Search results for "Leukocytes"

showing 10 items of 429 documents

Leukocyte subtypes, gelatinases, and their tissue inhibitors in a group of subjects with asymptomatic carotid atherosclerosis

2022

In a cohort of subjects with asymptomatic carotid atherosclerosis (ACA), we have evaluated the neutrophil and lymphocyte count and their ratio (NLR), the gelatinases (MMP-2 and MMP-9) and their tissue inhibitors (TIMP-1 and TIMP-2). At baseline, no difference was observed between ACA subjects and subject control group regarding neutrophil and lymphocyte count while was evident in ACA subjects a significant increase in MMP-2, MMP-9 and TIMP-2 associated to a significant decrease in TIMP-1. Dividing the ACA according to the number of cardiovascular risk factors (CRFs) we have observed an increase in lymphocyte count in the subgroup with 3–5 CRFs. Evaluating the leukocyte subtypes according to…

Carotid Artery Diseasescardiovascular risk factorslymphocytesTissue Inhibitor of Metalloproteinase-2Tissue Inhibitor of Metalloproteinase-1PhysiologyHematologyTIMP-2Asymptomatic carotid atherosclerosisTIMP-1Matrix Metalloproteinase 9neutrophilsinsulin resistancePhysiology (medical)LeukocytesHumansMatrix Metalloproteinase 2Cardiology and Cardiovascular MedicinegelatinasesBiomarkersResearch ArticleClinical Hemorheology and Microcirculation
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Different sensitivity of carp (Cyprinus carpio) and rainbow trout (Oncorhynchus mykiss) to the immunomodulatory effects of UVB irradiation.

2006

Abstract In order to study the sensitivity of two fish species, carp (Cyprinus carpio) and rainbow trout (Oncorhynchus mykiss), to the immunomodulatory effects of ultraviolet B (UVB) radiation, the fish were exposed to a single UVB dose of 50, 250, 500 or 1000 mJ cm−2. These species represent different phylogenetic groups of fish, and they differ also in their behaviour inhabitating often dark and turbid (carp) or clear and transparent waters (salmonids). Immune responses were studied on day 1 post-irradiation. Unexposed fish, and fish exposed to radiation depleted of UV wavelengths served as controls. UVB irradiation markedly enhanced the blood respiratory burst and cytotoxic activity in c…

CarpsHydrocortisoneUltraviolet Raysanimal diseasesZoologyAquatic ScienceKidneyCyprinusImmune systemLeukocytesEnvironmental ChemistryAnimalsUvb irradiationCarpRespiratory BurstHead Kidneyintegumentary systembiologyEcologyUltraviolet bDose-Response Relationship RadiationGeneral Medicinebiology.organism_classificationCytotoxicity Tests ImmunologicRespiratory burstImmunoglobulin MImmune SystemOncorhynchus mykissLuminescent MeasurementsRainbow troutSpectrophotometry UltravioletFishshellfish immunology
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Cladribine exerts an immunomodulatory effect on human and murine dendritic cells

2014

Cladribine is a purine nucleoside analog developed to treat lymphoid malignancies. Reported therapeutic benefits for the autoimmune disease multiple sclerosis indicate additional immunomodulatory effects beyond the well-characterized cytotoxic activity causing lymphopenia. Here, we demonstrate that cladribine reduces the secretion of inflammatory cytokines and chemokines by murine and human dendritic cells, the most potent antigen-presenting cells. This compound also modulates the expression of the activation markers CD86 and MHC II. Furthermore, cladribine affects the T cell priming capacity of dendritic cells, resulting in reduced induction of interferon-γ- and tumor necrosis factor-α-pro…

Cell SurvivalT-LymphocytesT cellImmunologyBiologyMicePhagocytosismedicineAnimalsHumansImmunologic FactorsImmunology and AllergyCytotoxic T cellAntigen-presenting cellCladribineCells CulturedCell ProliferationPharmacologyCD86ChemotaxisCell DifferentiationDextransDendritic CellsDendritic cellmedicine.diseaseMice Inbred C57BLLeukemiamedicine.anatomical_structureImmunologyLeukocytes MononuclearCancer researchCladribineCytokinesTumor necrosis factor alphaFluorescein-5-isothiocyanatemedicine.drugInternational Immunopharmacology
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Monocyte-derived dendritic cells of patients with coronary artery disease show an increased expression of costimulatory molecules CD40, CD80 and CD86…

2007

Background Atherosclerosis is a disease triggered by diverse exogenous stimuli and sustained by chronic inflammatory processes. Dendritic cells (DCs) are key regulatory antigen-presenting cells and play a crucial role in regulating the adaptive and innate immune system in any chronic inflammatory process. DCs are present in atherosclerotic lesions in the areas of the highest T-cell density. So far, their role in atherosclerosis has not been fully elucidated. We investigated the phenotypic properties of DCs in patients with coronary artery disease (CAD) in comparison to healthy individuals. Methods Peripheral blood monocytes were isolated from 50 patients with CAD and 19 healthy individuals …

Cellular differentiationchemical and pharmacologic phenomenaCoronary Artery DiseaseMonocytesFlow cytometryDownregulation and upregulationRisk FactorsMedicineHumansCD40 AntigensAgedRegulation of gene expressionCD86Innate immune systemCD40biologymedicine.diagnostic_testbusiness.industryhemic and immune systemsCell DifferentiationGeneral MedicineDendritic CellsMiddle AgedAtherosclerosisFlow CytometryC-Reactive ProteinGene Expression RegulationImmunologybiology.proteinB7-1 AntigenLeukocytes MononuclearB7-2 AntigenCardiology and Cardiovascular MedicinebusinessCD80Coronary artery disease
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Leukocyte Redistribution: Effects of Beta Blockers in Patients with Chronic Heart Failure

2009

BACKGROUND:Overproduction of pro-inflammatory cytokines is a well established factor in the progression of chronic heart failure (CHF). Changes in cellular immunity have not been widely studied, and the impact of standard medication is uncertain. Here we investigate whether a leukocyte redistribution occurs in CHF and whether this effect is influenced by beta-blocker therapy. METHODOLOGY:We prospectively studied 75 patients with systolic CHF (age: 68+/-11 years, left ventricular ejection fraction 32+/-11%, New York Heart Association class 2.5+/-0.7) and 20 age-matched healthy control subjects (age: 63+/-10 years). We measured the response of cells to endotoxin exposure in vitro, analysed su…

Cellular immunityAdrenergic beta-AntagonistsCardiovascular Disorders/Heart Failurelcsh:MedicineVentricular Function LeftAdrenergic beta-AntagonistsLeukocytesHumansMedicineIn patientProspective Studiescardiovascular diseaseslcsh:ScienceProspective cohort studyAgedHeart FailureMultidisciplinaryVentricular functionbusiness.industrylcsh:RCase-control studyMiddle AgedFlow Cytometrymedicine.diseaseCase-Control StudiesImmunology/Leukocyte ActivationHeart failureImmunologylcsh:QbusinessImmunology/Leukocyte DevelopmentResearch ArticlePLoS ONE
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Thy-1 (CD90) regulates the extravasation of leukocytes during inflammation.

2010

Human Thy-1 (CD90) has been shown to mediate adhesion of inflammatory cells to activated microvascular endothelial cells via interaction with Mac-1 (CD11b/CD18) in vitro. Since there are no data showing the physiological relevance of Thy-1 for the recruitment of inflammatory cells in vivo, different inflammation models were investigated in Thy-1-deficient mice and littermate controls. In thioglycollate-induced peritonitis, the number of neutrophils and monocytes was significantly diminished in Thy-1-deficient mice. During acute lung inflammation, the extravasation of eosinophils and monocytes into the lung was significantly reduced in Thy-1-deficient mice. Moreover, during chronic lung infl…

ChemokineMice 129 StrainNeutrophilsmedicine.medical_treatmentT-LymphocytesImmunologyInflammationCD18In Vitro TechniquesPeritonitisMonocytesMiceCell MovementCell AdhesionLeukocytesImmunology and AllergyMedicineAnimalsHumansCD90Thy-1InflammationMice KnockoutTransplantation Chimerabiologybusiness.industryInterleukinsEndothelial CellsPneumoniaExtravasationTransplantationEosinophilsMice Inbred C57BLCytokineIntegrin alpha MImmunologybiology.proteinThy-1 Antigensmedicine.symptomChemokinesbusinessextravasationPeptide HydrolasesEuropean journal of immunology
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Hypothermic preservation of lung allograft inhibits cytokine-induced chemoattractant-1, endothelial leucocyte adhesion molecule, vascular cell adhesi…

2007

Summary Organ dysfunction is a major clinical problem after lung transplantation. Prolonged cold ischaemia and reperfusion injury are believed to play a central role in this complication. The influence of cold preservation on subsequent warm reperfusion was studied in an isolated, ventilated and perfused rat lung. Rat lungs were flushed with cold Perfadex-solution and stored at 4°C for different time periods. Thereafter lungs were perfused and ventilated for up to 3 h. Physiological parameters, production of inflammatory mediators and leucocyte infiltration were measured before and after perfusion. Lungs subjected to a cold ischaemia time of up to 6 h showed stable physiological conditions …

ChemokinePathologymedicine.medical_specialtymedicine.medical_treatmentImmunologyIntercellular Adhesion Molecule-1Vascular Cell Adhesion Molecule-1Blood PressurePulmonary EdemaPulmonary ArteryBasic ImmunologyHypothermia InducedmedicineImmunology and AllergyAnimalsRespiratory systemRats WistarLungChemokine CCL2LungbiologyCell adhesion moleculemedicine.diseaseIntercellular Adhesion Molecule-1RatsEndothelial stem cellCytokinemedicine.anatomical_structureReperfusion InjuryImmunologybiology.proteinLeukocytes MononuclearTissue PreservationInflammation MediatorsE-SelectinReperfusion injuryCell Adhesion MoleculesLung Transplantation
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Atherosclerosis as an inflammatory disease.

2011

In many ways, atherosclerosis is a chronic inflammatory disorder and this issue is confirmed by recent investigations of that have focused on inflammation, providing new insight into mechanisms of disease. Several recent studies have addressed the role of chemokines in leukocyte accumulation in atherosclerosis, extending our knowledge and understanding of the complex and cell type-specific functions of chemokines in atherosclerosis. Activated T-lymphocytes within the atherosclerotic vessel wall express the CD40 ligand surface molecule, known to play a major role in several immunological pathways. In addition to activated T-lymphocytes, functional CD40 and CD40L are coexpressed by human vasc…

ChemokineSettore MED/09 - Medicina InternaEndotheliumT-LymphocytesInflammationDrug DiscoverymedicineLeukocytesAnimalsHumansCD154PharmacologyInflammationCD40Innate immune systembiologyCell adhesion moleculeMacrophagesAtherosclerosis inflammationAcquired immune systemAtherosclerosisPlaque Atheroscleroticmedicine.anatomical_structureDrug DesignImmunologybiology.proteinCancer researchCytokinesEndothelium Vascularmedicine.symptomChemokinesCurrent pharmaceutical design
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Tolerance towards resident intestinal flora in mice is abrogated in experimental colitis and restored by treatment with interleukin-10 or antibodies …

1996

There is now increasing evidence that hyperresponsiveness towards intestinal flora is a crucial event in the pathogenesis of inflammatory bowel disease (IBD). In support of this hypothesis, we recently described in humans that tolerance exists towards indigenous intestinal flora but is broken in active IBD lesions. In the present study, we have attempted to transfer this model into mice from different genetic backgrounds (BALB/c, SJL/J, C3H/HeJ). We found that mononuclear cells from spleen, small bowel and large bowel of mice do not proliferate, i.e. are tolerant when exposed to bacterial sonicates derived from autologous intestine (BsA) but do proliferate, i.e. are immune when exposed to b…

ColonImmunologySpleenBiologyLymphocyte ActivationInflammatory bowel diseaseMicrobiologyMicePeyer's PatchesImmune systemCrohn DiseaseSpecies SpecificityImmunityIntestine SmallImmune TolerancemedicineAnimalsHumansImmunologic FactorsImmunology and AllergyColitisMice Inbred BALB CMice Inbred C3HBacteriaAntibodies MonoclonalInterleukinColitismedicine.diseaseInterleukin-12Recombinant ProteinsInterleukin-10RatsSpecific Pathogen-Free OrganismsIntestinesDisease Models AnimalInterleukin 10medicine.anatomical_structureTrinitrobenzenesulfonic AcidImmunologyLeukocytes MononuclearInterleukin 12SpleenEuropean Journal of Immunology
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Efavirenz induces interactions between leucocytes and endothelium through the activation of Mac-1 and gp150,95

2013

The potential cardiovascular (CV) toxicity associated with combined antiretroviral therapy (cART) has been attributed mainly to the nucleoside reverse transcriptase inhibitors abacavir and didanosine. However, the other two components of cART--non-nucleoside reverse transcriptase inhibitors (NNRTIs) and protease inhibitors (PIs)--may also be implicated, either directly or by influencing the action of the other drugs. This study evaluates the acute direct effects of the NNRTIs efavirenz and nevirapine and one of the most widely employed PIs, lopinavir, on leucocyte-endothelium interactions, a hallmark of CV disease.Drugs were analysed in vitro in human cells (interactions of peripheral blood…

CyclopropanesMaleMicrobiology (medical)EfavirenzNevirapineEndotheliumAnti-HIV AgentsIntegrin alphaXbeta2Macrophage-1 AntigenPharmacologyBiologyLopinavirNucleoside Reverse Transcriptase InhibitorRats Sprague-Dawleychemistry.chemical_compoundimmune system diseasesAbacavirCell AdhesionLeukocytesmedicineAnimalsHumansPharmacology (medical)EndotheliumNevirapineDidanosineCells CulturedPharmacologyGene Expression Profilingvirus diseasesLopinavirFlow CytometryBenzoxazinesRatsInfectious Diseasesmedicine.anatomical_structurechemistryAlkynesToxicitymedicine.drugJournal of Antimicrobial Chemotherapy
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