalePathologymedicine.medical_specialtyLung NeoplasmsLysisPyran CopolymerUrologyG(M1) GangliosideGlycosphingolipidsCell LineMiceRenal cell carcinomamedicineAnimalsNeoplasm MetastasisCarcinoma Renal CellAntiserumMice Inbred BALB CLymphokine-activated killer cellLungbusiness.industryImmune SeraLiver NeoplasmsKiller activityCancermedicine.diseaseKidney NeoplasmsKiller Cells Naturalmedicine.anatomical_structureFemaleLymphbusinessNeoplasm TransplantationJournal of Urology
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Non-viral VEGF(165) gene therapy--magnetofection of acoustically active magnetic lipospheres ('magnetobubbles') increases tissue survival in an overs…

2008

Abstract Adenoviral transduction of the VEGF gene in an oversized skin flap increases flap survival and perfusion. In this study, we investigated the potential of magnetofection of magnetic lipospheres containing VEGF165-cDNA on survival and perfusion of ischemic skin flaps and evaluated the method with respect to the significance of applied magnetic field and ultrasound. We prepared perfluoropropane-filled magnetic lipospheres (‘magnetobubbles’) from Tween60-coated magnetic nanoparticles, Metafectene, soybean-oil and cDNA and studied the effect in an oversized random-pattern-flap model in the rats (n= 46). VEGF-cDNA-magnetobubbles were administered under a magnetic field with simultaneousl…

MalePathologymedicine.medical_specialtyNecrosismagneticAngiogenesisGenetic enhancementDermatologic Surgical ProceduresEnzyme-Linked Immunosorbent AssayTransfectionSurgical FlapsRats Sprague-DawleyMagneticsangiogenesismagnetobubblesmedicineAnimalsUltrasonicsSkinbusiness.industryVascular Endothelial Growth FactorsMusclesUltrasoundGraft SurvivalCell BiologyTransfectionGenetic TherapySkin TransplantationArticlesequipment and suppliesLipidsVEGFgene therapyMicrospheresRatsMicrovesselsModels AnimalMagnetofectionMolecular MedicineMagnetic nanoparticleslipospheresmedicine.symptombusinessPerfusionhuman activitiesmagnetofectionJournal of cellular and molecular medicine
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Ultrastructural studies of the retina in infantile neuronal ceroid-lipofuscinosis.

1988

A 9-year-old boy who had died of infantile neuronal ceroid-lipofuscinosis had experienced retina-derived visual failure. Ophthalmologically and morphologically, his retina was severely atrophic and scarred by a dense fibrillary gliosis while photoreceptor cells had completely disappeared, cells of the bipolar layer had decreased in number and had become atrophic beyond cytologic recognition. Retinal pigment epithelial cells had undergone either atrophy or proliferation. Disease-specific granular lipopigments had accumulated in perikarya and processes of remaining cells and were infrequently associated with melanin within huge melanolipofuscin bodies and RPE cells of sessile and migrating na…

MalePathologymedicine.medical_specialtygenetic structuresInfantile neuronal ceroid lipofuscinosisCytoplasmic GranulesRetinaLipofuscinMelaninchemistry.chemical_compoundAtrophyNeuronal Ceroid-LipofuscinosesCytologymedicineHumansChildMelaninsRetinaMembranesbusiness.industryRetinalGeneral MedicinePigments Biologicalmedicine.diseaseLipidseye diseasesOphthalmologymedicine.anatomical_structurechemistryUltrastructuresense organsbusinessRetinopathyRetina (Philadelphia, Pa.)
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Nutritional Status of Maintenance Dialysis Patients: Low Lean Body Mass Index and Obesity Are Common, Protein-Energy Wasting Is Uncommon.

2016

Background and Aims Maintenance dialysis patients are at increased risk of abnormal nutritional status due to numerous causative factors, both nutritional and non-nutritional. The present study assessed the current prevalence of protein-energy wasting, low lean body mass index and obesity in maintenance dialysis patients, and compared different methods of nutritional assessment. Methods In a cross-sectional study conducted in 2014 at Roskilde Hospital, Denmark, we performed anthropometry (body weight, skinfolds, mid-arm, waist, and hip circumferences), and determined plasma albumin and normalized protein catabolic rate in order to assess the prevalence of protein-energy wasting, low lean bo…

MalePediatricsProtein–energy malnutritionPhysiologyCross-sectional study030232 urology & nephrologySocial Scienceslcsh:Medicine030204 cardiovascular system & hematologyBiochemistryBody Mass IndexFats0302 clinical medicinePrevalenceMedicine and Health SciencesYoung adultlcsh:ScienceWastingAged 80 and overMultidisciplinaryAnthropometryMiddle AgedLipidsPhysiological ParametersNephrologyPhysical SciencesBody CompositionEngineering and TechnologyFemalePhysical AnthropologyWaist Circumferencemedicine.symptomResearch ArticleAdultmedicine.medical_specialtyMaterials ScienceNutritional StatusFuelsProtein-Energy MalnutritionYoung Adult03 medical and health sciencesRenal DialysisAlbuminsInternal medicineMedical DialysismedicineHumansObesityMaterials by AttributeAgedNutritionbusiness.industryBody Weightlcsh:RBiology and Life SciencesProteinsAnthropometrymedicine.diseaseObesityEnergy and PowerCross-Sectional StudiesEndocrinologyAnthropologyLean body massKidney Failure Chroniclcsh:QbusinessBody mass indexPLoS ONE
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Severe infantile colic and food intolerance: A long-term prospective study

1991

To determine the relationship between infantile colic and cow's milk protein intolerance (CMPI) in formula-fed infants, 70 infants (38 male, 32 female) were selected, with mean age 30.2 +/- 21.4 days, with severe colic (duration of crying greater than 4 h per day for 5 days per week). In 50 of the infants in the study group (71.4%) there was a remission of symptoms when cow's milk protein (CMP) was eliminated from the diet. Two successive challenges caused the return of symptoms in all these 50 infants. There was a positive anamnesis for atopy in 9 of 50 of the patients with CMP-related colic and in 1 of 20 of those with non-CMP-related colic (p greater than 0.05). A follow-up period of 18 …

MalePediatricsmedicine.medical_specialtyColicDiet therapydigestive systemInfantile colicAtopyHypersensitivitymedicineHumansProspective StudiesProspective cohort studyAnamnesisCryingbusiness.industryInfant NewbornGastroenterologyInfantMilk Proteinsmedicine.diseasefemale genital diseases and pregnancy complicationsdigestive system diseasesSurgerycarbohydrates (lipids)Food intoleranceDiarrheasurgical procedures operativePediatrics Perinatology and Child HealthFemaleInfant FoodMilk Hypersensitivitymedicine.symptombusinessFood HypersensitivityFollow-Up Studies
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Transcriptional induction of the fatty acid binding protein gene in mouse liver by bezafibrate

1993

AbstractThe mechanism by which hypolipidemic peroxisome proliferators of the fibrate family induce the liver fatty acid binding protein in liver of rodents is unknown. In order to delineate the level at which this protein is induced, the transcriptional activity of the specific gene encoding for liver fatty acid binding protein was measured in isolated hepatocyte nuclei obtained from male Swiss mice daily force-fed during 7 days with 400 mg/kg body weight bezafibrate. This treatment induced a 4-fold increase in the liver fatty acid binding protein transcription rate. Liver fatty acid binding protein mRNA level, measured by Northern blot analysis and cytosolic content of this protein, analyz…

MalePeroxisome proliferator activated receptorTranscription GeneticImmunoblottingBiophysicsPeroxisome proliferator-activated receptorNerve Tissue ProteinsFatty Acid-Binding ProteinsPeroxisome proliferator hypolipidemic drugBiochemistryFatty acid-binding proteinMiceStructural BiologyGeneticsmedicineAnimalsRNA Messengeradipocyte protein 2Molecular Biologychemistry.chemical_classificationLiver fatty acid binding proteinBezafibratebiologyBinding proteinBody WeightCell BiologyOrgan SizePeroxisomeBlotting NorthernMolecular biologyLipidsNeoplasm ProteinsGene regulationFatty acid synthasechemistryBiochemistryGene Expression RegulationLiverbiology.proteinElectrophoresis Polyacrylamide GelPeroxisome proliferator-activated receptor alphaBezafibrateCarrier ProteinsDNA ProbesFatty Acid-Binding Protein 7medicine.drugFEBS Letters
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Phenytoin-induced glutathione depletion in rat peripheral nerve

1995

Abstract Administration of high doses (150–250 mg/kg body weight) of phenytoin (DPH) promote a 40% decrease in glutathione (GSH) content of rat sciatic nerve. This DPH-induced GSH depletion is accompanied with an electrophysiological impairment of peripheral neuromuscular function. H7 (20 mg/kg body weight IP, 30 min prior to DPH), a protein kinase C inhibitor, was able to prevent the DPH-induced GSH depletion only at the lower DPH dose used. This same inhibitor completely prevented the electrophysiological impairment at the lower DPH dose, and only partially at the higher DPH dose used. These results confirm the hypothesis of a DPH-dependent activation of PKC (that might be triggered by, o…

MalePhenytoinAntioxidantmedicine.medical_treatmentAction PotentialsIn Vitro TechniquesPharmacologymedicine.disease_causeBiochemistryPiperazineschemistry.chemical_compound1-(5-Isoquinolinesulfonyl)-2-MethylpiperazinePhysiology (medical)polycyclic compoundsmedicineAnimalsEnzyme InhibitorsRats WistarMuscle SkeletalEvoked PotentialsProtein Kinase CProtein kinase CMotor NeuronsAnalysis of Variancetechnology industry and agricultureNeurotoxicityGlutathioneIsoquinolinesmedicine.diseaseGlutathioneSciatic NerveRatsKineticschemistryBiochemistryPhenytoinAnticonvulsantslipids (amino acids peptides and proteins)Sciatic nerveOxidative stressIntracellularmedicine.drugFree Radical Biology and Medicine
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