Search results for "Lectin"

showing 10 items of 495 documents

Secreted proteophosphoglycan of Leishmania mexicana amastigotes activates complement by triggering the mannan binding lectin pathway.

1997

Cutaneous lesions induced by infection of mice with the protozoan parasite, Leishmania mexicana, contain abundant amounts of a high molecular mass proteophosphoglycan (PPG), which is secreted by the amastigote stage residing in phagolysosomes of macrophages and can then be released into the tissue upon rupture of the infected cells. Amastigote PPG forms sausage-shaped but soluble particles and belongs to a novel class of serine-rich proteins that are extensively O-glycosylated by phosphooligosaccharides capped by mannooligosaccharides. The purified molecule is shown here to efficiently activate complement (C) and deplete hemolytic activity of normal serum and may prevent the opsonization of…

ImmunologyLeishmania mexicanaProtozoan ProteinsCollectinLeishmaniasis CutaneousLeishmania mexicanaMiceImmunology and AllergyAnimalsAmastigoteComplement ActivationMannan-binding lectinSerine proteaseMice KnockoutbiologyMacrophagesComplement C4Complement C3biology.organism_classificationCollectinsComplement systemAntibody opsonizationBiochemistryLectin pathwaybiology.proteinMice Inbred CBACalciumProteoglycansCarrier ProteinsEuropean journal of immunology
researchProduct

Human Siglec-10 can bind to vascular adhesion protein-1 and serves as its substrate

2009

AbstractLeukocytes migrate from the blood into areas of inflammation by interacting with various adhesion molecules on endothelial cells. Vascular adhesion protein-1 (VAP-1) is a glycoprotein expressed on inflamed endothelium where it plays a dual role: it is both an enzyme that oxidizes primary amines and an adhesin that is involved in leukocyte trafficking to sites of inflammation. Although VAP-1 was identified more than 15 years ago, the counterreceptor(s) for VAP-1 on leukocytes has remained unknown. Here we have identified Siglec-10 as a leukocyte ligand for VAP-1 using phage display screenings. The binding between Siglec-10 and VAP-1 was verified by different adhesion assays, and this…

ImmunologyReceptors Cell SurfaceInflammationCHO CellsPlasma protein bindingBiologyLigandsBiochemistryMice03 medical and health sciencesCricetulus0302 clinical medicinePeptide LibraryVascular BiologyCricetinaeLectinsLeukocyte TraffickingCell AdhesionmedicineAnimalsHumansEndotheliumLymphocytesProtein Structure QuaternaryCell adhesion030304 developmental biologyMice Knockout0303 health sciencesCell adhesion moleculeSoluble cell adhesion moleculesSIGLECCell BiologyHematologyAdhesionrespiratory systembacterial infections and mycosesRecombinant Proteinsrespiratory tract diseasesChemotaxis LeukocyteBiochemistry030220 oncology & carcinogenesisAmine Oxidase (Copper-Containing)medicine.symptomCell Adhesion MoleculesProtein BindingBlood
researchProduct

Increased susceptibility of complement factor B/C2 double knockout mice and mannan-binding lectin knockout mice to systemic infection with Candida al…

2008

Candida albicans is the major cause of systemic fungal infections in immunocompromised patients. We investigated the susceptibility of mice deficient in complement factor B and C2 (Bf/C2-/-), C1q (C1qa-/-), and mannan-binding lectin (MBL)-A (MBL-A) and MBL-C (MBL-A/C-/-) to systemic infection with C. albicans. Animals were infected i.p. with 10(8)C. albicans blastoconidia and monitored for mortality. Bf/C2-/- mice showed high mortality (over 90%) within the study period of 3 weeks. In contrast, mortality in C1qa-/- mice was below 15% whereas that of MBL-A/C-/- mice was 40% (P0.001). Intravenous infection of mice with 8x10(5) blastoconidia resulted in the same trend with Bf/C2-/- mice being …

Immunologychemical and pharmacologic phenomenaOpportunistic InfectionsMannose-Binding LectinBlastoconidiumComplement factor BMicrobiologyMicePhagocytosisSpecies SpecificityCandida albicansAnimalsGenetic Predisposition to DiseaseCandida albicansDouble knockoutComplement ActivationMolecular BiologyMannan-binding lectinMice KnockoutbiologyCandidiasisLectinComplement Pathway Mannose-Binding LectinComplement C2bacterial infections and mycosesbiology.organism_classificationCorpus albicansKnockout mousebiology.proteinComplement Factor BMolecular Immunology
researchProduct

Inflammatory Response of the Ascidian Ciona intestinalis

2016

Abstract The Ciona intestinalis inflammatory response to several irritants appears to be composed of a complex reaction set. The cellular reactions in the tunic involve hemocyte infiltration, hemocyte and epidermis activities, vacuolization, and cell disruption, while cell products can contribute to form capsule components and/or cause a tunic wound. In this response, the involvement of the pharynx, as the main immune-competent organ, has been disclosed by a lipopolysaccharide challenge that upregulates innate immunity genes and transcription activation genes. The pharynx responds through hemocyte recruitment into the pharynx vessels, enhancement of galectin-like lectins in the serum hemoly…

InflammationProphenoloxidaseInnate immune systemAscidianCiona intestinaliSettore BIO/05 - ZoologiaMedicine (miscellaneous)HemocyteLipopolysaccharideInflammationProphenoloxidaseBiologybiology.organism_classificationCell biologyImmune systemImmunologymedicineAlternative complement pathwayPharynxCiona intestinalismedicine.symptomCytokineLectinGeneGalectin
researchProduct

Functional analysis of the classical, alternative, and MBL pathways of the complement system: standardization and validation of a simple ELISA.

2004

Primary defence against invading microorganisms depends on a functional innate immune system and the complement system plays a major role in such immunity. Deficiencies in one of the components of the complement system can cause severe and recurrent infections, systemic diseases, such as systemic lupus erythematosus (SLE) and renal disease. Screening for complement deficiencies in the classical or alternative complement pathways has mainly been performed by haemolytic assays. Here, we describe a simple ELISA-based format for the evaluation of three pathways of complement activation. The assays are based on specific coatings for each pathway in combination with specific buffer systems. We ha…

Innate immune systemSystemic lupus erythematosusImmunologyComplement Pathway AlternativeComplement Pathway Mannose-Binding LectinEnzyme-Linked Immunosorbent AssayComplement System ProteinsBiologyComplement fixation testmedicine.diseaseMannose-Binding LectinComplement systemComplement (complexity)Immune systemImmunologymedicineImmunology and AllergyHumansLupus Erythematosus SystemicComplement Pathway ClassicalReagent Kits DiagnosticFicolinComplement ActivationMannan-binding lectinJournal of immunological methods
researchProduct

Cross-talk between minimally primed HL-60 cells and resting HUVEC reveals a crucial role for adhesion over extracellularly released oxidants

2011

This study demonstrates that a long-lasting co-culture of neutrophil surrogates (HL-60 cells), minimally primed by platelet activating factor (PAF), and resting endothelial cells (EC) results in the elaboration of an hyper-adhesive endothelial surface, as measured by the increase in the expression of endothelial adhesion molecules E-Selectin, VCAM-1, and ICAM-1. This endothelial dysfunction is mediated by the activation of the redox-sensitive transcription factor NF-κB through an exclusive adhesion-driven mechanism active in the endothelial cell: reactive oxygen and nitrogen species, extracellularly released by minimally primed HL-60 cells, are not involved in the induction of the endotheli…

Intercellular Adhesion Molecule-1Vascular Cell Adhesion Molecule-1HL-60 CellsInflammationNeutrophils Priming Endothelial cells Inflammation Adhesion Oxidants.BiologyBiochemistryCell Linechemistry.chemical_compoundSettore BIO/10 - BiochimicaE-selectinCell AdhesionmedicineHumansEndothelial dysfunctionCell adhesionPharmacologyPlatelet-activating factorCell adhesion moleculeNF-kappa BEndothelial CellsReceptor Cross-TalkIntercellular Adhesion Molecule-1Oxidantsmedicine.diseaseCoculture TechniquesCell biologyEndothelial stem cellchemistrybiology.proteinmedicine.symptomE-SelectinReactive Oxygen SpeciesBiochemical Pharmacology
researchProduct

Immunological and biological identification of tumour necrosis-like factor in sponges: Endotoxin that mediates necrosis formation in xenografts

1992

Xenografts of the sponge Geodia cydonium in its closely related species G. rovinjensis resulted in a rapid rejection of the graft within a period of 5 days. We identified an immunoreactive tumour necrosis factor (TNF)-like activity in the xenograft (Mr of 30,000) two days after grafting. In-vivo injection of 5 micrograms human recombinant TNF-alpha induced cytotoxicity in sponge cells in the same pattern and time course as during natural xenograft rejection. Anti-TNF-alpha polyclonals were found to react with xenograft extracts, by Western blot analysis, as from day 2 after grafting. Using ELISA we detected the TNF-like activity from day 2 after grafting with peak levels at days 4 and 5, wh…

Interferon InducersNecrosisTransplantation HeterologousImmunologyEnzyme-Linked Immunosorbent AssayBiochemistryInjectionslaw.inventionNecrosisWestern blotlawmedicineAnimalsImmunology and AllergyCytotoxicityReceptorMolecular BiologyGlycoproteinsCell Deathbiologymedicine.diagnostic_testTumor Necrosis Factor-alphaLectinHematologybiology.organism_classificationMolecular biologyExtracellular MatrixPoriferaEndotoxinsMolecular WeightSpongeImmunologybiology.proteinRecombinant DNATumor necrosis factor alphamedicine.symptomCytokine
researchProduct

Superior antitumor in vitro responses of allogeneic matched sibling compared with autologous patient CD8+ T cells.

2006

AbstractAllogeneic cell therapy as a means to break immunotolerance to solid tumors is increasingly used for cancer treatment. To investigate cellular alloimmune responses in a human tumor model, primary cultures were established from renal cell carcinoma (RCC) tissues of 56 patients. In three patients with stable RCC line and human leukocyte antigen (HLA)-identical sibling donor available, allogeneic and autologous RCC reactivities were compared using mixed lymphocyte/tumor cell cultures (MLTC). Responding lymphocytes were exclusively CD8+ T cells, whereas CD4+ T cells or natural killer cells were never observed. Sibling MLTC populations showed higher proliferative and cytolytic antitumor …

Interleukin 2Cytotoxicity ImmunologicCancer ResearchCD3 ComplexCell SurvivalLymphocyteCD8 AntigensEnzyme-Linked Immunosorbent AssayHuman leukocyte antigenBiologyCD8-Positive T-LymphocytesLymphocytes Tumor-InfiltratingAntigenAntibody SpecificityHLA AntigensCell Line TumormedicineTumor Cells CulturedCytotoxic T cellHumansL-SelectinCarcinoma Renal CellCell ProliferationTumor-infiltrating lymphocytesSiblingsAntibodies MonoclonalFlow CytometryKidney NeoplasmsCTL*medicine.anatomical_structureOncologyImmunologyCD8medicine.drugT-Lymphocytes CytotoxicCancer research
researchProduct

A novel galactose- and arabinose-specific lectin from the sponge Pellina semitubulosa: isolation, characterization and immunobiological properties.

1992

A new lectin from the sponge Pellina semitubulosa is derived which was extracted and purified to homogeneity. The purified lectin is probably a hexamer of polypeptide chains (each M(r) 34,000) which are covalently linked via disulfide linkages; the isoelectric point is 6.1. The lectin displays the following specificities: D-galactose (50% inhibition of hemagglutination at 0.2 mM) = L-arabinose (0.2 mM) greater than D-fucose (1.5 mM) greater than D-glucose (3.0 mM). It precipitates human erythrocytes (A1, A2, A1B, B, and O) with a titer between 2(8) and 2(11) and erythrocytes from sheep and rabbits with a titer between 2(5) and 2(10). The Pellina lectin displays a strong mitogenic effect on …

Interleukin 2HemagglutinationChemical PhenomenaLymphocyte ActivationBiochemistrySubstrate Specificitychemistry.chemical_compoundLectinsmedicineAnimalsLymphocytesAmino AcidsbiologyChemistry PhysicalMacrophagesInterleukinLectinGalactoseGeneral MedicineHemagglutination TestsMolecular biologyArabinosePoriferaTiterIsoelectric pointchemistryBiochemistryConcanavalin AGalactosebiology.proteinInterleukin-2medicine.drugInterleukin-1Biochimie
researchProduct

LES DISCRIMINATIONS A L'EMBAUCHE DANS LA SPHERE PUBLIQUE : EFFETS RESPECTIFS DE L'ADRESSE ET DE L'ORIGINE

2016

Cette étude évalue la discrimination dans l’accès à l’emploi dans trois professions présentes à la fois dans le secteur privé et dans la Fonction Publique. Deux dimensions sont examinées : l’effet de la réputation du lieu de résidence et l’effet de l’origine maghrébine. Elle est réalisée sur données expérimentales de testing réalisé dans trois professions en tension pour lesquelles la discrimination devrait a priori être très réduite : les responsables administratifs de catégorie A, les techniciens de maintenance de catégorie B et les aides soignantes de catégorie C. Pour chaque profession, nous avons construit 3 profils fictifs de candidats à l’emploi similaires en tout point à l’exception…

JEL : C - Mathematical and Quantitative Methods/C.C8 - Data Collection and Data Estimation Methodology • Computer Programs/C.C8.C81 - Methodology for Collecting Estimating and Organizing Microeconomic Data • Data AccessJEL: C - Mathematical and Quantitative Methods/C.C9 - Design of Experiments/C.C9.C93 - Field Experimentstesting.JEL: J - Labor and Demographic Economics/J.J4 - Particular Labor Markets/J.J4.J45 - Public Sector Labor Marketsfonction publiqueJEL: J - Labor and Demographic Economics/J.J7 - Labor Discrimination/J.J7.J71 - Discrimination[SHS.ECO]Humanities and Social Sciences/Economics and FinanceJEL : J - Labor and Demographic Economics/J.J4 - Particular Labor Markets/J.J4.J45 - Public Sector Labor Marketsoriginelieu de résidenceJEL : C - Mathematical and Quantitative Methods/C.C9 - Design of Experiments/C.C9.C93 - Field ExperimentsJEL : J - Labor and Demographic Economics/J.J1 - Demographic Economics/J.J1.J15 - Economics of Minorities Races Indigenous Peoples and Immigrants • Non-labor DiscriminationJEL: C - Mathematical and Quantitative Methods/C.C8 - Data Collection and Data Estimation Methodology • Computer Programs/C.C8.C81 - Methodology for Collecting Estimating and Organizing Microeconomic Data • Data Access[ SHS.ECO ] Humanities and Social Sciences/Economies and financesdiscrimination à l’embaucheJEL: J - Labor and Demographic Economics/J.J1 - Demographic Economics/J.J1.J15 - Economics of Minorities Races Indigenous Peoples and Immigrants • Non-labor DiscriminationJEL : J - Labor and Demographic Economics/J.J7 - Labor Discrimination/J.J7.J71 - Discrimination[SHS.ECO] Humanities and Social Sciences/Economics and Finance
researchProduct