Search results for "LIPIDS"

showing 10 items of 2228 documents

Replication of linkage of familial hypobetalipoproteinemia to chromosome 3p in six kindreds

2002

Familial hypobetalipoproteinemia (FHBL) is a genetically heterogeneous condition characterized by very low apolipoprotein B (apoB) concentrations in plasma and/or low levels of LDL-cholesterol (LDL-C) with a propensity to developing fatty liver. In a minority of cases, truncation-specifying mutations of the apoB gene (APOB) are etiologic, but the genetic basis of most cases is unknown. We previously reported linkage of FHBL to a 10 cM region on 3p21.1-22 in one kindred. The objectives of the current study were to identify other FHBL families with linkage to 3p and to narrow the FHBL susceptibility region on 3p. Six additional FHBL kindreds unlinked to the APOB region on chromosome 2 were ge…

Genetic MarkersAdultMaleMeiosiSettore MED/09 - Medicina InternaApolipoprotein BGenotypeGenetic LinkageQD415-436BiologyBiochemistryChromosomal crossoverHypobetalipoproteinemiasEndocrinologyQuantitative Trait HeritableGenetic linkageGenetic MarkerHaplotypeHumanslinkage analysisCrossing Over GeneticChildAgedAdult; Aged; Aged 80 and over; Child; Chromosome Mapping; Chromosomes Human Pair 3; Crossing Over Genetic; Female; Genetic Linkage; Genetic Markers; Genotype; Haplotypes; Humans; Hypobetalipoproteinemias; Male; Meiosis; Middle Aged; Pedigree; Quantitative Trait HeritableGeneticsAged 80 and overGenetic heterogeneityHaplotypeChromosomeChromosome MappingCell BiologyoligogenicMiddle AgedPedigreeMeiosisMarkov chain Monte CarloChromosome 3HaplotypesGenetic markerbiology.proteinvariance componentslipids (amino acids peptides and proteins)FemaleChromosomes Human Pair 3geneticHypobetalipoproteinemiaHuman
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Plasma clearance of human low-density lipoprotein in human apolipoprotein B transgenic mice is related to particle diameter.

2004

To test for intrinsic differences in metabolic properties of low-density lipoprotein (LDL) as a function of particle size, we examined the kinetic behavior of 6 human LDL fractions ranging in size from 251 to 265 A injected intravenously into human apolipoprotein (apo) B transgenic mice. A multicompartmental model was formulated and fitted to the data by standard nonlinear regression using the Simulation, Analysis and Modeling (SAAM II) program. Smaller sized LDL particles (251 to 257 A) demonstrated a significantly slower fractional catabolic rate (FCR) (0.050 +/- 0.045 h(-1)) compared with particles of larger size (262 to 265 A) (0.134 +/- -0.015 h(-1), P.03), and there was a significant …

Genetically modified mouseAdultMalemedicine.medical_specialtySimvastatinApolipoprotein BMetabolic Clearance RateEndocrinology Diabetes and MetabolismPlasma clearance low-density lipoprotein apolipoprotein B trangenic miceMice Transgenicchemistry.chemical_compoundMiceEndocrinologyInternal medicineBlood plasmamedicineAnimalsHumansParticle SizeApolipoproteins BPravastatinbiologyCatabolismMiddle AgedLipoproteins LDLEndocrinologychemistryLow-density lipoproteinModels Animalbiology.proteinRegression Analysislipids (amino acids peptides and proteins)Particle sizeNonlinear regressionLipoproteinMetabolism: clinical and experimental
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No effect of C-reactive protein on early atherosclerosis in LDLR-/- / human C-reactive protein transgenic mice

2008

summaryThe association between increased concentrations of C-reactive protein (CRP) and future cardiovascular events is well established. However, it is currently unclear whether this clinical observation represents an epiphenomenon or whether the pentraxin may actively promote the development of atherosclerosis. Experimental studies with knockout mice with a defect in apolipoprotein E (ApoE-/-) have been used to investigate the role of CRP in atherogenesis, but the results obtained have been contradictory so far. Since knockout mice with a defect in low density lipoprotein receptor (LDLR-/-) may represent a better model of atherogenesis compared to ApoE-/- animals, we undertook experiments…

Genetically modified mouseApolipoprotein ETime FactorsGenotypeLipoproteinsTransgeneMice TransgenicBiologyLesionMicemedicineAnimalsHumansComplement ActivationAortaCrosses GeneticMice KnockoutC-reactive proteinAcute-phase proteinHematologyAtherosclerosisDietary FatsLipidsDisease Models AnimalC-Reactive ProteinPhenotypeReceptors LDLImmunologyLDL receptorKnockout mousebiology.proteinlipids (amino acids peptides and proteins)medicine.symptomThrombosis and Haemostasis
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Cholesterol and Amyloid-β: Evidence for a Cross-Talk between Astrocytes and Neuronal Cells.

2011

Accumulating data supports the concept that alterations of cholesterol metabolism might influence the development of Alzheimer's disease (AD), a neurodegenerative disorder characterized by progressive accumulation of amyloid-β (Aβ) peptides in the brain. Changes in the neuronal production of Aβ have been described as a function of cholesterol levels, thus suggesting a causal link between cholesterol homeostasis dysregulation and AD pathogenesis. Under physiological conditions, cholesterol uptake in the brain is efficiently prevented by the blood-brain barrier, and mature neurons are thought to rely on glial cells for their cholesterol supply. In the present study, we tested the hypothesis t…

Genetically modified mouseBlotting WesternEnzyme-Linked Immunosorbent AssayMice TransgenicCell LinePathogenesisMicechemistry.chemical_compoundAlzheimer DiseasemedicineAnimalsHomeostasisHumansBrain ChemistryNeuronsAmyloid beta-PeptidesbiologyCholesterolGeneral NeuroscienceTransporterReceptor Cross-TalkGeneral Medicinemedicine.diseaseCoculture TechniquesPsychiatry and Mental healthClinical PsychologyCholesterolATP Binding Cassette Transporter 1chemistryAstrocytesABCA1biology.proteinATP-Binding Cassette Transporterslipids (amino acids peptides and proteins)Geriatrics and GerontologyAlzheimer's diseaseNeuroscienceHomeostasisATP Binding Cassette Transporter 1
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The role of plasma lipid transfer proteins in lipoprotein metabolism and atherogenesis.

2008

The plasma lipid transfer proteins promote the exchange of neutral lipids and phospholipids between the plasma lipoproteins. Cholesteryl ester transfer protein (CETP) facilitates the removal of cholesteryl esters from HDL and thus reduces HDL levels, while phospholipid transfer protein (PLTP) promotes the transfer of phospholipids from triglyceride-rich lipoproteins into HDL and increases HDL levels. Studies in transgenic mouse models and in humans with rare genetic deficiencies (CETP) or common genetic variants (CETP and PLTP) highlight the central role of these molecules in regulating HDL levels. Human CETP deficiency is associated with dramatic elevations of HDL cholesterol and apolipopr…

Genetically modified mousemedicine.medical_specialtyApolipoprotein BLipoproteinscholesteryl ester transfer proteinQD415-436BiochemistryLipoprotein Metabolismchemistry.chemical_compoundEndocrinologyPhospholipid transfer proteinInternal medicineCholesterylester transfer proteinmedicineAnimalsHumansCETP inhibitorPhospholipidsPolymorphism GeneticbiologyChemistryCholesterolTorcetrapibCell BiologyAtherosclerosisphospholipid transfer proteincarbohydrates (lipids)EndocrinologyBiochemistrylow density lipoproteinsToxicitybiology.proteinlipids (amino acids peptides and proteins)high density lipoproteinsCarrier ProteinsJournal of lipid research
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Effects of eight weeks of time-restricted feeding (16/8) on basal metabolism, maximal strength, body composition, inflammation, and cardiovascular ri…

2016

Background: Intermittent fasting (IF) is an increasingly popular dietary approach used for weight loss and overall health. While there is an increasing body of evidence demonstrating beneficial effects of IF on blood lipids and other health outcomes in the overweight and obese, limited data are available about the effect of IF in athletes. Thus, the present study sought to investigate the effects of a modified IF protocol (i.e. time-restricted feeding) during resistance training in healthy resistance-trained males. Methods: Thirty-four resistance-trained males were randomly assigned to time-restricted feeding (TRF) or normal diet group (ND). TRF subjects consumed 100 % of their energy needs…

Genetics and Molecular Biology (all)0301 basic medicineAdultMalemedicine.medical_specialtyCalorieTime FactorsNormal dietBody builders; Body composition; Fasting; Intermittent fasting; Resistance training; Time-restricted feeding; Medicine (all); Biochemistry Genetics and Molecular Biology (all)Blood lipidsPhysiology030209 endocrinology & metabolismIntermittent fastingOverweightBody buildersBiochemistryBody compositionGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineWeight lossRisk FactorsInternal medicineTime-restricted feedingIntermittent fastingmedicineHumansResting energy expenditureMuscle StrengthCaloric RestrictionMedicine(all)InflammationBiochemistry Genetics and Molecular Biology (all)030109 nutrition & dieteticsbusiness.industryBiochemistry Genetics and Molecular Biology(all)Medicine (all)ResearchResistance TrainingGeneral MedicineFeeding BehaviorFastingEndocrinologyCardiovascular DiseasesBasal metabolic rateBasal Metabolismmedicine.symptomBody builderbusinessJournal of translational medicine
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Familial combined hypolipidemia due to mutations in the ANGPTL3 gene

2013

The role of ANGPTL3 in lipoprotein metabolism emerged from studies in a mutant mouse strain characterized by severe hypotriglyceridemia and carrying a loss-of-function (LOF) mutation of the ANGPTL3 gene. ANGPTL3 was found to inhibit lipoprotein lipase and endothelial lipase. Genome-wide association studies in humans demonstrated the association of ANGPTL3 variants with plasma triglyceride levels and LOF mutations of ANGPTL3 were found in hypotriglyceridemic subjects in population studies. Recently, individuals originally classified as affected by familial hypobetalipoproteinemia were found to be homozygotes/compound heterozygotes for rare LOF mutations of ANGPTL3. They show a striking reduc…

GeneticsEndothelial lipaseMutationLipoprotein lipaseVery low-density lipoproteineducation.field_of_studySettore MED/09 - Medicina InternaEndocrinology Diabetes and MetabolismPopulationANGPTL3; ANGPTL8; endothelial lipase; familial combined hypolipidemia; HDL; LDL; lipoprotein lipaseBiologyCompound heterozygositymedicine.disease_causeANGPTL3medicinelipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicineeducationANGPTL3 ANGPTL8 endothelial lipase familial combined hypolipidemia HDL LDL lipoprotein lipaseLipoprotein
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The TaqIB and −629C>A polymorphisms at the cholesteryl ester transfer protein locus: associations with lipid levels in a multiethnic population. T…

2002

The Singapore population comprises Chinese, Malays and Asian Indians. Within this population, Asian Indians have the highest rates of coronary heart disease, whereas Chinese have the lowest. Conversely, Indians have the lowest high-density lipoprotein cholesterol (HDL-C) concentrations, followed by Malays and Chinese. We studied the TaqIB and -629C>A polymorphisms at the CETP locus in 1300 Chinese, 364 Malay and 282 Asian Indian men, and in 1558 Chinese, 397 Malay and 306 Asian Indian women, to determine whether these polymorphisms are responsible for the ethnic difference in HDL-C concentration. The frequency of the B2 allele in Chinese, Malays and Indians was 0.384, 0.339 and 0.449 in men…

GeneticsLinkage disequilibriumeducation.field_of_studybiologyAsian IndianPopulationLocus (genetics)language.human_languageGenotypeCholesterylester transfer proteinGeneticsbiology.proteinlanguagelipids (amino acids peptides and proteins)AlleleeducationGenetics (clinical)DemographyMalayClinical Genetics
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Hypobetalipoproteinemia

2011

Hypobetalipoproteinemias (HBL) represent a heterogeneous group of disorders characterized by reduced plasma levels of total cholesterol (TC), low density lipoprotein-cholesterol (LDL-C) and apolipoprotein B (apoB) below the 5th percentile of the distribution in the population. HBL are defined as primary or secondary according to the underlying causes. Primary monogenic HBL are caused by mutations in several known genes (APOB, PCSK9, MTP, SARA2) or mutations in genes not yet identified. Familial hypobetalipoproteinemia (FHBL) is the most frequent monogenic form of HBL with a dominant mode of inheritance. It may be due to loss-of-function mutations in APOB or, less frequently, in PCSK9 genes.…

Geneticseducation.field_of_studyApolipoprotein BPCSK9PopulationFatty liverAbetalipoproteinemiaBiologymedicine.diseaseBiochemistrymedicinebiology.proteinlipids (amino acids peptides and proteins)HypobetalipoproteinemiaeducationGeneChylomicron retention disease
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Characterization of a fractured basement reservoir using high-resolution 3D seismic and logging datasets: A case study of the Sab'atayn Basin, Yemen.

2018

The Sab'atayn Basin is one of the most prolific Mesozoic hydrocarbon basins located in central Yemen. It has many oil producing fields including the Habban Field with oil occurrences in fractured basement rocks. A comprehensive seismic analysis of fractured basement reservoirs was performed to identify the structural pattern and mechanism of hydrocarbon entrapment and reservoir characteristics. A 3D post-stack time migration seismic cube and logging data of 20 wells were used and several 2D seismic sections were constructed and interpreted. Depth structure maps were generated for the basement reservoir and overlying formations. The top of the basement reservoir is dissected by a set of NW-S…

Geologic SedimentsYemen010504 meteorology & atmospheric sciencesOutcropWater WellsDatasets as TopicGeographic Mappinglcsh:Medicine010502 geochemistry & geophysicsBiochemistry01 natural scienceschemistry.chemical_compoundJurassic PeriodOil and Gas FieldsPetrologylcsh:ScienceMaterialsSeismologyMineralsCretaceous PeriodMultidisciplinaryHydraulic FrackingPhysicsClassical MechanicsGeologyMineralogyLipidsPetroleum reservoirChemistryGeophysicsPetroleumBasement (geology)Source rockPhysical SciencesMesozoic EraPetroleumOrganic MaterialsPorosityGeologyResearch ArticleMaterials ScienceGraniteNatural GasStructural basinImaging Three-DimensionalEarthquakesHumans0105 earth and related environmental sciencesDamage Mechanicslcsh:RChemical CompoundsBiology and Life SciencesDrillingGeologic TimeHydrocarbonschemistryEarth SciencesGeographic Information Systemslcsh:QOilsOil shalePLoS ONE
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