Search results for "Leukocytes"

showing 10 items of 429 documents

Genetic and Environmental Effects on Telomere Length and Lung Function: A Twin Study.

2015

Background The purpose of the study was to estimate the heritability of leukocyte telomere length (LTL) and lung function and to examine whether LTL and lung function share genetic or environmental effects in common. Methods 386 monozygotic and dizygotic Finnish twin sisters (age 68.4±3.4 years) were included. Relative LTL was determined from peripheral blood DNA by qPCR. Lung function measures of FEV1, FVC, FEV1/FVC, and PEF were derived from spirometry. Genetic modeling was performed with MPlus statistical software. Results Univariate analysis revealed that in LTL, 62% (95% confidence interval 50-72) of the variance was explained by additive genetic and 38% (28-50) by unique environmental…

0301 basic medicineSpirometryAgingBivariate analysista3111Genetic correlation03 medical and health sciencesFEV1/FVC ratio0302 clinical medicineForced Expiratory VolumeLeukocytesTwins DizygoticMedicineHumansLungAgedmedicine.diagnostic_testbusiness.industryEnvironmental exposureta3142Environmental ExposureTwins Monozygoticrespiratory systemHeritabilityMiddle AgedTelomeretelomeresTwin studyConfidence intervalrespiratory tract diseases030104 developmental biology030228 respiratory systemSpirometrygenetic modelingFemaleGeriatrics and GerontologybusinessDemographyThe journals of gerontology. Series A, Biological sciences and medical sciences
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Formyl-peptide receptor 2 governs leukocyte influx in local Staphylococcus aureus infections

2017

Leukocytes express formyl-peptide receptors (FPRs), which sense microbe-associated molecular pattern (MAMP) molecules, leading to leukocyte chemotaxis and activation. We recently demonstrated that phenol-soluble modulin (PSM) peptides from highly pathogenic Staphylococcus aureus are efficient ligands for the human FPR2. How PSM detection by FPR2 impacts on the course of S. aureus infections has remained unknown. We characterized the specificity of mouse FPR2 (mFpr2) using a receptor-transfected cell line, homeobox b8 (Hoxb8), and primary neutrophils isolated from wild-type (WT) or mFpr2−/− mice. The influx of leukocytes into the peritoneum of WT and mFpr2−/− mice was analyzed. We demonstrat…

0301 basic medicineStaphylococcus aureusNeutrophilsBacterial Toxinsmedicine.disease_causeLigandsBiochemistryCell DegranulationFormyl peptide receptor 2MicrobiologyCell Line03 medical and health sciencesMice0302 clinical medicinePeritoneumCell MovementGeneticsmedicineLeukocytesAnimalsHumansCalcium SignalingReceptors LipoxinReceptorMolecular BiologyHomeodomain ProteinsMice KnockoutInnate immune systemChemistryResearchHOXB8Staphylococcal InfectionsReceptors Formyl PeptideMice Inbred C57BLDisease Models Animal030104 developmental biologymedicine.anatomical_structureCell cultureStaphylococcus aureusGenes BacterialMutationFemaleLeukocyte chemotaxis030215 immunologyBiotechnology
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Humanized mice in cutaneous leishmaniasis—Suitability analysis of human PBMC transfer into immunodeficient mice

2019

Humanized mice represent a suitable preclinical test system for example therapeutic interventions in various disease settings, including infections. Here, we intended to establish such system for cutaneous leishmaniasis by infecting T, B and NK cell-deficient mice adoptively transferred with human peripheral blood mononuclear cells (PBMC). L major infection led to the establishment of parasite lesions harbouring viable parasites and human T cells, but parasite elimination was not seen due to a species-specific activity of T cell-derived human IFNγ. In addition, up to 50% of infected mice succumbed to severe graft-versus-host disease. In summary, even though long-term disease outcome assessm…

0301 basic medicineT cellGraft vs Host DiseaseLeishmaniasis CutaneousDermatologyDiseaseBiochemistryPeripheral blood mononuclear cellLesionInterferon-gammaMice030207 dermatology & venereal diseases03 medical and health sciences0302 clinical medicineSpecies SpecificityCutaneous leishmaniasisT-Lymphocyte SubsetsIn vivoAnimalsHumansMedicineParasite hostingMolecular Biologybusiness.industryMacrophagesLeishmaniasismedicine.diseaseAdoptive Transfer030104 developmental biologymedicine.anatomical_structureModels AnimalImmunologyDisease ProgressionLeukocytes MononuclearHeterograftsmedicine.symptombusinessExperimental Dermatology
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Activation and selective IL-17 response of human Vγ9Vδ2 T lymphocytes by TLR-activated plasmacytoid dendritic cells.

2016

// Elena Lo Presti 1,2 , Nadia Caccamo 1,2 , Valentina Orlando 1,2 , Francesco Dieli 1,2 and Serena Meraviglia 1,2 1 Central Laboratory of Advanced Diagnosis and Biomedical Research (CLADIBIOR), University of Palermo, Palermo, Italy 2 Department of Biopathology and Medical Biotechnologies (DIBIMED), University of Palermo, Palermo, Italy Correspondence to: Serena Meraviglia, email: // Keywords : γδ T cells, plasmacytoid dendritic cells, IL-17, TLR activation, proliferation, Immunology and Microbiology Section, Immune response, Immunity Received : July 20, 2016 Accepted : August 02, 2016 Published :August 31, 2016 Abstract Vγ9Vδ2 T cells and plasmacytoid dendritic cells (pDCs) are two distinc…

0301 basic medicineTLR activationCellCell CommunicationLigandsLymphocyte Activation0302 clinical medicineT-Lymphocyte SubsetsCoculture TechniqueAntigen PresentationInterleukin-17Research Paper: Immunologyhemic and immune systemsIL-17medicine.anatomical_structurePhenotypeOncologyplasmacytoid dendritic cellsImmunology and Microbiology SectionInterleukin 17HumanCell typeproliferationCD40 LigandLigandBiologyDendritic Cellγδ T cells03 medical and health sciencesInducible T-Cell Co-Stimulator LigandInterferon-gammaImmune systemImmunityplasmacytoid dendritic cellmedicineHumansImmune responseCell Proliferationγδ T cellCD40Innate immune systemImmunityTLR9Dendritic CellsReceptors OX40Coculture TechniquesImmunity Innate030104 developmental biologyImmunologybiology.proteinLeukocytes MononuclearCpG IslandsCpG IslandImmunologic Memory030215 immunologyOncotarget
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Vitamin K antagonism impairs the bone marrow microenvironment and hematopoiesis

2018

Abstract Vitamin K antagonists (VKAs) have been used in 1% of the world’s population for prophylaxis or treatment of thromboembolic events for 64 years. Impairment of osteoblast function and osteoporosis has been described in patients receiving VKAs. Given the involvement of cells of the bone marrow microenvironment (BMM), such as mesenchymal stem cells (MSCs) and macrophages, as well as other factors such as the extracellular matrix for the maintenance of normal hematopoietic stem cells (HSCs), we investigated a possible effect of VKAs on hematopoiesis via the BMM. Using various transplantation and in vitro assays, we show here that VKAs alter parameters of bone physiology and reduce funct…

0301 basic medicineVitamin KImmunologyPopulationBone Marrow CellsPeriostinBiochemistryMice03 medical and health sciences0302 clinical medicineLeukocytesAnimalsMedicineeducationeducation.field_of_studyDose-Response Relationship Drugbusiness.industryMacrophagesMonocyteMesenchymal stem cellAnticoagulantsCell BiologyHematologyHematopoietic Stem CellsHematopoiesisTransplantationHaematopoiesis030104 developmental biologymedicine.anatomical_structureCellular MicroenvironmentMyelodysplastic Syndromes030220 oncology & carcinogenesisCancer researchWarfarinBone marrowStem cellbusinessCell Adhesion MoleculesBiomarkersBlood
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Myeloid-Derived Suppressor Cells Specifically Suppress IFN-γ Production and Antitumor Cytotoxic Activity of Vδ2 T Cells.

2018

γδ T cells represent less than 5% of circulating T cells; they exert a potent cytotoxic function against tumor or infected cells and secrete cytokines like conventional αβ T cells. As αβ T cells γδ T cells reside in the typical T cell compartments (the lymph nodes and spleen), but are more widely distributed in tissues throughout the body. For these reasons, some investigators are exploring the possibility of immunotherapies aimed to expand and activate Vδ2 T cells, or using them as Chimeric Antigen Receptor carriers. However, the role of immunosuppressive microenvironment on Vδ2 T cells during infections and cancers has not been completely elucidated. In particular, the effects of myeloid-…

0301 basic medicinelcsh:Immunologic diseases. AllergyCytotoxicity Immunologicγmedicine.medical_treatmentT cellδImmunologyAntitumoral activityT cellsSpleenLymphocyte ActivationJurkat cellsγδ T cellsImmunophenotyping03 medical and health sciencesInterferon-gamma0302 clinical medicineT-Lymphocyte SubsetsCell Line TumorNeoplasmsmedicineMyeloid-derived suppressor cellImmunology and AllergyCytotoxic T cellHumansIFN-γantitumoral activityArginaseChemistryMyeloid-Derived Suppressor CellsDegranulationReceptors Antigen T-Cell gamma-deltaImmunotherapy030104 developmental biologymedicine.anatomical_structureCell cultureCancer researchMyeloid-derived Suppressor CellLeukocytes MononuclearCytokinesImmunotherapyimmunotherapylcsh:RC581-607Biomarkers030215 immunologyFrontiers in immunology
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Novel anti-GARP antibody DS-1055a augments anti-tumor immunity by depleting highly suppressive GARP+ regulatory T cells

2021

Abstract Regulatory T (Treg) cells, which are essential for maintaining self-tolerance, inhibit anti-tumor immunity, consequently hindering protective cancer immunosurveillance, and hampering effective anti-tumor immune responses in tumor-bearing hosts. Here, we show that depletion of Treg cells via targeting glycoprotein A repetitions predominant (GARP) induces effective anti-tumor immune responses. GARP was specifically expressed by highly suppressive Treg cells in the tumor microenvironment (TME) of multiple cancer types in humans. In the periphery, GARP was selectively induced in Treg cells, but not in effector T cells, by polyclonal stimulation. DS-1055a, a novel afucosylated anti-huma…

0301 basic medicinemedicine.drug_classmedicine.medical_treatmentImmunologychemical and pharmacologic phenomenaMice SCIDBiologyMonoclonal antibodyT-Lymphocytes RegulatoryMice03 medical and health sciences0302 clinical medicineImmune systemCancer immunotherapyMice Inbred NODImmunityNeoplasmsImmune ToleranceTumor MicroenvironmentmedicineAnimalsHumansImmunology and AllergyMice KnockoutTumor microenvironmentImmunityAntibodies MonoclonalMembrane ProteinsFOXP3General MedicineImmunosurveillance030104 developmental biology030220 oncology & carcinogenesisLeukocytes MononuclearCancer researchbiology.proteinFemaleImmunotherapyAntibodyInternational Immunology
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Oral Unsaturated Fat Load Impairs Postprandial Systemic Inflammation in Primary Hypercholesterolemia Patients.

2021

Context: Primary hypercholesterolemia (PH) is a lipid disorder characterized by elevated levels of cholesterol and low-density lipoprotein (LDL). Low-grade systemic inflammation is associated with PH, which might explain the higher incidence of cardiovascular diseases in this setting.Objective: To evaluate the effect of an oral unsaturated fat load (OUFL) on different immune parameters and functional consequences in patients with PH in postprandial state.Design: A commercial liquid preparation of long-chain triglycerides (Supracal®; ω6/ω3 ratio >20/1, OUFL) was administered to 20 patients and 10 age-matched controls. Whole blood was collected before (fasting state) and 4 h after admi…

0301 basic medicinemedicine.medical_specialtyEndotheliumleukocytesinflammatory mediatorsRM1-950030204 cardiovascular system & hematologySystemic inflammationendothelial dysfunction03 medical and health scienceschemistry.chemical_compound0302 clinical medicineoral unsaturated fat loadInternal medicineprimary hypercholesterolemiamedicineplatelet activationPharmacology (medical)Platelet activationEndothelial dysfunctionOriginal ResearchPharmacologysystemic inflammationCholesterolbusiness.industryUnsaturated fatmedicine.disease030104 developmental biologyEndocrinologymedicine.anatomical_structurePostprandialchemistryTherapeutics. Pharmacologymedicine.symptombusinessLipoproteinFrontiers in pharmacology
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Metabolic syndrome enhances endoplasmic reticulum, oxidative stress and leukocyte–endothelium interactions in PCOS

2017

Polycystic ovary syndrome (PCOS) is associated with insulin resistance, which can lead to metabolic syndrome (MetS). Oxidative stress and leukocyte-endothelium interactions are related to PCOS. Our aim was to evaluate whether the presence of MetS in PCOS patients can influence endoplasmic reticulum (ER) and oxidative stress and leukocyte-endothelium interactions.This was a prospective controlled study conducted in an academic medical center. The study population consisted of 148 PCOS women (116 without/32 with MetS) and 112 control subjects (87 without / 25 with MetS). Metabolic parameters, reactive oxygen species (ROS) production, ER stress markers (GRP78, sXBP1, ATF6), leukocyte-endotheli…

0301 basic medicinemedicine.medical_specialtyXBP1Endocrinology Diabetes and Metabolism030209 endocrinology & metabolismBiologymedicine.disease_cause03 medical and health sciences0302 clinical medicineEndocrinologyInsulin resistanceInternal medicineCell AdhesionLeukocytesmedicineHumansProspective StudiesEndoplasmic Reticulum Chaperone BiPMetabolic SyndromeInterleukin-6ATF6Endoplasmic Reticulum StressIntercellular Adhesion Molecule-1medicine.diseasePolycystic ovaryOxidative Stress030104 developmental biologyEndocrinologyUnfolded protein responseHomeostatic model assessmentCytokinesFemaleEndothelium VascularMetabolic syndromeReactive Oxygen SpeciesCell Adhesion MoleculesOxidative stressPolycystic Ovary SyndromeMetabolism
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Leukocyte and Skeletal Muscle Telomere Length and Body Composition in Monozygotic Twin Pairs Discordant for Long-term Hormone Replacement Therapy

2017

Estrogen-based hormone replacement therapy (HRT) may be associated with deceleration of cellular aging. We investigated whether long-term HRT has effects on leukocyte (LTL) or mean and minimum skeletal muscle telomere length (SMTL) in a design that controls for genotype and childhood environment. Associations between telomeres, body composition, and physical performance were also examined. Eleven monozygotic twin pairs (age 57.6 ± 1.8 years) discordant for HRT were studied. Mean duration of HRT use was 7.3 ± 3.7 years in the user sister, while their co-twins had never used HRT. LTL was measured by qPCR and SMTLs by southern blot. Body and muscle composition were estimated by bioimpedance an…

0301 basic medicinemedicine.medical_specialtymedicine.drug_classMonozygotic twinmedicine.disease_cause03 medical and health sciencesGrip strengthJumpingInsulin resistanceInternal medicinebioimpedancemedicineElectric ImpedanceLeukocytesestrogenHumanstietokonetomografiapost-menopausalMuscle SkeletalExerciseGenetics (clinical)Hand Strengthbusiness.industryEstrogen Replacement TherapyObstetrics and GynecologySkeletal muscleta3141computed tomographyTwins MonozygoticMiddle AgedTelomeremedicine.diseasetelomeresObesitypercentage of fatfat-free mass030104 developmental biologyEndocrinologymedicine.anatomical_structuretwin designEstrogenTransgender hormone therapyPediatrics Perinatology and Child HealthBody CompositionFemaletelomeeritbusinessTwin Research and Human Genetics
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