Search results for "LUDI"

showing 10 items of 753 documents

The Mutational Landscape of Acute Myeloid Leukaemia Predicts Responses and Outcomes in Elderly Patients from the PETHEMA-FLUGAZA Phase 3 Clinical Tri…

2021

This article belongs to the Collection The Biomarkers for the Diagnosis and Prognosis in Cancer.

Neuroblastoma RAS viral oncogene homologOncologyCancer Researchgenetic riskMyeloid neoplasialeukemic cells0302 clinical medicinecytarabineclinical trials and observations:Other subheadings::/diagnosis [Other subheadings]MedicineComplete remissionazacytidineolder adultsRC254-282variantsLeukemiaAzacytidineHazard ratioleukemiaVariantsCytarabineacute:Neoplasms::Neoplasms by Histologic Type::Leukemia::Leukemia Myeloid::Leukemia Myeloid Acute [DISEASES]Neoplasms. Tumors. Oncology. Including cancer and carcinogensMyeloid leukemiaFludarabineLeukemiaOncology030220 oncology & carcinogenesisNGSOlder adultsLeucèmia mieloide aguda - Tractamentmyeloid neoplasiaMyelocyticmedicine.drugmedicine.medical_specialtymyelocyticcomplete remission:Otros calificadores::/diagnóstico [Otros calificadores]Subgroup analysisLeukemic cellsAcutePrognostic factorsArticle:neoplasias::neoplasias por tipo histológico::leucemia::leucemia mieloide::leucemia mieloide aguda [ENFERMEDADES]03 medical and health sciencesInternal medicineGenetic riskbusiness.industryprognostic factors:diagnóstico::pronóstico::resultado del tratamiento [TÉCNICAS Y EQUIPOS ANALÍTICOS DIAGNÓSTICOS Y TERAPÉUTICOS]Odds ratio:Diagnosis::Prognosis::Treatment Outcome [ANALYTICAL DIAGNOSTIC AND THERAPEUTIC TECHNIQUES AND EQUIPMENT]medicine.diseaseAvaluació de resultats (Assistència sanitària)CytarabinebusinessClinical trials and observations030215 immunology
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Detection of RAS mutations in circulating tumor DNA: a new weapon in an old war against colorectal cancer. A systematic review of literature and meta…

2019

Background: Tissue evaluation for RAS (KRAS or NRAS) gene status in metastatic colorectal cancer (mCRC) patients represent the standard of care to establish the optimal therapeutic strategy. Unfortunately, tissue biopsy is hampered by several critical limitations due to its invasiveness, difficulty to access to disease site, patient’s compliance and, more recently, neoplastic tissue spatial and temporal heterogeneity. Methods: The authors performed a systematic literature review to identify available trials with paired matched tissue and ctDNA RAS gene status evaluation. The authors searched EMBASE, MEDLINE, Cochrane, www.ClinicalTrials.gov , and abstracts from international meetings. In to…

Neuroblastoma RAS viral oncogene homologOncologymedicine.medical_specialtyStandard of careColorectal cancerSettore MED/06 - Oncologia Medicamedicine.disease_causelcsh:RC254-282meta-analysi03 medical and health sciences0302 clinical medicineInternal medicinemedicineLiquid biopsy030304 developmental biologyTherapeutic strategycirculating tumor DNAcirculating tumor DNA; diagnostic accuracy; liquid biopsy; meta-analysis; metastatic colorectal cancer; RAS0303 health sciencesliquid biopsybusiness.industrymetastatic colorectal cancermedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens3. Good healthmeta-analysisOncologyCirculating tumor DNA030220 oncology & carcinogenesisMeta-analysisdiagnostic accuracyKRASbusinessRAS
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Exact relativistic beta decay endpoint spectrum

2007

5 pages, 3 figures.-- PACS nrs.: 14.60.Pq; 13.30.-a; 23.40.-s; 23.40.Bw.-- ISI Article Identifier: 000250620900070.-- ArXiv pre-print available at: http://arxiv.org/abs/0706.0897

Nuclear and High Energy PhysicsParticle physicsPhysics::Instrumentation and DetectorsFOS: Physical sciences[PACS] Neutrino mass and mixingelectron and muon captureHigh Energy Physics - Phenomenology (hep-ph)FactorizationDouble beta decayNuclear Experiment (nucl-ex)Neutrino oscillationNuclear ExperimentPhysics[PACS] β decay[PACS] Decays of baryonsSpectrum (functional analysis)[PACS] β decay; double β decay; electron and muon captureFísicaBeta decay[PACS] Weak-interaction and lepton (including neutrino) aspects of β decayHigh Energy Physics - Phenomenologydouble β decayYield (chemistry)High Energy Physics::ExperimentNeutrinoKATRIN
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Glueball enhancement by color deconfinement

2007

5 pages, 4 figures.-- PACS nrs.: 14.80.-j; 24.80.+y; 25.75.Nq.-- ISI Article Identifier: 000245333000063.-- ArXiv pre-print available at: http://arxiv.org/abs/hep-ph/0609219

Nuclear and High Energy PhysicsParticle physics[PACS] Nuclear tests of fundamental interactions and symmetriesNuclear Theory[PACS] Quark deconfinement quark-gluon plasma production and phase transitions in heavy-ion collisionsHigh Energy Physics::LatticeFOS: Physical sciencesDeconfinementQuantum chromodynamics (QCD)Nuclear Theory (nucl-th)Nuclear physicsHigh Energy Physics - Phenomenology (hep-ph)Color confinementNuclear ExperimentNuclear theoryQuantum chromodynamicsPhysicsQuark confinementGlueball[PACS] Other particles (including hypothetical)High Energy Physics::PhenomenologyFísicaHigh Energy Physics - PhenomenologyColor modelHeavy ion-nucleus reactions
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Genetic predisposition to male breast cancer in Poland

2021

Abstract Background Breast cancer in men accounts for fewer than 1 % of all breast cancer cases diagnosed in men and women. Genes which predispose to male breast cancer include BRCA1 and BRCA2. The role of other genes is less clear. In Poland, 20 founder mutations in BRCA1, BRCA2, CHEK2, PALB2, NBN, RECQL are responsible for the majority of hereditary breast cancer cases in women, but the utility this genes panel has not been tested in men. Methods We estimated the prevalence of 20 alleles in six genes (BRCA1, BRCA2, CHEK2, PALB2, NBN, RECQL) in 165 Polish male breast cancer patients. We compared the frequency of selected variants in male breast cancer cases and controls. Results One of the…

OncologyAdultMaleCancer Researchmedicine.medical_specialtyendocrine system diseasesPALB2medicine.disease_causeBreast Neoplasms MaleBreast cancerSurgical oncologyInternal medicineNBNGeneticsmedicineGenetic predispositionBiomarkers TumorHumansGenetic Predisposition to DiseaseAlleleskin and connective tissue diseasesCHEK2RC254-282CHEK2AgedRetrospective StudiesAged 80 and overMutationbusiness.industryCarcinoma Ductal BreastNeoplasms. Tumors. Oncology. Including cancer and carcinogensMiddle Agedmedicine.diseasePrognosisBRCA1BRCA2Male breast cancerRECQLCarcinoma LobularOncologyMale breast cancerCase-Control StudiesPALB2MutationPolandbusinessFollow-Up StudiesResearch ArticleBMC Cancer
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Residual neurotoxicity in ovarian cancer patients in clinical remission after first-line chemotherapy with carboplatin and paclitaxel: the Multicente…

2006

Abstract Background Carboplatin/paclitaxel is the chemotherapy of choice for advanced ovarian cancer, both in first line and in platinum-sensitive recurrence. Although a significant proportion of patients have some neurotoxicity during treatment, the long-term outcome of chemotherapy-induced neuropathy has been scantly studied. We retrospectively assessed the prevalence of residual neuropathy in a cohort of patients in clinical remission after first-line carboplatin/paclitaxel for advanced ovarian cancer. Methods 120 patients have been included in this study (101 participating in a multicentre phase III trial evaluating the efficacy of consolidation treatment with topotecan, and 19 treated …

OncologyAdultmedicine.medical_specialtyCancer ResearchTime Factorsendocrine system diseasesPaclitaxelmedicine.medical_treatmentlcsh:RC254-282Severity of Illness IndexCarboplatinchemistry.chemical_compoundMedian follow-upInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineGeneticsHumansAgedRetrospective StudiesOvarian NeoplasmsChemotherapybusiness.industryCancerPeripheral Nervous System DiseasesRetrospective cohort studyMiddle Agedmedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensCarboplatinfemale genital diseases and pregnancy complicationsClinical trialchemistryOncologyTopotecanFemalebusinessOvarian cancerTopotecanmedicine.drugResearch Article
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The Assessment of Risk Factors for Long-term Survival Outcome in ypN0 Patients With Rectal Cancer After Neoadjuvant Therapy and Radical Anterior Rese…

2021

Abstract Background The main negative prognostic factors in patients with rectal cancer after radical treatment include regional lymph node involvement, lymphovascular invasion, and perineural invasion. However, some patients still develop cancer recurrence despite the absence of the above risk factors. The aim of the study was to assess clinicopathological factors influencing long-term oncologic outcomes in ypN0M0 rectal cancer patients after neoadjuvant therapy and radical anterior resection. Methods A retrospective survival analysis was performed on a group of 195 patients. We assessed clinicopathological factors which included tumor regression grade, number of lymph nodes in the specime…

OncologyAnterior rectal resectionmedicine.medical_specialtyRD1-811Lymphovascular invasionColorectal cancermedicine.medical_treatmentPerineural invasion030230 surgeryDisease-Free Survival03 medical and health sciences0302 clinical medicineRisk FactorsInternal medicinemedicineLymph node yieldHumansLymph nodeNeoadjuvant therapySurvival analysisRC254-282Neoplasm StagingRetrospective StudiesTumor Regression GradeUnivariate analysisbusiness.industryLate anastomotic leakRectal NeoplasmsResearchNeoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseasePrognosisNeoadjuvant Therapymedicine.anatomical_structureOncologyStage migration030220 oncology & carcinogenesisSurgeryNeoplasm Recurrence LocalbusinessCharlson comorbidity index
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Interassay and interobserver comparability study of four programmed death-ligand 1 (PD-L1) immunohistochemistry assays in triple-negative breast canc…

2021

Different immunohistochemical programmed death-ligand 1 (PD-L1) assays and scorings have been reported to yield variable results in triple-negative breast cancer (TNBC). We compared the analytical concordance and reproducibility of four clinically relevant PD-L1 assays assessing immune cell (IC) score, tumor proportion score (TPS), and combined positive score (CPS) in TNBC. Primary TNBC resection specimens (n = 104) were stained for PD-L1 using VENTANA SP142, VENTANA SP263, DAKO 22C3, and DAKO 28–8. PD-L1 expression was scored according to guidelines on virtual whole slide images by four trained readers. The mean PD-L1 positivity at IC-score ≥1% and CPS ≥1 ranged between 53% and 75% with th…

OncologyCPS combined positive scoreTC tumor cellsICI immune checkpoint inhibitorTriple Negative Breast NeoplasmsB7-H1 AntigenMedicineHER2 human epidermal growth factor receptor 2Triple-negative breast cancerRC254-282ICC intraclass correlation coefficientbiologyNeoplasms. Tumors. Oncology. Including cancer and carcinogensGeneral MedicineMSI microsatellite instabilityImmunohistochemistrypCR pathological complete responsePFS progression-free survivalImmunohistochemistryOriginal ArticleIC-ScoreIC immune cellsIHC immunohistochemistryProgrammed deathPD-L1medicine.medical_specialtyConcordanceTNBC triple-negative breast cancerOS overall survivalBreast cancerTriple-negative breast cancerPD-L1Internal medicineTPS tumor proportion scoreBiomarkers TumorHumansProgrammed death-ligand 1Reproducibilitybusiness.industryReproducibility of Resultsmedicine.diseaseITT intention to treatCI confidence intervalPD-L1 programmed death-ligand 1biology.proteinSurgeryCPSbusinessKappaTMB tumor mutational burdenBreast
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First Nationwide Molecular Screening Program in Spain for Patients With Advanced Breast Cancer: Results From the AGATA SOLTI-1301 Study

2021

Anàlisi de seqüències d'ADN; Subtipus PAM50; Genètica molecular Análisis de secuencias de ADN; Subtipo PAM50; Genética molecular DNA sequence analyses; PAM50 subtype; Molecular genetic Background: The SOLTI-1301 AGATA study aimed to assess the feasibility of a multi-institutional molecular screening program to better characterize the genomic landscape of advanced breast cancer (ABC) and to facilitate patient access to matched-targeted therapies in Spain. Methods: DNA sequencing of 74 cancer-related genes was performed using FFPE tumor samples in three different laboratories with three different gene panels. A multidisciplinary advisory board prospectively recommended potential targeted trea…

OncologyCancer ResearchDNA Alterationmedicine.medical_specialtymolecular targeted therapyAdvanced breast:Neoplasms::Neoplasms by Site::Breast Neoplasms [DISEASES]Cancer therapy:terapéutica::farmacoterapia::terapia molecular selectiva [TÉCNICAS Y EQUIPOS ANALÍTICOS DIAGNÓSTICOS Y TERAPÉUTICOS]:Therapeutics::Drug Therapy::Molecular Targeted Therapy [ANALYTICAL DIAGNOSTIC AND THERAPEUTIC TECHNIQUES AND EQUIPMENT]Breast cancerbreast cancerInternal medicineGene panelmolecular geneticmedicineDNA sequence analysesRC254-282Original ResearchFarmacologia molecular:neoplasias::neoplasias por localización::neoplasias de la mama [ENFERMEDADES]:Natural Science Disciplines::Biological Science Disciplines::Biology::Computational Biology::Genomics [DISCIPLINES AND OCCUPATIONS]Molecular screeningbusiness.industryCancerNeoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseOncologyMama - Càncer - Tractament:disciplinas de las ciencias naturales::disciplinas de las ciencias biológicas::biología::biología computacional::genómica [DISCIPLINAS Y OCUPACIONES]Mama - Càncer - Aspectes molecularsPAM50 subtypebusiness
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Identification of a genetic signature enriching for response to ibrutinib in relapsed/refractory follicular lymphoma in the DAWN phase 2 trial.

2021

Abstract Background The single‐arm DAWN trial (NCT01779791) of ibrutinib monotherapy in patients with relapsed/refractory follicular lymphoma (FL) showed an overall response rate (ORR) of 20.9% and a median response duration of 19.4 months. This biomarker analysis of the DAWN dataset sought to determine genetic classifiers for prediction of response to ibrutinib treatment. Methods Whole exome sequencing was performed on baseline tumor samples. Potential germline variants were excluded; a custom set of 1216 cancer‐related genes was examined. Responder‐ versus nonresponder‐associated variants were identified using Fisher's exact test. Classifiers with increasing numbers of genes were created …

OncologyCancer ResearchFollicular lymphomaBiochemistrychemistry.chemical_compoundGenetic signaturePiperidinesRecurrenceMedicineExomeLymphoma FollicularExome sequencingRC254-282Research ArticlesNeoplasms. Tumors. Oncology. Including cancer and carcinogensHematologyDNA-Binding ProteinsExact testOncologyIbrutinibRefractory Follicular LymphomaClin oncolResearch ArticleGenetic Markersmedicine.medical_specialtyImmunologyAntineoplastic AgentslymphomaBiologyGermline mutationInternal medicinePartial responseExome SequencingHumansRadiology Nuclear Medicine and imagingIn patientbusiness.industryAdeninegenetic variantsClinical Cancer ResearchbiomarkersCell Biologymedicine.diseasemutationsFANCAMutational analysisCARD Signaling Adaptor ProteinschemistryGuanylate CyclaseFamily medicineRelapsed refractoryMutationbusinessCancer medicine
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