Search results for "Leukotriene B"

showing 10 items of 68 documents

Effect of imidazo[1,2-α]pyrimidine derivatives on leukocyte function

2001

Objective and Design: A series of six imidazo[1,2-α]pyrimidine (IP) derivatives were evaluated for their effects on leukocyte functions in vitro as well as on the inflammatory response induced by zymosan in the mouse air pouch.¶Materials and Subjects: Human neutrophils and murine peritoneal macrophages were used for in vitro assays. Mouse air pouch was performed in Swiss mice.¶Treatment: Test compounds were incubated with either human neutrophils or mouse peritoneal macrophages at concentrations not showing cytotoxic effects. For in vivo experiments, IPs were injected into the air pouch.¶Methods: Elastase and myeloperoxidase release, superoxide generation and LTB4 production were assayed in…

Leukocyte migrationLipopolysaccharideNeutrophilsImmunologyAnti-Inflammatory AgentsLeukotriene B4Micechemistry.chemical_compoundSuperoxidesIn vivoAnimalsHumansPharmacologybiologySuperoxideElastaseZymosanImidazolesDegranulationMolecular biologyPyrimidineschemistryBiochemistryMyeloperoxidaseMacrophages Peritonealbiology.proteinFemaleInflammation Research
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Effects of some isoxazolpyrimidine derivatives on nitric oxide and eicosanoid biosynthesis

2000

Abstract The inhibitory effect of some isoxazolpyrimidine derivatives on iNOS and COX-2 endotoxin induction in mouse peritoneal macrophages has been studied. Three of these compounds inhibited nitrite and PGE2 accumulation in a concentration dependent-manner at μM range. None of these active compounds affected iNOS, COX-2, COX-1 or PLA2 activities, although some reduced iNOS or COX-2 expression. Besides, no effect was observed on human neutrophil inflammatory responses (LTB4 biosynthesis and Superoxide or elastase release). Active compounds were assayed by oral administration in the mouse air pouch model, where they inhibited nitrite accumulation without affecting PGE 2 levels or leukocyte …

Leukocyte migrationNeutrophilsBlotting WesternPharmacologyNitric OxideLeukotriene B4DinoprostonePhospholipases AGeneral Biochemistry Genetics and Molecular BiologyNitric oxideMicechemistry.chemical_compoundSuperoxidesOral administrationAnimalsHumansCyclooxygenase InhibitorsGeneral Pharmacology Toxicology and PharmaceuticsNitriteOxazolesInhibitory effectCyclooxygenase 2 InhibitorsPancreatic ElastaseSuperoxideElastaseMembrane ProteinsGeneral MedicineIsoenzymesPhospholipases A2PyrimidinesBiochemistrychemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesLuminescent MeasurementsCyclooxygenase 1EicosanoidsFemalelipids (amino acids peptides and proteins)Eicosanoid biosynthesis
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Beneficial effects of heme oxygenase-1 up-regulation in the development of experimental inflammation induced by zymosan.

2003

Heme oxygenase-1 (HO-1) is part of the integrated response to oxidative stress. This enzyme may exert anti-inflammatory effects in some animal models, although the precise mechanisms are not fully understood. We have examined the role of HO-1 in the inflammatory response induced by zymosan in the mouse air pouch. Zymosan administration induced HO-1 protein expression in leukocytes migrating to exudates, with maximal levels in the late phase of this response (24-48 h). This was accompanied by ferritin induction and bilirubin accumulation, indicating that this enzyme is active in our model. HO-1 expression by zymosan treatment was partly reduced by aminoguanidine, suggesting the participation…

Leukotriene B4Blotting WesternInflammationCell CountPharmacologymedicine.disease_causeLeukotriene B4Dinoprostonechemistry.chemical_compoundMicePhagocytosismedicineAnimalsHemePharmacologyInflammationTumor Necrosis Factor-alphaMacrophagesZymosanZymosanMembrane ProteinsBilirubinExudates and TransudatesFlow CytometryUp-RegulationHeme oxygenasechemistryBiochemistryHeme Oxygenase (Decyclizing)Molecular MedicineCytokinesEicosanoidsHeminTumor necrosis factor alphaFemalemedicine.symptomOxidative stressCell DivisionHeme Oxygenase-1HeminInterleukin-1The Journal of pharmacology and experimental therapeutics
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Effects of nordihydroguaiaretic acid on murine antibody-dependent cellular cytotoxicity.

1996

The purpose of this study was to analyze the effects of nordihydroguaiaretic acid, an inhibitor of the lipoxygenase pathway of arachidonic acid, on antibody-dependent cellular cytotoxicity. Antibody-dependent cellular cytotoxicity mediated by murine spleen cells was significantly inhibited by concentrations of nordihydroguaiaretic acid from 10(-5) to 10(-4) M (1C50 = 2 x 10(-5) M). The inhibitory effect of nordihydroguaiaretic acid was also observed on antibody-dependent cellular cytotoxicity mediated by macrophage-depleted spleen cells as well as isolated macrophages. Nordihydroguaiaretic acid was highly effective when added at the beginning of the assay and was always present throughout t…

Leukotriene B4Clinical BiochemistrySpleenIn Vitro TechniquesLeukotriene B4chemistry.chemical_compoundLipoxygenaseMicemedicineAnimalsMasoprocolLipoxygenase InhibitorsCytotoxicityAntibody-dependent cell-mediated cytotoxicityMice Inbred BALB CbiologyMacrophagesAntibody-Dependent Cell CytotoxicityNordihydroguaiaretic acidKineticsmedicine.anatomical_structurechemistryBiochemistryEnzyme inhibitorbiology.proteinArachidonic acidSpleenInternational journal of clinicallaboratory research
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Endogenous nitric oxide inhibits leukotriene B4 release from rat alveolar macrophages

1997

Effects of endogenous nitric oxide (NO) on the release of mediators of the lipoxygenase and cyclo-oxygenase pathway from rat alveolar macrophages were studied. Alveolar macrophages, freshly isolated or after 18-h culture, were incubated in (amino acid-free) Krebs medium and labelled with [3H]arachidonic acid. The release of [3H]leukotriene B4 and [3H]prostanoids (separated by high performance liquid chromatography) was determined. A 23187 was used as stimulus, as rising intracellular Ca2+ activates directly the phospholipase A2 and lipoxygenase pathway. A 23187 (10 microM) enhanced [3H]leukotriene B4 release from freshly prepared alveolar macrophages about 65-fold, but only 5- to 6-fold fro…

Leukotriene B4LipoxygenaseArachidonic AcidsBiologyNitric OxideLeukotriene B4Nitric oxideRats Sprague-Dawleychemistry.chemical_compoundLipoxygenasePhospholipase A2Macrophages AlveolarmedicineAnimalsEnzyme InhibitorsCalcimycinCells CulturedChromatography High Pressure LiquidPharmacologyomega-N-MethylarginineProstanoidMolecular biologyRatsmedicine.anatomical_structurechemistryBiochemistryProstaglandin-Endoperoxide SynthasesAlveolar macrophagebiology.proteinFemaleArachidonic acidNitric Oxide SynthasePulmonary alveolusEuropean Journal of Pharmacology
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Cigarette smoke increases BLT2 receptor functions in bronchial epithelial cells: in vitro and ex vivo evidence

2013

Summary Leukotriene B4 (LTB4) is a neutrophil chemotactic molecule with important involvement in the inflammatory responses of chronic obstructive pulmonary disease (COPD). Airway epithelium is emerging as a regulator of innate immune responses to a variety of insults including cigarette smoke, the major risk factor for COPD. In this study we have explored whether cigarette smoke extracts (CSE) or soluble mediators present in distal lung fluid samples (mini-bronchoalveolar lavages) from smokers alter the expression of the LTB4 receptor 2 (BLT2) and peroxisome proliferator-activated receptor-α (PPAR-α) in bronchial epithelial cells. We also evaluated the effects of CSE on the expression of i…

Leukotriene B4NeutrophilsImmunologyIntercellular Adhesion Molecule-1Blotting WesternReceptors Leukotriene B4Peroxisome proliferator-activated receptorSettore MED/41 - AnestesiologiaInflammationBronchiBiologychronic obstructive pulmonary diseasechemistry.chemical_compoundTobaccoacute lung injiurybronchial epithelial cellleukotriene B4.medicineCell AdhesionImmunology and AllergyHumansPPAR alphaReceptorPromoter Regions GeneticCells Culturedchemistry.chemical_classificationInnate immune systemPlant Extractscigarette smokeSmokingEpithelial CellsOriginal Articlesrespiratory systemFlow CytometryIntercellular Adhesion Molecule-1Neutrophiliarespiratory tract diseasesacute lung injiury; bronchial epithelial cells; cigarette smoke; chronic obstructive pulmonary disease; inflammation; leukotriene B4.STAT1 Transcription FactorchemistryinflammationImmunologyRespiratory epitheliumRNA Interferencemedicine.symptomBronchoalveolar Lavage FluidProtein Binding
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Monohydroxylated fatty acid substrate specificity of human leukocyte 5-lipoxygenase and ω-hydroxylase

1988

Various monohydroxylated fatty acids were synthesized from eicosapolyenoic acids, namely arachidonic (20:4 omega-6), timnodonic (20:5 omega-3), dihomogammalinolenic (20:3 omega-6) and mead (20:3 omega-9) acids. 12-Hydroxy derivatives, as well as 12-hydroxy-5,8,10-heptadecatrienoic acid (HHT), were produced with platelets as the enzyme source, and 15-hydroxy derivatives were produced by soya bean lipoxygenase treatment. Each monohydroxylated fatty acid was incubated with human leukocytes in the presence or absence of the calcium ionophore A23187, and dihydroxylated products were analysed by h.p.l.c. 12-Hydroxy derivatives of 20:4 omega-6, 20:5 omega-3 and 20:3 omega-9 were similarly oxygenat…

Leukotriene B4StereochemistryHydroxylationArachidonate LipoxygenasesBiochemistryMixed Function OxygenasesSubstrate SpecificityHydroxylationchemistry.chemical_compoundLipoxygenaseCytochrome P-450 Enzyme SystemHydroxyeicosatetraenoic AcidsLeukocytesHumansMolecular Biologychemistry.chemical_classificationArachidonate 5-LipoxygenasebiologyFatty AcidsFatty acidSubstrate (chemistry)Cell BiologyEnzymechemistryBiochemistryArachidonate 5-lipoxygenasebiology.proteinCytochrome P-450 CYP4AResearch ArticlePolyunsaturated fatty acidBiochemical Journal
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Effects of caffeoyl conjugates of isoprenyl-hydroquinone glucoside and quinic acid on leukocyte function.

2002

The activity of three prenylhydroquinone glucosides (1-3) and four caffeoylquinic esters (4-7), obtained from Phagnalon rupestre, on elastase release, myeloperoxidase activity and superoxide and leukotriene B(4) production from polymorphonuclear leukocytes was determined. 4,5-Dicaffeoylquinic acid strongly inhibited elastase release with an IC(50) value of 4.8 microM. Methylated caffeoylquinic derivatives were the most potent inhibitors of myeloperoxidase (IC(50) near 60 microM), whereas both methylated and free carboxyl isomers inhibited superoxide production with similar potency (IC(50) between 27 and 42 microM). The monocaffeoyl conjugate of prenylhydroquinone glucoside (3), the most pot…

Leukotriene B4StereochemistryNeutrophilsQuinic AcidAsteraceaeLeukotriene B4General Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundCaffeic AcidsGlucosideGlucosidesIsomerismPhenolsSuperoxidesHumansGeneral Pharmacology Toxicology and PharmaceuticsEnzyme InhibitorsCells CulturedPeroxidaseLeukotrieneHydroquinonebiologyMolecular StructurePancreatic ElastaseChemistrySuperoxidePlant ExtractsElastaseGeneral MedicineQuinic acidBiochemistryMyeloperoxidasebiology.proteinLife sciences
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Isolation of two triterpenoids and a biflavanone with anti-Inflammatory activity from Schinus molle fruits.

2003

Three compounds with anti-inflammatory activity were isolated from Schinus molle fruits. Two of the compounds were identified as 3- epi-isomasticadienolalic acid ( 1), isomasticadienonalic acid ( 2) and chamaejasmin ( 3). Triterpenes 1 and 2, and biflavanone 3 were tested on two models of mice paw inflammation: one of acute inflammation, induced by subcutaneous injection of either phospholipase A (2) (PLA (2)) or carrageenan in the paws of mice, and one of chronic inflammation in the form of eczema, provoked by repeated administration of TPA to the ears of mice. On the PLA (2)-induced mouse paw oedema, only 2 was active (30 mg/kg, 66 % inhibition at 60 min), whereas all compounds reduced th…

Leukotriene B4medicine.drug_classNeutrophilsAnacardiaceaePharmaceutical SciencePharmacologyCarrageenanAnti-inflammatoryPhospholipases AAnalytical ChemistrySchinus molleSubcutaneous injectionchemistry.chemical_compoundInhibitory Concentration 50MicePhospholipase A2TriterpeneDrug DiscoverymedicineAnimalsEdemaPharmacologychemistry.chemical_classificationFlavonoidsbiologyPlant ExtractsOrganic ChemistryAnti-Inflammatory Agents Non-Steroidalbiology.organism_classificationTriterpenesCarrageenanComplementary and alternative medicinechemistryBiochemistryFruitToxicitybiology.proteinMolecular MedicineTetradecanoylphorbol AcetateFemalePhytotherapyPlanta medica
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Effect of some hexahydroimidazo[1,2-c]pyrimidines in inflammatory responses involving leucocytes and macrophages.

2001

Abstract We have studied the effects of some hexahydroimidazo[1,2-c]pyrimidine derivatives (HIPs) on leucocyte functions in-vitro and we have assayed the anti-inflammatory activity of these compounds in two models of inflammation. All HIPs inhibited the human neutrophil degranulation process and superoxide generation at concentrations in the μM range. In mouse peritoneal macrophages stimulated with lipopolysaccharide, HIP-4 and HIP-5 inhibited nitrite production without affecting prostaglandin E2 (PGE2) accumulation. HIP-4 was also active in the zymosan-injected mouse air pouch model (at 100 nmol/pouch), with significant reductions in leucocyte migration and PGE2 and leukotriene B4 levels i…

LipopolysaccharideLeukotriene B4NeutrophilsPharmaceutical ScienceInflammationPharmacologyIn Vitro TechniquesCarrageenanLeukotriene B4Dinoprostonechemistry.chemical_compoundMiceSuperoxidesmedicineLeukocytesAnimalsEdemaHumansProstaglandin E2NitritesPeroxidasePharmacologyInflammationbiologyPancreatic ElastaseSuperoxideMacrophagesAnti-Inflammatory Agents Non-SteroidalDegranulationImidazolesZymosanCarrageenanPyrimidineschemistryBiochemistryMyeloperoxidasebiology.proteinMacrophages PeritonealFemalemedicine.symptommedicine.drugThe Journal of pharmacy and pharmacology
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