Search results for "Lip"

showing 10 items of 8306 documents

Metabolic adaptation in the human gut microbiota during pregnancy and the first year of life

2018

Abstract Background The relationship between the gut microbiome and the human host is dynamic and we may expect adjustments in microbiome function if host physiology changes. Metatranscriptomic approaches should be key in unraveling how such adjustments occur. Methods We employ metatranscriptomic sequencing analyses to study gene expression in the gut microbiota of infants through their first year of life, and of their mothers days before delivery and one year afterwards. Findings In infants, hallmarks of aerobic metabolism disappear from the microbial metatranscriptome as development proceeds, while the expression of functions related to carbohydrate transport and metabolism increases and …

AdultMale0301 basic medicineResearch paperCarbohydrate transportPregnancy Trimester ThirdPhysiologyFirst year of lifeButyrateGut floraGeneral Biochemistry Genetics and Molecular BiologyFeces03 medical and health sciences0302 clinical medicineBacterial ProteinsPregnancymedicineHumansGutMicrobiomeMetatranscriptomicsPregnancyBacteriabiologySequence Analysis RNAGene Expression ProfilingMicrobiotaInfant NewbornInfantGene Expression Regulation BacterialGeneral MedicineMetabolismLipid Metabolismbiology.organism_classificationmedicine.diseaseIntestinal epitheliumGastrointestinal MicrobiomeButyratesMetabolism030104 developmental biology030220 oncology & carcinogenesisFemaleMaternal AgeEBioMedicine
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Lipoprotein hydrophobic core lipids are partially extruded to surface in smaller HDL: "Herniated" HDL, a common feature in diabetes

2016

AbstractRecent studies have shown that pharmacological increases in HDL cholesterol concentrations do not necessarily translate into clinical benefits for patients, raising concerns about its predictive value for cardiovascular events. Here we hypothesize that the size-modulated lipid distribution within HDL particles is compromised in metabolic disorders that have abnormal HDL particle sizes, such as type 2 diabetes mellitus (DM2). By using NMR spectroscopy combined with a biochemical volumetric model we determined the size and spatial lipid distribution of HDL subclasses in a cohort of 26 controls and 29 DM2 patients before and after two drug treatments, one with niacin plus laropiprant a…

AdultMale0301 basic medicineSimvastatinmedicine.medical_specialtyIndolesMagnetic Resonance SpectroscopySurface Properties030204 cardiovascular system & hematologyMicroscopy Atomic ForceNiacinArticle03 medical and health scienceschemistry.chemical_compound0302 clinical medicineFenofibrateInternal medicinemedicineHumansTriglyceridesAgedFluorescent DyesMultidisciplinaryFenofibrateCholesterolCholesterol HDLType 2 Diabetes Mellitusnutritional and metabolic diseasesLipid metabolismMiddle AgedLipid MetabolismLipids030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2chemistryBiochemistryCardiovascular DiseasesSimvastatinFemalelipids (amino acids peptides and proteins)LaropiprantNiacinLipoproteinmedicine.drug
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Stimulatory TSH-Receptor Antibodies and Oxidative Stress in Graves Disease

2018

CONTEXT: We hypothesized that TSH-receptor (TSHR) stimulating antibodies (TSAbs) are involved in oxidative stress mechanisms in patients with Graves disease (GD). METHODS: Nicotinamide adenine dinucleotide phosphate oxidase, isoform 2 (NOX2); oxidative parameters; and oxidative burst were measured in serum, urine, and whole blood from patients with GD and control subjects. Superoxide production was investigated in human embryonic kidney (HEK)-293 cells stably overexpressing the TSHR. Lipid peroxidation was determined by immunodot-blot analysis for protein-bound 4-hydroxy-2-nonenal (4-HNE) in human primary thyrocytes and HEK-293–TSHR cells. RESULTS: Serum NOX2 levels were markedly higher in …

AdultMale0301 basic medicineendocrine systemmedicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and MetabolismGraves' diseaseClinical Biochemistry030209 endocrinology & metabolismContext (language use)medicine.disease_causeBiochemistryLipid peroxidation03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyInternal medicinemedicineHumansEuthyroidClinical Research ArticlesTriiodothyroninebusiness.industryBiochemistry (medical)Toxic nodular goiterReceptors ThyrotropinMiddle AgedPrognosismedicine.diseaseGraves DiseaseRespiratory burstOxidative StressHEK293 Cells030104 developmental biologyEndocrinologychemistryFemaleLipid PeroxidationbusinessBiomarkershormones hormone substitutes and hormone antagonistsOxidative stressFollow-Up StudiesImmunoglobulins Thyroid-StimulatingThe Journal of Clinical Endocrinology & Metabolism
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The rs2294918 E434K variant modulates patatin-like phospholipase domain-containing 3 expression and liver damage

2016

The patatin-like phosholipase domain-containing 3 (PNPLA3) rs738409 polymorphism (I148M) is a major determinant of hepatic fat and predisposes to the full spectrum of liver damage in nonalcoholic fatty liver disease (NAFLD). The aim of this study was to evaluate whether additional PNPLA3 coding variants contribute to NAFLD susceptibility, first in individuals with contrasting phenotypes (with early-onset NAFLD vs. very low aminotransferases) and then in a large validation cohort. Rare PNPLA3 variants were not detected by sequencing coding regions and intron-exon boundaries either in 142 patients with early-onset NAFLD nor in 100 healthy individuals with alanine aminotransferase22/20 IU/mL. …

AdultMale0301 basic medicinemedicine.medical_specialtyAdolescentPolymorphism Single Nucleotide03 medical and health sciences0302 clinical medicineNon-alcoholic Fatty Liver DiseaseLipid dropletInternal medicineNonalcoholic fatty liver diseasemedicineHumansGenetic Predisposition to DiseaseAlleleChildGeneticsHepatologybiologyMembrane ProteinsAlanine TransaminaseLipaseMiddle AgedHepatologyLipid Metabolismmedicine.diseasedigestive system diseases030104 developmental biologyEndocrinologyHaplotypesLiverAlanine transaminasePatatin-like phospholipaseadolescent; adult; alanine transaminase; case-control studies; child; female; genetic predisposition to disease; haplotypes; humans; lipase; lipid metabolism; liver; male; membrane proteins; middle aged; non-alcoholic fatty liver disease; polymorphism; single nucleotide; hepatologyCase-Control Studiesbiology.proteinFemale030211 gastroenterology & hepatologySteatosisSteatohepatitis
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Metabolic disorders and inflammation are associated with familial combined hyperlipemia

2019

Background: Familial Combined Hyperlipidemia (FCH) is related to different metabolic disorders. The objective of this study was to evaluate the presence of alterations of hydrocarbonated metabolism and lipid profile together with inflammatory and adhesion molecules in subjects with FCH compared to controls. Methods: 75 HFC patients and 75 healthy individuals were studied. Glucose, insulin, HOMA-IR index and lipid parameters, in addition to anti-oxidized LDL antibodies (Anti ox-LDL), small and dense LDL (sdLDL) and HDL subfractions, proinflammatory cytokines and adhesion molecules were measured. Results: FCH patients showed higher levels of hydrocarbonated metabolism parameters, total choles…

AdultMale0301 basic medicinemedicine.medical_specialtyApolipoprotein Bmedicine.medical_treatmentPhenotype B of LDL HDL subclassesClinical BiochemistryHyperlipidemia Familial CombinedInflammationBiochemistryProinflammatory cytokine03 medical and health sciences0302 clinical medicineInsulin resistanceInternal medicinemedicineHumansInflammationmedicine.diagnostic_testbiologybusiness.industryCell adhesion moleculeInsulinCholesterol HDLFCHBiochemistry (medical)Insulin resistanceGeneral MedicineMetabolismmedicine.diseasePhenotype030104 developmental biologyEndocrinology030220 oncology & carcinogenesisbiology.proteinCytokinesFemalelipids (amino acids peptides and proteins)medicine.symptombusinessLipid profileAdhesion moleculesClinica Chimica Acta
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Hepatic and circulating levels of PCSK9 in morbidly obese patients: Relation with severity of liver steatosis

2020

Non-alcoholic fatty liver disease (NAFLD) is becoming the main cause of liver disease in Western countries, especially in morbidly obese patients (MOPs). The proprotein convertase subtilisin/kexin type 9 (PCSK9) has been recently studied because of its possible involvement in the pathogenesis of NAFLD, but its role, at least in MOPs, is still controversial. The aim of this study was to clarify the correlation between the circulating levels of the PCSK9 protein (cPCSK9) and its hepatic expression with the severity of liver damage in a population of MOPs with NAFLD undergoing bariatric surgery. PCSK9 mRNA was positively correlated with FASN, PPARγ and PPARα mRNAs, while no significant differe…

AdultMale0301 basic medicinemedicine.medical_specialtySettore MED/06 - Oncologia MedicaPopulationBariatric SurgeryInflammation030204 cardiovascular system & hematologySeverity of Illness IndexPathogenesisNon-alcoholic fatty liver disease (NAFLD)03 medical and health sciencesBallooning degenerationLiver disease0302 clinical medicineInternal medicinemedicineHumansMorbidly obese patients (MOPs)educationMolecular Biologyeducation.field_of_studybusiness.industryPCSK9Fatty liverCell BiologyMiddle AgedLipid Metabolismmedicine.diseaseObesity MorbidFatty LiverProprotein convertase subtilisin/kexin type 9 (PCSK9)030104 developmental biologyEndocrinologyLiverFemaleProprotein Convertase 9medicine.symptomSteatosisbusiness
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Lomitapide affects HDL composition and function

2016

Abstract Background Lomitapide reduces low-density lipoprotein-cholesterol (LDL-C) but also high-density lipoprotein-cholesterol (HDL-C) levels. The latter may reduce the clinical efficacy of lomitapide. We investigated the effect of lomitapide on HDL-C levels and on cholesterol efflux capacity (CEC) of HDL in patients with homozygous familial hypercholesterolemia (HoFH). Methods and results Four HoFH patients were treated with increasing dosages of lomitapide. Lomitapide decreased LDL-C (range −34 to −89%). Total HDL-C levels decreased (range −16 to −34%) with a shift to buoyant HDL. ABCA1-mediated CEC decreased in all patients (range −39 to −99%). The changes of total, ABCG1- and SR-BI-me…

AdultMale0301 basic medicinemedicine.medical_specialtySettore MED/09 - Medicina InternaHDLHomozygous familial hypercholesterolemiaFamilial hypercholesterolemia030204 cardiovascular system & hematologyHyperlipoproteinemia Type IIYoung Adult03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineHumansMedicineIn patientClinical efficacyHyperlipoproteinemia Type IICholesterolbusiness.industryCholesterol HDLHomozygotenutritional and metabolic diseasesCholesterol LDLCholesterol efflux capacityAtherosclerosismedicine.diseaseCholesterol lowering drugsLomitapideLomitapideCholesterolPhenotypeTreatment Outcome030104 developmental biologyEndocrinologychemistryBenzimidazolesFemalelipids (amino acids peptides and proteins)Composition (visual arts)Cholesterol lowering drugHydroxymethylglutaryl-CoA Reductase InhibitorsLipoproteins HDLCardiology and Cardiovascular MedicinebusinessATP Binding Cassette Transporter 1Atherosclerosis
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Does higher education protect against obesity? Evidence using Mendelian randomization

2017

Objectives. The aim of this explorative study was to examine the effect of education on obesity using Mendelian randomization. Methods. Participants (N = 2011) were from the on- going nationally representative Young Finns Study (YFS) that began in 1980 when six cohorts (aged 30, 33, 36, 39, 42 and 45 in 2007) were recruited. The average value of BMI (kg/m(2)) measurements in 2007 and 2011 and genetic information were linked to comprehensive register based information on the years of education in 2007. We first used a linear regression (Ordinary Least Squares, OLS) to estimate the relationship between education and BMI. To identify a causal relationship, we exploited Mendelian randomization …

AdultMale0301 basic medicinemedicine.medical_specialtyobesityschoolingEpidemiology515 PsychologyProtective factorBody Mass Index03 medical and health sciencesbody weightBMI0302 clinical medicineWaist–hip ratioInternal medicineLinear regressionMendelian randomizationmedicineHumans030212 general & internal medicineFinlandGenetic associationeducationbusiness.industryPublic Health Environmental and Occupational Healthta3141ylipainota3142Mendelian Randomization Analysista3121medicine.diseaseObesityConfidence interval030104 developmental biologyEndocrinologykoulutusOrdinary least squaresEducational StatuslihavuusFemalewaist-hip ratiobusinessGenome-Wide Association StudyDemographyPreventive Medicine
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THE VITAMIN D RECEPTOR TAQ I POLYMORPHISM IS ASSOCIATED WITH REDUCED VDR AND INCREASED PDIA3 PROTEIN LEVELS IN HUMAN INTESTINAL FIBROBLASTS

2020

The synonymous single nucleotide polymorphism (SNP) rs731236, located in the vitamin D receptor (VDR) gene (Taq I) has been associated with both decreased levels of the protein in peripheral blood mononuclear cells and a fibrosis-related complication in Crohn´s disease (CD). Interactions between VDR and a protein-disulfide isomerase-associated 3 (PDIA3) in the regulation of extracellular matrix have been reported and we aim to analyze the relevance of the VDR genotypes and the effects of Vitamin D (VD) in the expression of VDR, PDIA3 and proliferation of intestinal fibroblasts. Human intestinal fibroblasts were isolated from the non-affected surgical resections of colorectal patients and cl…

AdultMale0301 basic medicinemusculoskeletal diseasesAdolescentGenotypeEndocrinology Diabetes and MetabolismClinical BiochemistryProtein Disulfide-IsomerasesPDIA3BiologyPDIA3Polymorphism Single NucleotideBiochemistryPeripheral blood mononuclear cellCalcitriol receptorFibroblast migrationExtracellular matrixYoung Adult03 medical and health sciences0302 clinical medicineEndocrinologyVitamin D and neurologypolycyclic compoundsHumansGene silencingVitamin DMolecular BiologyAllelesCells CulturedCell ProliferationVDRdigestive oral and skin physiologyCell BiologyTransfectionFibroblastsMolecular biologySingle nucleotide polymorphismIntestines030104 developmental biologyCrohn ' s disease030220 oncology & carcinogenesisReceptors CalcitriolMolecular MedicineFemalelipids (amino acids peptides and proteins)Crohn´s diseaseTaq IJournal of steroid biochemistry and molecular biology
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Laquinimod dampens IL-1β signaling and Th17-polarizing capacity of monocytes in patients with MS

2020

ObjectiveTo assess the impact of laquinimod treatment on monocytes and to investigate the underlying immunomodulatory mechanisms in MS.MethodsIn this cross-sectional study, we performed in vivo and in vitro analyses of cluster of differentiation (CD14+) monocytes isolated from healthy donors (n = 15), untreated (n = 13), and laquinimod-treated patients with MS (n = 14). Their frequency and the expression of surface activation markers were assessed by flow cytometry and the viability by calcein staining. Cytokine concentrations in the supernatants of lipopolysaccharide (LPS)-stimulated monocytes were determined by flow cytometry. The messenger ribonucleic acid (mRNA) expression level of gene…

AdultMale41Lipopolysaccharide[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyCD14medicine.medical_treatmentInterleukin-1betaQuinolonesLymphocyte ActivationMonocytesArticleFlow cytometrychemistry.chemical_compoundMultiple Sclerosis Relapsing-RemittingDownregulation and upregulationmedicineHumansCD86medicine.diagnostic_testbusiness.industry[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyInterleukin[SDV.IMM.IMM]Life Sciences [q-bio]/Immunology/ImmunotherapyMiddle AgedMolecular biologyCross-Sectional StudiesCytokineNeurologychemistryTh17 CellsFemaleNeurology (clinical)[SDV.IMM.IMM] Life Sciences [q-bio]/Immunology/ImmunotherapybusinessLaquinimodSignal TransductionNeurology - Neuroimmunology Neuroinflammation
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