Search results for "LPE"

showing 10 items of 493 documents

Vapor–liquid equilibria in the systems 3-methylpentane+methyl 1,1-dimethylethyl ether and 3-methylpentane+methyl 1,1-dimethylpropyl ether at 101.3 kPa

1999

Abstract Pure-component vapor pressure of 3-methylpentane and vapor–liquid equilibrium (VLE) for the binary systems of 3-methylpentane with methyl 1,1-dimethylethyl ether (MTBE) and with methyl 1,1-dimethylpropyl ether (TAME) at 101.3 kPa have been measured. Both systems exhibit small positive deviations from ideality. They can be described as regular solutions and do not present azeotrope. The activity coefficients and boiling points of the solutions were correlated with its composition by Wilson, UNIQUAC, NRTL, and Wisniak–Tamir equations.

Activity coefficientUNIQUACVapor pressureGeneral Chemical EngineeringGeneral Physics and AstronomyEtherchemistry.chemical_compoundBoiling pointchemistryAzeotropeNon-random two-liquid modelPhysical chemistryOrganic chemistryPhysical and Theoretical Chemistry3-MethylpentaneFluid Phase Equilibria
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Acute Nonlymphocytic Leukemia in Adults: Pathophysiology, Status of Current Therapy, and New Approaches

1987

Recent information concerning the cell biology of leukemias has provided new insights into the pathophysiology and pathogenesis of acute leukemia, involving the detection of leukemia viruses, oncogenes and their products, and the discovery of factors supporting clonal leukemic growth. Murine, avian, and cat leukemia viruses are well characterized. To date, only HTLV I appears to be a likely candidate as a human leukemia virus. For both avian and murine viruses, there is a fundamental classification distinction between long-latency viruses (LLV) and acute transforming viruses (ATV). The ATV are replication defective and must be propagated with a helper virus. They have within their genome an…

Acute leukemiaHaematopoiesisLeukemiaAcute myeloblastic leukemiaHelper virusmedicineHairy cell leukemiaBiologymedicine.diseaseVirologyVirusLong terminal repeat
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Follow‐up of patients subjected to direct and indirect pulp capping of young permanent teeth. A retrospective study

2021

Abstract Objective A retrospective study of the success rate of direct pulp capping (DPC) and indirect pulp capping (IPC) was carried out in children between 6–14 years‐old, considering separately primary caries or caries affecting teeth with molar incisor hypomineralization (MIH). Material and methods Data were collected in a dental public health service. Following the inclusion criteria, 232 treatments were analyzed. Success was defined by the presence of a functional tooth without clinical signs or symptoms of pulpal or periapical disease. The success rate was correlated to patient gender, the affected tooth and the indication of therapy using the chi‐squared and Fisher exact test. The s…

AdolescentDentistryDental Pulp Cappingsymbols.namesakeIndirect Pulp Cappingstomatognathic systemyoung permanent teethHumansMedicineChildGeneral DentistryFisher's exact testRetrospective StudiesPermanent teethbusiness.industrySilicatesOxidesRetrospective cohort studyRK1-715Original ArticlesCalcium CompoundsPrimary cariesMolar Incisor HypomineralizationUnited StatesPulp cappingindirect pulp cappingstomatognathic diseasesTreatment OutcomeDentistrydirect pulp cappingsymbolsOriginal ArticleVital pulp therapybusinessPulp Capping and Pulpectomy AgentsDental public healthFollow-Up StudiesClinical and Experimental Dental Research
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Genetic proof for the transient nature of the Th17 phenotype

2010

IL-17-producing CD4(+) T cells (Th17) have been classified as a new T helper cell subset. Using an IL-17 fate mapping mouse strain, which genetically fixes the memory of IL-17 expression, we demonstrate that IL-17A/F-expressing T helper cells generated either in vitro or in vivo are not a stable T-cell subset. Upon adoptive transfer of IL-17F-reporter-positive Th17 cells to RAG-deficient or WT animals, encephalitogenic Th17 cells partially lose IL-17 expression and upregulate IFN-γ. Additionally, we show that Th1 cells can convert in vivo to IL-17A/IFN-γ-coexpressing cells in the mesenteric lymph nodes (mLN). Our data classify IL-17A and IL-17F as cytokines produced transiently in response …

Adoptive cell transferEncephalomyelitis Autoimmune ExperimentalGenes RAG-1TransgeneImmunologyReceptors Antigen T-CellMice TransgenicBiologyLymphocyte ActivationInterferon-gammaMiceInterleukin 21AntigenGenes ReporterT-Lymphocyte SubsetsIn vivomedicineAnimalsImmunology and AllergyCytotoxic T cellMesenteric lymph nodesMice KnockoutIntegrasesCell DifferentiationT helper cellTh1 CellsAdoptive TransferCell biologyMice Inbred C57BLmedicine.anatomical_structureGene Expression RegulationImmunologyTh17 CellsEuropean Journal of Immunology
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RORgamma-expressing Th17 cells induce murine chronic intestinal inflammation via redundant effects of IL-17A and IL-17F.

2008

Background and Aims IL-17–producing CD4 + T-helper cells (Th17) contribute to chronic autoimmune inflammation in the brain, and levels of Th17-derived cytokines increase in patients with colitis, suggesting a role in pathogenesis. We analyzed the roles of Th17 cells and the transcription factor retinoic acid receptor-related organ receptor (ROR)γ, which regulates Th17 differentiation, in chronic intestinal inflammation. Methods Using an adoptive transfer model of colitis, we compared the colitogenic potential of wild-type, interleukin-17A (IL-17A)–, IL-17F–, IL-22–, and RORγ-deficient CD4 + CD25 − T cells in RAG1-null mice. Results Adoptive transfer of IL-17A–, IL-17F–, or IL-22–deficient T…

Adoptive cell transferNeutrophilsReceptors Retinoic Acidmedicine.medical_treatmentBiologyInflammatory bowel diseasePathogenesisMiceInterferonCell MovementmedicineAnimalsIL-2 receptorColitisCells CulturedReceptors Thyroid HormoneHepatologyInterleukinsInterleukin-17GastroenterologyDendritic CellsT-Lymphocytes Helper-InducerNuclear Receptor Subfamily 1 Group F Member 3medicine.diseaseColitisAdoptive TransferMice Inbred C57BLCytokineImmunologyChronic Diseasebiology.proteinCytokinesAntibodymedicine.drugGastroenterology
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The influence of major histocompatibility complex class II genes and T-cell Vbeta repertoire on response to immunization with HBsAg.

1998

Nonresponsiveness to HBsAg vaccination is observed in 5-10% of vaccine recipients and is possibly caused by a defect in the T helper cell compartment. The immune response to HBsAg is influenced by genes of the major histocompatibility complex. We have investigated MHC class I and class II antigens in 53 adult responders and 73 nonresponders. Results obtained in this first study were tested in a second study with 56 responders and 62 nonresponders from an infant vaccination trial. In addition, the peripheral Vbeta-chain T-cell receptor repertoire was investigated using monoclonal antibodies and flow-cytometry in 26 adult responders and 38 nonresponders. As previously reported, nonresponsiven…

AdultHBsAgT cellReceptors Antigen T-Cell alpha-betaImmunologyGenes MHC Class IIMajor histocompatibility complexCohort StudiesImmune systemGene FrequencyMHC class ImedicineImmunology and AllergyHumansHepatitis B VaccinesAllelesDiphtheria-Tetanus-Pertussis VaccineHepatitis B Surface AntigensbiologyT-cell receptorInfantGeneral MedicineT helper cellHLA-DR AntigensVirologyVaccinationmedicine.anatomical_structureImmunologybiology.proteinImmunizationHLA-DRB1 ChainsHuman immunology
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Quantitative and functional analysis of core-specific T-helper cell and CTL activities in acute and chronic hepatitis B

2008

Aims/background CD4+ T-helper cell (Th) responses to hepatitis B virus (HBV) core antigen (HBc) are increased during exacerbations in acute and chronic hepatitis B (AHB, CHB) and might influence the induction of CD8+ cytotoxic T lymphocytes (CTL) that are important for viral clearance. Methods HBc-specific proliferative responses and cytokine release of blood mononuclear cells (PBMC) were studied in patients with AHB or CHB, as well as responders and non-responders to interferon-alpha treatment (IFN-R, IFN-NR), by [3H]-thymidine-uptake, enzyme-linked immunosorbent assay (ELISA) and Elispot assay and were compared to peptide HBc18 27-specific CTL precursor frequencies among CD8+ T cells deri…

AdultMaleAdolescentLymphocyte Activationmedicine.disease_causePolymerase Chain ReactionHepatitis B ChronicmedicineHumansCytotoxic T cellHepatitis B virusHepatologybiologybusiness.industryELISPOTInterferon-alphaT-Lymphocytes Helper-InducerT helper cellMiddle AgedHepatitis Bbiology.organism_classificationmedicine.diseaseHepatitis B Core AntigensVirologyCTL*medicine.anatomical_structureHepadnaviridaeAcute DiseaseImmunologyLeukocytes MononuclearFemalebusinessCD8T-Lymphocytes CytotoxicLiver
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T cells involved in psoriasis vulgaris belong to the Th1 subset

1994

Although the pathogenesis of psoriasis vulgaris is still unknown, several characteristics point to an immunologically mediated process. Epidermal psoriatic lesions are characterized by a hyperproliferation of keratinocytes and an infiltration of T lymphocytes and granulocytes. Because the former may be mediated in part by lymphokines secreted by T cells, we have focused our interest on the in vivo and in vitro cytokine secretion patterns of T lymphocytes from psoriatic lesions. In five patients T lymphocytes were obtained from epidermal specimens. The cells were propagated with lectin and irradiated feeder cells and subsequently cloned by limiting dilution. The resulting T-cell clones were …

AdultMaleBiopsyCD8 AntigensT-Lymphocytesmedicine.medical_treatmentMolecular Sequence DataDermatologyBiologyPolymerase Chain ReactionBiochemistryInterferon-gammaT-Lymphocyte SubsetsPsoriasisSynovial FluidmedicineHumansPsoriasisRNA MessengerMolecular BiologyCells CulturedAgedSkinAged 80 and overBase SequenceTumor Necrosis Factor-alphaArthritis PsoriaticLymphokineInterleukinT-Lymphocytes Helper-InducerCell BiologyT lymphocyteMiddle Agedmedicine.diseaseInterleukin-10PhenotypeCytokineepidermal T cellsCD4 AntigensImmunologyCytokinesInterleukin-2cytokine productionFemaleCytokine secretionTumor necrosis factor alphaInterleukin-4CD8
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Liver follicular helper T-cells predict the achievement of virological response following interferon-based treatment in HCV-infected patients.

2012

Background Here, we assessed the presence of intrahepatic follicular helper T-cells (TFH) in a cohort of consecutive genotype 1 (G1) chronic hepatitis C (CHC) patients comprising non-responders (NRs), relapsers (RRs) or those with sustained virological response (SVR) to pegylated interferon and ribavirin, and tested their relation with the response to antiviral treatment. Methods A total of 78 patients with G1 CHC (30 SVR, 15 RR and 33 NR), comparable for sex, age, viral load and fibrosis were evaluated by immunohistochemistry for liver content of PD1+Bcl6+ TFH cells. The number of TFH cells in the immunostained sections was counted out of five representative high-power microscopic fields (…

AdultMaleGenotypeHepacivirusSettore MED/08 - Anatomia PatologicaAntiviral AgentsPolymorphism Single NucleotideBiomarkers PharmacologicalPolyethylene GlycolsCohort StudiesPharmacotherapyInterferonRibavirinGenotypeFollicular phaseHumansMedicinePharmacology (medical)liver biopsy Interferon-alpha follicular helper T-cellsPharmacologySettore MED/12 - Gastroenterologiabusiness.industryInterleukinsInterferon-alphaT-Lymphocytes Helper-InducerHepatitis CHepatitis C ChronicMiddle AgedViral LoadPrognosismedicine.diseaseImmunohistochemistryRecombinant ProteinsCD4 Lymphocyte CountInfectious DiseasesLiverImmunologyCohortRNA ViralImmunohistochemistryDrug Therapy CombinationFemalehcv immunohistochemistryInterferonsbusinessCohort studymedicine.drug
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Peripheral blood dendritic cells are phenotypically and functionally intact in chronic hepatitis B virus (HBV) infection

2007

Summary Persistence of hepatitis B virus (HBV) infection is associated with reduced anti-viral T cell responses. Impaired dendritic cell (DC) function was suggested as the cause of reduced T cell stimulation in chronic HBV carriers. Thus, we compared myeloid (mDC) and plasmacytoid DC (pDC) from chronic HBV carriers and controls. Frequency and phenotype of isolated DC were analysed by fluorescence activated cell sorter staining, DC function by mixed lymphocyte reaction, cytokine bead array, intracellular cytokine staining, enzyme-linked immunosorbent assay and enzyme-linked immunospot. Expression of HBV DNA and mRNA was studied by polymerase chain reaction (PCR). Circulating total DC, mDC or…

AdultMaleHepatitis B virusHeterozygoteTranslational StudiesT cellImmunologyBone Marrow CellsLymphocyte Activationmedicine.disease_causeStatistics NonparametricVirusHepatitis B ChronicmedicineHumansImmunology and AllergyCytotoxic T cellLymphocyte CountHepatitis B virusbiologyDendritic CellsDendritic cellT helper cellHepatitis BFlow Cytometrybiology.organism_classificationmedicine.diseasemedicine.anatomical_structureHepadnaviridaeCase-Control StudiesDNA ViralImmunologyCytokinesRNA ViralFemaleLymphocyte Culture Test MixedT-Lymphocytes CytotoxicClinical and Experimental Immunology
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